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1 not low-volume aSAH (multivariable logistic regression).
2 ings was examined using multivariable linear regression.
3 detectable or undetectable with log-binomial regression.
4 ciated with PIR were assessed using logistic regression.
5 ictors of cPR were identified using logistic regression.
6 e using bivariate and multivariable logistic regression.
7 nalysis and by univariable and multivariable regression.
8 Risks of death were analyzed using Cox regression.
9 e carried out with ANOVA and multiple linear regression.
10 ching and multilevel, multivariable logistic regression.
11 (>=2 falls) before evaluation using adjusted regression.
12 etween phenotypes was assessed with logistic regression.
13 tervals (CIs) were determined using logistic regression.
14 ere pooled using 3-level random-effects meta-regression.
15 mortality using backwards stepwise logistic regression.
16 mortality between HEU and HUU using Poisson regression.
17 67, 95%CI: 0.45-0.98) only by univariate Cox regression.
18 Pearson's chi-square test, and simple linear regression.
19 cellular adenomas had long-term stability or regression.
20 %) was compared using unconditional logistic regression.
21 regression and with glaucoma using logistic regression.
22 esults generated from multivariable logistic regression.
23 TR was evaluated with multivariable logistic regression.
24 were investigated using logistic and linear regression.
25 as modeled by using Cox proportional hazards regression.
26 Models were tested with use of Cox regression.
29 ere estimated using robust logistic quantile regression, adjusting for age, sex, ethnicity, education
32 rk in murine eyes, which naturally undergoes regression after birth, to gain mechanistic insights tha
35 ardiometabolic markers using multiple linear regression among 15,612 adults aged 40-78 y at baseline
37 o treat by means of multilevel random effect regression analyses adjusting for clustering in health c
40 r-regions, with adjusted linear and logistic regression analyses examining associations with immune p
42 e analyses and multivariable binary logistic regression analyses were conducted on weighted data.
48 d for the entire lung, and multiple logistic regression analyses with areas under the curve (AUCs) as
49 ive, sequence pattern analyses, and logistic regression analyses) aimed to detect any combinations of
61 transcriptomic datasets through elastic net regression analysis to identify a gene signature that ca
83 ally-weighted scatterplot smoothing (LOWESS) regression and change-point analyses and Spearman correl
84 lied stratified linkage disequilibrium score regression and evaluated heritability enrichment in 64 g
85 lopment and validation cohorts: the logistic regression and gradient boosting machine models were tra
86 e tradeoff of quantile g-computation and WQS regression and how these quantities are impacted by the
88 tive propensity score-matched, survival (Cox regression and Kaplan-Meier), and center effects analyse
92 ce of the ANN was evaluated against logistic regression and the standard grading system by analysing
93 -adjusted associations with IOP using linear regression and with glaucoma using logistic regression.
95 s, Spearman correlation coefficients, linear regression, and generalized estimating equation models.
97 around 90 minutes; however, local quadratic regression around the 90-minute cutoff did not reveal ev
98 D) score at waitlist removal for "too sick." Regression assessed the association between social deter
103 The Wilcoxon signed rank test, orthogonal regression, Bland-Altman analysis, and coefficients of v
107 95% confidence intervals (CIs) using Poisson regression, controlling for potential confounding factor
108 imensional classification task with a larger regression dataset, allowing for the creation of deeper
109 units, calculated using proportional hazards regression, declined steadily with age at BMI assessment
111 that incumbent tests (e.g. t-test and linear regression) do not consider, which can lead to false pos
115 ion of Gaussian distributed errors in linear regression for eQTL detection, which results in increase
117 ch 38 loci would be missed within a logistic regression framework with a binary phenotype defined as
121 mmed cell death 1 (PD1) can result in tumour regression in preclinical models and can improve antican
122 mated using mixed-effects linear or logistic regression, including a random effect to adjust for with
123 ter early left ventricular mass index (LVMi) regression is associated with fewer hospitalizations 1 y
125 atform differences by a linear mixed effects regression (LMER) model, and estimate them from matched
127 the number of markers, a penalized multiple regression method can be adopted by fitting all bins to
130 ) over time was assessed with a linear mixed regression model adjusting for the effects of baseline M
135 est; Spearman's correlation and log-binomial regression model estimated the association between MMPs
140 Measurements and predictions of a land-use regression model indicate moderate spatial correlation b
141 an be achieved using a multivariate logistic regression model of MRI parameters after thresholding th
145 The method is based on a piecewise linear regression model that was developed to predict the bound
151 ch of the response using a univariate linear regression model, and to select predictors that meet a p
156 94.2%, 96.9%, 97%, and 94% for the logistic regression model; 92.7%, 100%, 100%, and 92.9% for the I
161 d PROs were investigated using mixed-effects regression models adjusted for clinically-relevant confo
162 5,276) and 15 (n = 3,446) years using linear regression models adjusted for potential confounders.
163 statin C) and ACR with cancer risk using Cox regression models adjusted for potential confounders.
167 2) and persistence from 12 to 24 months into regression models and tested for the mediating effect of
172 ransition status, and multivariable logistic regression models examined factors associated with satis
178 tive outcomes, we used multivariate logistic regression models to adjust for clinical and demographic
179 vidual descriptors and clusters, we used Cox regression models to assess associations with time from
180 nd, in a post-hoc analysis, we used logistic regression models to assess the association between demo
183 or glaucoma versus nonglaucoma from logistic regression models using MRW-BMO values from all sectors
184 permutational multivariate ANOVA and hurdle regression models using the negative binomial distributi
186 idual univariable and multivariable logistic regression models were assessed for each time-weighted-a
200 results among outpatients using mixed-effect regression models with a random effect for study site ho
201 s for mortality were calculated by using Cox regression models with emphysema as the main predictor.
203 rion in a stepwise fashion to build logistic regression models, which were then translated into predi
210 d predicted response to ICS through logistic regression models.Measurements and Main Results: We iden
215 ment due to high response rates and profound regression of malignant melanomas carrying BRAF(V600E) m
216 with various neuromuscular diseases, such as regression of motor neuron axons, motor neuron death, an
218 ndent mouse and primary human cells, causing regression of the malignant clones in vivo, and inducing
220 stry-specific measures, a significant linear regression of total mortality rate (as well as PCa speci
221 ioselectivity, including multivariate linear regression of TS energy, were carried out and the obtain
222 , (ROC analysis, followed by binary logistic regression) only Ultrasound depth is a significant predi
223 fter adjustment using multivariable logistic regression, patients in the high-risk group were more li
225 We compared, by Cox proportional hazards regression, progression-free survival (PFS) after relaps
226 lness and should be dosed "enough," logistic regression, propensity score matching, and inverse proba
227 ses using traditional multivariable logistic regression, propensity score matching, propensity score
228 algorithms were developed based on logistic regression, random forests, gradient boosted trees and a
231 problem: sparse label-noise-robust logistic regression (Rlogreg), robust elastic net based on the le
233 ceptor reflectivity and intact RPE after SDD regression should be seen in the larger context of outer
235 mically scaled tumor volume are estimated as regression splines in a generalized additive mixed model
236 150 minutes by the residuals of a nonlinear regression that predicted TG at 150 minutes as a functio
237 and Basenji, by applying stratified LD score regression to 41 diseases and traits (average N = 320K),
241 xacerbation groups and then used statistical regression to compare this VDP threshold against convent
243 odels were built using Partial Least Squares regression to determine dry matter (DM), soluble solids
246 crossover analysis with conditional logistic regression to estimate the association between hourly pa
247 DVS sales data and difference-in-differences regression to evaluate how WIC authorization affected sa
249 ds during the study period and used logistic regression to examine sociodemographic and clinical fact
252 AD/PD followingly was determined by the Cox regression to identify potential confounding factors.
253 ks and computational paradigms, ranging from regression to image classification and reinforcement lea
254 imputation for missing covariates, logistic regression to model the association between PFAS exposur
255 clones and with Partial Least Squares (PLS) regressions to predict its contents in soluble solids (S
257 nent was developed, and a classification and regression tree was used to stratify patients into diffe
258 t-offs estimated from the classification and regression tree, patients were stratified into different
260 s associated with a significant pathological regression (TRG1-2 = 44% vs 8%, P < 0.001) and a trend t
261 of pre-eclampsia and GHTN with log-binomial regression using generalized estimating equations to acc
266 le case of CLL relapse following spontaneous regression was associated with increased BCR signaling,
268 re performed for each comparison, and a meta-regression was conducted to adjust for use and duration
279 ful changes were defined a priori and linear regression was used to model PCI scores on baseline PCI,
283 riptive statistics and multivariate logistic regression, we examined the association (P < .05) betwee
284 e-crossover design, and conditional logistic regression, we examined the association between source-s
286 Backward selection and multivariate logistic regression were conducted to assess risk of GI adverse e
288 alysis of covariance and multivariate linear regressions were conducted with sleep-related variables
292 arkers were assessed by multivariable linear regression, whereas associations between TMAO and the fe
293 thod is based on Bayesian variable selection regression, which not only accounts for cis- and trans-e
294 tions to predict GGG 1 vs >1, using logistic regression with a nested leave-pair out cross validation
295 life-EQ-5D-5L on a 0 to 1 scale-using linear regression with adjustment for patient, tumor, and treat
297 e applied multiexposure linear mixed-effects regressions with participant-level random intercept to i
299 s (VE) was estimated by conditional logistic regression, with adjustment for reported contact with ch
300 ves were estimated by weighted least squares regression (WLS), confirming heteroscedasticity for all