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1 CD4(+)T/Treg and CD8(+)T/Treg ratios (Treg, regulatory T cells).
2 and number of thymic and peripheral Foxp3(+) regulatory T cells.
3 on in the number and suppressive activity of regulatory T cells.
4 resenting cells to innate lymphoid cells and regulatory T cells.
5 ecome anergic or differentiate into Foxp3(+) regulatory T cells.
6 T cells but also increases the frequency of regulatory T cells.
7 y CD4 T cells and constrained in severity by regulatory T cells.
8 in vitro that mainly drives lesions, but not regulatory T cells.
9 amma production and expanding CD4(+)IL-10(+) regulatory T cells.
10 hey contribute to the generation of Th17 and regulatory T cells.
11 expanded Foxp3(+)CXCR5(+)PD-1(+) follicular regulatory T cells.
12 een groups receiving wild-type or NLRX1(-/-) regulatory T cells.
13 al tolerance and enhances differentiation of regulatory T cells.
14 mune-suppressive cytokine mainly produced by regulatory T cells.
15 ell numbers and supported differentiation of regulatory T cells.
16 lly contributed to the protective actions of regulatory T cells.
17 s aspect here, particularly as it relates to regulatory T cells.
18 are more similar to effector T cells than to regulatory T cells.
19 itors markedly suppress the proliferation of regulatory T cells.
20 pathogens by driving the differentiation of regulatory T cells.
21 uppression of proliferation and cytokines by regulatory T cells.
22 ly on cytotoxic T cells, T-helper cells, and regulatory T cells.
23 the effector subsets Th1, Th17, and induced regulatory T cells.
24 ity to differentiate into induced/peripheral regulatory T cells.
25 namely in transplantation, requires Foxp3(+) regulatory T cells.
26 nflammatory phenotype of Aspergillus-induced regulatory T-cells.
35 crophage infiltration and higher presence of regulatory T cell after ischemia-reperfusion injury.
36 y primed antigen-specific forkhead box P3(+) regulatory T cells after in vitro coincubation with OVA/
37 expansion of forkhead box protein 3-positive regulatory T cells along with suppression of existing TH
38 , along with increased numbers of intestinal regulatory T cells and a higher production of luminal Ig
39 2s by co-stimulatory molecules such as ICOS, regulatory T cells and by compounds such as nicotine.
40 ia governing the balance between T helper 17/regulatory T cells and by regulating the formation of ef
41 expression of Gal-9 drives the expansion of regulatory T cells and contraction of effector T cells,
42 nsion of effector T cells and instability of regulatory T cells and contributes to T cell-dependent t
43 characterized by high levels of intrahepatic regulatory T cells and expression of inhibitory molecule
44 ning, but were accompanied by suppression of regulatory T cells and greater influx of activated CD8(+
45 d the cutaneous microenvironment by limiting regulatory T cells and IFN-gamma and inhibiting IL-10 pr
46 mechanism operating on the surface of human regulatory T cells and involving membrane protein GARP,
47 96 is essential for CD11c(+) cells to induce regulatory T cells and maintain gut homeostasis, illustr
48 ppressive cells at the tumor site, including regulatory T cells and myeloid-derived suppressor cells,
49 of profoundly decreased CTLA4 expression on regulatory T cells and probably contributes to autoimmun
50 se in the frequency of mTGFbeta(+)Foxp3(int) regulatory T cells and the persistence of CD206(+) macro
52 function in peripheral CD4(+)CD25(+)FOXP3(+) regulatory T cells, and an impaired capacity of CD4(+)CD
53 on in naive T cells, helper Th17 T cells and regulatory T cells, and Ca(2+) signals mediated in T cel
54 ulations including hematopoietic stem cells, regulatory T cells, and facilitating cells are being con
56 ntrol TFH cell maturation, expand follicular regulatory T cells, and inhibit the TFH cell-mediated an
57 o autoimmunity by promoting anergy, inducing regulatory T cells, and inhibiting effector T cells.
58 lterations in the percentage of lymphocytes, regulatory T cells, and T-helper 1 and 17 cells, all maj
59 DK4/6 inhibitors both on tumour cells and on regulatory T cells are associated with reduced activity
60 apy mediated via the sustained generation of regulatory T cells arguably represents the ideal therape
64 ociated with significantly higher numbers of regulatory T cells both in aortic sinus and spleen with
67 of CD25 function abolished the induction of regulatory T cells by IL-10DCs, indicating a critical ro
68 e-positive thymocytes, CD4(+)CD8(-) T cells, regulatory T cells, CD4(+) T cell marker expression, lif
71 demonstrated that soluble CD137 produced by regulatory T cells contributed to their autoimmune-suppr
76 n of naive T cells into T helper 17 cells or regulatory T cells creates subtype-specific enhancer-pro
77 induction increased numbers of lung and gut regulatory T cells, decreased lung TH2 responses, and am
78 ion-chemokine induction, T-cell stimulation, regulatory T cell depletion-was observed at all dose lev
82 RGC-32(-/-) mice have normal Th1, Th2, and regulatory T cell differentiation but show defective Th1
83 resented premature defects in mTEC-dependent regulatory T-cell differentiation and thymocyte maturati
84 inflamed islets and suggest that therapeutic regulatory T cells directly or indirectly regulate their
87 oups of patients, tolDCs were able to induce regulatory T cells from autologous naive CD4(+) T cells.
88 guished Vbeta2(+), Vbeta6(+) and Vbeta8.2(+) regulatory T cells from conventional T cells and also di
90 gnificant increase in CD4CD25forkhead box P3 regulatory T cells (from 18% to 25%, P = 0.0002) and a c
91 FOXP3 is the transcription factor ruling regulatory T cell function and maintenance of peripheral
92 findings identify Batf as critical to tissue regulatory T cell function and suggest that polymorphism
96 atment combined with systemic elimination of regulatory T cells had potent synergistic effects agains
97 atory proteins CD80/CD86, signals needed for regulatory T cell homeostasis, are upregulated less on N
100 -driven inflammatory responses and increased regulatory T cells in atherosclerotic arteries and lymph
102 Interestingly, the percentage of Foxp3+ regulatory T cells in liver transplant patients was stab
104 ntial role for lactationally transferred dam regulatory T cells in suppression of the ex vivo respons
108 R transgenic mice, we show that depletion of regulatory T cells in the setting of lymphatic injury re
109 t interferon gamma production by a subset of regulatory T cells in the tumor microenvironment trigger
110 leukin in the Adaptive Study of IL-2 Dose on Regulatory T Cells in Type 1 Diabetes (DILT1D), a single
111 tion of antigen-specific, inducible FoxP3(+) regulatory T cells in vitro and in vivo, and by reduced
112 G3)(+), CD49b(-), forkhead box P3 (Foxp3)(-) regulatory T cells in vitro, such that beta subunit of I
116 ession of IL-33 promoted expansion of ST2(+) regulatory T cells, increased Th2 cytokine milieu, and i
117 ells, and CD70 limits T cell expansion via a regulatory T cell-independent mechanism that involves ca
118 ts and CD21(low) B cells, as well as TH2 and regulatory T cells, indicating a common disease pathogen
119 Emerging evidence reveals that a group of regulatory T cells, induced Tregs (iTregs), effectively
120 y in a mouse strain-dependent manner through regulatory T cell induction among lymphocytes in the int
121 the gut as well as loss of antigen-specific regulatory T cell induction in the mesenteric lymph node
122 e to allogeneic antigens, strongly promoting regulatory T-cell induction and inhibiting T-cell tumour
123 f the blood-brain barrier, we confirmed that regulatory T cells inhibited tPA-induced endothelial exp
127 herapeutic group, the frequencies of induced regulatory T cells (iTregs: CD4(+)CD25(-)Fopx3(+)) and C
131 of PD-L1, and contained within their stroma regulatory T cells, M2 (protumor) macrophages, and exhau
132 We show that a population of CD4CD127CD45RA Regulatory T cell may offer the best compromise between
133 Foxp3Treg cell was performed in DEpletion of REGulatory T cells mice at day 80 posttransplantation.
134 rgies and suggest that induction of Foxp3(-) regulatory T cells might be a useful strategy for tolera
135 hair follicle development, commensals induce regulatory T cell migration to the skin to ensure cutane
137 ve failed to investigate the role of natural regulatory T cells (nTregs) on the mitigation of GvHD by
139 CD8(+) T cells, but showed no differences in regulatory T-cell numbers relative to patients without I
142 cific T cells; induction of antigen-specific regulatory T cells; or immune deviation in favor of TH1
143 , had a lower percentage of CD4+CD25hiFoxP3+ regulatory T cells (P < .01), but higher proportions of
146 T cells and to increase their sensitivity to regulatory T-cell polarization signals, both processes r
148 elper type 2 cell (Th2 cell), Th17 cell, and regulatory T cell populations while suppressing Th1 cell
150 ces in the percentages of immune-suppressive regulatory T cells, pro-inflammatory Th17 cells, or thei
152 y T cells in immunosuppression and show that regulatory T-cell proliferation in tissues distal to sit
154 of oral tolerance by suppressing peripheral regulatory T cell (pTreg) conversion and promoting TH1 i
157 hemagglutinin (HA) which potentially enhance regulatory T cell response due to conservation with the
158 rleukin-17/21 inhibition, kinase inhibition, regulatory T-cell restoration, and CSF-1 inhibition.
159 dependent IL-10 production by progranulin in regulatory T cells restrains inflammatory arthritis.
167 er, simultaneous expansion of CD4(+)Foxp3(+) regulatory T cells (T reg cells) impedes protective resp
169 ependent and is mediated by the induction of regulatory T cells that share a similar transcriptional
170 cular regulatory (Tfr) cells are a subset of regulatory T cells that suppress T follicular helper (Tf
171 h autophagy favours the lineage stability of regulatory T cells, the contribution of autophagy to the
172 therapeutics that can rebalance the ratio of regulatory T cells to effectors are a relevant issue.
174 sociated with higher IL-10 concentration and regulatory T cell-to-leukocyte ratio in RIPK3 knockout m
176 tigate the therapeutic potential of adoptive regulatory T cell transfer against tPA-induced haemorrha
178 but not conventional CD4(+) T-cell subsets [regulatory T cell (Treg) and conventional T cell (Tconv)
179 own whether these factors are needed for the regulatory T cell (Treg) arm of MHC class II responses.
180 receptor under the Foxp3 promoter, transient regulatory T cell (Treg) depletion results in long-lasti
182 terferon regulatory factor 4 (IRF4) promotes regulatory T cell (Treg) generation by enhancing express
184 sion of CTL killing by CD4(+)CD25(+)Foxp3(+) regulatory T cell (Treg) is in part mediated by TGFbeta-
186 regulatory T (TFR) cells are a newly defined regulatory T cell (Treg) subset that suppresses follicul
188 L4 deficiency compromised the development of regulatory T cells (Treg cells) and resulted in a substa
189 balance in the lineages of immunosuppressive regulatory T cells (Treg cells) and the inflammatory TH1
191 NKG2D, a decrease in CD4(+) CD25(+) FOXP3(+) regulatory T cells (Treg cells) as well as CD25(+) CD4(+
192 sgenic mice are predominantly CD4(+)Foxp3(+) regulatory T cells (Treg cells) expressing tolerogenic c
199 identified as a key transcription factor for regulatory T cells (Treg cells), was also expressed in m
201 Approaches that enhance the function of regulatory T cells (Treg) and dampen effector T cells ca
203 atic ILC2s but also expanded CD4(+) Foxp3(+) regulatory T cells (Treg) expressing the IL-33 receptor
210 together with increased Th1, Th2, Th17, and regulatory T-cell (Treg) CD4(+) subsets; (2) significant
212 Direct mutation in CTLA-4 leads to defective regulatory T-cell (Treg) function associated with impair
213 pulation of immunosuppressive T-cells called regulatory T-cells (Treg) regulates proliferation of imm
215 on of the Foxp3 gene leads to the absence of regulatory T cells (Tregs) and a fatal systemic autoimmu
217 lergy season by increasing the percentage of regulatory T cells (Tregs) and inducing tolerance.The ob
218 AE) as a result of an increase of protective regulatory T cells (Tregs) and reduction of encephalitog
219 constitutively expressed on CD4(+) Foxp3(+) regulatory T cells (Tregs) and TNF-TNFR2 interaction is
226 feration, the distinct metabolic features of regulatory T cells (Tregs) are less well established.
230 e demonstrated that the adoptive transfer of regulatory T cells (Tregs) can mediate the inflammation
234 GVHD risk and appears to initially depend on regulatory T cells (Tregs) followed by gradual, presumab
236 es might be harnessed to selectively promote regulatory T cells (Tregs) for therapies in autoimmunity
237 to their ability to suppress effector cells, regulatory T cells (Tregs) have been proposed as a cellu
242 ecent studies showed that the dysfunction of regulatory T cells (Tregs) is directly linked to the ini
243 tivation of T cells and their suppression by regulatory T cells (Tregs) is dysregulated in autoimmune
254 ng tumor-associated macrophages and FoxP3(+) regulatory T cells (Tregs) within the tumor microenviron
255 s (myeloid-derived suppressor cells (MDSCs), regulatory T cells (Tregs), and programmed death-1 (PD-1
256 iously generated engineered antigen-specific regulatory T cells (Tregs), created by transduction of a
257 unosuppression in association with increased regulatory T cells (Tregs), making this transcription fa
258 results in expansion of Foxp3(+) and LAP(+) regulatory T cells (Tregs), suggesting oral delivery of
259 metry, we found that the TME was enriched in regulatory T cells (Tregs), tissue resident memory CD8(+
260 actor that drives lineage differentiation of regulatory T cells (Tregs), was thought to imprint a uni
261 articles is known to induce antigen-specific regulatory T cells (Tregs), we adapted this approach to
278 by increasing the stability and function of regulatory T cells using the DNA methyltransferase inhib
279 ction of peripheral T-cell subsets including regulatory T cells was observed after the first dose of
280 ugh most tolerance-inducing regimens rely on regulatory T cells, we found that induction of tolerance
285 T cells, which share phenotypic markers with regulatory T cells, were enriched in SIV DNA in blood, l
286 ic cells, Th2 cells, follicular T cells, and regulatory T cells, where IL-33 influences the developme
287 tolerance to atherogenic peptides increases regulatory T cells which activate M2 macrophages, preven
288 ATP citrate lyase, induced the depletion of regulatory T cells (which dampen anticancer immunity) fr
290 or immune cells, and decreased the number of regulatory T cells, which in turn killed a larger number
291 This in turn promotes the activation of regulatory T cells while impairing Th1 response; however
293 rogram inducing the accumulation of Foxp3(+) regulatory T cells, while decreasing the levels of Th17
295 n conclusion, our results suggest a role for regulatory T-cells with a pro-inflammatory TH17-like phe
298 ly, this reduction resulted in a decrease in regulatory T cells, with an increase in myelin basic pro
299 on was paralleled by an increase in FoxP3(+) regulatory T cells within the cellular infiltrates aroun
300 ually repopulated the numbers of circulating regulatory T cells within the first 7 days after stroke,
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