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1 =.036 and P =.01, respectively) but not MDS refractory anemia with excess blasts.
2 th multilineage dysplasia (RCMD)-RS, 11 with refractory anemia with excess blasts-1 (RAEB1)-RS, and 5
3 tish classification: refractory anemia (20), refractory anemia with excess blasts (35), refractory an
5 diagnosis, the FAB distinctions between MDS (refractory anemia with excess blasts and refractory anem
6 ts (RCMD-RS) and UPD4q24, and five patients (refractory anemia with excess blasts-II, n = 1; 5q- synd
7 lasts, refractory anemia with excess blasts, refractory anemia with excess blasts in transformation (
8 ewly diagnosed acute myeloid leukemia (AML), refractory anemia with excess blasts in transformation (
9 ory anemia with ringed sideroblasts (9), and refractory anemia with excess blasts in transformation (
10 ewly diagnosed acute myeloid leukemia (AML), refractory anemia with excess blasts in transformation (
11 In current medical practice, patients with refractory anemia with excess blasts in transformation (
12 rthermore, we demonstrate that patients with refractory anemia with excess blasts in transformation (
14 DS (refractory anemia with excess blasts and refractory anemia with excess blasts in transformation)
15 emia with excess blasts and one patient with refractory anemia with excess blasts in transformation.
17 ractory anemia with ringed sideroblasts (5), refractory anemia with excess blasts (RAEB) (4), and RAE
18 ced complementary DNA from bone marrow of 47 refractory anemia with excess blasts (RAEB) patients, 29
23 mic MDS patients (refractory anemia [RA] and refractory anemia with excess blasts [RAEB-1]) with ATG
24 or high-risk myelodysplastic syndrome (MDS) (refractory anemia with excess blasts [RAEB] or RAEB-t, 6
26 refractory anemia with ringed sideroblasts, refractory anemia with excess blasts, refractory anemia
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