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1 ially overlap with those of Reelin-deficient reeler mice.
2 elin, and radial glial fibers in control and reeler mice.
3 oth control (wild-type and heterozygous) and reeler mice.
4  and GAD67 mRNAs in both WT and heterozygous reeler mice.
5  transplanted HNSCs in wild-type, but not in reeler mice.
6 rent from both wild-type mice and homozygous reeler mice.
7 ruption of cell positioning in the retina of reeler mice.
8 a developmental event that fails to occur in reeler mice.
9 lacement of neurons and associated ataxia in reeler mice.
10 ssed in normal mouse brain that is absent in reeler mice.
11 basal forebrain markers Dlx1/2 in normal and reeler mice.
12 ese interneurons is not generally altered in reeler mice.
13 e disrupted cortical lamination phenotype in reeler mice and subsequent identification of the Reelin
14 ld be inverted in hem-ablated animals, as in reeler mice, deficient in reelin signaling.
15            Surprisingly, network activity in reeler mice displays similar characteristics and pharmac
16                            Reelin-deficient (Reeler) mice exhibited impaired respones to hypoxia comp
17                                 Heterozygous reeler mice (HRM) haploinsufficient for reelin express a
18 ative study in the cerebella of heterozygous reeler mice (HRM), in which reelin expression is down-re
19 te abnormalities are present in the brain of reeler mice lacking Reelin.
20                                           In reeler mice, most of the injected HNSCs failed to migrat
21 yonic and postnatal layer 1 in wild-type and reeler mice, mutant in the production of reelin.
22 ding that the brains of developing and adult reeler mice of both sexes contained a markedly reduced n
23  the extracellular matrix protein missing in reeler mice, plays an important role in neuronal migrati
24                              In heterozygous reeler mice, reelin bound to DSPSD, and the expression o
25 isease phenotypes of the spastic and Orleans reeler mice respectively.
26 reelin and GAD67 in both WT and heterozygous reeler mice, suggesting an epigenetic action through the
27                                 Heterozygous reeler mice that exhibit a 50% downregulation of reelin
28                           In both normal and reeler mice the period of neurogenesis of SPN was simila
29                                           In reeler mice, the endogenous NSC population in the hippoc
30 rons migrated and differentiated normally in reeler mice, the migrations of both sympathetic (SPNs) a
31 r initial migration, SPN in both control and reeler mice were closely apposed to radial glial fibers
32   This callosal phenotype is not detected in reeler mice, which also exhibit defects in cortical lami

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