ライフサイエンスコーパス: regurgitant

1 two injured leaves treated with caterpillar regurgitant.

2 ponse to simulated attacks and produced less regurgitant.

3 he potential of providing direct measures of regurgitant and shunt flow.

4 lvular heart disease is the primary cause of regurgitant and stenotic valvular lesion in the U.S.

5 the natural history of combined stenotic and regurgitant aortic valve disease.

6 Repair of regurgitant aortic valves is not widely accepted, but in

7 te a new approach to bicommissural repair of regurgitant aortic valves.

8 volatile organic compounds after caterpillar regurgitant application.

9 This variant of the purely regurgitant BAV may cause either chronic AR (when the an

10 ventricular outflow tract forward and aortic regurgitant blood flows.

11 On the basis of the finding that caterpillar regurgitant can reduce the amount of toxic nicotine rele

12 The regurgitant contains a series of these compounds with fa

13 The presence of IAN in larval regurgitant contributes to reduced oviposition by adult

14 s replaced the mitral valve in patients with regurgitant disease.

15 rs (EMFs) as reference standards, and aortic regurgitant effective orifice areas (EOAs) were determin

16 ion remains the leading cause of stenotic or regurgitant failure in native human and porcine bioprost

17 the volume of regurgitation, the pattern of regurgitant flow across the mitral valve was not signifi

18 clinically observed midsystolic decrease in regurgitant flow and orifice area as transmitral pressur

19 have identified distinct patterns of mitral regurgitant flow disturbances in patients with mitral pr

20 UBM showed increasing abnormal regurgitant flow in the aorta and extending into the emb

21 s study was to quantify and characterize the regurgitant flow pattern and regurgitant orifice area in

22 e the effect of dynamic variations of mitral regurgitant flow rate (MRFR) and effective regurgitant o

23 In functional mitral regurgitation (MR), regurgitant flow rate and orifice area display a unique

24 The continuity principle dictates that regurgitant flow rate can be calculated as the product o

25 size, and transmitral pressure, with direct regurgitant flow rate measurement, to test the hypothesi

26 r = 0.95, SEE = 0.05 cm, p < 0.0001 for peak regurgitant flow rate; r = 0.85, SEE = 0.08 cm, p < 0.00

27 =0.01 cm2) and between 3D and reference peak regurgitant flow rates and regurgitant stroke volumes (r

28 Peak regurgitant flow rates and regurgitant stroke volumes we

29 rifice areas (EOAs) were determined from EMF regurgitant flow rates divided by continuous-wave (CW) D

30 Instantaneous regurgitant flow rates were obtained by aortic and pulmo

31 Instantaneous regurgitant flow rates were obtained by aortic and pulmo

32 MR was quantified as peak and mean regurgitant flow rates, regurgitant stroke volumes and r

33 lied by the aliasing velocity to obtain peak regurgitant flow rates.

34 an be used to quantify both aortic EROAs and regurgitant flow rates.

35 er workstation, the RV forward and pulmonary regurgitant flow volumes were obtained by a program that

36 integral (VTI) = midsystolic MRFR by PISA x regurgitant flow VTI/peak velocity; 2) simplified PISA =

37 R caused similar color jet area, midsystolic regurgitant flow, and peak velocity (P>0.40).

38 a provides an accurate direct measurement of regurgitant flow, overcoming the limitations of existing

39 vena contracta (VC-W), the smallest area of regurgitant flow, reflects the degree of valvular regurg

40 flow tract forward flow and ascending aortic regurgitant flow.

41 data for color Doppler imaging of the aortic regurgitant flows were transferred into a TomTec compute

42 h high accuracy: 85% of the 39 subjects with regurgitant fraction >33% progressed to surgery (mostly

43 s) in comparison with 8% of 74 subjects with regurgitant fraction </= 33% (P<0.0001); the area under

44 A similar separation was observed for regurgitant fraction </=40% and >40%.

45 t pulmonary regurgitation (less than mild or regurgitant fraction <10% on magnetic resonance imaging

46 Peak E wave velocity correlated with regurgitant fraction (r = 0.52, p < 0.001).

47 The MR Index correlated with regurgitant fraction (r = 0.76, p < 0.0001).

48 icantly correlated with reductions in mitral regurgitant fraction (r = 0.77, p < 0.001).

49 e quantitation of regurgitant volume (RVol), regurgitant fraction (RF) and effective regurgitant orif

50 AR was quantified using regurgitant fraction (RF) measured by phase-contrast vel

51 ejection fraction, RV volumes, and pulmonary regurgitant fraction (RF).

52 us intraoperative flow probe measurements of regurgitant fraction (RgF) and regurgitant volume (RgV).

53 re at 90 days corrected the volume overload (regurgitant fraction 6 +/- 5% versus 27 +/- 16% for late

54 nd C statistics) was performed, with HDR and regurgitant fraction as independent predictors.

55 volume from 40 +/- 20 ml to 24 +/- 17 ml and regurgitant fraction from 40 +/- 12% to 25 +/- 14% (both

56 to quantitate aortic regurgitant volume and regurgitant fraction in a chronic animal model with surg

57 Pulmonary regurgitant fraction increased from 32.8+/-15% to 49.6+/

58 Over time, aortic regurgitant fraction increased slightly but significantl

59 In animals with PI, pulmonary regurgitant fraction progressed more in the presence of

60 8), but the combination of this measure with regurgitant fraction provided the best discriminatory po

61 c magnetic resonance imaging RVol r=0.84 and regurgitant fraction r=0.80.

62 evaluation by an expert and quantitation of regurgitant fraction using two-dimensional and Doppler e

63 evaluation by an expert and quantitation of regurgitant fraction using two-dimensional and Doppler e

64 lar ejection fraction was 60+/-8%, pulmonary regurgitant fraction was 34+/-17%, and right ventricular

65 ed across the pulmonary valve, the pulmonary regurgitant fraction was 37%; this was not seen in the a

66 In the PI group, pulmonary regurgitant fraction was 49.2+/-5.9% at 3-month follow-u

67 Regurgitant fraction was consistently >50% over the cour

68 A strong decline in pulmonary regurgitant fraction was observed after hTPV implantatio

69 Pulmonary flow volumes and regurgitant fraction were quantified by velocity-encoded

70 1.2 +/- 7.6% vs. 19 +/- 13%, p < 0.0001 for regurgitant fraction).

71 ume; r = 0.90, SEE = 0.07 cm, p < 0.0001 for regurgitant fraction).

72 ted leaflet closure and mild-to-moderate MR (regurgitant fraction, 25.2+/-2.8%).

73 tricle-to-left atrial shunt implanted in 12 (regurgitant fraction, 30%).

74 ume, 62+/-45 mL and r=.80 (P<.0001); and for regurgitant fraction, 45+/-17% and r=.78 (P<.0001).

75 mL, 50%, and 40 mm2 for regurgitant volume, regurgitant fraction, and orifice, respectively.

76 is feasible, significantly reduces pulmonary regurgitant fraction, facilitates right ventricular volu

77 ble right ventricular function and pulmonary regurgitant fraction, on exercise stress test the 22q11.

78 added little to the discriminatory power of regurgitant fraction/volume alone.

79 The RSVs and regurgitant fractions (RFs) obtained by the DCD method u

80 Regurgitant volumes and regurgitant fractions by the new method agreed well with

81 As a result of these measurements, the regurgitant fractions derived by the 3D method agreed we

82 t flow rates, regurgitant stroke volumes and regurgitant fractions determined using mitral and aortic

83 rial shunt implanted, consistently producing regurgitant fractions of approximately 30%.

84 m 7.0 to 48.0 (26.9+/-12.2) mL/beat, and the regurgitant fractions varied from 23% to 78% (55+/-16%).

85 ides accurate aortic regurgitant volumes and regurgitant fractions without cumbersome measurements.

86 etermining pulmonary regurgitant volumes and regurgitant fractions.

87 oderate mitral regurgitation) and changes in regurgitant grade at each heart valve were evaluated.

88 rgitant orifice causes, despite a decline in regurgitant gradient, a notable increase in regurgitant

89 ied 27 patients with severe TR, defined by a regurgitant index (RI) >33%, who underwent PTE.

90 ntinuous wave Doppler characteristics of the regurgitant jet and tricuspid regurgitant jet-derived pu

91 x echocardiographic variables: color Doppler regurgitant jet penetration and proximal isovelocity sur

92 Patients with a tricuspid-valve regurgitant jet velocity >/=3.2 m/s (3.6%) on transthora

93 ive predictive value for the tricuspid-valve regurgitant jet velocity >/=3.2 m/s threshold for the di

94 An elevated tricuspid regurgitant jet velocity (TRV) is associated with hemoly

95 y associated with PH, defined as a tricuspid regurgitant jet velocity (TRV) of at least 2.5 m/s.

96 Abnormal tricuspid regurgitant jet velocity (TRV) was defined as more than

97 d vascular complications (elevated tricuspid regurgitant jet velocity [TRV], microalbuminuria, leg ul

98 g echocardiography to measure peak tricuspid regurgitant jet velocity and by evaluating plasma levels

99 nsion and correlated directly with tricuspid regurgitant jet velocity in the NIH cohort (R = 0.50, P<

100 A tricuspid regurgitant jet velocity of at least 2.5 m per second, a

101 ion was prospectively defined as a tricuspid regurgitant jet velocity of at least 2.5 m per second.

102 e of the dichotomous variable of a tricuspid regurgitant jet velocity of less than 2.5 m per second o

103 Elevated tricuspid regurgitant jet velocity, pulmonary hypertension, diasto

104 Methods to directly measure the regurgitant jet vena contracta area are presented, along

105 The mitral regurgitant jet was central in origin in 12 group 1 pati

106 nar flow acceleration zone and the turbulent regurgitant jet, has been reported to be a clinically us

107 ristics of the regurgitant jet and tricuspid regurgitant jet-derived pulmonary artery pressure, pulse

108 of the mitral leaflets at the origin of the regurgitant jet.

109 lic volume index, and the area of the mitral regurgitant jet; increased the left ventricular ejection

110 precise anatomy, and visualization of mitral regurgitant jets in mitral valve prolapse.

111 There were multiple regurgitant jets in three of five (60%) patients in this

112 hat this held only for laminar flow, not for regurgitant jets, in which turbulence and fluid entrainm

113 also to help discern the consequences of the regurgitant lesion on left ventricular performance.

114 accuracy and outcome implications of mitral regurgitant lesions assessed by echocardiography.

115 Recent efforts into the inflow position and regurgitant lesions, with transcatheter repair and repla

116 s synergism suggests that contents in animal regurgitants making their way into plant tissue during f

117 accepted method for the surgical repair of a regurgitant mitral valve.

118 Currently, over 90% of regurgitant mitral valves of varying etiologies are amen

119 us, exercise duration, LVOT gradient, mitral regurgitant (MR) volume, LV pre-A pressure and LA volume

120 We identified bacteria from the regurgitant of field-collected Helicoverpa zea larvae us

121 (17-hydroxylinolenoyl-l-Gln) present in the regurgitant of Spodoptera exigua (beet armyworm caterpil

122 lass of compounds has been isolated from the regurgitant of the grasshopper species Schistocerca amer

123 r, there were only low detectable amounts of regurgitant or bacteria on H. zea-damaged tomato leaves.

124 tion was demonstrated by increased effective regurgitant orifice (0.21 cm(2); 25th to 75th percentile

125 rgitant volume increased less than effective regurgitant orifice (120 [25th to 75th percentile, 78.6

126 esence of diabetes, and increasing effective regurgitant orifice (adjusted risk ratio per 10-mm2 incr

127 01) were obtained between VC-W and effective regurgitant orifice (ERO) area and regurgitant volume re

128 nts, regurgitant volume (RVol) and effective regurgitant orifice (ERO) area were 36+/-24 mL/beat and

129 The VCW correlated well with the effective regurgitant orifice (ERO) by the flow convergence method

130 Whether effective regurgitant orifice (ERO) by the flow convergence method

131 a (PISA) method for calculation of effective regurgitant orifice (ERO) of aortic regurgitation (AR).

132 ltaneously by echocardiography the effective regurgitant orifice (ERO) of FMR by using 2 methods: mit

133 ol), regurgitant fraction (RF) and effective regurgitant orifice (ERO) to define progression of MR.

134 sions (all P>0.55), and mitral regurgitation regurgitant orifice (P=0.62).

135 ms, 48.8 [14.8 to 161]) and mitral effective regurgitant orifice (r = 0.50, p = 0.0001; odds ratio [9

136 hic quantitation of IMR (measuring effective regurgitant orifice [ERO] and regurgitant volume).

137 ocardiographic quantitation of MR (effective regurgitant orifice [ERO]) and left ventricular (LV) sys

138 does not require spatial localization of the regurgitant orifice and can be performed semiautomatedly

139 ethod that eliminated the need to locate the regurgitant orifice and that could be performed semiauto

140 Direct measurements of the effective regurgitant orifice are also feasible and serve as an al

141 ated with a regurgitant volume > 60 mL and a regurgitant orifice area > 0.4 cm2.

142 predicted a regurgitant volume < 60 mL and a regurgitant orifice area < 0.4 cm2 in 24 of 29 patients.

143 1 ml to 3.1 +/- 0.5 ml, p < 0.05), effective regurgitant orifice area (0.130 +/- 0.010 cm(2) to 0.040

144 The mean anatomic regurgitant orifice area (0.35+/-0.10 cm(2)) was underes

145 ume (69 +/- 47 to 69 +/- 56 ml) or effective regurgitant orifice area (0.5 +/- 0.4 to 0.5 +/- 0.6 cm2

146 Significant reductions in effective regurgitant orifice area (0.9+/-0.3cm(2) versus 0.4+/-0.

147 eduction in annular area (57%) and effective regurgitant orifice area (53%) measured with 3-dimension

148 the accuracy of determining aortic effective regurgitant orifice area (EROA) and aortic regurgitant v

149 ion of severe secondary MR from an effective regurgitant orifice area (EROA) of 0.4 to 0.2 cm(2), and

150 l regurgitant flow rate (MRFR) and effective regurgitant orifice area (EROA) on mitral regurgitant st

151 egurgitant volume (r = .85, SEE = 20 mL) and regurgitant orifice area (r = .86, SEE = 0.15 cm2).

152 egurgitant volume (r = .85, SEE = 19 mL) and regurgitant orifice area (r = .88, SEE = 0.14 cm2).

153 Regurgitant orifice area (ROA) is an important measure o

154 graphic measurements (TA diameter, effective regurgitant orifice area [EROA], left ventricular stroke

155 Using an in vitro model of MR, the effective regurgitant orifice area and regurgitant volume (RVol) w

156 VCW, effective regurgitant orifice area and regurgitant volume were mea

157 ide was concordant with changes in effective regurgitant orifice area and regurgitant volume, and was

158 of heart failure through a reduction in the regurgitant orifice area but not through a change in the

159 patients (n=30, functional MR), 3D effective regurgitant orifice area correlated well with cardiac ma

160 In all patients, the regurgitant orifice area decreased with therapy from 0.5

161 determined as well as regurgitant volume and regurgitant orifice area derived from color M-mode and D

162 ly, increased transmitral pressure decreased regurgitant orifice area for any geometric configuration

163 ught to validate direct planimetry of mitral regurgitant orifice area from three-dimensional echocard

164 ted significantly with regurgitant volume or regurgitant orifice area in a multivariate analysis.

165 haracterize the regurgitant flow pattern and regurgitant orifice area in patients undergoing therapy

166 mmary, the time course and rate of change of regurgitant orifice area in patients with functional MR

167 Reduction of the regurgitant orifice area is likely related to decreased

168 Similarly, the rate of change of regurgitant orifice area more strongly related to that o

169 All patients had midsystolic decreases in regurgitant orifice area that mirrored increases in tran

170 A similar regurgitant orifice area time course was observed in fou

171 ved tented-leaflet configuration and dynamic regurgitant orifice area variation can be reproduced in

172 ve annular area of 14.1 cm(2), and effective regurgitant orifice area was 1.35 cm(2).

173 Effective regurgitant orifice area was calculated by dividing the

174 In 30 patients with functional MR, regurgitant orifice area was obtained as flow (from M-mo

175 vere primary degenerative MR (mean effective regurgitant orifice area, 0.45 +/- 0.25 cm)(2) with no c

176 te to severe mitral regurgitation (effective regurgitant orifice area, 38+/-18 mm(2)) and preserved l

177 d transmitral pressure on dynamic changes in regurgitant orifice area.

178 ricuspid regurgitation (TR) by occupying the regurgitant orifice area.

179 e acting to close the leaflets decreases the regurgitant orifice area.

180 transmitral pressure significantly affected regurgitant orifice area; however, transmitral pressure

181 g inspiration, a large increase in effective regurgitant orifice causes, despite a decline in regurgi

182 Effective regurgitant orifice changes are independently linked to

183 Effective regurgitant orifice during inspiration was independently

184 end-systolic dimension, and mitral effective regurgitant orifice increased the C-statistic for longer

185 tion does not predict accurately whether the regurgitant orifice is fixed or dynamic.

186 Patients with an effective regurgitant orifice of at least 40 mm2 had a five-year s

187 Patients with an effective regurgitant orifice of at least 40 mm2 should promptly b

188 As compared with patients with a regurgitant orifice of less than 20 mm2, those with an o

189 Mitral effective regurgitant orifice size (n=84) influenced RV EF (beta=-

190 ation with angiographic grades for effective regurgitant orifice were 43+/-37 mm and r=.79 (P<.0001);

191 ional mitral regurgitation (larger effective regurgitant orifice).

192 itant volume, 66+/-40 ml per beat; effective regurgitant orifice, 40+/-27 mm2).

193 tricular ejection fraction, mitral effective regurgitant orifice, indexed LV end-diastolic volume, an

194 Mean LVEF, mitral effective regurgitant orifice, indexed LV end-systolic diameter (L

195 tricular ejection fraction, mitral effective regurgitant orifice, resting right ventricular systolic

196 endocarditis were considered to have a fixed regurgitant orifice, whereas patients with mitral valve

197 ange in the force balance acting to create a regurgitant orifice, with rising transmitral pressure co

198 r ischemia were considered to have a dynamic regurgitant orifice.

199 gurgitation caused by dynamic changes in the regurgitant orifice.

200 not be load independent because of a dynamic regurgitant orifice.

201 ng forces acting on the leaflets creates the regurgitant orifice.

202 nd Id-FTR were also matched for TR effective-regurgitant-orifice (ERO).

203 r alias contour with velocity va) divided by regurgitant peak velocity (obtained by continuous wave [

204 Vena contracta width correlated well with regurgitant severity determined by electromagnetic flowm

205 The pre-surgical estimate of regurgitant severity was correlated with the postoperati

206 AV-Vel, which reflects both stenosis and regurgitant severity, provides an objective and easily a

207 ve regurgitant orifice area (EROA) on mitral regurgitant stroke volume (MRSV) quantification using 4

208 ate; r = 0.85, SEE = 0.08 cm, p < 0.0001 for regurgitant stroke volume; r = 0.90, SEE = 0.07 cm, p <

209 nd reference peak regurgitant flow rates and regurgitant stroke volumes (r=0.99, difference=0.11 L/mi

210 Thirteen different forward and regurgitant stroke volumes (RSVs) across the noncircular

211 ied as peak and mean regurgitant flow rates, regurgitant stroke volumes and regurgitant fractions det

212 Peak regurgitant flow rates and regurgitant stroke volumes were calculated as the produc

213 right ventricular (RV) forward and pulmonary regurgitant stroke volumes.

214 column for direct visual recording of mitral regurgitant SV (MRSV).

215 nically damaged and induced with caterpillar regurgitant than seedlings not exposed to GLV.

216 Tricuspid regurgitant (TR) jet velocity and its relationship to pu

217 d can be used in conjunction with quantified regurgitant values obtained from velocity-encoded MR ima

218 h of diagnosis, surgery was performed for 13 regurgitant valves in 11 patients (24%).

219 ves seen in 34 4D phase-contrast studies, 29 regurgitant valves were identified, with good agreement

220 linically to assess severity of stenotic and regurgitant valves.

221 Surgical treatment of stenotic or regurgitant valvular lesions can alter the natural histo

222 w, or pulmonary hypertension (peak tricuspid regurgitant velocity >2.5 m/s) should alert clinicians o

223 Elevated tricuspid regurgitant velocity (> or = 2.5 m/sec), a measure of pu

224 ic pressure (PASP) >35 mmHg and/or tricuspid regurgitant velocity (TRV) >2.5 m/s.

225 e cell disease (SCD), an increased tricuspid regurgitant velocity (TRV) measured by Doppler echocardi

226 to test whether the ratio of peak tricuspid regurgitant velocity (TRV, ms) to the right ventricular

227 Tricuspid regurgitant velocity and creatinine levels also did not

228 by Doppler echocardiography (using tricuspid regurgitant velocity), and left ventricular systolic and

229 Baseline tricuspid regurgitant velocity, a measure of pulmonary systolic pr

230 nificant tricuspid regurgitation with a high regurgitant velocity.

231 ted with anemia, endothelin-1, and tricuspid regurgitant velocity; the latter is reflective of peak p

232 gnificantly more reduced in patients in whom regurgitant vena contracta area was reduced by >50% comp

233 h > or = 0.5 cm was always associated with a regurgitant volume > 60 mL and a regurgitant orifice are

234 without surgery compared with only 21% with regurgitant volume >55 mL (P<0.0001).

235 na contracta width < or = 0.3 cm predicted a regurgitant volume < 60 mL and a regurgitant orifice are

236 ndications for surgery: 91% of subjects with regurgitant volume </=55 mL survived to 5 years without

237 versus -37+/-21%), and percent mitral valve regurgitant volume (-99+/-2% versus -52+/-56%) for the X

238 /LA area 43 +/- 4% to 8 +/- 5%, p < 0.0001), regurgitant volume (14.7 +/- 2.1 ml to 3.1 +/- 0.5 ml, p

239 versus 426+/-50 ms; P<0.0001) yielded lower regurgitant volume (24.8+/-13.4 versus 48.6+/-25.6 mL; P

240 +/-0.5 cm versus 0.6+/-0.3 cm; P=0.001), and regurgitant volume (57.2+/-12.8 mL/beat versus 30.8+/-6.

241 ues for VCW (0.5 +/- 0.2 to 0.5 +/- 0.2 cm), regurgitant volume (69 +/- 47 to 69 +/- 56 ml) or effect

242 In the patients, mitral regurgitant volume (MRV) by ACMm-ACMa agreed with PD-2D

243 color Doppler methods for determining mitral regurgitant volume (MRV) have prevented their widespread

244 width from apical views correlated well with regurgitant volume (r = .85, SEE = 19 mL) and regurgitan

245 asternal long-axis view correlated well with regurgitant volume (r = .85, SEE = 20 mL) and regurgitan

246 hepatic venous flow (r = 0.79, p < 0.0001), regurgitant volume (r = 0.77, p<0.0001) and right atrial

247 asurements of regurgitant fraction (RgF) and regurgitant volume (RgV).

248 All patients underwent MRI to quantify regurgitant volume (RV) of OMR by subtracting the aortic

249 In IMR patients, regurgitant volume (RVol) and effective regurgitant orif

250 We hypothesized that CMR measurement of regurgitant volume (RVol) is more reproducible than TTE.

251 area (EROA) of 0.4 to 0.2 cm(2), and from a regurgitant volume (RVol) of 60 to 30 ml.

252 , the effective regurgitant orifice area and regurgitant volume (RVol) were measured by the PISA tech

253 diographic methods allow the quantitation of regurgitant volume (RVol), regurgitant fraction (RF) and

254 itutions and presenting moderate SMR (mitral regurgitant volume 30 to 45 mL/beat) not considered for

255 had a mean ejection fraction 64 +/- 9%, mean regurgitant volume 67 +/- 31 ml, and low mean Charlson c

256 s, 60% men) in sinus rhythm with organic MR (regurgitant volume 68 +/- 42 ml/beat) and performed at b

257 al TEE demonstrates significant reduction of regurgitant volume after PMVR.

258 Mitral regurgitant volume and orifice area did not correlate wi

259 f the present study was to quantitate aortic regurgitant volume and regurgitant fraction in a chronic

260 raphic dimensions were determined as well as regurgitant volume and regurgitant orifice area derived

261 valve closure, increased the early systolic regurgitant volume before complete coaptation, and decre

262 Mitral valve regurgitant volume by color Doppler 3D TEE was determine

263 because the heart compensates for increasing regurgitant volume by left-atrial enlargement, causes le

264 orifice area was calculated by dividing the regurgitant volume by the continuous-wave Doppler veloci

265 e regurgitant orifice area (EROA) and aortic regurgitant volume by using the color Doppler-imaged ven

266 Mitral regurgitant volume calculated from R(calc) and R(meas) c

267 The mitral regurgitant volume decreased from 47+/-27 ml before ther

268 ric method consistently decreased after CRT: regurgitant volume from 40 +/- 20 ml to 24 +/- 17 ml and

269 ) to be independently associated with mitral regurgitant volume improvement.

270 The added regurgitant volume in MR increases the left atrial to le

271 As a result of reduced TR driving force, regurgitant volume increased less than effective regurgi

272 sociated with postoperative change in mitral regurgitant volume on univariable analysis were entered

273 tive MR grade, correlated significantly with regurgitant volume or regurgitant orifice area in a mult

274 ted in context, and in mid-late systolic MR, regurgitant volume provides information more reflective

275 effective regurgitant orifice (ERO) area and regurgitant volume recorded by quantitative Doppler (r=0

276 Regurgitant volume was reduced from 84.1+/-38.3 mL prein

277 Mitral regurgitant volume was then calculated according to the

278 VCW, effective regurgitant orifice area and regurgitant volume were measured by quantitative Doppler

279 ht ventricular stroke volume minus pulmonary regurgitant volume) after BMS remained unchanged (33.8+/

280 ring effective regurgitant orifice [ERO] and regurgitant volume).

281 ice were 43+/-37 mm and r=.79 (P<.0001); for regurgitant volume, 62+/-45 mL and r=.80 (P<.0001); and

282 tified according to current recommendations (regurgitant volume, 66+/-40 ml per beat; effective regur

283 es in effective regurgitant orifice area and regurgitant volume, and was not different between dynami

284 However, shorter MR yields lower regurgitant volume, consequences, and benign outcomes.

285 on (grade 4) were 60 mL, 50%, and 40 mm2 for regurgitant volume, regurgitant fraction, and orifice, r

286 ted by pulsed-Doppler technique to determine regurgitant volume.

287 RO was not linked to outcome, in contrast to regurgitant volume.

288 regurgitant gradient, a notable increase in regurgitant volume.

289 red with those with no PPM (change in mitral regurgitant volume: -11+/-4 versus -17+/-5 mL, respectiv

290 ce = 0.51 +/- 1.89 ml/beat for the pulmonary regurgitant volume; and r = 0.91, mean difference = -0.2

291 +/- 2.9 ml vs. 11 +/- 5.8 ml, p < 0.0001 for regurgitant volume; mean difference 1.2 +/- 7.6% vs. 19

292 Regurgitant volumes and regurgitant fractions by the new

293 olor Doppler method provides accurate aortic regurgitant volumes and regurgitant fractions without cu

294 a promising method for determining pulmonary regurgitant volumes and regurgitant fractions.

295 Pulmonary regurgitant volumes and RV forward stroke volumes comput

296 and agreements between peak and mean RFR and regurgitant volumes per beat as determined by Doppler ec

297 Rs varied from 0.7 to 4.9 (2.7+/-1.3) L/min, regurgitant volumes per beat varied from 7.0 to 48.0 (26

298 Peak and mean RFRs and regurgitant volumes per beat were calculated from vena c

299 Regurgitant volumes ranged from 2 to 191 mL.

300 imal study, using strictly quantified aortic regurgitant volumes, demonstrated that the digital color