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1 conducted using the Cox proportional hazards regression model.
2 ed using a multivariate proportional hazards regression model.
3 were assessed using a hierarchical, logistic regression model.
4 ored in a similarly adjusted multiple linear regression model.
5 we refer to as the latent Dirichlet process regression model.
6 score was calculated and then entered into a regression model.
7 ed by the ensemble predictions obtained in a regression model.
8 having early or late tears using a segmented regression model.
9 rate was assessed using a negative binomial regression model.
10 incident HF was analyzed by using a Poisson regression model.
11 ing regimen, were included in a multivariate regression model.
12 structure to directly improve the log-linear regression model.
13 compared by using a Cox proportional hazards regression model.
14 mpared with reference patients in a logistic regression model.
15 implementation, analyzed using the segmented regression model.
16 residual analysis based on a multiple linear regression model.
17 independent predictors of gait speed in the regression model.
18 Results were analyzed using a logistic regression model.
19 lanomas was analyzed using an exact logistic regression model.
20 risks of outcomes were estimated by Poisson regression models.
21 ined predictors of outcomes with appropriate regression models.
22 ay on treatment effectiveness using logistic regression models.
23 of higher AE rates using generalized linear regression models.
24 nivariate analysis and multivariate logistic regression models.
25 d-use data into seasonally adjusted land-use regression models.
26 risk factors for ESRD using multivariate Cox regression models.
27 HD in offspring were analyzed using logistic regression models.
28 ed using univariate and multivariable linear regression models.
29 triacylglycerols through multivariate linear regression models.
30 he output of DHR models to static sinusoidal regression models.
31 al group were calculated with the use of Cox regression models.
32 nd asthma (n = 1405) were investigated using regression models.
33 linear and generalized linear mixed-effects regression models.
34 ases using propensity score matching and Cox regression models.
35 r of diagnosis, were estimated using Poisson regression models.
36 sex, were examined using linear and Poisson regression models.
37 dence rates were assessed by fitting Poisson regression models.
38 mes between 1994 and 2012 using adjusted Cox regression models.
39 in the TD/CTD cohort were studied using Cox regression models.
40 and LTL was evaluated using multiple linear regression models.
41 lasma scores directly from food intake using regression models.
42 nalyzed in 381 participants by using Poisson regression models.
43 to all phenotypes using logistic and linear regression models.
44 d nonstatin LLT use in hierarchical logistic regression models.
45 midlife using quantile, linear, and logistic regression models.
46 tested using univariate and multivariate Cox regression models.
47 tion (PSD), were analyzed with multivariable regression models.
48 ion method based on latent Dirichlet Process regression models.
49 nd function by multivariable-adjusted linear regression models.
50 lities for four common variants of threshold regression models.
51 from multivariable Cox proportional hazards regression models.
52 motional outcome were calculated in multiple regression models.
53 followed by generalized estimating equation regression modeling.
54 s and controls with Cox proportional hazards regression modeling.
55 year were quantified by multivariate linear regression modeling.
56 assessed by generalized linear mixed method regression modeling.
57 thods using 2 independently derived logistic regression models (a de novo model and an a priori model
59 otic melanomas were evaluated using logistic regression models adjusted for age, sex, study center, a
61 d with the use of proportional probabilities regression models adjusted for potential confounders.
62 ere assessed using linear fixed-effect panel regression models adjusted for smoke-free policies, gros
66 h outcome, we estimated conditional logistic regression models adjusting for race/ethnicity, history
69 tality was obtained from multilevel logistic regression model, adjusting for demographics, mechanism,
70 ly (per 5% of energy) were obtained from Cox regression models, adjusting for demographic factors, me
71 ed using multivariable hierarchical logistic regression models, adjusting for important prognostic fa
74 new pipeline was built based on a meta-Lasso regression model and the proof-of-concept study was perf
76 trends from 2009-2014 were calculated using regression models and compared with claims-based estimat
78 nsity score-matched Cox proportional hazards regression models and Kaplan-Meier survival analysis.
79 d a series of linear continuous and logistic regression models and non-linear restricted cubic spline
83 109) were investigated using multiple linear regression models and random intercept and random slope
85 binary indicator (exposed/nonexposed) to the regression model, and a method based on fractional polyn
86 sed Cox proportional hazard models, logistic regression models, and Fine-Gray competing risk regressi
88 operating characteristic curve for the full regression model applied to the NLST database demonstrat
92 lative and fish quality control aspects, PLS regression model based on spectral range 1139.9-1643.7cm
93 al severity with linear and ordinal logistic regression models before and after adjusting for covaria
95 Such method consists of fitting whole-genome regression models by subsampling observations in each ro
99 ng the relative magnitude of the exponential regression model coefficients of independent predictors
100 iosensitive variable improved lasso logistic regression models compared to model performance without
101 We used published data to create logistic regression models comparing annual trends in the represe
109 atios (HR) and 95% CIs with multivariate Cox regression models fitting stromal TILs as a continuous v
114 as the independent variable in multivariable regression models for association with primary intracere
116 id tool for screening huge numbers of linear regression models for significant interaction terms in a
121 timate PM2.5 concentrations, many parametric regression models have been developed, while nonparametr
127 oxidative stress, and the utility of complex regression models in capturing mediated associations whe
128 t day 28 were retrospectively analyzed using regression models in different subgroups of patients.
136 t test, the Mann-Whitney test, and logistic regression modeling of sample adequacy were performed.
137 e contributions of these pathways using meta-regression models of published data on dust pesticide co
142 s used to identify covariates for a logistic regression model predictive of severe ADAMTS13 deficienc
147 hat our previous work using RSA based linear regression model resulted out higher prediction quality
152 In addition, a two-step hierarchical linear regression model showed that significant predictors of B
154 whole, our final analysis based on logistic regression models showed that higher NDVI was a predicto
156 parasite prevalence, and multilevel Poisson regression models showed that such differences were infl
162 nivariable and multivariable stepwise linear regression models, taking family structure into account.
165 bined flow-cytometry variables in a multiple regression model that identified individuals with celiac
166 relative risk was observed for the land-use regression model that included traffic information (RR =
167 in early childhood, and we estimate flexible regression models that allow for nonlinearities in the r
168 sed risk metrics have been based on logistic regression models that incorporate aspects of the medica
169 analyzed these data using mixed-effects meta-regression models that weighted each summary statistic b
178 nscripts, we used a cross-validated logistic regression model to identify the presence of HCC-derived
181 ly downscaled using an asynchronous regional regression model to provide finer resolution future clim
182 dataset and then used mixed-effects logistic regression modeling to determine the effect of NAI treat
183 by applying generalized linear least-squares regression modelling to components of the multiproxy dat
186 on kernel and were then fitted with logistic regression models to classify steatosis, that were then
187 meters over time, and used repeated-measures regression models to define their association with LVEF
188 were tested by using multivariable logistic regression models to determine which combination of metr
190 diabetes mellitus (GDM), and we used linear regression models to estimate associations with first-tr
193 ansport models, and satellite-based land use regression models to estimate neighborhood annual averag
197 We used multivariable time-dependent Cox regression models to evaluate vaccine effectiveness, acc
198 types, and covariates, we used robust linear regression models to examine associations of prenatal le
201 METHODS AND We fit mixed-effects logistic regression models to routine surveillance data recording
204 zard ratios were estimated with weighted Cox regression models using Barlow weights to account for th
209 To adjust for selection bias, a logistic regression model was created to estimate odds ratios for
216 A facility-level fixed-effects quasi-Poisson regression model was used to examine the incidence of ur
218 mong this cohort, a Cox proportional hazards regression model was used to identify predictors of surv
237 etween potato consumption and mortality, Cox regression models were constructed to estimate HRs with
243 e, sex, and body mass index-adjusted) linear regression models were fitted to study the association b
251 CIs calculated from Cox proportional hazards regression models were used to assess the relationship b
256 Meier estimates and Cox proportional hazards regression models were used to calculate hazard ratios.
262 >/=3 months, and population average logistic regression models were used to determine risk factors fo
272 ntially confounding covariates, and logistic regression models were used to estimate the risk of pre-
281 d sex-adjusted and multivariate-adjusted Cox regression models, whatever the significance threshold r
283 t identifying relevant pathways; 2) Use of a regression model whose covariates embed all method-drive
287 ) were calculated using conditional logistic regression models with adjustment for important covariat
288 rcent effect changes in conditional logistic regression models with and without additional adjustment
289 Data were analyzed using multiple logistic regression models with backward stepwise elimination (Pr
290 ts on use of coping strategies and mediation regression models with bias-corrected bootstrapping to e
291 HOD: We estimate cross-sectional, ecological regression models with data from 27 European countries o
293 ned Gaussian process (GP) classification and regression models with expression and localization data
294 VL > 40 copies/mL) were estimated by Poisson regression models with generalized estimating equations
295 eline (time 0) biomarker profiles-both using regression models with interaction terms for treatment a
296 st episode of self-harm were analyzed in Cox regression models with time-varying treatment, adjusted
297 waitlisted patients using a multivariate Cox regression model, with a competing risk approach as a se
298 on with QOL was then assessed using a linear regression model, with binocular 10-2 VF sensitivity as
299 coabundance groups were used as outcomes in regression models, with prenatal/birth and demographic c
300 easures at 35 years (wave 10) using logistic regression models, with progressive adjustment: (1) adju
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