ライフサイエンスコーパス: reflux esophagitis

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1 atus and acid-related disease (peptic ulcer, reflux esophagitis).

2 -to-columnar epithelial metaplasia caused by reflux esophagitis.

3 ntestinal-like columnar metaplasia linked to reflux esophagitis.

4 propria papillae, characteristic of chronic reflux esophagitis.

5 ant condition arising in response to chronic reflux esophagitis.

6 e subjects with and control subjects without reflux esophagitis.

7 In total, 230 reflux esophagitis, 224 Barrett's esophagus, and 227 eso

8 s. 6%), a difference that was not found with reflux esophagitis (24% vs. 32%).

9 ts should be considered high risk to develop reflux esophagitis and advised of the management conside

10 R1 is seen at high frequency in inflammatory reflux esophagitis and at all subsequent stages, whereas

11 s of the esophageal epithelium (EE), such as reflux esophagitis and cancer, are rising in incidence.

12 se) or is a significant risk factor, such as reflux esophagitis and gallstones.

13 ht loss, GBP reduces GERD symptoms, improves reflux esophagitis, and decreases esophageal acid exposu

14 Patients with reflux esophagitis are at an increased risk of harboring

15 onship between the use of NSAIDs and risk of reflux esophagitis, Barrett's esophagus, and esophageal

16 have challenged the traditional notion that reflux esophagitis develops when esophageal surface epit

17 For uncomplicated reflux esophagitis, environmental factors appear more im

18 No significant difference was found for the reflux esophagitis group (33% vs. 29%).

19 reflux was defined by abnormal pH-testing or reflux esophagitis >Los Angeles grade A.

20 tinguished patients with EoE from those with reflux esophagitis, identified by pH-impedance testing.

21 arly histologic events in the development of reflux esophagitis in a rat model and performed in vitro

22 n alternative concept for the development of reflux esophagitis in which refluxed gastric juice does

23 hree events (drug inefficacy, gastritis, and reflux esophagitis) in two patients were considered rela

24 c volunteers and 36 patients with endoscopic reflux esophagitis, in basal and parafilm chewing-stimul

25 Reflux esophagitis is believed to be caused by the caust

26 se findings suggest that the pathogenesis of reflux esophagitis may be cytokine-mediated rather than

27 signed to groups with histologic evidence of reflux esophagitis (n = 50), or normal histology results

28 (n = 27), Barrett's esophagus (n = 40), and reflux esophagitis (n = 55) were recruited from tertiary

29 mong GERD cases, we identified patients with reflux esophagitis (RE).

30 Mastication in reflux esophagitis significantly increased salivary volu

31 Reflux esophagitis started at postoperative day 3 with l

32 preliminary study of 12 patients with severe reflux esophagitis successfully treated with PPI therapy

33 llas Veterans Affairs Medical Center who had reflux esophagitis successfully treated with proton pump

34 ated quality of life (HRQoL) with healing of reflux esophagitis to further derive insights into the u

35 verall, 5 % of the on-demand group developed reflux esophagitis versus none in the continuous group (

36 Reflux esophagitis was detected in 24 (45%), 17 (32%), a

37 Reflux esophagitis was seen in 17 cases (32.7%) of SSc a

38 Patients with reflux esophagitis were approached laparoscopically usin

39 626 patients with reflux esophagitis were enrolled into one of two treatme

40 gastric vessel offers effective therapy for reflux esophagitis with > 90% patient satisfaction.

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