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1 l drug ecstasy, is a potent serotonin (5-HT) releaser.
2 of the DAT to other uptake blockers, but not releasers.
5 as a rich pharmacology including inhibitors, releasers (amphetamines, which promote the exchange mode
6 ur 4-year long study and cycling between the releaser and nonreleaser phenotypes occurred at least on
11 brid' activity as a SERT substrate (ie, 5-HT releaser) and DAT blocker, whereas 4-MePPP is a blocker
12 trosoglutathione (GSNO, widely used as an NO releaser) and sodium nitroprusside (SNP, which is a NO+
13 ethyl 4-MA was an efficacious substrate-type releaser at DAT that evoked an inward depolarizing curre
15 Methylone, MDC, and HHMC were substrate-type releasers at monoamine transporters as determined in vit
18 tions of a newly identified water-soluble CO releaser (CORM-A1) that, unlike the first prototypic mol
19 Apis mellifera, possess unrelated primer and releaser functions for the workers and act as a sex attr
20 mine, an indirect presynaptic norepinephrine releaser, into dorsal hand veins of 49 normotensive men
22 creased stereotypic response to the dopamine releaser, methamphetamine, and an insensitivity to the a
24 pase A2alpha (cPLA2alpha), the rate-limiting releaser of arachidonic acid used for pro-inflammatory e
25 (HYCOs) that combine an Nrf2 inducer with a releaser of carbon monoxide (CO), an anti-inflammatory p
30 igated the effect of FK409, a spontaneous NO releaser, on the development of allograft vasculopathy,
31 inhibitor and as a prodrug for the monoamine-releaser phenmetrazine (PM), and it may serve as a clini
33 nteraction with the DAT as a pure blocker or releaser rather than their structural similarity to coca
34 given these compounds function as primer and releaser signals that regulate the social organization o
35 dilation in the piglet, responses to the NO releasers SNP and SNAP are unchanged by the Kca channel
36 The potencies of amphetamine-like dopamine releasers such as 3,4-methylenedioxymethamphetamine, met
38 methamphetamine (MDMA) is a potent monoamine releaser that produces an acute euphoria in most individ
39 ty of different dopamine uptake blockers and releasers to inhibit dopamine uptake, measured using fas
40 ed c-fos induction by the specific serotonin releaser-uptake inhibitor dexfenfluramine and alteration
43 intrinsic biochemical behavior of a slow CO releaser, which may be advantageous in the treatment of
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