ライフサイエンスコーパス: relaxation time

1 ical tissue, causing a shortening of the T2* relaxation time.

2 calculating nerve proton spin density and T2 relaxation time.

3 resulting in a measureable change in the T1-relaxation time.

4 is substantially smaller than the structural relaxation time.

5 s, this is an upper bound to the equilibrium relaxation time.

6 by calculation of proton spin density and T2 relaxation time.

7 -terminal domains with congruent with 15 mus relaxation time.

8 DPN, more so than proton spin density or T2 relaxation time.

9 with reduced ejection fraction and prolonged relaxation time.

10 find a significant slowing down of the alpha-relaxation time.

11 quences sensitive to ultra-short transversal relaxation times.

12 spectra show that there is a distribution of relaxation times.

13 e synthesis of paramagnetic probes with long relaxation times.

14 the total Stokes shift and the Stokes shift relaxation times.

15 ls each characterized by a range of electron relaxation times.

16 in a minor contribution to the electron spin relaxation times.

17 ST detection, including pH, temperature, and relaxation times.

18 changes the polariton transient spectra and relaxation times.

19 ngle-fiber modulus, extensibility, or stress relaxation times.

20 fs and is asymmetric, with a tail at slower relaxation times.

21 Areas under the curve of native T1 relaxation times (0.94) were higher compared with those

22 tandard deviation], P = .006) and longer T2* relaxation time (1041 musec +/- 424, P = .016) were foun

23 elevated in HIV-infected patients (native T1 relaxation times, 1128.3+/-53.4 ms versus 1086.5+/-54.5

24 ter in fibromyalgia to be associated with T1 relaxation times, a surrogate marker of water content, b

25 of MECVF relies on quantification of the T1 relaxation time after contrast enhancement, which can be

26 tion; here, [Formula: see text] is the phase-relaxation time and [Formula: see text] is the turbulenc

27 ompanied by a rapid growth of the structural relaxation time and a concomitant decrease of configurat

28 oefficient (ADC) were calculated, as were T2 relaxation time and proton spin density obtained from DT

29 h the water in the sheath has a reduced T(2) relaxation time and spin density relative to its surroun

30 Gel relaxation time and the mechanical response to dynamic s

31 vide a measurement of the g-factor, the spin relaxation time and the sub-lattice degeneracy splitting

32 ezable water content, (1)H proton transverse relaxation time and water self-diffusivity determined by

33 SAs) possess small modulation depth and slow relaxation time and, therefore, are incapable of ensurin

34 results in a threefold enhancement in qubit relaxation times and a comparable reduction in coherence

35 t on T2-weighted TSE images and DW images T2 relaxation times and ADC values of the liver and FLLs we

36 gadoxetic acid disodium (Gd-EOB-DTPA) on T2 relaxation times and apparent diffusion coefficient (ADC

37 namics remains challenging due to their long relaxation times and associated computational cost.

38 eous, to define the relationships between T1 relaxation times and balanced salt solution (BSS), to si

39 es: eCPMG and eDiff, by modulating spin-spin relaxation times and diffusion of MBC molecular componen

40 ing as an alternative due to their long spin-relaxation times and ease of processing, but, with the n

41 strong relationship between quantitative MRI relaxation times and hepatic iron content.

42 Relaxation times and parametric relaxation maps were gen

43 n exchange rates, we measured the (15)N T(2) relaxation times and simulated them for chemical-shift e

44 there is little, if any, connection between relaxation times and thermal conductivity.

45 parameters, (1)H rotating-frame spin-lattice relaxation times and water-to-protein spin diffusion exp

46 ters E-wave deceleration time, isovolumetric relaxation time, and E'-wave velocity improved similarly

47 magnetic resonance neurography (DTI-MRN), T2 relaxation time, and proton spin density can detect and

48 time that is short relative to electron spin relaxation times, and data are processed to obtain the a

49 atio, late gadolinium enhancement, native T1 relaxation times, and extracellular volume fraction.

50 hat permit high spin number, long electronic relaxation times, and labile water exchange, we evaluate

51 ompatibility, and whose spectral properties, relaxation times, and sensitivity are promising for in v

52 (1)H spectral lineshapes, T2 relaxation times, and two-dimensional (2D) (1)H-(13)C co

53 Quantification of T1 relaxation time appears to be a good predictor of the su

54 y scale and the concomitant huge increase of relaxation time approaching the glass transition.

55 Analysed with the relaxation time approximation model using phonon dispers

56 combined with Boltzmann transport method and relaxation time approximation.

57 ombined with Boltzmann transport method with relaxation time approximation.

58 in venous thrombi and whether changes in T1 relaxation time are informative of the susceptibility to

59 of dynamically heterogeneous regions and the relaxation time are very different in two and three dime

60 That is, observed relaxation times are not simply proportional to the solv

61 Separate electron and hole relaxation times are observed as a function of hot carri

62 the existence of a characteristic molecular relaxation time around 0.1 ms.

63 mm(3)) were applied to determine transverse relaxation time as affected by magnetic field heterogene

64 Changes in the quantitative MRI relaxation times as well as severe splenomegaly were obs

65 chemical shift overlap, and short transverse relaxation times (associated with slow tumbling) render

66 h C(60) increases the zero-field 4f electron relaxation time at 2 K to several hours.

67 ents in a step-wise fashion, we measured the relaxation time at each temperature and, above the ficti

68 T1 relaxation times at dGEMRIC showed strong correlation wi

69 Our computed electron-phonon relaxation times at the onset of the Gamma, L, and X val

70 increasing cell rigidity, whereas the shape relaxation time becomes longer and longer due to the cel

71 for rearrangement (temperature dependence of relaxation times) becomes smaller than the activation en

72 max even after inclusion of LV stiffness and relaxation time (beta=0.80; P<0.01).

73 level of polarization and long spin-lattice relaxation time-both of which are necessary for future c

74 f optimizing both EPR linewidth and electron relaxation times by studying direct DNP of (13)C using S

75 Furthermore, the results of MD based relaxation time calculations suggest that in amorphous m

76 sufficiently high surface disorder, the spin relaxation time can be extended via the Dyakonov-Perel m

77 We then show that the relaxation time can be robustly computed from structure

78 T1 relaxation times can in turn be used to calculate the co

79 nting for 90-92% of the total signal, with a relaxation time centred at 47-60 ms and a broad band wit

80 We found that magnetic resonance imaging T2 relaxation time changes in subjects commenced on lithium

81 XPCS study on a colloidal suspension with a relaxation time comparable to the SACLA free-electron la

82 1-5 media showed significantly shortened T2 relaxation times compared with unlabeled control cells (

83 ncrease the apparent spin-label phase memory relaxation time, complemented by high sensitivity afford

84 Longitudinal (1)H(2)O NMR relaxation time constant (T(1)) values were measured in

85 al cross-sectional area (CSAmax), transverse relaxation time constant (T2), and lipid fraction were c

86 Results reveal a defect relaxation time constant of 10-0.2 ms, which decreases

87 er density of up to 1323 W cm(-3) with a low relaxation time constant of 0.27 ms.

88 The relaxation time constant of the fluctuations was in the

89 ollow-up quantitative MR imaging (transverse relaxation time constant; MRI-T2 ), MR spectroscopy (fat

90 P<0.01) and impaired relaxation (isovolumic relaxation time: control, 0.21 ms [interquartile range,

91 3 [1]) and diastolic dysfunction (isovolumic relaxation time: controls 44 ms [6] versus FGR 52 [9]).

92 a of BALB/C mice, and temporal changes in T1 relaxation time correlated with thrombus composition.

93 and diagnostic accuracy (91%) for native T1 relaxation times (cutoff, 1140 msec) were equivalent com

94 d T2 relaxation effects across this range of relaxation times did not account for the findings.

95 T1rho relaxation times did not correlate with cartilage sGAG c

96 After excitation at 800 nm, the measured relaxation time distribution of multiple complexes has a

97 r minutes, we find that complexes sample the relaxation time distribution on a timescale of seconds.

98 generated at high fields dominated the T(2) relaxation time distributions and biofilm growth could n

99 Instead, the microscopic relaxation times diverge so rapidly that, upon further c

100 t specific identifiers including microscopic relaxation times diverging on a Vogel-Tammann-Fulcher (V

101 t of 81.6 ns is needed as an addition to all relaxation times due to intrachain friction sources.

102 The fast relaxation time during dynamic relaxation is obtained as

103 The dynamics of T2 relaxation times during the first post-infarction week w

104 lating configurational entropy to structural relaxation time) established in earlier numerical studie

105 The slow relaxation time exhibits an Arrhenius behavior with no s

106 The average T2 relaxation time for all-cause rejection versus no reject

107 The average T2 relaxation time for grade 0R (n=46) and grade 1R (n=17)

108 revealed 15-fold lower SNR and much shorter relaxation times for (35)Cl than (23)Na.

109 mputed using the WIEN2k code and the carrier relaxation times for electrons and holes are calculated

110 T2* relaxation times for normal cartilage (Beck score 1, 35.

111 rons and phonons saturates a quantum thermal relaxation time [Formula: see text].

112 Long magnetic relaxation times have been demonstrated for single lanth

113 By analogy to linear polymers, shorter T(1) relaxation times have been traditionally associated to l

114 e report a strong enhancement in the optical relaxation time in Cu by direct growth of few-layer grap

115 ults show that the history dependence of the relaxation time in glasses requires knowledge only of th

116 values) for absolute signal intensity and T2 relaxation time in healthy subjects, their relativised v

117 In humans, T2 relaxation time in the ischemic myocardium declines sign

118 From T(1) (spin-lattice relaxation time in the laboratory frame) and T(1rho) (sp

119 T2 relaxation time in the myocardium (T2 mapping) and the e

120 laboratory frame) and T(1rho) (spin-lattice relaxation time in the rotating frame) measurements, it

121 is decoupled from that of the translational relaxation time in two dimensions but not in three dimen

122 ons as in solids over almost entire range of relaxation time in which liquids exist as such, and demo

123 rong anharmonicity, high reproducibility and relaxation times in excess of 40 mus at its flux-insensi

124 w the status of efforts to achieve long spin-relaxation times in graphene with its weak spin-orbit co

125 atter decreases were largely explained by T1 relaxation times in gray matter, a surrogate measure of

126 togenesis and found that reduced amygdala T2 relaxation times in high-magnetic-field MRI hours after

127 the association of global and regional brain relaxation times in patients with prior exposure to line

128 associated with a significant increase in T2 relaxation times in the ischemic region.

129 Therefore, the electron- and hole-spin relaxation times in these systems with zero or minimal s

130 isotope effect and the unusual variation of relaxation times in water at low temperatures can be exp

131 The relaxation time increase can be explained by correlated

132 nm, with a similar size dependence where the relaxation times increased from 140 +/- 10 to 310 +/- 15

133 the buffer solution the protein's rotational relaxation time increases exponentially, taking values i

134 diastolic filling, decreases, and isovolumic relaxation time increases, indicating that both active a

135 The discrepancy in the relaxation times increases with increasing temperatures,

136 Transverse relaxation times indicated a lower degree of muscle dena

137 of proton spin density and a decrease of T2 relaxation time, indicating changes in the microstructur

138 BAA receptor concentration in addition to T1 relaxation times, indicating perhaps increased neuronal

139 The slow hot carrier relaxation time is 0.5 ps.

140 The T1 relaxation time is a robust noninvasive imaging biomarke

141 lease in our model is that the matrix stress relaxation time is comparable to the time scale for wate

142 ynamic behaviour reveals that the structural relaxation time is substantially reduced in these adapti

143 tra further suggest that the distribution of relaxation times is temperature independent at low frequ

144 Isovolumic relaxation time (IVRT), early diastolic filling (E/A), m

145 se from observing hyperpolarized (1)H, short relaxation times limit the utility of prepolarizing this

146 ies, E:A ratio, deceleration, and isovolumic relaxation times; LV systolic function was preserved.

147 transversal (T2) nuclear magnetic resonance relaxation time mapping.

148 Quantitative MR imaging relaxation time maps demonstrated up to a twofold variat

149 The authors reviewed T2* relaxation time maps of 28 hips from 26 consecutive pati

150 T(2) and T(2)* relaxation time maps were created from the multiecho seq

151 s a suppression of the Knight shift and spin relaxation time measured in nuclear magnetic resonance (

152 The relaxation times measured at variable temperature provid

153 The longest relaxation times measured correspond to estimated viscos

154 ch as MR spectroscopy, T1rho calculation, T2 relaxation time measurement, diffusion quantitative imag

155 translational diffusion and proton T1rho/T2 relaxation-time measurements for rotational diffusion, t

156 ed mitral E deceleration time, LV isovolumic relaxation time, mitral E/E', and pulmonary vein A wave

157 itative spin-lattice (T1) and spin-spin (T2) relaxation time MR imaging mapping was performed before

158 spectroscopy we resolve loss peaks yielding relaxation times near 100 s at 126 K for low-density amo

159 ated via analysis of the NMR line shapes and relaxation times observed between 12 and 400 K.

160 (2)H NMR MAS spectra and T1 relaxation times obtained from the deuterated phenylalan

161 ar10 s(-1), which corresponds to the inverse relaxation time of a healthy red blood cell.

162 c environments by measuring the longitudinal relaxation time of a single-spin probe as it is systemat

163 re than one order of magnitude in the energy relaxation time of a superconducting artificial atom.

164 ve at room temperature with a characteristic relaxation time of about one month.

165 by the hydrogen bonds did not influence the relaxation time of cytosine significantly due to the gen

166 values upon annealing, but the difference in relaxation time of density and hardness, which is usuall

167 red in stroked hemispheres; and longitudinal relaxation time of lactic acid was found to increase by

168 haracteristic times of the system, e.g., the relaxation time of local excitations.

169 he Lorentzian noise component determines the relaxation time of molecular fluctuations, which, in tur

170 Moreover, the temperature dependence of the relaxation time of orientational correlations is decoupl

171 We determine an energy relaxation time of T1 approximately 100 microseconds and

172 The singlet relaxation time of the (13)C(2) pair in [1-(18)O,(13)C(2

173 We show that the relaxation time of the confined DNA is >10 min, which is

174 ggests a decoupling of the amplitude and the relaxation time of the membrane thickness fluctuations,

175 (1)H T2 relaxation time of the most abundant population (populat

176 ased on causality - on the comparison of the relaxation time of the order parameter with the "time di

177 Changes in the spin relaxation time of the sensor located in the lipid bilay

178 The relaxation time of the solvent protons in 3 mM solutions

179 The mean T1 relaxation time of thrombus was shortest at 7 days follo

180 different from surrounding liver parenchyma relaxation times of 840 msec +/- 113 and 28 msec +/- 3 (

181 s reached the limit of the microsecond-scale relaxation times of biological molecules bound to a forc

182 Relaxation times of healthy thigh muscle and brain tissu

183 T2 relaxation times of in vivo-labeled MSC transplants and

184 Mean T1 and T2 relaxation times of lymphatic fluid at 3.0 T were 3100 m

185 CPMG) sequence was used to measure spin-spin relaxation times of proton pools representing major yolk

186 aging is based on the dependence of the spin relaxation times of protons in water molecules in a host

187 and that the extracted temperature dependent relaxation times of the assemblies follow the Vogel-Fulc

188 The mean T2 relaxation times of the liver and focal hepatic lesions

189 The mean T2 relaxation times of the liver and focal liver lesions as

190 T(2) relaxation times of the majority proton population sugge

191 The relative intensities and the transverse relaxation times of the NMR signal components associated

192 er, we show that both the magnitudes and the relaxation times of these backbone breathing fluctuation

193 )H-SABRE experiments, and we record (15)N T1 relaxation times of up to 2 min.

194 shear viscosity, plateau modulus, and stress relaxation time) of the shear-thickening precursor are s

195 ure the longitudinal relaxation time, the T1 relaxation time, of protons in a magnetic field after ex

196 tionship between postcontrast ventricular T1 relaxation time on CMR and freedom from AF after pulmona

197 A shorter postcontrast ventricular T1 relaxation time on CMR is associated with reduced freedo

198 re probed, enabling us to determine the spin relaxation time on the island.

199 r myocardial water content at 120 min and T2 relaxation times on 120 min cardiac magnetic resonance t

200 The impact of electron relaxation times on the DNP enhancement (epsilon) is exa

201 approximately half of the longitudinal spin relaxation time over a wide range of temperatures, which

202 correlate the above g-shifts to spin-lattice relaxation times over four orders of magnitude, from 200

203 ersus controls (288 +/- 13.4), but not of T2 relaxation time (p = 0.49).

204 een different proton pools with different T1 relaxation times, particularly when the starch gelatiniz

205 t longer inversion recovery and phase memory relaxation times provide larger epsilon.

206 al shortening, ejection fraction, isovolumic relaxation time, rate of pressure development and rate o

207 ation approach to obtain backbone (15)N spin relaxation time ratios T1/T2 for a monomeric EIAV-CA in

208 compared with control patients, half maximal relaxation time (RT50) at 60 per minute was prolonged by

209 s substantially larger than the viscoelastic relaxation time scale of the biofilms, and this appearan

210 We extend the NMR spin relaxation time scale sensitivity deeper into the nanose

211 Isovolumic relaxation time shortened (baseline 109.1 +/- 21.7 ms vs

212 paramagnetic Fe3+, which causes thrombus T1 relaxation time shortening.

213 t long-lived states (LLSs) in solution, with relaxation times substantially longer than the conventio

214 relation between regional gray matter and T1 relaxation times suggests decreased tissue water content

215 scales depending on the nuclear spin-lattice relaxation time T(1) in the range of seconds.

216 te is 2-3 times longer than the spin-lattice relaxation time T(1).

217 rally identifiable and shortening of the MRI relaxation times T 1 and [Formula: see text].

218 Since both isotopes have long relaxation times T(1), the hyperpolarized NMR signal of

219 er of magnitude compared to the spin lattice relaxation time (T 1), but they have to be prevented fro

220 nce was used to calculate post-contrast T(1) relaxation time (T(1) time) of the LV myocardium as an i

221 iradical having a long electron spin-lattice relaxation time (T(1e)) has been developed as an exogeno

222 rticles, specifically by measuring spin-spin relaxation time (T(2)), is reported.

223 ic solids characterized by long spin-lattice relaxation times (T(1)((1)H) > 200 s), (1)H-(1)H spin di

224 sion using the biexponential analysis of the relaxation times (T(21), T(22)) and amplitudes (A(21), A

225 We report measurements of the longitudinal relaxation time T1 of brain tissue, blood, and scalp fat

226 ly longer than the conventional spin-lattice relaxation time T1.

227 Qubit relaxation times T1 across 22 qubits are consistently ma

228 -weighted MR imaging, quantitation of native relaxation times T1 and T2, the relaxation rate R2*, and

229 2) min(-1)] and in-cell (129)Xe spin-lattice relaxation time (T1 = 2.19 +/- 0.06 h) for 1000 Torr Xe

230 Quantitative relaxation time (T1 and T2) measurements were made in ex

231 l to the study of intelligence: longitudinal relaxation time (T1) and magnetisation transfer ratio.

232 (35)Cl longitudinal relaxation time (T1) and T2* of healthy human brain and

233 quantities in spintronics are the population relaxation time (T1) and the phase memory time (T2): T1

234 Gd-DTPA did not alter vitreous longitudinal relaxation time (T1) at baseline or at 3 hours after NMD

235 The magnetic resonance longitudinal relaxation time (T1) changes with thrombus age in humans

236 Reduction of the (19)F spin-lattice relaxation time (T1) enables rapid imaging and an improv

237 is "turned on" by altering the longitudinal relaxation time (T1) of bulk water protons.

238 e liquid state according to the spin-lattice relaxation time (T1) of the nucleus.

239 er of magnitude compared to the spin lattice relaxation time (T1), but they have to be prevented from

240 lifetime of the qubit, and the spin-lattice relaxation time (T1), the thermally defined upper limit

241 (1)H NMR relaxation times (T1 and T2) were measured at low field

242 Based on their spin-lattice relaxation times (T1), two dimensional (1)H NMR spectros

243 oach to correlate spin-lattice and spin-spin relaxation times (T1-T2) including acquisition of the FI

244 meters (concentration [rho] and longitudinal relaxation time [T1]) of human cortical bone in vivo.

245 ity of lifetime of spin states (spin-lattice relaxation time, T1) and coherences (spin-spin relaxatio

246 hically inequivalent carbon and spin-lattice relaxation times, T1, yield characteristic information t

247 ect of B1(+) correction on the native tissue relaxation time (T10) of fat, parenchyma, and malignant

248 ance (MR) relaxometry demonstrating short T1 relaxation time (T1R) in the basal ganglia reflects exce

249 r present 2D images of T1 and the transverse relaxation time T2 of the brain and show that, as expect

250 by microstructure, influence the transverse relaxation time (T2) in an orientation-dependent fashion

251 etic resonance imaging (MRI)-based spin-spin relaxation time (T2) mapping has been shown to be associ

252 Quantitatively, transverse relaxation time (T2) of CSS increased non-linearly with

253 Diffusion tensor imaging and transverse relaxation time (T2) relaxometry were performed at basel

254 ance imaging showed a significant transverse relaxation time (T2) shortening in the pancreata of mice

255 es (50-80 K) to increase the short spin-spin relaxation time (T2) upon which the technique relies.

256 The MRI outcomes-fat fraction, transverse relaxation time (T2), and magnetisation transfer ratio (

257 by downhill running (DR) by using transverse relaxation time (T2)-weighted magnetic resonance imaging

258 he characteristic distribution of transverse relaxation times (T2) within dendrimers (shorter values

259 ulations were identified measuring spin-spin relaxation times (T2).

260 laxation time, T1) and coherences (spin-spin relaxation time, T2) to the immediate environment was ut

261 Regional native T1/T2 relaxation time, T2-weighted ratio, and extracellular vo

262 llenging analysis of previtreous behavior of relaxation time (tau(T)) in ultraviscous low molecular w

263 binding kinetics of these measurements, the relaxation time (tau) was obtained, where higher tau val

264 s attributed to an increase in the carrier's relaxation time (tau).

265 quilibrium shape fluctuations that contain a relaxation time, tau, which is robust and independent of

266 nt study shows that I1 converts to I2 with a relaxation time tau1=0.1s at 25 degrees C in 25 mM KCl.

267 n transition metal ions with long electronic relaxation times (taus) is demonstrated.

268 oup 1 medium showed significantly shorter T2 relaxation times than hMSCs labeled with group 2-5 media

269 form a glass that behaves as a solid with a relaxation time that grows exponentially with decreasing

270 ostructures have longitudinal and transverse relaxation times that are on par with commonly used heav

271 d phosphonated trityl radical possesses long relaxation times that are sensitive to probe the microen

272 ging method used to measure the longitudinal relaxation time, the T1 relaxation time, of protons in a

273 show by a Redfield theory calculation of the relaxation times, the distribution shape corresponds to

274 ensemble, we demonstrate that, regarding the relaxation times, the ensemble can be considered ergodic

275 ental effects on the spin-label phase memory relaxation time Tm .

276 study underlines the potential of native T1 relaxation times to complement current cardiac MR approa

277 weak anti-localization effect and to a spin-relaxation time two to three orders of magnitude smaller

278 The relaxation time values and their associated relative pop

279 erred from an Arrhenius plot of the magnetic relaxation time versus the temperature.

280 Quantification of T1 relaxation time was a good predictor of successful throm

281 Mean T1 relaxation time was increased in HCM and DCM (HCM 1209+/

282 T2* relaxation time was measured before and after maternal h

283 T1 relaxation time was related to thrombus methemoglobin fo

284 In contrast, T2 relaxation time was significantly higher in controls (ti

285 The volume of lung tissue with increased relaxation times was determined by using a threshold-bas

286 nt (ADC), fractional anisotropy (FA), and T2 relaxation time were calculated.

287 e mono- and bi-exponential short and long T2 relaxation times were 24.7 ms, 4.2 ms (fraction 15%) and

288 On day 7 CMR, T2 relaxation times were as high as those observed at reper

289 or-encoded parametric maps of T2* transverse relaxation times were calculated on a pixel-by-pixel bas

290 (n = 12), tissue-specific magnetic resonance relaxation times were compared before and after lithium

291 tial and bi-exponential short and long T1rho relaxation times were estimated to be 26.9 ms, 4.6 ms (f

292 T1 relaxation times were measured before and after gadolini

293 The T2 relaxation times were measured using the 16-echo Carr-Pu

294 The mean log T2 relaxation times were reduced by 62% and 43%, respective

295 Native T1 relaxation times were significantly longer in patients w

296 TAM)-based spin label with a relatively long relaxation time where the protein is immobilized by atta

297 ecedentedly observed field dependence of the relaxation time, which was modeled with three contributi

298 greatly facilitated the interpretation of T1 relaxation times, which have been interpreted rather nar

299 l T1 nuclear magnetic resonance (NMR) proton relaxation times, which is proportional to amount of car

300 t proton spatial distributions and different relaxation times, which may also provide information abo