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1 ion, 43% +/- 8%) and 17.5% of patients had a regional wall motion abnormality.
2 ection fraction less than 50% or presence of regional wall motion abnormality.
3 ions, left ventricular (LV) thrombus, and LV regional wall-motion abnormality.
4 s in 20 normal subjects and 40 patients with regional wall motion abnormalities.
5 te, automated, and quantitative diagnosis of regional wall motion abnormalities.
6 al/rest flow) to 1.1 to 1.9 without baseline regional wall motion abnormalities.
7 in 12 individuals and > 8% in 5; 3 developed regional wall motion abnormalities.
8 s defined as: 1) development or worsening of regional wall motion abnormality; 2) left ventricular ej
9 g/BSA(1.3); p < 0.001), higher frequency of regional wall motion abnormalities (52% vs. 28%; p = 0.0
10 onal denervation were more likely to have LV regional wall-motion abnormalities (92% versus 52%, P=0.
11 with a dilated left ventricle with global or regional wall motion abnormalities and aneurysm formatio
12 ined echocardiographic parameters, including regional wall motion abnormalities and pulmonary hyperte
13 onin assay, left-ventricular dysfunction and regional wall motion abnormalities, and left-ventricular
14 angiographic collaterals may prevent resting regional wall motion abnormalities but do not appear to
15 h stress cardiomyopathy had left ventricular regional wall motion abnormalities but normal coronary a
16 ) were greater in patients who initially had regional wall motion abnormalities compared to patients
17 or the objective automated interpretation of regional wall motion abnormalities, defined as deviation
22 fusion data are complementary when assessing regional wall motion abnormalities in postischemic myoca
24 r than hibernation is the principal cause of regional wall motion abnormalities in these patients.
25 CT strain analysis has potential to identify regional wall-motion abnormalities in cardiomyopathy tha
26 SIMI, defined as development or worsening of regional wall motion abnormality; left ventricular eject
27 (impaired global RV systolic function and RV regional wall motion abnormalities on cardiac magnetic r
28 Group I had a higher frequency of resting regional wall motion abnormalities on left ventriculogra
29 positive for development of new or worsening regional wall motion abnormality or failure of augmentat
30 right ventricular dysfunction (P=0.01), and regional wall motion abnormalities (P<0.01) were associa
31 s defined as the development or worsening of regional wall motion abnormality, reduction of left vent
32 correlated with LV ejection fraction (LVEF), regional wall motion abnormalities (RWMAs), and peak fil
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