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1 ulate the release of the endothelium-derived relaxing factor.
2 um to cause synthesis of endothelium-derived relaxing factor.
3  to influence release of endothelium-derived relaxing factor.
4 members of the family of endothelium-derived relaxing factors.
5                    As an endothelium-derived relaxing factor, a mediator of immune responses, a neuro
6 NO) is identified as the endothelium-derived relaxing factor and a neurotransmitter with a superfusio
7 e degradation of NO, the endothelium-derived relaxing factor and a primary mediator of endothelial fu
8  considered an important endothelium-derived relaxing factor and may function to protect blood vessel
9 ide radical can scavenge endothelium-derived relaxing factor and produce potent oxidizing agents, whi
10 e biological activity of endothelium-derived relaxing factor and that this decrease can be reversed b
11    The identification of endothelium-derived relaxing factor as nitric oxide (NO) dramatically altere
12 counts in large part for endothelium-derived relaxing factor bioactivity.
13 sporine, wortmannin, the endothelium-derived relaxing factor congener S-nitroso-N-acetylcysteine (SNA
14         The discovery of endothelium-derived relaxing factor (EDRF) and its importance in the identif
15 sitive, and synthesis of endothelial-derived relaxing factor (EDRF), therefore, may be most important
16 um ADMA (S(ADMA)) and/or endothelium-derived relaxing factor (EDRF)/NO.
17 n the endothelium (e.g., endothelium-derived relaxing factor, endothelium-derived hyperpolarizing fac
18 hey also are physiologic endothelial-derived relaxing factors for blood vessels.
19 dulin, eNOS produces NO, endothelial-derived relaxing factor, from l-arginine (l-Arg) by means of ele
20 ally active component of endothelium-derived relaxing factor, has critical roles in the maintenance o
21              A number of endothelium-derived relaxing factors have been identified including nitric o
22 cardiac tissue, is known to act as a soluble relaxing factor in fast skeletal muscle fibers by acting
23 t results in impaired release of endothelial relaxing factors including NO.
24 iscover and identify new endothelium-derived relaxing factors involved in the regulation of erectile
25 ensed and transduced to cause the release of relaxing factors is poorly understood.
26 ght to determine whether endothelium-derived relaxing factor (nitric oxide) exerts a tonic vasodilati
27 oxide synthase-dependent endothelium-derived relaxing factor response and paradoxically enhances cont
28 orelaxant effects of the endothelium-derived relaxing factor S-nitrosocysteine (SNC) may not be simpl
29 hat NO is the prototypic endothelium-derived relaxing factor, this signaling molecule was implicated

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