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1 ical tissue, causing a shortening of the T2* relaxation time.
2 calculating nerve proton spin density and T2 relaxation time.
3  resulting in a measureable change in the T1-relaxation time.
4 is substantially smaller than the structural relaxation time.
5 s, this is an upper bound to the equilibrium relaxation time.
6 by calculation of proton spin density and T2 relaxation time.
7 -terminal domains with congruent with 15 mus relaxation time.
8  DPN, more so than proton spin density or T2 relaxation time.
9 with reduced ejection fraction and prolonged relaxation time.
10 find a significant slowing down of the alpha-relaxation time.
11 quences sensitive to ultra-short transversal relaxation times.
12 spectra show that there is a distribution of relaxation times.
13 e synthesis of paramagnetic probes with long relaxation times.
14  the total Stokes shift and the Stokes shift relaxation times.
15 ls each characterized by a range of electron relaxation times.
16 in a minor contribution to the electron spin relaxation times.
17 ST detection, including pH, temperature, and relaxation times.
18  changes the polariton transient spectra and relaxation times.
19 ngle-fiber modulus, extensibility, or stress relaxation times.
20  fs and is asymmetric, with a tail at slower relaxation times.
21           Areas under the curve of native T1 relaxation times (0.94) were higher compared with those
22 tandard deviation], P = .006) and longer T2* relaxation time (1041 musec +/- 424, P = .016) were foun
23 elevated in HIV-infected patients (native T1 relaxation times, 1128.3+/-53.4 ms versus 1086.5+/-54.5
24 ter in fibromyalgia to be associated with T1 relaxation times, a surrogate marker of water content, b
25  of MECVF relies on quantification of the T1 relaxation time after contrast enhancement, which can be
26 tion; here, [Formula: see text] is the phase-relaxation time and [Formula: see text] is the turbulenc
27 ompanied by a rapid growth of the structural relaxation time and a concomitant decrease of configurat
28 oefficient (ADC) were calculated, as were T2 relaxation time and proton spin density obtained from DT
29 h the water in the sheath has a reduced T(2) relaxation time and spin density relative to its surroun
30                                          Gel relaxation time and the mechanical response to dynamic s
31 vide a measurement of the g-factor, the spin relaxation time and the sub-lattice degeneracy splitting
32 ezable water content, (1)H proton transverse relaxation time and water self-diffusivity determined by
33 SAs) possess small modulation depth and slow relaxation time and, therefore, are incapable of ensurin
34  results in a threefold enhancement in qubit relaxation times and a comparable reduction in coherence
35 t on T2-weighted TSE images and DW images T2 relaxation times and ADC values of the liver and FLLs we
36  gadoxetic acid disodium (Gd-EOB-DTPA) on T2 relaxation times and apparent diffusion coefficient (ADC
37 namics remains challenging due to their long relaxation times and associated computational cost.
38 eous, to define the relationships between T1 relaxation times and balanced salt solution (BSS), to si
39 es: eCPMG and eDiff, by modulating spin-spin relaxation times and diffusion of MBC molecular componen
40 ing as an alternative due to their long spin-relaxation times and ease of processing, but, with the n
41 strong relationship between quantitative MRI relaxation times and hepatic iron content.
42                                              Relaxation times and parametric relaxation maps were gen
43 n exchange rates, we measured the (15)N T(2) relaxation times and simulated them for chemical-shift e
44  there is little, if any, connection between relaxation times and thermal conductivity.
45 parameters, (1)H rotating-frame spin-lattice relaxation times and water-to-protein spin diffusion exp
46 ters E-wave deceleration time, isovolumetric relaxation time, and E'-wave velocity improved similarly
47 magnetic resonance neurography (DTI-MRN), T2 relaxation time, and proton spin density can detect and
48 time that is short relative to electron spin relaxation times, and data are processed to obtain the a
49 atio, late gadolinium enhancement, native T1 relaxation times, and extracellular volume fraction.
50 hat permit high spin number, long electronic relaxation times, and labile water exchange, we evaluate
51 ompatibility, and whose spectral properties, relaxation times, and sensitivity are promising for in v
52                 (1)H spectral lineshapes, T2 relaxation times, and two-dimensional (2D) (1)H-(13)C co
53                         Quantification of T1 relaxation time appears to be a good predictor of the su
54 y scale and the concomitant huge increase of relaxation time approaching the glass transition.
55                            Analysed with the relaxation time approximation model using phonon dispers
56 combined with Boltzmann transport method and relaxation time approximation.
57 ombined with Boltzmann transport method with relaxation time approximation.
58  in venous thrombi and whether changes in T1 relaxation time are informative of the susceptibility to
59 of dynamically heterogeneous regions and the relaxation time are very different in two and three dime
60                            That is, observed relaxation times are not simply proportional to the solv
61                   Separate electron and hole relaxation times are observed as a function of hot carri
62  the existence of a characteristic molecular relaxation time around 0.1 ms.
63  mm(3)) were applied to determine transverse relaxation time as affected by magnetic field heterogene
64              Changes in the quantitative MRI relaxation times as well as severe splenomegaly were obs
65 chemical shift overlap, and short transverse relaxation times (associated with slow tumbling) render
66 h C(60) increases the zero-field 4f electron relaxation time at 2 K to several hours.
67 ents in a step-wise fashion, we measured the relaxation time at each temperature and, above the ficti
68                                           T1 relaxation times at dGEMRIC showed strong correlation wi
69                 Our computed electron-phonon relaxation times at the onset of the Gamma, L, and X val
70  increasing cell rigidity, whereas the shape relaxation time becomes longer and longer due to the cel
71 for rearrangement (temperature dependence of relaxation times) becomes smaller than the activation en
72 max even after inclusion of LV stiffness and relaxation time (beta=0.80; P<0.01).
73  level of polarization and long spin-lattice relaxation time-both of which are necessary for future c
74 f optimizing both EPR linewidth and electron relaxation times by studying direct DNP of (13)C using S
75         Furthermore, the results of MD based relaxation time calculations suggest that in amorphous m
76 sufficiently high surface disorder, the spin relaxation time can be extended via the Dyakonov-Perel m
77                        We then show that the relaxation time can be robustly computed from structure
78                                           T1 relaxation times can in turn be used to calculate the co
79 nting for 90-92% of the total signal, with a relaxation time centred at 47-60 ms and a broad band wit
80  We found that magnetic resonance imaging T2 relaxation time changes in subjects commenced on lithium
81  XPCS study on a colloidal suspension with a relaxation time comparable to the SACLA free-electron la
82  1-5 media showed significantly shortened T2 relaxation times compared with unlabeled control cells (
83 ncrease the apparent spin-label phase memory relaxation time, complemented by high sensitivity afford
84                    Longitudinal (1)H(2)O NMR relaxation time constant (T(1)) values were measured in
85 al cross-sectional area (CSAmax), transverse relaxation time constant (T2), and lipid fraction were c
86                      Results reveal a defect relaxation time constant of 10-0.2 ms, which decreases
87 er density of up to 1323 W cm(-3) with a low relaxation time constant of 0.27 ms.
88                                          The relaxation time constant of the fluctuations was in the
89 ollow-up quantitative MR imaging (transverse relaxation time constant; MRI-T2 ), MR spectroscopy (fat
90  P<0.01) and impaired relaxation (isovolumic relaxation time: control, 0.21 ms [interquartile range,
91 3 [1]) and diastolic dysfunction (isovolumic relaxation time: controls 44 ms [6] versus FGR 52 [9]).
92 a of BALB/C mice, and temporal changes in T1 relaxation time correlated with thrombus composition.
93  and diagnostic accuracy (91%) for native T1 relaxation times (cutoff, 1140 msec) were equivalent com
94 d T2 relaxation effects across this range of relaxation times did not account for the findings.
95                                        T1rho relaxation times did not correlate with cartilage sGAG c
96     After excitation at 800 nm, the measured relaxation time distribution of multiple complexes has a
97 r minutes, we find that complexes sample the relaxation time distribution on a timescale of seconds.
98  generated at high fields dominated the T(2) relaxation time distributions and biofilm growth could n
99                     Instead, the microscopic relaxation times diverge so rapidly that, upon further c
100 t specific identifiers including microscopic relaxation times diverging on a Vogel-Tammann-Fulcher (V
101 t of 81.6 ns is needed as an addition to all relaxation times due to intrachain friction sources.
102                                     The fast relaxation time during dynamic relaxation is obtained as
103                           The dynamics of T2 relaxation times during the first post-infarction week w
104 lating configurational entropy to structural relaxation time) established in earlier numerical studie
105                                     The slow relaxation time exhibits an Arrhenius behavior with no s
106                               The average T2 relaxation time for all-cause rejection versus no reject
107                               The average T2 relaxation time for grade 0R (n=46) and grade 1R (n=17)
108  revealed 15-fold lower SNR and much shorter relaxation times for (35)Cl than (23)Na.
109 mputed using the WIEN2k code and the carrier relaxation times for electrons and holes are calculated
110                                          T2* relaxation times for normal cartilage (Beck score 1, 35.
111 rons and phonons saturates a quantum thermal relaxation time [Formula: see text].
112                                Long magnetic relaxation times have been demonstrated for single lanth
113  By analogy to linear polymers, shorter T(1) relaxation times have been traditionally associated to l
114 e report a strong enhancement in the optical relaxation time in Cu by direct growth of few-layer grap
115 ults show that the history dependence of the relaxation time in glasses requires knowledge only of th
116 values) for absolute signal intensity and T2 relaxation time in healthy subjects, their relativised v
117                                In humans, T2 relaxation time in the ischemic myocardium declines sign
118                      From T(1) (spin-lattice relaxation time in the laboratory frame) and T(1rho) (sp
119                                           T2 relaxation time in the myocardium (T2 mapping) and the e
120  laboratory frame) and T(1rho) (spin-lattice relaxation time in the rotating frame) measurements, it
121  is decoupled from that of the translational relaxation time in two dimensions but not in three dimen
122 ons as in solids over almost entire range of relaxation time in which liquids exist as such, and demo
123 rong anharmonicity, high reproducibility and relaxation times in excess of 40 mus at its flux-insensi
124 w the status of efforts to achieve long spin-relaxation times in graphene with its weak spin-orbit co
125 atter decreases were largely explained by T1 relaxation times in gray matter, a surrogate measure of
126 togenesis and found that reduced amygdala T2 relaxation times in high-magnetic-field MRI hours after
127 the association of global and regional brain relaxation times in patients with prior exposure to line
128 associated with a significant increase in T2 relaxation times in the ischemic region.
129       Therefore, the electron- and hole-spin relaxation times in these systems with zero or minimal s
130  isotope effect and the unusual variation of relaxation times in water at low temperatures can be exp
131                                          The relaxation time increase can be explained by correlated
132 nm, with a similar size dependence where the relaxation times increased from 140 +/- 10 to 310 +/- 15
133 the buffer solution the protein's rotational relaxation time increases exponentially, taking values i
134 diastolic filling, decreases, and isovolumic relaxation time increases, indicating that both active a
135                       The discrepancy in the relaxation times increases with increasing temperatures,
136                                   Transverse relaxation times indicated a lower degree of muscle dena
137  of proton spin density and a decrease of T2 relaxation time, indicating changes in the microstructur
138 BAA receptor concentration in addition to T1 relaxation times, indicating perhaps increased neuronal
139                         The slow hot carrier relaxation time is 0.5 ps.
140                                       The T1 relaxation time is a robust noninvasive imaging biomarke
141 lease in our model is that the matrix stress relaxation time is comparable to the time scale for wate
142 ynamic behaviour reveals that the structural relaxation time is substantially reduced in these adapti
143 tra further suggest that the distribution of relaxation times is temperature independent at low frequ
144                                   Isovolumic relaxation time (IVRT), early diastolic filling (E/A), m
145 se from observing hyperpolarized (1)H, short relaxation times limit the utility of prepolarizing this
146 ies, E:A ratio, deceleration, and isovolumic relaxation times; LV systolic function was preserved.
147  transversal (T2) nuclear magnetic resonance relaxation time mapping.
148                      Quantitative MR imaging relaxation time maps demonstrated up to a twofold variat
149                     The authors reviewed T2* relaxation time maps of 28 hips from 26 consecutive pati
150                               T(2) and T(2)* relaxation time maps were created from the multiecho seq
151 s a suppression of the Knight shift and spin relaxation time measured in nuclear magnetic resonance (
152                                          The relaxation times measured at variable temperature provid
153                                  The longest relaxation times measured correspond to estimated viscos
154 ch as MR spectroscopy, T1rho calculation, T2 relaxation time measurement, diffusion quantitative imag
155  translational diffusion and proton T1rho/T2 relaxation-time measurements for rotational diffusion, t
156 ed mitral E deceleration time, LV isovolumic relaxation time, mitral E/E', and pulmonary vein A wave
157 itative spin-lattice (T1) and spin-spin (T2) relaxation time MR imaging mapping was performed before
158  spectroscopy we resolve loss peaks yielding relaxation times near 100 s at 126 K for low-density amo
159 ated via analysis of the NMR line shapes and relaxation times observed between 12 and 400 K.
160                  (2)H NMR MAS spectra and T1 relaxation times obtained from the deuterated phenylalan
161 ar10 s(-1), which corresponds to the inverse relaxation time of a healthy red blood cell.
162 c environments by measuring the longitudinal relaxation time of a single-spin probe as it is systemat
163 re than one order of magnitude in the energy relaxation time of a superconducting artificial atom.
164 ve at room temperature with a characteristic relaxation time of about one month.
165  by the hydrogen bonds did not influence the relaxation time of cytosine significantly due to the gen
166 values upon annealing, but the difference in relaxation time of density and hardness, which is usuall
167 red in stroked hemispheres; and longitudinal relaxation time of lactic acid was found to increase by
168 haracteristic times of the system, e.g., the relaxation time of local excitations.
169 he Lorentzian noise component determines the relaxation time of molecular fluctuations, which, in tur
170  Moreover, the temperature dependence of the relaxation time of orientational correlations is decoupl
171                       We determine an energy relaxation time of T1 approximately 100 microseconds and
172                                  The singlet relaxation time of the (13)C(2) pair in [1-(18)O,(13)C(2
173                             We show that the relaxation time of the confined DNA is >10 min, which is
174 ggests a decoupling of the amplitude and the relaxation time of the membrane thickness fluctuations,
175                                      (1)H T2 relaxation time of the most abundant population (populat
176 ased on causality - on the comparison of the relaxation time of the order parameter with the "time di
177                          Changes in the spin relaxation time of the sensor located in the lipid bilay
178                                          The relaxation time of the solvent protons in 3 mM solutions
179                                  The mean T1 relaxation time of thrombus was shortest at 7 days follo
180  different from surrounding liver parenchyma relaxation times of 840 msec +/- 113 and 28 msec +/- 3 (
181 s reached the limit of the microsecond-scale relaxation times of biological molecules bound to a forc
182                                              Relaxation times of healthy thigh muscle and brain tissu
183                                           T2 relaxation times of in vivo-labeled MSC transplants and
184                               Mean T1 and T2 relaxation times of lymphatic fluid at 3.0 T were 3100 m
185 CPMG) sequence was used to measure spin-spin relaxation times of proton pools representing major yolk
186 aging is based on the dependence of the spin relaxation times of protons in water molecules in a host
187 and that the extracted temperature dependent relaxation times of the assemblies follow the Vogel-Fulc
188                                  The mean T2 relaxation times of the liver and focal hepatic lesions
189                                  The mean T2 relaxation times of the liver and focal liver lesions as
190                                         T(2) relaxation times of the majority proton population sugge
191  The relative intensities and the transverse relaxation times of the NMR signal components associated
192 er, we show that both the magnitudes and the relaxation times of these backbone breathing fluctuation
193 )H-SABRE experiments, and we record (15)N T1 relaxation times of up to 2 min.
194 shear viscosity, plateau modulus, and stress relaxation time) of the shear-thickening precursor are s
195 ure the longitudinal relaxation time, the T1 relaxation time, of protons in a magnetic field after ex
196 tionship between postcontrast ventricular T1 relaxation time on CMR and freedom from AF after pulmona
197        A shorter postcontrast ventricular T1 relaxation time on CMR is associated with reduced freedo
198 re probed, enabling us to determine the spin relaxation time on the island.
199 r myocardial water content at 120 min and T2 relaxation times on 120 min cardiac magnetic resonance t
200                       The impact of electron relaxation times on the DNP enhancement (epsilon) is exa
201  approximately half of the longitudinal spin relaxation time over a wide range of temperatures, which
202 correlate the above g-shifts to spin-lattice relaxation times over four orders of magnitude, from 200
203 ersus controls (288 +/- 13.4), but not of T2 relaxation time (p = 0.49).
204 een different proton pools with different T1 relaxation times, particularly when the starch gelatiniz
205 t longer inversion recovery and phase memory relaxation times provide larger epsilon.
206 al shortening, ejection fraction, isovolumic relaxation time, rate of pressure development and rate o
207 ation approach to obtain backbone (15)N spin relaxation time ratios T1/T2 for a monomeric EIAV-CA in
208 compared with control patients, half maximal relaxation time (RT50) at 60 per minute was prolonged by
209 s substantially larger than the viscoelastic relaxation time scale of the biofilms, and this appearan
210                       We extend the NMR spin relaxation time scale sensitivity deeper into the nanose
211                                   Isovolumic relaxation time shortened (baseline 109.1 +/- 21.7 ms vs
212  paramagnetic Fe3+, which causes thrombus T1 relaxation time shortening.
213 t long-lived states (LLSs) in solution, with relaxation times substantially longer than the conventio
214 relation between regional gray matter and T1 relaxation times suggests decreased tissue water content
215 scales depending on the nuclear spin-lattice relaxation time T(1) in the range of seconds.
216 te is 2-3 times longer than the spin-lattice relaxation time T(1).
217 rally identifiable and shortening of the MRI relaxation times T 1 and [Formula: see text].
218                Since both isotopes have long relaxation times T(1), the hyperpolarized NMR signal of
219 er of magnitude compared to the spin lattice relaxation time (T 1), but they have to be prevented fro
220 nce was used to calculate post-contrast T(1) relaxation time (T(1) time) of the LV myocardium as an i
221 iradical having a long electron spin-lattice relaxation time (T(1e)) has been developed as an exogeno
222 rticles, specifically by measuring spin-spin relaxation time (T(2)), is reported.
223 ic solids characterized by long spin-lattice relaxation times (T(1)((1)H) > 200 s), (1)H-(1)H spin di
224 sion using the biexponential analysis of the relaxation times (T(21), T(22)) and amplitudes (A(21), A
225   We report measurements of the longitudinal relaxation time T1 of brain tissue, blood, and scalp fat
226 ly longer than the conventional spin-lattice relaxation time T1.
227                                        Qubit relaxation times T1 across 22 qubits are consistently ma
228 -weighted MR imaging, quantitation of native relaxation times T1 and T2, the relaxation rate R2*, and
229 2) min(-1)] and in-cell (129)Xe spin-lattice relaxation time (T1 = 2.19 +/- 0.06 h) for 1000 Torr Xe
230                                 Quantitative relaxation time (T1 and T2) measurements were made in ex
231 l to the study of intelligence: longitudinal relaxation time (T1) and magnetisation transfer ratio.
232                          (35)Cl longitudinal relaxation time (T1) and T2* of healthy human brain and
233 quantities in spintronics are the population relaxation time (T1) and the phase memory time (T2): T1
234  Gd-DTPA did not alter vitreous longitudinal relaxation time (T1) at baseline or at 3 hours after NMD
235          The magnetic resonance longitudinal relaxation time (T1) changes with thrombus age in humans
236          Reduction of the (19)F spin-lattice relaxation time (T1) enables rapid imaging and an improv
237  is "turned on" by altering the longitudinal relaxation time (T1) of bulk water protons.
238 e liquid state according to the spin-lattice relaxation time (T1) of the nucleus.
239 er of magnitude compared to the spin lattice relaxation time (T1), but they have to be prevented from
240  lifetime of the qubit, and the spin-lattice relaxation time (T1), the thermally defined upper limit
241                                     (1)H NMR relaxation times (T1 and T2) were measured at low field
242                  Based on their spin-lattice relaxation times (T1), two dimensional (1)H NMR spectros
243 oach to correlate spin-lattice and spin-spin relaxation times (T1-T2) including acquisition of the FI
244 meters (concentration [rho] and longitudinal relaxation time [T1]) of human cortical bone in vivo.
245 ity of lifetime of spin states (spin-lattice relaxation time, T1) and coherences (spin-spin relaxatio
246 hically inequivalent carbon and spin-lattice relaxation times, T1, yield characteristic information t
247 ect of B1(+) correction on the native tissue relaxation time (T10) of fat, parenchyma, and malignant
248 ance (MR) relaxometry demonstrating short T1 relaxation time (T1R) in the basal ganglia reflects exce
249 r present 2D images of T1 and the transverse relaxation time T2 of the brain and show that, as expect
250  by microstructure, influence the transverse relaxation time (T2) in an orientation-dependent fashion
251 etic resonance imaging (MRI)-based spin-spin relaxation time (T2) mapping has been shown to be associ
252                   Quantitatively, transverse relaxation time (T2) of CSS increased non-linearly with
253      Diffusion tensor imaging and transverse relaxation time (T2) relaxometry were performed at basel
254 ance imaging showed a significant transverse relaxation time (T2) shortening in the pancreata of mice
255 es (50-80 K) to increase the short spin-spin relaxation time (T2) upon which the technique relies.
256    The MRI outcomes-fat fraction, transverse relaxation time (T2), and magnetisation transfer ratio (
257 by downhill running (DR) by using transverse relaxation time (T2)-weighted magnetic resonance imaging
258 he characteristic distribution of transverse relaxation times (T2) within dendrimers (shorter values
259 ulations were identified measuring spin-spin relaxation times (T2).
260 laxation time, T1) and coherences (spin-spin relaxation time, T2) to the immediate environment was ut
261                        Regional native T1/T2 relaxation time, T2-weighted ratio, and extracellular vo
262 llenging analysis of previtreous behavior of relaxation time (tau(T)) in ultraviscous low molecular w
263  binding kinetics of these measurements, the relaxation time (tau) was obtained, where higher tau val
264 s attributed to an increase in the carrier's relaxation time (tau).
265 quilibrium shape fluctuations that contain a relaxation time, tau, which is robust and independent of
266 nt study shows that I1 converts to I2 with a relaxation time tau1=0.1s at 25 degrees C in 25 mM KCl.
267 n transition metal ions with long electronic relaxation times (taus) is demonstrated.
268 oup 1 medium showed significantly shorter T2 relaxation times than hMSCs labeled with group 2-5 media
269  form a glass that behaves as a solid with a relaxation time that grows exponentially with decreasing
270 ostructures have longitudinal and transverse relaxation times that are on par with commonly used heav
271 d phosphonated trityl radical possesses long relaxation times that are sensitive to probe the microen
272 ging method used to measure the longitudinal relaxation time, the T1 relaxation time, of protons in a
273 show by a Redfield theory calculation of the relaxation times, the distribution shape corresponds to
274 ensemble, we demonstrate that, regarding the relaxation times, the ensemble can be considered ergodic
275 ental effects on the spin-label phase memory relaxation time Tm .
276  study underlines the potential of native T1 relaxation times to complement current cardiac MR approa
277  weak anti-localization effect and to a spin-relaxation time two to three orders of magnitude smaller
278                                          The relaxation time values and their associated relative pop
279 erred from an Arrhenius plot of the magnetic relaxation time versus the temperature.
280                         Quantification of T1 relaxation time was a good predictor of successful throm
281                                      Mean T1 relaxation time was increased in HCM and DCM (HCM 1209+/
282                                          T2* relaxation time was measured before and after maternal h
283                                           T1 relaxation time was related to thrombus methemoglobin fo
284                              In contrast, T2 relaxation time was significantly higher in controls (ti
285     The volume of lung tissue with increased relaxation times was determined by using a threshold-bas
286 nt (ADC), fractional anisotropy (FA), and T2 relaxation time were calculated.
287 e mono- and bi-exponential short and long T2 relaxation times were 24.7 ms, 4.2 ms (fraction 15%) and
288                             On day 7 CMR, T2 relaxation times were as high as those observed at reper
289 or-encoded parametric maps of T2* transverse relaxation times were calculated on a pixel-by-pixel bas
290 (n = 12), tissue-specific magnetic resonance relaxation times were compared before and after lithium
291 tial and bi-exponential short and long T1rho relaxation times were estimated to be 26.9 ms, 4.6 ms (f
292                                           T1 relaxation times were measured before and after gadolini
293                                       The T2 relaxation times were measured using the 16-echo Carr-Pu
294                              The mean log T2 relaxation times were reduced by 62% and 43%, respective
295                                    Native T1 relaxation times were significantly longer in patients w
296 TAM)-based spin label with a relatively long relaxation time where the protein is immobilized by atta
297 ecedentedly observed field dependence of the relaxation time, which was modeled with three contributi
298 greatly facilitated the interpretation of T1 relaxation times, which have been interpreted rather nar
299 l T1 nuclear magnetic resonance (NMR) proton relaxation times, which is proportional to amount of car
300 t proton spatial distributions and different relaxation times, which may also provide information abo

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