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1 dal reentry tachycardia and atrioventricular reentrant tachycardia).
2 substantiated by induction of orthodromic AV reentrant tachycardia.
3 give rise to localized reentry and AV nodal reentrant tachycardia.
4 f adenosine in patients with typical AV node reentrant tachycardia.
5 dose adenosine led to initiation of AV node reentrant tachycardia.
6 occurred in 13 of 139 patients with AV node reentrant tachycardia.
7 herapeutic approach in patients with AV node reentrant tachycardia.
8 ent symptomatic episodes of atrioventricular reentrant tachycardia.
9 ; PPI-TCL) is a useful tool in mapping macro-reentrant tachycardias.
11 rrence, including sustained atrioventricular reentrant tachycardia (132 patients) or atrial fibrillat
12 f initial episodes of atrioventricular nodal reentrant tachycardia (95% confidence interval 21% to 67
14 dual AV node physiology and typical AV node reentrant tachycardia and 10 control patients were given
15 ents with CHD who had atrioventricular nodal reentrant tachycardia and were treated with catheter abl
16 ral isthmus is a critical part of perimitral reentrant tachycardia, as well as an important substrate
17 doxically AH(SVT)<AH(NSR), differentiates NF reentrant tachycardia/atrioventricular nodal reentrant t
18 of junctional reciprocating tachycardia, NF reentrant tachycardia/atrioventricular nodal reentrant t
19 nce, data on atypical atrioventricular nodal reentrant tachycardia (AVNRT) are scarce, and the optima
20 ulmonary veins (PVs), atrioventricular nodal reentrant tachycardia (AVNRT) can also cause or coexist
22 r patients with the typical form of AV nodal reentrant tachycardia (AVNRT) underwent selective radiof
23 th atrial flutter and atrioventricular nodal reentrant tachycardia (AVNRT) who underwent fluoroscopic
24 pathways serve as the substrate for AV node reentrant tachycardia (AVNRT), ablation of the slow path
25 undergone ablation of atrioventricular nodal reentrant tachycardia (AVNRT), an accessory pathway (AP)
31 en consecutive patients with 26 intra-atrial reentrant tachycardias complicating surgery for congenit
32 cating tachycardia or atrioventricular nodal reentrant tachycardia), excluding Wolff-Parkinson-White,
34 7% to 99%) have remained free of intraatrial reentrant tachycardia for a mean of 7.5 +/- 5.3 months.
35 reentrant tachycardia/atrioventricular nodal reentrant tachycardia from permanent form of junctional
37 reentrant tachycardia/atrioventricular nodal reentrant tachycardia had longer AH (29 ms versus 10 ms;
38 th dual AV node pathways and typical AV node reentrant tachycardia has not previously been studied.
39 ent decades in curing atrioventricular nodal reentrant tachycardias has intensified efforts to provid
43 possible factors for inducible intra-atrial reentrant tachycardia (IART) in a group of patients afte
47 ion sites remains difficult for intra-atrial reentrant tachycardias (IART) in congenital heart diseas
48 duced in 27 of 30 patients; atrioventricular reentrant tachycardia in 25 (93%) of 27 and intraatrial
51 We tested the hypotheses that intra-atrial reentrant tachycardia in patients who had undergone prio
52 er ablation for the treatment of intraatrial reentrant tachycardia in patients with previous atrial s
53 que for definitive management of intraatrial reentrant tachycardia in patients with previous atrial s
55 1 atrioventricular (AV) block during AV node reentrant tachycardia induced in the electrophysiology l
56 actory period (P<0.001) and atrioventricular reentrant tachycardia initiating atrial fibrillation (P<
59 l in blocked beats, 2:1 block during AV node reentrant tachycardia is due to functional infranodal bl
60 s observed in 6 (8%) of the atrioventricular reentrant tachycardia mediated by septal AP (P<0.001; se
62 al reciprocating tachycardia; n=4] or NF [NF reentrant tachycardia; n=2]) accessory pathway underwent
66 may underlie the acceleration of functional reentrant tachycardias paced by a clinician or an antita
67 rkinson-White patients with atrioventricular reentrant tachycardia referred for electrophysiological
68 N) electrophysiology in the treatment of AVN reentrant tachycardias rely on empirical findings, such
69 (1.6%) ablations for atrioventricular nodal reentrant tachycardia resulted in atrioventricular block
71 In patients with 2:1 AV block during AV node reentrant tachycardia, the absence of a His bundle poten
72 th dual AV node pathways and typical AV node reentrant tachycardia, the fast pathway is more sensitiv
74 The relationship of atrioventricular nodal reentrant tachycardia to congenital heart disease (CHD)
75 nversion of new-onset atrioventricular nodal reentrant tachycardia to sinus rhythm in critically ill
76 actory period (P<0.001) and atrioventricular reentrant tachycardia triggering sustained pre-excited a
77 ory pathways (P<0.001), and atrioventricular reentrant tachycardia triggering sustained pre-excited a
78 y of atrial surgery and clinical intraatrial reentrant tachycardia underwent electrophysiologic testi
79 admission to onset of atrioventricular nodal reentrant tachycardia was 4.5 +/- 5 days (median 2.5).
81 ith 20 SVTs (atypical atrioventricular nodal reentrant tachycardia without [n=11]/with [n=3] a bystan
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