ライフサイエンスコーパス: reepithelialization

1 blast migration and proliferation, and wound reepithelialization.

2 omitant with activation of keratinocytes for reepithelialization.

3 trix and dynamic cell-matrix adhesion during reepithelialization.

4 injured lung, one of which is to facilitate reepithelialization.

5 calization of CD44 in the rat corneas during reepithelialization.

6 eractions that mediate cell migration during reepithelialization.

7 nism to maintain their directionality during reepithelialization.

8 xpress collagenase-1 during normal cutaneous reepithelialization.

9 m eccrine sweat glands parallels the rate of reepithelialization.

10 of corneal haze were apparent shortly after reepithelialization.

11 d of subepithelial haze begins shortly after reepithelialization.

12 proximal keratinocytes and maintained during reepithelialization.

13 sociated with a trend toward delayed corneal reepithelialization.

14 activation, limited keratinization, and slow reepithelialization.

15 important for skin differentiation and wound reepithelialization.

16 with no difference in collagen expression or reepithelialization.

17 ved cells from the follicles aiding in wound reepithelialization.

18 cutaneous wounds heal with an acute delay in reepithelialization.

19 stasis, epithelial cell migration, and wound reepithelialization.

20 .9- to 7.7-week specimens exhibited complete reepithelialization.

21 e genes not previously associated with wound reepithelialization.

22 an increase in inflammation and the rate of reepithelialization, a finding consistent with the pheno

23 scle atrophy, osteoporosis, and reduction of reepithelialization ability in wound-healing.

24 Effective reepithelialization after injury is essential for correc

25 orts have been focused on accelerating wound reepithelialization and closure.

26 One treatment with Ac-PHSRN-NH(2) stimulates reepithelialization and contraction of dermal wounds in

27 MRL mice showed accelerated reepithelialization and decreased corneal opacity compar

28 icacy of keratinocyte growth factor (KGF) in reepithelialization and elastin in dermal wound healing.

29 ffective as a wound healing agent, enhancing reepithelialization and granulation tissue deposition by

30 use skin showed a decrease in wound healing (reepithelialization and granulation) compared to the wil

31 r(db) diabetic mice these particles enhanced reepithelialization and granulation, by 2- and 3-fold re

32 ofoundly accelerated wound closure mainly by reepithelialization and increased keratinocyte migration

33 betes are at higher risk for delayed corneal reepithelialization and infection.

34 und repair, new tissue formation starts with reepithelialization and is followed by granulation tissu

35 fter lesion healing in concert with complete reepithelialization and loss of HSV DNA from skin biopsi

36 EMD and P2 promoted reepithelialization and neovascularization in full-thick

37 Reepithelialization and neovascularization were assessed

38 was quantified through area measurements of reepithelialization and neovascularization.

39 Rapid reepithelialization and reduced keratitis/iritis were al

40 Wound healing kinetics, including wound reepithelialization and wound contraction as well as mic

41 The wound closure, reepithelialization, and collagen deposition were accele

42 s their healing by stimulating angiogenesis, reepithelialization, and collagen deposition, and by sup

43 aged and specific pattern of cell migration, reepithelialization, and cytokine expression.

44 d CF integrated into the dermis, accelerated reepithelialization, and improved the outcome of CK tran

45 ediated modulation of KC migration and wound reepithelialization, and may aid the development of nove

46 efect, the number of specimens with complete reepithelialization, and rate of closure were evaluated

47 nocyte colonies in the wound and accelerated reepithelialization as compared with CK alone.

48 Transplantation of CF accelerated reepithelialization as determined from wound histologies

49 diminution in size at 3 months, and complete reepithelialization at 6 months.

50 EMD maintained a significant acceleration of reepithelialization at day 3 (P = 0.004).

51 Cx43 decline was delayed until 48 h, when reepithelialization began.

52 Secondary outcomes included rate of reepithelialization, best spectacle-corrected visual acu

53 keratinocyte migration is not essential for reepithelialization but suggest instead that alpha3beta1

54 ays, in part by increasing the rate of wound reepithelialization by 35% compared to control.

55 ta4 topically or intraperitoneally increased reepithelialization by 42% over saline controls at 4 d a

56 ize of the index lesion by 42 days, complete reepithelialization by 98 days, and absence of relapse b

57 e extracellular matrix (ECM), and stimulates reepithelialization by keratinocytes.

58 , is required in the epidermis to facilitate reepithelialization by remodeling the basement membrane,

59 receptors in in vitro and in vivo models of reepithelialization by subtype-selective antagonists, sm

60 Abnormalities in corneal reepithelialization caused by second-hand cigarette smok

61 tory cell content, growth factor production, reepithelialization, collagen synthesis, and angiogenesi

62 The rate of wound reepithelialization, collagen synthesis, and angiogenesi

63 at were receiving NTX had an acceleration in reepithelialization compared with diabetic animals that

64 re, increasing epidermal differentiation and reepithelialization, despite the reduced proliferation.

65 Keratinocyte migration is critical to reepithelialization during wound repair.

66 d a reduced median time to 50 and 100% ulcer reepithelialization for ACT1-treated ulcers.

67 The primary end point was mean percent ulcer reepithelialization from baseline to 12 weeks.

68 stripping, MK6a(-/-) mice showed a delay in reepithelialization from the hair follicle.

69 Wound closure, reepithelialization, granulation tissue formation, and r

70 Once allograft reepithelialization has occurred, immunosuppression can

71 nce of events from the point of injury until reepithelialization in axolotl skin explant model and sh

72 Blockade of VEGF also blocked reepithelialization in both the postmenstrual endometriu

73 one (NTX; 30 mg/kg, twice daily) can restore reepithelialization in diabetic cornea, we induced diabe

74 vivo blockade of Smad7 increased the rate of reepithelialization in Itga3(-/-) and WT wounds to simil

75 We have shown here that retarded reepithelialization in Itga3(-/-) mouse skin wounds is d

76 cts in keratinocyte proliferation and tissue reepithelialization in the absence of wild-type DETCs.

77 hydroxamate inhibitor, and in mouse tissue, reepithelialization in trachea from matrilysin-null mice

78 umented function for Smad7 as a regulator of reepithelialization in vivo and implicates Smad7 as a po

79 rs, including EGFR ligands, accelerate wound reepithelialization in vivo and in vitro.

80 However, the precise function of MMPs during reepithelialization in vivo has been elusive in mammalia

81 particularly MMP-1 in migration and possibly reepithelialization in vivo.

82 llular protein, has an important role during reepithelialization in wound healing and is critical for

83 EGFR-stimulated reepithelialization is highly dependent on Slug, as demo

84 Wounds were histologically processed, and reepithelialization, leukocyte infiltration, and angioge

85 This resulted in a novel extending shield reepithelialization mechanism, which we confirmed by com

86 sion was examined in vivo by using a corneal reepithelialization model in mice heterozygous for a Pax

87 nisms of lung repair, which include alveolar reepithelialization, myofibroblast differentiation/activ

88 During reepithelialization of cutaneous wounds, keratinocytes d

89 y, expression of wild-type MEKK1 accelerates reepithelialization of full-thickness skin and corneal d

90 Reepithelialization of induced full-thickness skin wound

91 of molecules reported to be associated with reepithelialization of mucosal and cutaneous wounds.

92 activin on granulation tissue formation and reepithelialization of skin wounds nor its protumorigeni

93 ergo a dramatic phenotypic conversion during reepithelialization of skin wounds to become hyperprolif

94 underlying matrix may influence the rate of reepithelialization of skin wounds.

95 ptor-mediated, negative growth factor during reepithelialization of the abraded human cornea.

96 Reepithelialization of the Col1a1(r/r) wounds took 7 d l

97 estoration of transepithelial resistance and reepithelialization of the denuded epithelium.

98 sary for the morphogenesis, homeostasis, and reepithelialization of the stratified epidermis.

99 factor (PAF), and interferes with the normal reepithelialization of wounded cornea.

100 oporation, the wound bed showed an increased reepithelialization rate, collagen synthesis, and angiog

101 MRL mice than in B6 mice, by means of rapid reepithelialization, reduced inflammation, and reduced f

102 pression is necessary for an EGFR-stimulated reepithelialization response.

103 In vitro reepithelialization stimulated by ectopic Slug expressio

104 e expression of CD44 correlates with corneal reepithelialization, suggesting that CD44 may be involve

105 of keratinocyte alpha3beta1 integrin during reepithelialization suggests that this adhesion molecule

106 These results indicate a delay in reepithelialization that is dependent on the duration of

107 plied topically every hour while awake until reepithelialization, then 4 times daily for at least 3 w

108 rticipates in skin healing and corneal wound reepithelialization through enhancement of epithelial mi

109 The process of reepithelialization was apparent by 12 h, however, the w

110 Wound reepithelialization was assessed using immunohistochemis

111 tion of CD44 on cell surfaces during corneal reepithelialization was consistent with the pattern of m

112 At 4 and 8 weeks, corneal reepithelialization was markedly subnormal, with delays

113 Reepithelialization was measured by fluorescein staining

114 Reepithelialization was monitored by fluorescein stainin

115 Reepithelialization was the dominant mode of wound closu

116 By adapting HSEs to study wound reepithelialization, we found that the extended passage

117 KC migration and wound reepithelialization were facilitated by M4 and inhibited

118 upregulation of Cx43 and doubled the rate of reepithelialization, which exceeded control levels.

119 We hypothesize that reepithelialization will also prevent chronic rejection

120 on PRCP(gt/gt) mice had delayed closure and reepithelialization with reduced PECAM staining, but inc