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1 evant to the association between obesity and reflux disease.
2 formed surgical therapy for gastroesophageal reflux disease.
3 vel potential treatment for gastroesophageal reflux disease.
4 ently overlap with those of gastroesophageal reflux disease.
5 cribed for the treatment of gastroesophageal reflux disease.
6 ormation, 11 (41%) had objective evidence of reflux disease.
7 the presence of concomitant gastroesophageal reflux disease.
8 vely distinguishing it from gastroesophageal reflux disease.
9 to those of patients with gastro-oesophageal reflux disease.
10 an effective treatment for gastroesophageal reflux disease.
11 ognized disease that mimics gastroesophageal reflux disease.
12 rritable bowel syndrome and gastroesophageal reflux disease.
13 developing complications of gastroesophageal reflux disease.
14 ease commonly confused with gastroesophageal reflux disease.
15 disorders and treatment of gastroesophageal reflux disease.
16 between BMI and symptoms of gastroesophageal reflux disease.
17 iologic system is perturbed in subjects with reflux disease.
18 ccurrence and management of gastroesophageal reflux disease.
19 s are at increased risk for gastroesophageal reflux disease.
20 dicative of severe types of gastroesophageal reflux disease.
21 ts with symptomatic GERD do not have erosive reflux disease.
22 and a reliable indicator of gastroesophageal reflux disease.
23 e research and treatment of gastroesophageal reflux disease.
24 l ulcer, gastric ulcer, and gastroesophageal reflux disease.
25 esophagus or other types of gastroesophageal reflux disease.
26 commonly associated with gastro-oesophageal reflux disease.
27 heartburn in patients with gastro-esophageal reflux disease.
28 formed for the treatment of gastroesophageal reflux disease.
29 ients with mild-to-moderate gastroesophageal reflux disease.
30 and stricture secondary to gastroesophageal reflux disease.
31 ardia are manifestations of gastroesophageal reflux disease.
32 cinoma are complications of gastroesophageal reflux disease.
33 ure by waist belt on reflux in patients with reflux disease.
34 o clear and buffer the refluxate, leading to reflux disease.
35 long-lasting treatment for gastroesophageal reflux disease.
36 s associated with a risk of gastroesophageal reflux disease.
37 in pediatric patients with gastroesophageal reflux disease.
38 r the surgical treatment of gastroesophageal reflux disease.
44 pnea may be associated with gastroesophageal reflux disease, a strong risk factor for Barrett's esoph
45 , chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep apnea, anxiety,
46 factors evaluated included gastroesophageal reflux disease, alcohol consumption, smoking, chronic op
47 a complication of chronic gastro-oesophageal reflux disease, although asymptomatic individuals might
48 and duration of symptoms of gastroesophageal reflux disease among randomly selected participants in t
50 quality of life related to gastroesophageal reflux disease and a 50% or greater reduction in the use
51 of peptic ulcer disease and gastrosophageal reflux disease and acts by irreversibly blocking ATP4A,
52 ecreased prevalence of both gastroesophageal reflux disease and adenocarcinoma of the esophagus and c
54 niques in the management of gastroesophageal reflux disease and constipation also may have an impact
56 ties is similar except for gastro-esophageal reflux disease and dyslipidemia that appear to be more s
57 both procedures except for gastro-esophageal reflux disease and dyslipidemia, which were more success
59 evolving definition and its relationship to reflux disease and functional gastrointestinal disorders
61 osis, including the role of gastroesophageal reflux disease and proton pump inhibitor-responsive esop
62 e to design new surgical strategies to treat reflux disease and reduce complications of fundoplicatio
63 The potential roles of undiagnosed venous reflux disease and the military physical training enviro
64 Approximately 20% have gastro-oesophageal reflux disease and this can be effectively treated with
65 n with a medical history of gastroesophageal reflux disease and type II diabetes presented to the hos
66 acid reflux in cough due to gastroesophageal reflux disease, and 3) developing reliable and reproduci
69 ma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other types of pneumon
70 ological manifestation of gastro-oesophageal reflux disease, and is a major risk factor for the devel
73 isk factor for oesophageal adenocarcinoma is reflux disease, and the rising incidence of this coincid
74 is usually due to asthma, gastro-oesophageal reflux disease, and upper airway conditions, and that it
75 Laparoscopic procedures for gastroesophageal reflux disease appear to be as effective as those done b
78 regarding the treatment of gastroesophageal reflux disease are developed, based on a review of studi
82 f gastric disorders such as gastroesophageal reflux disease, autoimmune gastritis, gastric cancer, an
83 have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, obesity, and tobacc
84 assessed 100 patients with gastroesophageal reflux disease before and after sphincter augmentation.
85 e and effective therapy for gastroesophageal reflux disease, but its effect on the LES has not been e
86 s frequently mimic those of gastroesophageal reflux disease, but the diseases are distinct in their h
87 of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophageal pH study (body
88 ar-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost effective and would
89 that is distinguished from gastroesophageal reflux disease by the expression of a unique esophageal
92 in the small subset of patients with severe reflux disease causing a shortened esophagus and strictu
97 se of these technologies in gastroesophageal reflux disease continues to accelerate, and the last 2 y
98 nterval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95% CI: 1.33-1.96).
99 oesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and population-based
100 chest pain associated with gastroesophageal reflux disease, correlates abnormal ambulatory pH monito
101 , regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, laryngitis, GERD, GOR
102 , regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, laryngitis, GERD, GOR
103 ssive surgical treatment of gastroesophageal reflux disease decreases the rate of bronchiolitis and i
104 te chronic tissue injury in gastroesophageal reflux disease differentially affects mechanosensitive a
105 rugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive drugs, sleep aids
106 sses (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, antihypertensive d
107 as functional dyspepsia and gastroesophageal reflux disease (e.g. vomiting, disordered lower esophage
108 n the last 2 years for many gastroesophageal reflux disease endotherapies, providing some insight int
110 onse rates compared to patients with erosive reflux disease (ERD); pH metry contributes to GERD diagn
111 l complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Barrett's esophagus
114 ides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis, treatment, and pr
115 th those from subjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls
116 e interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI, 1.4-2.6), dyspep
117 thophysiological factor in gastro-esophageal reflux disease (GERD) and as a target for GERD treatment
119 incidence and predictors of gastroesophageal reflux disease (GERD) and dyspepsia and their associatio
120 tential association between gastroesophageal reflux disease (GERD) and extraesophageal manifestations
122 an alternative treatment of gastroesophageal reflux disease (GERD) and may provide durable reflux con
123 ential for the treatment of gastroesophageal reflux disease (GERD) and other esophagogastric diseases
124 the Montreal definition of gastroesophageal reflux disease (GERD) and the Rome III definition of fun
125 sophageal manifestations of gastroesophageal reflux disease (GERD) and to compare the most recent tec
126 ion of the cardia indicates gastroesophageal reflux disease (GERD) and/or is a manifestation of panga
127 recipients with documented gastroesophageal reflux disease (GERD) are at increased risk for graft dy
128 urrent diagnostic tests for gastroesophageal reflux disease (GERD) are suboptimal and do not accurate
129 ia who were thought to have gastroesophageal reflux disease (GERD) but who did not respond to medical
134 iding physicians diagnose gastro-oesophageal reflux disease (GERD) have not been evaluated in terms o
135 anges associated with acute gastroesophageal reflux disease (GERD) have not been studied prospectivel
137 as to compare recurrence of gastroesophageal reflux disease (GERD) in children randomized to laparosc
138 ly used in the treatment of gastroesophageal reflux disease (GERD) in children; however, their effica
139 cation for the treatment of gastroesophageal reflux disease (GERD) in comparison with a sham procedur
142 se or eliminate features of gastroesophageal reflux disease (GERD) in some patients whose symptoms pe
149 It has been suggested that gastroesophageal reflux disease (GERD) is a risk factor for developing rh
153 As the economic burden of gastroesophageal reflux disease (GERD) is largely weighted to maintenance
154 he Montreal classification, gastroesophageal reflux disease (GERD) is much more than heartburn and pa
162 iteria included symptoms of gastroesophageal reflux disease (GERD) more than once a month, use of med
164 cid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it preclude a diagnosis
165 y used for the treatment of gastroesophageal reflux disease (GERD) or completing Heller's myotomy and
166 Treatment-refractory gastro-oesophageal reflux disease (GERD) remains a significant problem in t
168 It has been speculated that gastroesophageal reflux disease (GERD) represents a risk factor for the o
169 ed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite proton pump inhib
170 ects a higher prevalence of gastroesophageal reflux disease (GERD) symptoms or a higher degree of eso
174 re matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's esophagus and to
175 f objective measurements of gastroesophageal reflux disease (GERD) would improve management of patien
178 ease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic esophagitis (EoE
179 table bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive bladder syndrome (
180 ysfunction syndrome (RUDS), gastroesophageal reflux disease (GERD), and rare cases of inflammatory pu
181 r three common indications: gastroesophageal reflux disease (GERD), Barrett's esophagus (BE), and non
182 proposed for patients with gastroesophageal reflux disease (GERD), but there is little evidence of i
183 pylori may protect against gastrointestinal reflux disease (GERD), but these results could be due to
184 ation and for patients with gastroesophageal reflux disease (GERD), diabetes mellitus, depression, an
186 ysiological states, such as gastroesophageal reflux disease (GERD), functional dyspepsia and, possibl
187 tion of body mass index and gastroesophageal reflux disease (GERD), including its complications (esop
188 sophagus, a complication of gastroesophageal reflux disease (GERD), predisposes patients to esophagea
189 previous fundoplication for gastroesophageal reflux disease (GERD), underwent reoperative surgery.
214 treatment of patients with gastroesophageal reflux disease (GERD); similarly, laparoscopic technique
215 o included 10 patients with gastroesophageal reflux disease (GERD; age, 32-60 y; 7 women) without tro
216 ere matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to population control
217 ntified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting, and pain) align
219 ardia, the contributions of gastroesophageal reflux disease, H. pylori infection, and other factors t
220 in pediatric patients with gastroesophageal reflux disease have shown good to excellent results; how
221 in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality of Life score 18.2
222 mptom that can be caused by gastroesophageal reflux disease; however, treatment outcome has been diff
223 degenerative joint disease, gastroesophageal reflux disease, hypertension, urinary stress incontinenc
225 associated with symptoms of gastroesophageal reflux disease in both normal-weight and overweight wome
226 in meters - and symptoms of gastroesophageal reflux disease in persons of normal weight has not been
227 cidic environment caused by gastroesophageal reflux disease in the gastroesophageal junction and asso
228 ic approach to patients with extraesophageal reflux disease involved the use of insensitive tools, wh
235 the pathogenesis of heartburn in nonerosive reflux disease is a reaffirmation of the definition of r
238 hagus epithelium related to gastroesophageal reflux disease, is the strongest known risk factor for t
239 ough heartburn is the most common symptom of reflux disease, it is unclear whether the severity of he
240 on "Endoscopic Therapy for Gastroesophageal Reflux Disease." It was approved by the Clinical Practic
242 e esophagus associated with gastroesophageal reflux disease may result in sensitization of afferent p
243 s (asthma, sinusitis, and gastro-oesophageal reflux disease), mental health disorders (depression, po
244 for an esophageal etiology-gastroesophageal reflux disease, motility abnormalities, or esophageal hy
245 rcinogen treatment leads to gastroesophageal reflux disease, multilayered epithelium (MLE, a presumed
246 lication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF vs. n = 28 360 de
247 ere matched to persons with gastroesophageal reflux disease (n = 308) without Barrett's esophagus and
248 indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esophagus (n = 21).
250 t symptom burden in patients with nonerosive reflux disease (NERD) compared with patients with erosiv
253 h improved understanding of gastroesophageal reflux disease, newer developments in diagnostic techniq
254 , including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunctional breathing.
255 cter (LES) in patients with gastroesophageal reflux disease often has a low resting pressure and a sh
256 orically distinguished from gastroesophageal reflux disease on the basis of histology and lack of res
258 proton pump inhibitors for gastroesophageal reflux disease or in patients with any of the nongastroi
259 indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and was correlated to
261 ry of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal bleeding, and prior
262 This article is a review of gastroesophageal reflux disease, other types of esophagitis, benign and m
264 We randomly assigned 64 gastroesophageal reflux disease patients to radiofrequency energy deliver
265 on for selected symptomatic gastroesophageal reflux disease patients who are intolerant of, or desire
266 Too much acid can lead to gastroesophageal reflux disease, peptic ulcer disease, and stress-related
267 eosinophilic bronchitis, gastro-oesophageal reflux disease, postnasal drip syndrome or rhinosinusiti
268 a >50% improvement in their gastroesophageal reflux disease quality of life score (n = 19 [61%] vs. n
269 objective is to evaluate the ability of the Reflux Disease Questionnaire (RDQ) to identify GERD acco
270 ary outcome was symptom control evaluated by Reflux Disease Questionnaire and Reflux Symptom Index.
272 pter reviews the biology of gastroesophageal reflux disease, relating pathophysiology to medical and
273 Endoscopic therapies for gastroesophageal reflux disease represent a minimally invasive alternativ
274 vely high rate of recurrent gastroesophageal reflux disease requiring treatment, diminishing some of
275 f non-drug treatments for gastro-oesophageal reflux disease, safety of long-term drug treatment, and
276 igated whether patients with supraesophageal reflux disease (SERD) have impaired UES and esophageal b
277 ence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infections of the respi
278 fied version of a validated gastroesophageal reflux disease-specific QOL tool to patients before and
279 ty of life, measured by the gastroesophageal reflux disease-specific QOL tool, and recurrence, define
281 proved in group A, with the Gastroesophageal Reflux Disease Symptom Assessment Scale score decreasing
282 very significantly improved gastroesophageal reflux disease symptoms and quality of life compared wit
283 ue in patients with chronic gastroesophageal reflux disease symptoms is of unproven value, and recomm
284 y for patients with chronic gastroesophageal reflux disease symptoms to assess for Barrett's esophagu
285 d Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the prevalence of Barr
286 rs in patients with chronic gastroesophageal reflux disease that involves recurring cycles of inflamm
288 pertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabetes, osteoporosis)
289 tly cleared new endoluminal gastroesophageal reflux disease treatments; however, no controlled trials
293 nocturnal heartburn due to gastroesophageal reflux disease, was approved by the US Food and Drug Adm
296 uamous epithelium caused by gastroesophageal reflux disease, whereas intestinal metaplasia in the dis
300 (BEAC) is a complication of gastroesophageal reflux disease, with no effective chemotherapy and poor
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