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1 -resolution analysis of mouse eye growth and refractive development.
2 n interaction of genetics and environment in refractive development.
3 aminergic activity play a role in regulating refractive development.
4 ng a potential role for circadian rhythms in refractive development.
5 ated the retina as a principal controller of refractive development.
6 assumed that signals from the fovea dominate refractive development.
7 ssessing the effects of visual experience on refractive development.
8 nd GABA(B) receptors modulate eye growth and refractive development.
9 h a history of mild ROP and known courses of refractive development.
10 o develop novel mechanistic hypotheses about refractive development.
11 ID on chicken accommodation, eye growth, and refractive development.
12 us can influence both peripheral and central refractive development.
17 ual processing, retinal nAChRs may influence refractive development and ocular pathologies such as ne
18 al vision is not sufficient to ensure normal refractive development, and the fovea is not essential f
20 ll other subjects with ROP, whose courses of refractive development had been toward emmetropia, and t
23 nfrared photorefraction were used to analyze refractive development in postnatal day (P)21 to P89 C57
26 heses to understand the mechanisms governing refractive development, not only in experimental animals
28 visual factors may influence the growth and refractive development of the human eye after puberty an
29 from the fovea are not essential for normal refractive development or the vision-induced alterations
30 ion of abnormal rod-mediated sensitivity and refractive development suggests that rod-mediated retina
40 visual conditions, only minor differences in refractive development were observed between the nob and
41 t eye or systemic conditions known to affect refractive development were recruited from four geograph
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