ライフサイエンスコーパス: redox

1 ich may be driven by fluctuations in luminal redox.

2 e Ln/Fe oxygen coordination and the Fe(2/3+) redox.

3 or new high-capacity electrodes with anionic redox, a still unanswered question was remaining regardi

4 Herein we describe a quantitative redox-activatable sensor (qRAS) for the real-time monito

5 s, these results outline an approach for the redox activation of small molecules at mild potentials b

6 ron transfer (PCET) steps along a pathway of redox active amino acids (Y122beta <--> [W48beta?] <-->

7 ailed to account for roughly half of NADPH's redox active hydrogen.

8 ions on the reduction of cobalt complexes of redox active ligands and explored the reactivity of redu

9 level due to self-charge injection into the redox active polymeric system.

10 r electrodes with kinetic selectivity toward redox active species and help guide synthetic approaches

11 s synthetic approaches to prepare functional redox-active and conjugated homopolymers as well as the

12 Diverse organisms secrete redox-active antibiotics, which can be used as extracell

13 he use of a chiral amine catalyst, bearing a redox-active carbazole unit, which could rapidly reduce

14 characterization and bioelectrocatalysis of redox-active cyclodextrin-coated nanoparticles.

15 the rate constant of the oxidation of PDI's redox-active Cys residues (Cys(53) and Cys(397)) by hydr

16 (AhpF), catalyzes the rapid reduction of the redox-active disulfide center of the antioxidant protein

17 due to the energy splitting between two key redox-active dpi* frontier molecular orbitals (FMOs).

18 N-hydroxyphthalimide (NHPI) based redox-active esters were found to be convenient starting

19 work, gold electrodes were modified using a redox-active layer based on dipyrromethene complexes wit

20 A family of neodymium complexes featuring a redox-active ligand in three different oxidation states

21 in engineering approaches aimed at designing redox-active proteins for diverse biotechnological appli

22 Iron complexes bound by redox-active pyridine dialdimine (PDAI) ligands catalyze

23 The eDMA allows for the detection of redox-active reporter molecules irrespective of their el

24 alysis as a generic platform to target other redox-active side chains for native conjugation.

25 is supported on a surface, the diffusion of redox-active species to the electrode is partially block

26 molecules were designed to contain a pair of redox-active substituents, quinone and hydroquinone grou

27 regulator, SurR, is among a handful of known redox-active transcriptional regulators.

28 Redox-active tyrosines (Ys) play essential roles in enzy

29 provide new tools for mechanistic studies on redox-active Ys in proteins and on functional and aberra

30 These cubanes are redox-active, and calculations reveal that the Co ions b

31 ribe a single-molecule junction comprising a redox-active, atomically precise cobalt chalcogenide clu

32 actor ligands: Hemilabile, MN2S2 ligands and redox-active, nitrosyl ligands, whose interplay guides t

33 emical and biochemical methods to assess the redox activity of the [4Fe4S](2+) cluster in Saccharomyc

34 n phosphate acts effectively as a reversible redox agent for the regeneration of the dye.

35 variation of the address-directed flux of a redox analyte, ferrocenedimethanol (FDM).

36 Zircon Ce/Ce* and Eu/Eu* are sensitive to redox and fractionation respectively, and here are used

37 siological abnormalities, including abnormal redox and mitochondrial metabolism.

38 ydA1) at different pH values, we resolve the redox and protonation events in the catalytic cycle and

39 de positive electrodes offer access to anion redox at high potentials, thereby promising high energy

40 rgets encoding proteins involved in cellular redox balance and DNA replication, including the Mcm rep

41 ntermediate and play a key role in anaerobic redox balance in many fermenting bacteria.

42 t to be involved in maintaining the cellular redox balance, producing NADPH for biosynthesis by recyc

43 le are able to maintain the intraperoxisomal redox balance.

44 ncing metabolic pathways and thiol/disulfide redox balance.

45 lular redox status in particular glutathione redox balance.

46 ules and providing electrochemical access to redox-based cell signals and behaviours.

47 d to diminish drug-resistance in Mtb through redox-based interventions.

48 dependent manner, via S-nitrosation (SNO), a redox-based modification of cysteine thiols.

49 Greater insight into such redox biology may enable precisely targeted manipulation

50 approach couples the detection of a cellular redox biomarker with the ability to release a small-mole

51 ests biogeochemical cycling across a dynamic redox boundary, with primary productivity fuelled by che

52 cult-to-handle modifications to the cellular redox buffer which can impair proper cellular function.

53 evant levels by label free impedance derived redox capacitance.

54 synthesis by recycling the two other primary redox carriers, NADH and ferredoxin.

55 se from the cellular inside to the binuclear redox center (BNC) can occur through two distinct pathwa

56 ting/deactivating the very first step in the redox chain.

57 In particular, a one-electron redox change at a distal metal site leads to a change in

58 s platform exhibits reversible, two-electron redox chemistry at mild potentials and reacts with O2, C

59 ork presents evidence that classical d-block redox chemistry can be performed reversibly by f-block m

60 perties of porous graphitic carbons with the redox chemistry of iodine to produce iodine-carbon batte

61 The redox chemistry of magnesium and its application in rech

62 polysaccharide monooxygenases (LPMO10s) use redox chemistry to cleave glycosidic bonds in the two fo

63 Here we couple records of ocean redox chemistry with nitrogen isotope ((15)N/(14)N) valu

64 x, including seawater pH, pCO2, temperature, redox chemistry, irradiance and nutrient availability.

65 Capacity for electron transfer among redox cofactors versus charge recombination with nearby

66 two juxtaposed FAD molecules per monomer in redox communication with an active disulfide bridge in a

67 Both redox condition and size fraction segregated bacterial t

68 Electron donor amendments, different redox conditions (anaerobic, aerobic, sequential anaerob

69 he MS data were analyzed in conjunction with redox conditions and iron availability within the source

70 its depth-specific distribution depending on redox conditions is a result of a nitrate-triggered roll

71 flecting a wider range of system designs and redox conditions.

72 es distributions in connection with variable redox conditions.

73 d persistent in groundwater under a range of redox conditions.

74 of thioredoxin-interacting protein, a major redox control molecule, and consequent formation of reac

75 nd proteins; it is sustained by a variety of redox-controlled metabolic reactions.

76 on protocol that uses potassium hydride as a redox-controlled reducing agent to access the PAH dianio

77 OCP moves toward the formal potential of the redox couple and eventually becomes poised at this value

78 the voltammetric response of an outer sphere redox couple can be used to track changes in the structu

79 ation was analyzed through monitoring of the redox couple Fe(2+)/Fe(3+) by electrochemical impedance

80 Several redox-coupled proton translocation mechanisms have been

81 , XRD, FTIR, XPS, TGA, BET, and CV using the redox couples [Fe(CN)6](-3/-4) and [Ru(NH3)6](+3/+2) res

82 electrode surface, thus causing a change in redox current upon voltammetric interrogation.

83 The futile redox cycle rapidly consumes O2, rendering standard assa

84 s has implications for the marine phosphorus redox cycle, and might aid the use of phosphite as an al

85 ld by surface relaxation via electrochemical redox cycling is reported.

86 We reasoned that targeting a redox cycling nitroxide to mitochondria could prevent re

87 yrite (FeS2) plays a significant role in the redox cycling of iron and sulfur on Earth and is the pri

88 en in part by fungal respiration and/or iron redox cycling.

89 The redox-cycling behavior of this device was modeled using

90 Here, we address the redox-dependent conformational dynamics of hPDI through

91 dinating the activity of beta-lactamase in a redox-dependent manner to tolerate AG.

92 roxy cyclopentenones and their corresponding redox derivatives, such as thapsigargin, a cytotoxic nat

93 FAD/(FAD + NAD(P)H), revealed three distinct redox distributions and significant differences in their

94 This gigantic redox-driven enzyme employs the energy from quinone redu

95 nts of soils that can be strongly altered by redox-driven processes.

96 R-dependent glutathione oxidation influences redox-driven salicylic acid accumulation.

97 holes and thus enable the color switching of redox dyes using visible light.

98 solves the inherent cross-diffusion issue of redox ECs and has the added benefit of greatly stabilizi

99 the assembly factor depends on the cellular redox environment.

100 FMNH2 The results suggest that mitoNEET is a redox enzyme that may promote oxidation of NADH to facil

101 (GDH) is a thermostable, oxygen insensitive redox enzyme used in bioelectrochemical applications.

102 MICAL Redox enzymes are important post-translational effectors

103 benzoquinones, which are interchangeable via redox equilibria, contribute to both thermal and photoch

104 oxoammonium salt solutions is explained by a redox equilibrium as shown between oxoammonium salts and

105 is relevant for light-driven accumulation of redox equivalents, because it exemplifies how the buildu

106 oscopic studies, we have identified the last redox event as being the turnover-limiting step of the o

107 owledge, the first example of a one-electron redox event causing concerted change in multiple iron ce

108 Redox flow batteries have the potential to revolutionize

109 The main advantage of redox flow batteries is their ability to decouple power

110 electron storage anolyte for aqueous organic redox flow battery (AORFB) applications.

111 ic-solvent is investigated as an anolyte for redox-flow batteries.

112 erconversions and only one case of catalytic redox function are seen.

113 In this way, a fine-tuned redox gradient is established to power a unidirectional,

114 al electron transfer processes or simulating redox gradients as they exist in microbial biofilms.

115 This study indicates that vertical redox gradients exert a major control on the quantity an

116 solid-phase Fe speciation along the vertical redox gradients of floodplains, which exhibited a succes

117 tified as biogeochemical hotspots with steep redox gradients.

118 gate voltage to the molecule, we switch the redox group between the oxidized and reduced states, lea

119 etal alloys in bulk rock, possibly producing redox heterogeneities in subducting slabs.

120 phosphate (NADPH) production and imbalanced redox homeostasis in erythrocytes.

121 entral importance of oxygen, energetics, and redox homeostasis in immune cell metabolism, and how the

122 ting in the regulation of NAD(P)(+) :NAD(P)H redox homeostasis in various prokaryotic and eukaryotic

123 chondrial respiratory function, not abnormal redox homeostasis, distinguishes ASD from unaffected LCL

124 obal protein synthesis, and perturbations in redox homeostasis, including increased endogenous ROS le

125 synthesis Pathway (HBP), as well as cellular redox homeostasis, resulting in global changes in protei

126 tential across the cell membrane and disrupt redox homeostasis, thereby inhibiting bacterial growth.

127 cell invasion and anoikis resistance through redox homeostasis.

128 maintenance of mitochondrial respiration and redox homeostasis.

129 e anaplerosis, macromolecule production, and redox homeostasis.

130 oxin reductase (TrxR) system maintains thiol redox homeostasis.

131 -hydrogenase embedded in a viologen-modified redox hydrogel for the fabrication of a sensitive hydrog

132 The connections between the redox imbalance in post-mortem muscle, early protein oxi

133 Like porphyrins, corrole derivatives with a redox-inactive coordinated atom follow the Gouterman fou

134 n (1) and salinomycin (4) harbor a number of redox-inactive ketoreductase (KR(0)) domains that are im

135 The AgNF was electrodeposited as redox indicator on a gold electrode, which was then func

136 ce in electrochemical signals from a soluble redox indicator, ferricyanide, on nitrocellulose films t

137 The in situ control of redox insult in human organs is of major clinical releva

138 unction (i.e., scalp hair follicles) against redox insult.

139 ich allow the enantiomerization to occur via redox-interconversion.

140 tron transfer for spectroscopic detection of redox intermediates during catalytic proton reduction.

141 intermolecular water nucleophilic attack and redox isomerization of {[LCu(III)]2-(mu-O)2}(2+) are ene

142 is approach, called SNOxICAT (S-nitrosothiol redox isotope-coded affinity tag), we found that exposur

143 ng that as little as a few tens of copies of redox-labeled macromolecules immobilized on individual n

144 f such a complex mechanism suggests that the redox level of the environment regulates the BCAA biosyn

145 ing microbial respiration rates due to lower redox levels in the soil.

146 -catalyzed C(sp(3))-H activation via various redox manifolds, including Pd(0)/Pd(II), Pd(II)/Pd(IV),

147 These results are consistent with a redox mechanism for propagation of membrane polarity asy

148 Collectively, this study reveals a redox mechanism for regulating tankyrase activity and im

149 e results highlight a material operating via redox mechanism that may find utility in the storage and

150 ceuticals are probably also metabolized by a redox mechanism.

151 gaseous HCl electrolysis with Fe(3+) /Fe(2+) redox-mediated cathode is demonstrated for Cl2 regenerat

152 osphorylates Mical to directly amplify Mical Redox-mediated F-actin disassembly.

153 y drug leflunomide, which likewise undergoes redox-mediated Kemp elimination by P450-BM3.

154 Here we demonstrate that the choice of redox mediator in these solar cells has a profound influ

155 sis is suggested to involve an intracellular redox mediator, which is released during light irradiati

156 electrode in three different charges of the redox mediators (i.e., neutral FcCH2OH, cationic Ru(NH3)

157 ntal medicine, and molecular knowledge-based redox medicine.

158 ole for TRMT1-catalyzed tRNA modification in redox metabolism and show that individuals with TRMT1-as

159 tabolic processes such as photosynthesis and redox metabolism.

160 Post-translational redox modification of methionine residues often triggers

161 The redox-modulated change in DNA-binding affinity regulates

162 The functionalized redox nanomaterial exhibits reversible electrocatalytic

163 mediated O2 reduction are observed with the redox nanoparticle system compared to equivalent bioelec

164 These reactions are redox-neutral and completely atom-economical, exhibit br

165 ith quinone diazides under Rh(III)-catalyzed redox-neutral conditions.

166 An operationally simple, mild, redox-neutral method for the cross-coupling of alpha-hyd

167 This method achieves one-step, redox-neutral synthesis of catechols with diverse substi

168 mechanical motion and guest binding with the redox noninnocent and valence tautomerism properties of

169 The redox operations switch the independent alpha and beta r

170 erface (OAI) of hyporheic zones subjected to redox oscillations, VC is degraded via coexisting aerobi

171 We first examined redox (oxidation/reduction) properties and stability of

172 r electrochemical response from a reversible redox pair was obtained.

173 cal responses of this device to a reversible redox pair were examined.

174 Cytochrome P450 reductase (CPR) is the redox partner for most human cytochrome P450 enzymes.

175 s proposed to convert MftA into the external redox partner mycofactocin.

176 "fast" phase of the enzyme reduction by the redox partner NADPH-cytochrome P450 oxidoreductase, and

177 res of cytochrome P450cam complexed with its redox partner, putidaredoxin (Pdx), shows that P450cam a

178 ble NAD cofactor and may rely on an external redox partner, rather than cofactor exchange, for multip

179 Thus, the ER redox poise is tuned by reciprocal control of glutathion

180 pplied potentials are believed to affect the redox potential across the cell membrane and disrupt red

181 igration, while soil characteristics such as redox potential and surface salinity developed later in

182 ca 3.2 m in the anolyte and a relatively low redox potential of 2.2 V vs. Li(+) /Li.

183 Furthermore, the higher redox potential of copper and the enhanced weakening of

184 ristic sigmoid plots when represented versus redox potential suggesting that all changes are the resu

185 Severe changes in the environmental redox potential, and resulting alterations in the oxidat

186 Higher radiation doses increased the redox potential, promoted the lipid oxidation and elevat

187 for free and Brine Releasable (BR) VSCs and redox potential.

188 , the concentrations of ions, the pH and the redox potential.

189 ) ), this Mn-oxide is predicted to show high redox potentials ( approximately 4.2 V vs Na/Na(+) ) wit

190 st-principles calculations of proton-coupled redox potentials and magnetizations reveal that the Ni-o

191 ve as the reservoir for electrons, but their redox potentials are tuned by the choice of ligand at Mn

192 ations indicate that both UQ and MQ have low redox potentials around -260 and -230 mV, respectively,

193 fine-tuning of the electronic properties and redox potentials of the photocatalyst in both the excite

194 the entropic and enthalpic contributions to redox potentials with Q and with the identity and hydrop

195 n differential pulse voltammetry signal of a redox probe ([Fe(CN)6](3-)/[Fe(CN)6](4-)) that is altere

196 iciently restricted the electron transfer of redox probe Fe(CN)6(4-/3-) were utilized to detect BoNT/

197 erent properties, for the first time, as the redox probe in the development of HCV core antigen elect

198 ule with inherent properties was used as the redox probe in the development of the TNT aptasensor was

199 cyclic voltammetry of an ideal outer-sphere redox probe, reversible ferrocene methanol oxidation.

200 d by using potassium hexacyanoferrate(II) as redox probe.

201 s significant dominated by the charge of the redox probe.

202 gged with a ferrocene molecule, which acts a redox probe.

203 e use of DCFH2-DA, as many other fluorogenic redox probes, is mainly confined to the detection of int

204 Compared to the other redox probes, riboflavin is superior in its oxidization

205 Remarkably, the anionic redox process occurs jointly with the oxidation of Ir(4+

206 ue to structural damage occurring during the redox process.

207 irst examples of room-temperature reversible redox processes for s-block metal complexes.

208 on of long-chain fatty acids and main energy-redox processes is able to simulate the relationship bet

209 Redox processes of molybdenum-sulfide (Mo-S) compounds a

210 ion of carbon uptake, catabolism, energy and redox production, and growth), while allowing a large de

211 e over the basal growth requirement, and (3) redox production, which also scales with nutrient uptake

212 ensively in this role due to their intrinsic redox properties and reactivity, but more recently, stra

213 The synthesis, metalation, and redox properties of an acyclic bis(iminothienyl)methene

214 tting are largely controlled by the inherent redox properties of the materials.

215 IF-67 and ZIF-8, were interrogated for their redox properties using Fourier transformed alternating c

216 We show that the redox-protective protein thioredoxin-1 (TRX1) increases

217 and ferredoxin-NADP(+) reductases (FNR) are redox proteins that mediate electron metabolism in vivo,

218 We extended the thiol-redox proteomic technique, isotope-coded affinity tag la

219 Histograms of pixel-wise optical redox ratio, defined as FAD/(FAD + NAD(P)H), revealed th

220 Using the anionic redox reaction (O(2-) /O(-) ), this Mn-oxide is predicte

221 es an effective platform for elucidating the redox reaction and oxygen diffusion within transition me

222 n complex I at which energy generated by the redox reaction is used to initiate proton translocation.

223 Through a fundamental understanding of the redox reaction mechanism in Li2 MnO3 , Na(Li1/3 Mn2/3 )O

224 amine layers were confirmed by the increased redox reaction.

225 ed organic matter (DOM) affects mercury (Hg) redox reactions and anaerobic microbial methylation in t

226 ironmental conditions, it can participate in redox reactions and influence the sorption processes at

227 The combination of ion intercalation with redox reactions of iodine allows for developing recharge

228 t performance for high-temperature catalytic redox reactions such as water splitting.

229 carbon has yet to be coupled with extrinsic redox reactions to develop rechargeable batteries.

230 It is less clear, however, how redox reactions would contribute to acidification.

231 ffer sustainable alternatives to traditional redox reactions, but strategies are needed to enhance th

232 nd environmental applications exploiting the redox reactions.

233 The redox regulated C-terminal extension (CTE) and the assoc

234 ntifies an important role for ACC enzymes in redox regulation and cell survival.

235 t not STIM1, at least in part as a result of redox regulation of cytokine gene expression.

236 tioxidant function, but also participates in redox regulation of metabolic pathways previously establ

237 eotide pathways, and metabolites involved in redox regulation were greatly affected 4 hours post-expo

238 hat NTRC plays a pivotal role in chloroplast redox regulation, being necessary for the activity of di

239 enetic alterations and to cell signaling and redox regulation.

240 ed here thus reveal the mechanism underlying redox-regulation of AP-1 Fos/Jun transcription factors a

241 ) counterpart (GSSH) have been recognized as redox regulators, some of which were previously ascribed

242 An enantioselective redox-relay Heck alkynylation of di- and trisubstituted

243 plated in situ gelation process, whereas the redox-responsiveness was achieved by using a disulfide b

244 oxidative stress required the canonical CXXC redox-sensing motif.

245 ajor phosphatase revealed that the enzyme is redox sensitive.

246 TRPA1 activation was dependent on essential redox-sensitive cysteine and lysine residues within N-te

247 f benzoxazoles is compatible with a range of redox-sensitive functional groups.

248 Two-photon imaging of redox-sensitive GFP corroborated the finding that mitoch

249 d H2O2 production resulted in disturbance of redox-sensitive signaling including Akt and MAPKs pathwa

250 Here we present iron-speciation, redox-sensitive trace element, and nitrogen isotope data

251 Here, we present a record of redox-sensitive uranium from the central equatorial Paci

252 These redox sensitivity changes were confirmed using biotinyla

253 Redox separations for multiple Fc molecules are based on

254 ning (fertilizer-derived) nitrate drives the redox shift from originally reducing toward oxidizing en

255 thway whereby FABP4/aP2 regulates macrophage redox signaling and inflammasome activation via control

256 This could participate in the redox signaling in hyperglycemic heart and contribute to

257 ein kinase S-glutathionylation as a means of redox signaling in plant cells.

258 t has focused on the intricate ways by which redox signaling integrates these converse properties.

259 re responsible for fine-tuning physiological redox signaling.

260 sential for governing life processes through redox signaling.

261 ial dysfunction is characterised by aberrant redox signalling and an inflammatory phenotype.

262 oxidation or reduction in situ, and thus the redox species are not what are observed before and after

263 the ion current displays high sensitivity to redox species, suggesting the possibility of trace-level

264 the calculated concentration profiles of the redox species.

265 y and deliver nitric oxide with a controlled redox state and rate is crucial for its pharmaceutical/m

266 mediated by epigenetic re-programming of the redox state in the CB chemosensory reflex pathway.

267 nd elucidation of the mechanisms whereby the redox state influences circadian regulation.

268 These data suggest that the redox state of bacteria during infection differs signifi

269 act as oxidants to balance the intracellular redox state of cells in anoxic biofilm subzones.

270 he steady-state level of ROS, as well as the redox state of each compartment, is different at any giv

271 rce microscopy (AFM) to demonstrate that the redox state of the [4Fe4S] clusters regulates the abilit

272 conditions where we are able to control the redox state of the enzyme precisely.

273 lar metabolism, including on the metabolome, redox state, and glucose utilization.

274 t of residues around heme a changes with the redox state, hence suggesting that the H channel could p

275 ransfer reactions, leading to the changes in redox state.

276 but differ with respect to the relevant iron redox state.

277 Different redox states have a profound effect on domain orientatio

278 s opportunities for evaluating intracellular redox states in biochemical investigations.

279 (-) ligands of the (2FeH) site for different redox states of the H-cluster.

280 change in the energy level alignment of the redox states relative to the Fermi level of the electrod

281 Still, the mitochondrial redox status did not change with Z-3-hexenol, another ab

282 A; and MEcPP-mediated alteration of cellular redox status in particular glutathione redox balance.

283 perception of E-2-hexenal, which changes the redox status of the mitochondria.

284 anges in mitochondrial enzyme activities and redox status that lead to apoptosis, necrosis, and autop

285 ys associated with amino acid metabolism and redox status.

286 ironment, with tissue-specific variations in redox stress and oxygen concentration.

287 phisticated mechanisms to sense and adapt to redox stress in nature and within the host.

288 activation of NRF2, the master regulator of redox stress tolerance.

289 et through its protection against AR-induced redox stress.

290 The use of a reversible ternary redox switch enables us to set the pKa to three differen

291 he [4Fe4S] cluster in Pol delta can act as a redox switch for activity, and we propose that this swit

292 air function of mitoNEET is based on an Fe/S redox switch mechanism: under normal cellular conditions

293 ments elucidate the electronic structure and redox thermodynamics of Ni-only and mixed NiFe oxyhydrox

294 l SAM containing PEG moieties and a tethered redox thiol, both markers are detectable across clinical

295 tion complex may be a necessary step for the redox transformation via catalytic or direct oxidation p

296 aters or toxicology exposure media, the same redox transformations can occur, causing altered behavio

297 Adsorption and redox transformations on clay mineral surfaces are preva

298 idazolate ligand is responsible for multiple redox transformations.

299 chaeal and fungal enzymes, which peak in two redox transition zones (12) .

300 ium (Se) geochemistry, which is sensitive to redox transitions across suboxic conditions.

301 aelectron transfer chain between neighboring redox units of clustered particles (Dh,DLS = 195 nm) and