ライフサイエンスコーパス: release

1 impairs decidualisation and alters chemokine release.

2 icularly at C-domain of CaM, enabling Ca(2+) release.

3 ferents and can negatively regulate dopamine release.

4 harge trapping and optically-mediated charge release.

5 t suppression of tumor necrosis factor alpha release.

6 that epithelial polarity impacts directional release.

7 optimize linker stability for mitochondrial release.

8 F96365) also reduced MSU crystal-induced NET release.

9 e the structural basis of allosteric product release.

10 g gemcitabine after heat mediated controlled release.

11 -dependent manner to cause virus budding and release.

12 e of negative potential (-2.0 V) induces the release.

13 neous release as well as synchronous, evoked release.

14 tion, sialic acid modification, and N-glycan release.

15 enotype-Tissue Expression (GTEx) project v6p release.

16 hich may selectively inhibit cell-free virus release.

17 mechanism underlying KCC2-dependent insulin release.

18 X7 receptor activation to stimulate IL-1beta release.

19 ulation of polypeptide substrate binding and release.

20 nization and Ca(2+) cooperativity of vesicle release.

21 gestion, 77% carotenoids and 67% tocols were released.

22 the same accuracy as fully hand-curated SCOP releases.

23 ty chemicals involved in accidental chemical releases.

24 e reduction, an important control on methane releases.

25 When activated to degranulate, they release a plethora of bioactive compounds from their sec

26 CalQuo (2) differentiates cells that release a single calcium response and those that release

27 We are releasing a tool to allow users to identify areas of the

28 exhibited increased caffeine-induced Ca(2+) release across a wide range of concentrations in the abs

29 trated that adiponectin and nitric oxide are released after activation of adipocyte-expressed beta3 a

30 a key role in METH dysregulation of dopamine release and dopamine-related behaviors.

31 have opposing physiological roles in insulin release and glucose homeostasis.

32 -cells of adult mice greatly impairs insulin release and glucose tolerance in mice fed with a calorie

33 onship between novelty-induced acetylcholine release and later contextual associability.

34 inly metabolized via glycerol production and release and lipid synthesis (particularly FFA, triglycer

35 n had no influence on short-term fluorescein release and pre-coating of beads with body fluids did no

36 t their stability after processing and their release and solubility during digestion.

37 of synaptic transmission via massive vesicle release and subsequent failure to endocytose lost vesicl

38 or proteins necessary for GABA synthesis and release and that sensory neurons released GABA in respon

39 was taken complemented by detection of N2 O released and nitrogen isotope determinations of fern bio

40 ar vesicle biology, including cargo sorting, release, and bioactivity.

41 contents, free fatty acid (FFA) content and release, and cholesterol and cholesterol esters increase

42 ns such as ATP production, Ca(2+) uptake and release, and substrate accumulation depend on the proton

43 om BMP group showed >1,000-fold higher BMP-2 release, and the majority of them stained intensely for

44 elial cell activation, matricellular protein release, and tissue damage were measured at admission.

45 trongest IL-6 and matrix metalloproteinase-3 release, and was even more pronounced compared with supp

46 We anticipate that the suggested pull and release approach to graphene folding may find applicatio

47 Here we show that glutamate-releasing ARC neurons expressing oxytocin receptor, unli

48 that calcium-dependent processes involved in release are altered with age.

49 t and action potential-dependent spontaneous release as well as synchronous, evoked release.

50 Cerium oxide released as a result of the combustion of diesel fuel co

51 ide (MTT) assay, lactate dehydrogenase (LDH) release assay, Hoechst 33342 staining, annexin V/PI stai

52 shed light on the question-the site of BDNF release at the synapse.

53 ore, desferrioxamine B (DFOB), iron (Fe) was released at higher rates and to greater extents relative

54 The flame retardant BDE-209 may release BDE-99 and other lower brominated BDEs through b

55 duced degradation of self-immolative linkers released BENSpm from the DSS-BEN polymers.

56 In addition to releasing bradykinin from HK, Kal directly activated mon

57 ydrogel was also designed to encapsulate and release bulky globular proteins, such as mCherry, in a l

58 m, involving variations not only in dopamine release but also in dopamine neuron connectivity, cotran

59 Here, we investigated whether VEGF release by HPMCs is controlled by IL-6 in combination wi

60 role of pannexin-1/ATP/P2X7 axis in IL-1beta release by human monocytes.

61 in, Nef, which is suspected of extracellular release by infected CD4+ T cells on protein quality cont

62 has been shown to be neuroprotective and is released by astrocytes.

63 our work identified the miRNAs specifically released by different human CD4(+) T cell subsets and st

64 Exosomes are vesicles released by many eukaryotic cells; their cargo includes

65 Acyl groups released by PLIP1 are exported from the chloroplast, rei

66 accumulated in the TT region and then can be released by resuming a conventional TW in the ST region.

67 pendent of IL-26, because both natural IL-26 released by Th17 clones and rhIL-26 lacked antimicrobial

68 ulated their C5aR and FcRIII expression, and released C5a.

69 omote divisions by loss of E-cadherin, which releases cadherin-associated beta-catenin (Armadillo in

70 sing transition metals; however, ring-strain release can provide the necessary thermodynamic driving

71 s of HeLa cells an ATP-dependent factor that releases Cdc20 from MCC and identified it as chaperonin

72 ntrol electrical properties and drug-loading/release characteristics.

73 ed that RAG1/2 causes aberrant insertions by releasing cleaved antibody gene fragments that subsequen

74 -1beta] secretion, and lactate dehydrogenase release) compared to that with the hypha-competent contr

75 n corresponds to deprotonation of the proton release complex (PRC), a complex in the extracellular do

76 the excited state of the E:THF:NADPH product release complex, the reduced nicotinamide ring of the co

77 naling proteins, neuropeptides and vesicular release components, and transcription factors.

78 er processing, the release was lower and the released compounds were not degraded.

79 Over longer timescales, CO2 release could act as a negative feedback, limiting progr

80 omparison of experimental and predicted drug release data revealed that in addition to surface area,

81 Furthermore, loss of release did not disrupt the morphogenesis of presynaptic

82 llate ATTM represents a new class of sulfide-releasing drugs.

83 rdiac myocytes should inhibit further Ca(2+) release during the action potential plateau.

84 We observed that exosomes released during a mouse M. tuberculosis infection contri

85 Extracellular vesicles (EVs) released during cell stress, or demise, can contain a ba

86 Histones released during influenza induced cytotoxicity and showe

87 ol-sensitive proteins that control glutamate release (e.g., SV2A, synaptogyrin-1) and postsynaptic si

88 hboring cells by the enzymatic action of the released endolysin.

89 reviously found that neutrophils produce and release Eosinophil Cationic Protein and histamine, two i

90 es spontaneous and inhibits Ca(2+)-triggered release events.

91 lized particle-based system can specifically release Ex4 while immobilizing GOx as a result of the di

92 bserved abnormal expression of corticotropin-releasing factor receptor type 2 (CRFR2) to be associate

93 We found that corticotropin-releasing factor type 1 receptor within the paraventricu

94 buting ions, removing neurotransmitters, and releasing factors to influence blood flow and neuronal a

95 ormation of arbitrarily shaped structures of released film and locally specified thickness for each r

96 vated by extracellular ATP and UTP molecules released following injury/stress.

97 an effective platform to produce controlled release formulation of anti-cancer drugs, and ATRA-PLLA

98 pidomic analyses indicate that TAG lipolysis releases free fatty acids at a time that correlates well

99 Abeta release from and transport across HBMEC were significant

100 and cell yield as L1 medium and observed DIP release from ATP into the medium, suggesting that K. mik

101 harmacological studies on chemokine/cytokine release from human macrophages, the prostanoid EP1 recep

102 ction potentials (bpAPs) could elicit Ca(2+) release from lysosomes in the dendrites.

103 next amperometrically analyzed catecholamine release from PC12 cells, revealing that charge neutraliz

104 Desmoglein 2 modulates extracellular vesicle release from squamous cell carcinoma keratinocytes.

105 tuations at the soma affect neurotransmitter release from synaptic boutons.

106 The mechanisms of metal release from the articulation at the head cup bearing an

107 cription factors that are required for sperm release from the pollen tube to the female gametes, a cr

108 L offsets increased SR load such that Ca(2+) release from the SR was maintained during ageing.

109 The release from the water-based coating was comparably high

110 the key to HSPC maintenance and suggest that release from this suppressive mechanism is a fundamental

111 tment not only increases interleukin (IL)-33 released from breast tumor cells, which is crucial for t

112 ns can be explained if ACh and glutamate are released from common vesicles onto spatially segregated

113 on is a key process for silver nanoparticles released from consumer products in the environment.

114 les and damage-associated molecular patterns released from host cells.

115 Previous work showed that Mad2 is released from MCC by the joint action of the TRIP13 AAA-

116 via a number of endocytotic pathways and are released from membrane-enclosed endocytotic organelles,

117 ATP is also released from sensory-motor nerves during antidromic ref

118 are affected by spore density and chemicals released from spores, germination interactions were quan

119 EGF in circulation, yet how EGF is stored or released from stimulated cells is undefined.

120 Upon DNA damage, p53 mRNA is released from stress granules and associates with polyri

121 N2 O was not detectably released from surface-sterilized ferns with the Rhizobia

122 Full-length uPAR is released from the cell surface, but the mechanism and si

123 They are released from the contractile ring as it disassembles an

124 On mitotic entry, NuMA is released from the nucleus and competes LGN from E-cadher

125 ug/L) that were five times higher than those released from the simulated full LSL.

126 roken, PM "bubbles" with exposed PS that are released from the surface of the otherwise intact cell.

127 Total silver released from the test sample and the evaluated AgNP sta

128 cently, lipids have been identified that are released from tissues and act locally or systemically to

129 urface environments, or due to anthropogenic releases from waste rich in antimony, a component used i

130 the NPC whose barrier, transport, and cargo release functionalities establish a continuum under a me

131 nthesis and release and that sensory neurons released GABA in response to depolarization.

132 nd that LPS-activated BV-2 microglia rapidly released Gal-3, which was blocked by calcineurin inhibit

133 ISPR-mediated genome editing to controllably release GLP-1 (glucagon-like peptide 1), a critical incr

134 PSMA's carboxypeptidase activity releases glutamate from vitamin B9 and other glutamated

135 The encapsulated lactase was released gradually during the simulated digestions to hy

136 th inadequate compensation by Growth hormone-releasing hormone (GHRH) and Growth hormone (GH), undera

137 fects of agonistic analogs of growth hormone-releasing hormone (GHRH) and their mechanism of action w

138 ticotrophin-releasing hormone or thyrotropin-releasing hormone and do not express arginine vasopressi

139 these neurons largely express corticotrophin-releasing hormone or thyrotropin-releasing hormone and d

140 eptors, and by the hypothalamic gonadotropin-releasing hormone through activation of PKA.

141 de homologous to the vertebrate gonadotropin-releasing hormone, is downregulated as workers become ga

142 ect of a biodegradable bimatoprost sustained-release implant (Bimatoprost SR).

143 iod for Ca(2+) spark initiation after Ca(2+) release in cardiac myocytes should inhibit further Ca(2+

144 was performed to test the necessity of BDNF release in driving scopolamine-induced behavioral respon

145 replication, entry, spread, production, and release in hepatoma cells and HFLCs.

146 IX more efficiently attenuated PGE2 and IL-6 release in HG+IL-1beta-treated cells than in NG+IL-1beta

147 iding an improved opportunity for controlled release in higher-stage patients.

148 nvolved in FXa-mediated intracellular Ca(2+) release in HUVEC and FXa reactive IgG from patients with

149 ot only blocked exendin-4-stimulated insulin release in islets but also lowered insulin levels while

150 ac muscle cBIN1, both of which support cBIN1 release in MPs from human hearts.

151 prominent photothermal effect and quick drug release in response to NIR irradiation.

152 to meaningful predictions of near-future CH4 release in the Arctic.

153 Lower dopamine release in the associative striatum correlated with inat

154 Wheat flour particles of more than 60mum (released in air by sifting) dropped mainly in the perpen

155 ich would be expected from a substance to be released in response to RIPC and to protect the myocardi

156 that synaptotagmin-7-dependent asynchronous release indeed does not produce a prolonged synaptic sig

157 roscopy) and biophysical measurements of ATP release indicate that G100V/C103V retards initial fusion

158 ic beta cells are functionally programmed to release insulin in response to changes in plasma glucose

159 ous matrix acting like a filter favoring the release into water of carboxylic and fulvic acid-like co

160 the intermembrane space of mitochondria and released into bloodstream during pathological conditions

161 both genes are found at the cell surface and released into media.

162 on the human body and the ecosystem, can be released into soils, ground-, and surface waters either

163 tic and temporal variations, how ENMs may be released into the environment, and the effect of compart

164 Following aquarium releases, invasive lionfishes have colonized large areas

165 es a major absorption peak at 422 nm without releasing iron or sulfide from the clusters.

166 LC/MS analytics that the intracellular cargo release is controlled by the sequence of the peptide lin

167 ticles taken orally, in particular, the drug release kinetics and interaction with the gastrointestin

168 ed and 2 generic drugs) on-market, immediate-release lamotrigine drug products.

169 l during the Siberian flood-basalt eruptions released large amounts of CO2 and CH4 into the atmospher

170 cause ZnO-NPs formed larger aggregations and released less zinc ions (Zn(2+)) at greater temperature

171 genic rice lines overexpressing (OX) OsALMT4 released malate from the roots constitutively and had 2-

172 differs by HIV-1 status and compartment, and releases matrix degradation products.

173 ent on our Article "Evidence of the hydrogen release mechanism in bulk MgH2", Surrey et al. assert th

174 Sterilized, impacted, spill-site sediment released minor amounts of cis- and up to 35 mug/L of tra

175 light to introduce compounds into cells, to release molecular species from cells or to selectively i

176 The masking release (MR; i.e., better speech recognition in fluctuat

177 m the HCCS active site; thus, we term these "release mutants."

178 ypocretin and GABA (gamma-aminobutyric-acid)-releasing neurons of the lateral hypothalamus, which pro

179 n and a neutron n (), resulting in an energy release of 12 MeV.

180 ymatic activity of PARN is necessary for the release of 18S-E from Bystin-associated pre-40S particle

181 ranulation of mast cells and basophils, with release of agents of the allergic response, ensues when

182 ry system has been fabricated for controlled release of alendronate (AL).

183 ulin, and downstream signaling regulated the release of antibacterial myeloperoxidase and lactoferrin

184 and Zn-AMSs enhanced maturation and cytokine release of bone marrow dendritic cells in vitro.

185 e under near-UV irradiation, led to complete release of carbonyls.

186 s with the plasma membrane, resulting in the release of Cathepsin B.

187 In vitro study showed more sustained drug release of CM-AL-containing scaffolds than these of CM/n

188 In contrast, the release of d-[(14) C]aspartate was preferentially sensit

189 We hypothesize that the local release of drug into the tumor vasculature and resulting

190 queous phase showed a high stability and low release of encapsulated compounds over time.

191 bination of failed clearance and exaggerated release of glutamate by glial cells during immune activa

192 oendocrine circuit that evokes the pulsatile release of gonadotropin hormones (luteinizing hormone an

193 d ursodeoxycholic acid on the expression and release of HbetaD1 and HbetaD2 from colonic epithelial c

194 to describe the generation, consumption and release of heat from landfills, to predict landfill temp

195 gen exposure through several steps including release of IL-33, which promotes cytokine (IL-5, IL-13)

196 by acting on myeloid cells and promoting the release of inflammatory molecules, including IL-1beta.

197 ntly respire nitrate without the significant release of intermediates, such as nitrous oxide.

198 r signalling and PI3K-AKT-mTOR axis leads to release of MCL cells from TME, reversal of drug resistan

199 le geometries for the controlled loading and release of medicines is presented.

200 We suggest that upregulation and release of miR-21 contribute to sensory neuron-macrophag

201 duced mitochondrial respiratory capacity and release of mitochondrial DNA into the cytosol.

202 As an example, we simulated ten years of release of nano CeO2, CuO, TiO2, and ZnO in the San Fran

203 vered that IkappaBalpha enhances the rate of release of nuclear factor kappa B (NFkappaB) from DNA ta

204 The pattern of release of oil from the microcapsules and co-microcapsul

205 e by steady-state analyses claiming that the release of one cationic species as product requires the

206 transcriptional enhancers might modulate the release of paused RNAPII via 3D chromatin looping.

207 National release of PFASs was estimated by coupling measured conc

208 econd phase of the biphasic force decay upon release of phosphate from caged phosphate was previously

209 d significantly lower metabolic activity and release of pro-inflammatory cytokines than CFC tissue, b

210 idoglycan (PG), pose a major problem for the release of progeny virions.

211 The resulting hydrogel provides sustained-release of protein for more than a week.

212 ble polymeric microspheres for the sustained release of proteinaceous drugs.

213 ned, processively copied, and regenerated by release of single-stranded product DNA.

214 sure of these cells to allergens induces the release of soluble mediators causing allergic symptoms.

215 Early steps in infection include release of the capsid into the cytoplasm, docking of the

216 hat I2 is required for virion morphogenesis, release of the D13 scaffold, and the association of EFC

217 hey are activated in cancer cells, involving release of the I(-) ligand in the presence of glutathion

218 the first domain is a rate limiting step for release of the inactivation domain, and highlights the f

219 t the target compound ee and the synchronous release of the indicator results in a nonenantioselectiv

220 the intermembrane space likely involves the release of the protein precursor within the lipid bilaye

221 molecules at the membrane and the continuous release of the proteins from the vesicle to the plasma m

222 -binding site, respectively, suggesting that release of the Q toward the membrane is coupled to an en

223 okines IL-6 and IL-12p40 while enhancing the release of the regulatory/anti-inflammatory cytokine IL-

224 es for a range of applications, with in situ release of the required hydrogen from a stable liquid of

225 lation involving the growth hormone-mediated release of the transcriptional blockade of genes associa

226 4 detected annihilation events from a single release of the trapped anti-atoms accumulated from five

227 docking of the capsid at a nuclear pore, and release of the viral genome into the nucleus.

228 the capsid engages the NPC and what triggers release of the viral genome into the nucleus.

229 ed contraction can be explained by a greater release of thromboxane from PVAT from female animals and

230 Blood-brain barrier disruption (BBB) and release of toxic blood molecules into the brain contribu

231 Release of Tr-A from both formulations was greatly accel

232 y pulsed electric field showed a significant release of trans-(4.01+/-0.48) and cis-(5.04+/-0.26mug/g

233 rimary trophoblasts through the constitutive release of type III IFNs (IFNlambda1 and IFNlambda2) and

234 Direct releases of CO2 from litter layer only accounted for 19

235 s with body fluids did not affect short-term release or antibacterial activity.

236 ase a single calcium response and those that release oscillatory calcium fluxes.

237 1-mediated transduction of VEGF signaling to release paused RNAPII.

238 le moths compete to arrive first at a female releasing pheromone.

239 n of greater than two as a glucose acceptor, releasing phosphate from glucose 1-phosphate.

240 ion through the impairment of endogenous NPY release, potentially contributing to heightened anxiety.

241 Thus, substrate is released prior to ATP hydrolysis.

242 Moreover, curcumin exhibited sustained release profile from crosslinked nanogels in simulated g

243 nt growth was further enlightened through Cu release profile of Cu-chitosan NPs.

244 ormulation through comprehensive analyses of release profiles and cellular-uptake and cell viability

245 uoles to mature in an acidic environment and release progeny virions in a membrane-mediated cell-to-c

246 sis is the reduced capacity of leukocytes to release proinflammatory cytokines in response to ex vivo

247 se mechanisms is that LCR exhibit complex Ca release propagation patterns (including merges and separ

248 aser with particularly pronounced serotonin- releasing properties, has unique subjective effects that

249 dent fashion thereby indirectly reducing the released protein.

250 sampling, detection, and characterization of release rate and form were applied: Transformation of th

251 age forms, implants or stents to enhance the release rate of eluting drug from polymer-rich structure

252 arameters, the diffusion coefficient and the release rate parameter, are automatically estimated from

253 llikrein generation and excessive bradykinin release resulting from cleavage of high-molecular-weight

254 rs in IVIGs and to ensure consistent product release, revaccination of plasma donors was investigated

255 Subsequent treatment with RNase H releases RNA-templated ligation products into solution f

256 he cytochrome bc family limit the amounts of released ROS to a low, perhaps just signaling, level thr

257 Standardized protocols/methods for release sampling, detection, and characterization of rel

258 First, locally generated DA release signals are organized over more than four orders

259 nally differentiated, neuronal SH-SY5Y cells release significantly less extracellular HSV-1 by 24 h p

260 lassifies the majority of protein structures released since SCOP development concluded in 2009, using

261 segregation of cholinergic and glutamatergic release sites is also possible.

262 Release sites were distributed throughout the AZ and und

263 cement of the LSL was predicted to result in releasing spikes with significantly high concentrations

264 y HCCS mutants such as E159A are enhanced in release (step 4) of cyt c from the HCCS active site; thu

265 t at the cost of biogeochemical changes that release stored contaminants.

266 Release studies found that solid polymeric samples lost

267 lucose (up to +81%) and xylose (up to +153%) release, suggesting that down-regulating CAD1 is a promi

268 c receptors had no effect on evoked dopamine release, suggesting that feedback inhibition of acetylch

269 A prolonged-release tacrolimus, basiliximab, and mycophenolate mofet

270 l tone) is induced by brainstem neurons that release the monoamines serotonin and noradrenaline, and

271 ng; D1R-) spiny projection neurons (SPNs) co-release the opioid neuropeptide dynorphin, which acts at

272 rins bind to the prodomain, apply force, and release the TGF-beta growth factor.

273 PlexinD1 binding to GIPC1 releases the autoinhibition, promoting its interaction w

274 oteasomes, but not standard 26S proteasomes, releasing the 20S catalytic immunoproteasome.

275 on of the receptive synergid and PT rupture, releasing the sperm cells for double fertilization.

276 rate constant for actin-activated phosphate release, the biochemical step in myosin's ATPase cycle a

277 slower phase (>10 s), when stalled complexes release their short RNA and make another without escapin

278 red by caspase-1-dependent interleukin-1beta release, though this phenotype could be suppressed by ph

279 subject to quality assurance review prior to release to ensure high quality.

280 over BA circuitry via 5-HT and glutamate co-release to inhibit the BA output.SIGNIFICANCE STATEMENT

281 sulation effectively controlled carbohydrate release to simulated gastric, intestinal and colonic flu

282 lationship probably reflects arsenobetaine's release to water from marine animals associated with the

283 Coupled C-S cleavage and CO2 release to yield [(bpy)3Pd3(micro3-S)2](NO3)2 (bpy, 2,2'

284 This Ca(2+) release triggered the fusion of lysosomes with the plasm

285 However, glucose utilization, glycerol release, triglyceride and glycogen contents, free fatty

286 It is released under an Open source MIT license.

287 that magnetic reconnection causes the energy release via 'magnetic breakout'-a positive-feedback mech

288 Apical ATP release was attenuated in Calhm1 knockout cultures follo

289 After processing, the release was lower and the released compounds were not de

290 NET and ROS release was monitored after the addition of NADP (NADPH)

291 ng that feedback inhibition of acetylcholine release was not involved.

292 receptors known to inhibit presynaptic GABA release was significantly reduced in the RVM of CFA-trea

293 ptures unzip faults in the Earth's crust and release waves that cause destructive shaking.

294 rier-Transform Infrared spectroscopy (FTIR); releases were quantified by Inductively Coupled Plasma M

295 hemical reaction prompting the active-drug's release, which effectively controls mGlu5 receptor activ

296 s has been shown to reduce cortical dopamine release, which is critically involved in the reinforcing

297 leading to caspase-1 activation and cytokine release, which mediate protective innate immune response

298 he emergency department, and the patient was released with a diagnosis of a fifth digit sprain.

299 1alpha gradient, leading to ectopic platelet release within the bone marrow.

300 ridaforolimus-eluting stents (RESs) and slow-release zotarolimus-eluting stents among 1919 patients u