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1 mone 44 (DH44), an ortholog of corticotropin-releasing factor.
2 alize with the stress hormone, corticotropin releasing factor.
3 y active clinical candidate of corticotropin-releasing factor 1 (CRF 1) antagonist 1 showed a signifi
4 ally active antagonists of the corticotropin-releasing factor 1 (CRF 1) receptor.
5 de), D2R agonist (quinpirole), corticotropin-releasing factor 1 (CRF1) antagonist (antalarmin), and a
6 ne domains of the glucagon and corticotropin releasing factor 1 (CRF1) receptors to develop improved
7                       Ras-guanine nucleotide-releasing factor 1 (Ras-GRF1) and Ras-GRF2 are highly si
8           Ablation of Ras-guanine nucleotide-releasing factor 1 (Ras-GRF1), a neuronal activator of R
9 n of Ras protein-specific guanine nucleotide releasing factor 1 (RasGrf1), a Ras activator (5-fold, P
10 with Ras protein-specific guanine nucleotide-releasing factor 1 (RASGRF1), leading to impaired activa
11 armacologic inhibition (with a corticotropin-releasing factor 1 receptor antagonist) of pain-related
12  earlier lead pyrazinone-based corticotropin-releasing factor-1 (CRF(1)) receptor antagonist, reveale
13 al-like state characterized by corticotropin-releasing factor-1 (CRF(1)) receptor antagonist-reversib
14         Evidence suggests that corticotropin-releasing factor-1 (CRF(1)) receptor antagonists may off
15 ed as potent and orally active corticotropin-releasing factor-1 (CRF(1)) receptor antagonists.
16 s to palatable food results in corticotropin-releasing factor-1 (CRF1) receptor antagonist-reversible
17 26907 in the ras-specific guanine-nucleotide releasing factor 2 (RASGRF2) gene, encoding a protein th
18 e processes; Ras-specific guanine nucleotide releasing factor 2 (RasGRF2).
19 ene (Ras protein-specific guanine nucleotide-releasing factor 2) with all clinical events except stro
20                        After considering the releasing factor (66.6%) from the swab, the final accura
21                                Corticotropin-releasing factor, a stress-related neuropeptide implicat
22 id peptide function, increased corticotropin-releasing factor activity).
23 ess neurotransmitters, such as corticotropin-releasing factor and dynorphin, in the neurocircuitry of
24 ing by increased expression of corticotropin-releasing factor and its feedback regulation of TLR4 exp
25  and Hcrt systems in which the corticotropin-releasing factor and N/OFQ systems coordinately modulate
26 sensitizes the LC-NE system to corticotropin releasing factor and stress.
27 (especially neuropeptide Y and corticotropin-releasing factor) are modulated by alcohol drinking duri
28                                Corticotropin releasing factor binding protein (CRF-BP) was originally
29                                Corticotropin releasing factor-binding protein (CRF-BP) binds CRF and
30 ed Crk SH3 domain-binding guanine-nucleotide releasing factor (C3G) binding to CrkII, whereas inhibit
31                     Infusion of hypothalamic-releasing factors can reactivate the thyroid axis in pat
32 othalamus and primarily in non-corticotropin releasing factor-containing neurons of the bed nucleus o
33 os activation were observed in corticotropin releasing factor-containing neurons of the paraventricul
34 at baseline and in response to corticotropin-releasing factor (CRF) (0.5 microg kg(1)) plus arginine
35 BDNF signaling is mediated by corticotrophin-releasing factor (CRF) acting in the NAc.
36 s, including a coordination of corticotropin-releasing factor (CRF) actions at both of its receptors,
37                 We report that corticotropin-releasing factor (CRF) acts in the ventral tegmental are
38 also posit a central role for corticotrophin-releasing factor (CRF) and an interaction between CRF an
39 tine SA on the coexpression of corticotropin-releasing factor (CRF) and arginine vasopressin (AVP) mR
40 gh it has long been known that corticotropin-releasing factor (CRF) and CRF receptors are prominent i
41 esent review is on the role of corticotropin-releasing factor (CRF) and CRF-related peptides in the d
42 ng-term alterations in central corticotropin-releasing factor (CRF) and glucocorticoid receptor (GR)
43 ion between the stress hormone corticotropin releasing factor (CRF) and glutamate release onto dopami
44 d to partially colocalize with corticotropin-releasing factor (CRF) and growth hormone-releasing horm
45 xtrahypothalamic expression of corticotropin-releasing factor (CRF) and its G-protein-coupled CRF1 an
46        The stress neuropeptide corticotropin releasing factor (CRF) and its receptors (CRF-R1, CRF-R2
47                                Corticotropin-releasing factor (CRF) and nociceptin/orphanin FQ (nocic
48 europharmacological actions of corticotropin-releasing factor (CRF) and norepinephrine in extrahypoth
49   Release of the neuropeptides corticotropin-releasing factor (CRF) and orexin-A in the ventral tegme
50                                Corticotropin-releasing factor (CRF) and related peptides play a role
51      The circadian activity of corticotropin releasing factor (CRF) and the hypothalamic-pituitary-ad
52                            The corticotropin releasing factor (CRF) antagonist, alpha-helical CRF(9-4
53 e report the identification of corticotropin-releasing factor (CRF) as a critical component of the ca
54                      Exogenous corticotropin-releasing factor (CRF) but not corticosterone mimicked t
55 ne arm of the stress response, corticotropin-releasing factor (CRF) can act in the brain to modulate
56 ase of neuromodulators such as corticotropin-releasing factor (CRF) can drive drug-dependent behavior
57                 To investigate corticotropin-releasing factor (CRF) contributions to fear memory in L
58                                Corticotropin releasing factor (CRF) coordinates the brains responses
59 e stress-related neuropeptide, corticotropin-releasing factor (CRF) directs this by a bimodal regulat
60  the central extrahypothalamic corticotropin-releasing factor (CRF) expression is associated with str
61 of an acute colitis on central corticotropin-releasing factor (CRF) expression.
62                            The corticotropin releasing factor (CRF) family of ligands and their recep
63                           The corticotrophin-releasing factor (CRF) family of neuropeptides and their
64 enced by the highly conserved corticotrophin-releasing factor (CRF) family of peptides and receptors
65                            The corticotropin-releasing factor (CRF) family of peptides includes CRF a
66 tin 3 (Ucn 3), a member of the corticotropin-releasing factor (CRF) family of peptides, is strongly e
67 tin 2 (Ucn2), a peptide of the corticotropin-releasing factor (CRF) family, binds with high affinity
68 n of the stress neuromediator, corticotropin-releasing factor (CRF) has been implicated in these diso
69 levant PI cells identified the corticotropin-releasing factor (CRF) homolog, DH44, as a circadian out
70 e self-administration, reduced corticotropin-releasing factor (CRF) immunodensity in the paraventricu
71 ence of somatostatin (SS) and corticotrophin-releasing factor (CRF) in forebrain neurons that project
72 lation of cytosolic calcium by corticotropin-releasing factor (CRF) in midbrain dopamine neurons.
73 k indicates a crucial role for corticotropin-releasing factor (CRF) in neurobiological responses asso
74  support a modulatory role for corticotropin-releasing factor (CRF) in regulating the dorsal raphe nu
75 ole of the stress neurohormone corticotropin-releasing factor (CRF) in stress-induced binge eating in
76  work hypothesizing a role for corticotropin-releasing factor (CRF) in the IC during craving and rela
77 tionship between dynorphin and corticotropin-releasing factor (CRF) in the induction of dysphoria, th
78 ith the stress-related peptide corticotropin-releasing factor (CRF) in the LC.
79 ibution of the stress hormone, corticotropin-releasing factor (CRF) in the mouse brain.
80 tly described the existence of corticotropin-releasing factor (CRF) in the mouse cochlea.
81 thasone (DEX) inhibition, and corticotrophin-releasing factor (CRF) induced activation.
82 d that the stress neurohormone corticotropin-releasing factor (CRF) inhibits 5-HT neurons in the dors
83 ggests that catecholamines and corticotropin-releasing factor (CRF) interact in a serial manner to ac
84 ces the release of the peptide corticotropin-releasing factor (CRF) into the ventral tegmental area (
85                                Corticotropin-releasing factor (CRF) is a key mediator of the behavior
86                                Corticotropin-releasing factor (CRF) is also known as a stress-related
87                                Corticotropin-releasing factor (CRF) is an important link between stre
88               The neuropeptide corticotropin-releasing factor (CRF) is believed to play a role in a n
89                                Corticotropin-releasing factor (CRF) is critical for the endocrine, au
90 emonstrate that, in the vBNST, corticotropin releasing factor (CRF) is expressed in neurons that inne
91                                Corticotropin-releasing factor (CRF) is expressed in the Ce, has a rol
92                               Corticotrophin-releasing factor (CRF) is expressed in the central nucle
93                                Corticotropin releasing factor (CRF) is the primary mediator of stress
94                                Corticotropin-releasing factor (CRF) mediates anxiogenic responses by
95 t experiments examined whether corticotropin-releasing factor (CRF) modulates memory consolidation vi
96                               Corticotrophin-releasing factor (CRF) modulates the influence of stress
97 5, we measured AT(1) receptors corticotropin-releasing factor (CRF) mRNA and immunoreactive CRF in th
98                                Corticotropin-releasing factor (CRF) mRNA expression in the hypothalam
99 eliable increases in Fos-ir or corticotropin-releasing factor (CRF) mRNA in the PVH.
100 duction and relative levels of corticotropin-releasing factor (CRF) mRNA.
101 on, potentially by activating corticotrophin releasing factor (CRF) neurons in the anterolateral cell
102 d activates a subpopulation of corticotropin-releasing factor (CRF) neurons in the bed nucleus of the
103 pecifically recruited GABA and corticotropin-releasing factor (CRF) neurons in the mPFC and produced
104 hip between corticosterone and corticotropin-releasing factor (CRF) on both beta-amyloid (Abeta) and
105 of an intravenous injection of corticotropin-releasing factor (CRF) on fructose malabsorption and the
106  was to examine the ability of corticotropin releasing factor (CRF) or antibody to insulin growth fac
107 ry but did not increase either corticotropin-releasing factor (CRF) or corticosterone.
108 , arginine-vasopressin (AVP), corticotrophin-releasing factor (CRF) or tyrosine hydroxylase (TH).
109                   Injection of corticotropin-releasing factor (CRF) or urocortin III, key mediators o
110                                Corticotropin-releasing factor (CRF) orchestrates the stress response,
111                            The corticotropin-releasing factor (CRF) peptide hormone family members co
112                                Corticotropin releasing factor (CRF) plays a major role in CNS respons
113                        Because corticotropin-releasing factor (CRF) plays a role in stress responses,
114 derable evidence suggests that corticotropin-releasing factor (CRF) plays an important role in regula
115                            The corticotropin-releasing factor (CRF) receptor 1 (CRFR1) is a target fo
116 eta elevation are dependent on corticotropin-releasing factor (CRF) receptor 1 signaling and an intac
117                                Corticotropin-releasing factor (CRF) receptor activation within the ba
118                            The corticotropin-releasing factor (CRF) receptor CRFR2 is expressed widel
119 awal was mediated by increased corticotropin releasing factor (CRF) receptor-1 expression and signall
120                                Corticotropin-releasing factor (CRF) receptors are found in stress-rel
121                        Because corticotropin-releasing factor (CRF) receptors are implicated, we comp
122   Natural peptide agonists of corticotrophin-releasing factor (CRF) receptors bind to the receptor by
123 lation increases the levels of corticotropin-releasing factor (CRF) receptors in the serotonergic dor
124                                Corticotropin-releasing factor (CRF) receptors-which bind the hormone
125                                Corticotropin releasing factor (CRF) regulates physiological and behav
126 e stress-related neuropeptide, corticotropin-releasing factor (CRF) regulates the dorsal raphe nucleu
127 e through alterations in brain corticotropin-releasing factor (CRF) regulation of neurocircuitry invo
128                                Corticotropin-releasing factor (CRF) signaling at the CRF1 receptor (C
129                 Alterations in corticotropin-releasing factor (CRF) signaling pathways have been impl
130     Over the past few decades, corticotropin-releasing factor (CRF) signaling pathways have been show
131                  Activation of corticotropin-releasing factor (CRF) signaling pathways mediates both
132 nforcement is a recruitment of corticotropin-releasing factor (CRF) signaling within the amygdala.
133 dies have implicated the brain corticotropin-releasing factor (CRF) stress systems in mediating the n
134 VN) have been shown to inhibit corticotropin releasing factor (CRF) synthesis via GABA(A) receptors.
135 trahypothalamic stress peptide corticotropin-releasing factor (CRF) system and activation of CRF(1) r
136 H secretion by activating the corticotrophin-releasing factor (CRF) system and sympathoadrenal pathwa
137           Dysregulation of the corticotropin-releasing factor (CRF) system has been implicated in man
138                            The corticotropin releasing factor (CRF) system in the central amygdala (C
139                            The corticotropin releasing factor (CRF) system in the central amygdala (C
140                            The corticotropin-releasing factor (CRF) system is the primary central med
141 ncluding the extrahypothalamic corticotropin-releasing factor (CRF) system, following long-term drug
142                            The corticotropin-releasing factor (CRF) system, which is involved in stre
143 gdala (Neo-A) lesions on brain corticotropin-releasing factor (CRF) systems and hypothalamic-pituitar
144                            The corticotropin-releasing factor (CRF) type 1 receptor (CRF1R) for the 4
145           Systemic blockade of corticotropin-releasing factor (CRF) type 1 receptors (CRFR1s) reduces
146 gdala (CeA), ethanol acts via corticotrophin-releasing factor (CRF) type 1 receptors to enhance GABA
147                                Corticotropin releasing factor (CRF) within the ventral tegmental area
148                                Corticotropin-releasing factor (CRF), a 41 amino acid peptide, mediate
149           We investigated how corticotrophin releasing factor (CRF), a critical stress response media
150            Here we report that corticotropin-releasing factor (CRF), a neuropeptide released in respo
151 e number of neurons expressing corticotropin releasing factor (CRF), a neuropeptide that has a promin
152 imolecular interaction between corticotropin-releasing factor (CRF), a neuropeptide, and its type 1 r
153 ited different sensitivity to corticotrophin-releasing factor (CRF), a stress hormone that has been i
154 this, the CEA highly expresses corticotropin-releasing factor (CRF), an important modulator of stress
155 excitatory amino acids (EAAs), corticotropin-releasing factor (CRF), and endogenous opioids acting at
156 opeptide related to vertebrate corticotropin-releasing factor (CRF), and its receptor, Dh44R1.
157  regional, CCK-8, substance P, corticotropin releasing factor (CRF), and neuropeptide Y levels in adu
158 r modulation by dopamine (DA), corticotropin-releasing factor (CRF), and their combination (DA plus C
159     The neuroactive substances corticotropin-releasing factor (CRF), arginine-vasopressin (AVP), hist
160  is thought to communicate via corticotropin-releasing factor (CRF), but studies have yet to examine
161              The neuropeptide, corticotropin-releasing factor (CRF), coordinates the physiological an
162                                Corticotropin-releasing factor (CRF), encoded by the CRH gene, is a ke
163                               Corticotrophin releasing factor (CRF), enkephalin (ENK), and dynorphin
164 ed receptor B1 family includes corticotropin-releasing factor (CRF), growth hormone-releasing hormone
165  axis-activating neuropeptide, corticotropin-releasing factor (CRF), may be the keystone in drug-indu
166 nd measured mRNA expression of corticotropin releasing factor (CRF), neuropeptide Y (NPY) and proopio
167 es indicate the involvement of corticotropin-releasing factor (CRF), noradrenaline, dopamine, glutama
168                                Corticotropin-releasing factor (CRF), originally characterized as the
169                                Corticotropin-releasing factor (CRF), the major stress peptide in the
170 e focus is on the receptor for corticotropin-releasing factor (CRF), the orchestrator of the stress r
171                                Corticotropin-releasing factor (CRF), the stress-related neuropeptide,
172 oradrenergic substrates [via a corticotropin-releasing factor (CRF)-dependent mechanism] that regulat
173 nd inhibitory transmission in corticotrophin-releasing factor (CRF)-expressing dorsal-medial (mpd) ne
174 en shown previously to cause a corticotropin-releasing factor (CRF)-mediated increase in tonic locus
175                                Corticotropin-releasing factor (CRF)-mediated mechanisms in the bed nu
176 tin 1 (Ucn 1) is an endogenous corticotropin releasing factor (CRF)-related peptide.
177                                Corticotropin-releasing factor (CRF)-related peptides can modulate str
178 e prototypical stress hormone, corticotropin releasing factor (CRF).
179 e stress-related neuropeptide, corticotropin-releasing factor (CRF).
180 the anxiogenic neuromodulator, corticotropin releasing factor (CRF).
181 he stress-related neurohormone corticotropin-releasing factor (CRF).
182 erminate of these responses is corticotropin-releasing factor (CRF).
183 parabrachial nucleus coexpress corticotropin-releasing factor (CRF).
184  that produce the neuropeptide corticotropin-releasing factor (CRF).
185 uding neuropeptide Y (NPY) and corticotropin-releasing factor (CRF).
186  the stress-sensitive peptide, corticotropin releasing factor (CRF).
187 ocorticoid receptors (GRs) and corticotropin-releasing factor (CRF).
188 opaminergic neurons expressing corticotropin-releasing factor (CRF).
189                  The mammalian corticotropin releasing factor (CRF)/urocortin (Ucn) peptide hormones
190 nal aggression) is impaired by corticotropin-releasing factor-(CRF) related peptides, but where these
191  density, stress neuropeptide (corticotropin releasing factor--CRF) levels, and plasma corticosterone
192 tested the hypothesis that the corticotropin-releasing factor (CRF1) antagonist GSK561679 differentia
193 ticoliberin family include the corticotropin releasing factors (CRFs), sauvagine, the urotensins, and
194 transcriptionally activated in corticotropin releasing factor-expressing cells.
195 region-specific alterations of corticotropin-releasing factor expression and promoter methylation, ch
196  neuropeptides vasopressin and corticotropin-releasing factor facilitate, while serotonin inhibits, a
197 rs, which bind peptides of the corticotropin releasing factor family and are key mediators in the str
198 NGF and the hormone urotensin (corticotropin-releasing factor family ligand).
199 ently identified member of the corticotropin-releasing factor family, is expressed in discrete neuroe
200                RasGRP1, a guanine nucleotide-releasing factor for Ras, plays an important role in pos
201                  Human recombinant histamine-releasing factor has also recently been shown to induce
202 n of cholecystokinin and secretin release by releasing factors have also been elucidated.
203  al. provide a causal link between histamine-releasing factor (HRF) interactions with IgE and food al
204 lacks a secreted factor related to histamine-releasing factor (HRF).
205                                    Histamine-releasing factor (HRF; also known as translationally con
206 ther suggest that an activated corticotropin-releasing factor/hypothalamic-pituitary-adrenal axis sys
207 thropin-releasing hormone, and corticotropin-releasing factor immunoreactive cells in the paraventric
208 s the physiological actions of corticotropin-releasing factor in the anterior pituitary gland and the
209 l axis, including signaling by corticotropin-releasing factor, in the pathophysiology of PTSD.
210  A1 and aromatase enhanced local bone marrow-releasing factors, including androgen receptor, estrogen
211                                Corticotropin-releasing factor infusions into LA impair the consolidat
212                                Corticotropin-releasing factor infusions into the central nucleus of t
213                         While corticotrophin releasing factor is the prototypic member of this class,
214 cleave both factor IX peptide bonds prior to releasing factor IX abeta.
215 rebrospinal fluid cortisol and corticotropin-releasing factor levels.
216 s in restraint-induced Fos and corticotropin-releasing factor mRNA expression in the neurosecretory r
217 cting to vasopressinergic and corticotrophin releasing factor neurons in the paraventricular nucleus,
218 acts with the neuromodulators, corticotropin-releasing factor, norepinephrine, dopamine, and serotoni
219  role of brain stress systems (corticotropin-releasing factor, norepinephrine, orexin [hypocretin], v
220 l impact of treatment with such hypothalamic releasing factors on recovery from critical illness as w
221 lockade of the stress hormone corticotrophin-releasing factor or of peripheral GC action, as well as
222 ior hypothalamus that may gate corticotropin-releasing factor output from the amygdala to the anterio
223 n of the brain stress system's corticotropin-releasing factor outside of the hypothalamus in the exte
224       Cohesin dynamics are controlled by the releasing factors Pds5 and Wapl and the cohesin stabiliz
225  this region of receptors for corticotrophin-releasing factor, pituitary adenylate cyclase-activating
226 appetite regulation, including corticotropin releasing factor, pro-opiomelanocortin B, and glucose tr
227 elopmental stage Ras-GRF (guanine nucleotide-releasing factor) proteins are not involved.
228 logical consequence of central corticotropin-releasing factor receptor (CRF-R) activation.
229 ed differential involvement of corticotropin-releasing factor receptor (CRFR) 1 and 2 in acute stress
230 f signaling through the type 1 corticotropin-releasing factor receptor (CRFR1).
231 dministration of the selective corticotropin-releasing factor receptor 1 (CRF(1)) antagonist R121919
232 n, we examined the role of the corticotropin-releasing factor receptor 1 (CRF(1)) in this behavior as
233 haracterize the effects of the corticotropin-releasing factor receptor 1 (CRF-R1) antagonist, GW87600
234  (NE) receptors (alpha1) via a corticotropin-releasing factor receptor 1 (CRF-R1)-dependent mechanism
235 g the efficacy of GSK561679, a corticotropin-releasing factor receptor 1 (CRF1 receptor) antagonist i
236            We describe a novel corticotropin-releasing factor receptor 1 (CRF1) antagonist with advan
237                    Blockade of corticotropin-releasing factor receptor 1 (CRF1) suppresses stress-ind
238  member of a class B GPCR, the corticotropin releasing factor receptor 1 (CRF1R).
239                                Corticotropin-releasing factor receptor 1 (CRFR1) mediates the physiol
240 what has been observed for the corticotropin-releasing factor receptor 1 (CRFR1), SAP97 expression is
241 r-activated receptor gamma and corticotropin-releasing factor receptor 1 were notable exceptions.
242 ivated by either neurokinin I, corticotropin-releasing factor receptor 1, or dopamine D(1) receptors.
243              Mice deficient in corticotropin-releasing factor receptor 2 (CRF2) (C57BL/6J:129Sv backg
244 ed significant upregulation of corticotropin releasing factor receptor 2 (CrfR2) in the amygdala of m
245 constitutive activation of the corticotropin-releasing factor receptor family homologue SEB-3.
246 ent study examines the role of corticotropin releasing factor receptor subtypes 1 and 2 (CRFR1, CRFR2
247  CRF(2(a))R and the homologous corticotropin-releasing factor receptor type 1 (CRF(1)R) possessing a
248 vestigated whether blockade of corticotropin-releasing factor receptor type 1 (CRF-R1) could prevent
249 ctions in anandamide driven by corticotropin-releasing factor receptor type 1 (CRF1) potentiation of
250 re the interaction between the corticotropin releasing factor receptor type 1 (CRF1R) and its native
251 ed interactions of the class B corticotropin-releasing factor receptor type 1 (CRF1R) with two peptid
252                                Corticotropin-releasing factor receptor type 1 (CRFR1) is involved in
253 compartments, we show that the corticotropin-releasing factor receptor type 1 has a specific monomer/
254 nsmembrane domain of the human corticotropin-releasing factor receptor type 1 in complex with the sma
255                            The corticotropin-releasing factor receptor type 1 is a class B receptor w
256 librium is established for the corticotropin-releasing factor receptor type 1.
257                                Corticotropin-releasing factor receptor type 2 (CRFR2) has been implic
258 central stress response, while corticotropin-releasing factor receptor type 2 (CRFR2) has been sugges
259 bserved abnormal expression of corticotropin-releasing factor receptor type 2 (CRFR2) to be associate
260                            The corticotropin-releasing factor receptor type 2a (CRF(2(a))R) possesses
261                                Corticotropin-releasing factor receptor-1 (CRF(1)) mediates the stress
262 pyrazolo[1,5-a]-1,3,5-triazine corticotropin releasing factor receptor-1 (CRF(1)) receptor antagonist
263 pyrazolo[1,5-a]-1,3,5-triazine corticotropin releasing factor receptor-1 (CRF(1)) receptor antagonist
264 is unaffected by alpha1-AR and corticotropin-releasing factor receptor-1 (CRFR(1)) antagonists, but i
265 tention were attenuated by the corticotropin-releasing factor receptor-1 antagonist antalarmin but no
266 application of urocortin I (a corticotrophin-releasing factor receptor-2 agonist) to the ventromedial
267 of the analogous domain of the corticotropin-releasing factor receptor.
268                                Corticotropin-Releasing Factor Receptors (CRFRs) are class B1 G-protei
269                                Corticotropin-releasing factor receptors (CRFRs), class B G protein-co
270                             Codon selection, releasing factor recognition sequence and specific intro
271      Urocortin 3 (Ucn 3) is a corticotrophin-releasing factor related neuropeptide highly expressed i
272 he peptide antagonist, cyclic corticotrophin-releasing factor residues 30-41 (cCRF(30-41)), was calcu
273 ion-like protein (RepA-WH1) into the E. coli releasing factor RF1 promotes its aggregation and enable
274         Alterations in central corticotropin-releasing factor signaling pathways have been implicated
275 n-gamma (IFN-gamma) and induce cytolysis via releasing factors such as perforin, which permeabilizes
276 s), (4) the (gastrointestinal) corticotropin-releasing factor system, and (5) the intestinal response
277                   In addition, corticotropin-releasing factor systems were shown to facilitate memory
278 s probed using a dexamethasone/corticotropin-releasing factor test.
279   These results reveal PAR polymer as an AIF-releasing factor that plays important roles in PARP-1-de
280 s circadian rhythms of locomotor behavior by releasing factors that act on receptor sites near the th
281 uscle laceration sites on a delivery vehicle releasing factors that induce cell activation and migrat
282  role in maintaining vascular homeostasis by releasing factors that regulate local blood flow, system
283 counteracting the functions of corticotropin-releasing factor, the primary stress-mediating neuropept
284 lin, nesfatin-1, somatostatin, corticotropin-releasing factor, thyrotropin-releasing hormone and calc
285 buting ions, removing neurotransmitters, and releasing factors to influence blood flow and neuronal a
286 ation between histamine release to histamine releasing factor/translationally controlled tumor protei
287 hesis was investigated for the corticotropin-releasing factor type 1 (CRF(1)) receptor using a novel
288                                Corticotropin releasing factor type 1 (CRF1) is a key component in the
289                  Activation of corticotropin-releasing factor type 1 (CRF1) receptors in the CeA play
290 ve effect is mediated via the corticotrophin-releasing factor type 1 receptor (CRF1R, also known as C
291 peated social stress decreased corticotropin-releasing factor type 1 receptor and increased mu-opioid
292                  We found that corticotropin-releasing factor type 1 receptor within the paraventricu
293 LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s) mediate alcoho
294 LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s) mediate alcoho
295 o central anorectic effects of corticotropin-releasing factor type 2 (CRF(2)) receptor stimulation.
296                            The corticotropin-releasing factor type 2 receptor (CRFR2) is suggested to
297 hat the mechanism involved the corticotropin-releasing factor type 2 receptor, cAMP elevation, and ac
298 , which required activation of corticotropin-releasing factor type-1 (CRF-R1) receptors.
299 ombined administration of those hypothalamic releasing factors, which have been identified as suppres
300 plantation of blank scaffolds, and scaffolds releasing factors without cells.

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