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1 achineries to liberate this key hypothalamic releasing hormone.
2 ropin-releasing hormone (CRH) or thyrotropin-releasing hormone.
3 eus was identified as containing thyrotropin-releasing hormone.
4 s to leptin and a suppression of thyrotropin-releasing hormone.
5 normalities in HIV, including growth hormone-releasing hormone.
6 ulating hormone and hypothalamic thyrotropin-releasing hormone.
7 n episodic, excitatory drive to gonadotropin-releasing hormone 1 (GnRH) neurons, the synaptic mechani
10 udes suppression of hypothalamic thyrotropin-releasing hormone, accounting for persistently reduced s
11 sex hormone fluctuation using a gonadotropin-releasing hormone agonist (GnRHa) and evaluated if emerg
12 ly that after 1-year follow up, gonadotropin-releasing hormone agonist (GnRHa) did not prevent chemot
16 rian suppression induced by the gonadotropin-releasing hormone agonist leuprolide acetate, leuprolide
17 reased after treatment with the gonadotropin-releasing hormone agonist leuprolide, which inhibits the
18 rchiectomy or receipt of luteinising hormone-releasing hormone agonist or antagonist therapy started
21 2 healthy, young males received gonadotropin-releasing hormone agonist treatment 1 month prior to 3 o
22 o cixutumumab added to a luteinizing hormone-releasing hormone agonist with bicalutamide versus AD al
23 every-3-months depot of luteinizing hormone-releasing hormone agonist) to primary radiotherapy (RT)
24 ndomly assigned 2:1 to a luteinizing hormone-releasing hormone agonist, bicalutamide and bevacizumab
25 rian suppression induced by the gonadotropin-releasing hormone agonist, leuprolide acetate; leuprolid
26 premenopausal women receiving a gonadotropin-releasing hormone agonist, with estrogen receptor- and/o
27 authors examined the effects of gonadotropin-releasing hormone agonist-induced ovarian suppression on
28 as achieved with the use of the gonadotropin-releasing-hormone agonist triptorelin, oophorectomy, or
30 4.04 [95% CI, 1.88-8.69]), and gonadotropin-releasing hormone agonists (OR, 1.93 [95% CI, 1.20-3.10]
31 rivation therapy in the form of gonadotropin-releasing hormone agonists and newer antagonists, examin
33 androgen blockade consisting of gonadotropin-releasing hormone agonists with oral antiandrogens (OR,
34 such as surgical oophorectomy, gonadotropin-releasing hormone agonists, chemotherapy-induced ovarian
35 of 6 mutually exclusive groups: gonadotropin-releasing hormone agonists, oral antiandrogens, combined
39 ter or higher received a luteinizing hormone-releasing hormone analogue and an antiandrogen agent for
42 intrauterine system (LNG-IUD), gonadotropin-releasing hormone analogues (GnRHa; nafarelin, leuprolid
43 er the administration of luteinizing hormone-releasing hormone analogues (LHRHa) during chemotherapy
45 (castration via orchiectomy or gonadotropin-releasing hormone analogues) suppresses circulating test
47 corticotropin-releasing factor, thyrotropin-releasing hormone and calcitonin gene-related peptide) i
48 ticotrophin-releasing hormone or thyrotropin-releasing hormone and do not express arginine vasopressi
49 ediated by reduced secretion of gonadotropin-releasing hormone and our results support the involvemen
50 ed hypothalamic expression of corticotrophin-releasing hormone and oxytocin, neuropeptides known to c
51 mber of DMV neurones excited by thyrotrophin-releasing hormone and the gastric motility response to D
52 the endogenous natural ligand, gonadotropin releasing hormone, and an agonist that is specific for t
53 tocin, vasopressin, enkephalin, thyrothropin-releasing hormone, and corticotropin-releasing factor im
54 .e., C-reactive protein (CRP), corticotropin-releasing hormone, and cytokines) were compared among te
55 prohormone processing of proinsulin, pro-GH-releasing hormone, and proghrelin in association with re
56 mitters such as glutamate, GABA, thyrotropin releasing hormone, and substance P encoded by the Tachyk
57 tide markers (cholecystokinin, corticotropin-releasing hormone, and tachykinin 1) label sleep-promoti
59 ive hormone production using a gonadotrophin-releasing hormone antagonist (GnRHant) for 10 days, with
60 g hormone and administration of gonadotropin-releasing hormone antagonist to prevent premature ovulat
64 rs28365143 variant within the corticotropin-releasing hormone binding protein (CRHBP) gene predicted
66 creased somewhat by exposure to gonadotropin-releasing hormone but are not necessarily linked to Cga
68 hat encode the stress hormones corticotropin-releasing hormone (CRH) and adrenocorticotropic hormone.
69 ysiological concentrations of corticotrophin-releasing hormone (CRH) and arginine vasopressin (AVP) c
70 o hypothalamic secretagogues, corticotrophin-releasing hormone (CRH) and arginine vasopressin (AVP) t
71 o hypothalamic secretagogues, corticotrophin-releasing hormone (CRH) and arginine vasopressin (AVP) t
73 and stress molecules including corticotropin-releasing hormone (CRH) and neurotensin (NT), secreted i
74 ormone (ACTH) by hypothalamic corticotrophin-releasing hormone (CRH) and then stimulation of the adre
75 show that a constant level of corticotrophin-releasing hormone (CRH) can activate a dynamic pituitary
76 sh with transgenically labeled corticotropin-releasing hormone (CRH) cells, which represent a major r
77 l-genetic approaches to reduce corticotropin-releasing hormone (Crh) expression in the central nucleu
78 the production and release of corticotropin-releasing hormone (CRH) from the paraventricular nucleus
79 he suppression of hypothalamic corticotropin-releasing hormone (CRH) gene expression and potently enh
81 cluster of neurons expressing corticotropin-releasing hormone (Crh) in the pontine micturition cente
86 atory and inhibitory inputs to corticotropin-releasing hormone (CRH) neurons in the hypothalamic para
87 xiety-related behaviours, and corticotrophin releasing hormone (crh) neurons, key regulators of the s
88 ansmit signals to hypothalamic corticotropin-releasing hormone (CRH) neurons, which control stress ho
89 atory synaptic strength in PVN corticotropin-releasing hormone (CRH) neurons, with GLP-1R activation
90 actions of corticosterone and corticotropin-releasing hormone (CRH) on synaptic physiology and dendr
91 xytocin, arginine vasopressin, corticotropin-releasing hormone (CRH) or thyrotropin-releasing hormone
92 corticosterone and hippocampal corticotropin-releasing hormone (CRH) permeate memory-forming hippocam
94 adigm posits that hypothalamic corticotropin-releasing hormone (CRH) regulates neuroendocrine functio
98 n, increased activation of the corticotropin-releasing hormone (CRH) system within the extended amygd
99 (WM) deficits; changes to the corticotropin-releasing hormone (CRH) system; and structural, function
101 nd release of the neuropeptide corticotropin-releasing hormone (CRH) within the hippocampus during st
102 ive preclinical data implicate corticotropin-releasing hormone (CRH), acting through its CRH1 recepto
104 ected and processed to measure corticotropin-releasing hormone (CRH), urocortin (Ucn), beta-endorphin
105 stimulating the expression of corticotropin-releasing hormone (CRH), urocortin, proopiomelanocortin
106 for vasotocin, mesotocin (MT), corticotropin-releasing hormone (CRH), vasoactive intestinal polypepti
107 4), a homolog of the mammalian corticotropin-releasing hormone (CRH), were specifically activated by
108 e stress-released neuropeptide corticotropin-releasing hormone (CRH), which influence the integrity o
109 regulator of the HPA axis is corticotrophin-releasing hormone (CRH), which is made in the parventric
110 have described the effects of corticotropin-releasing hormone (CRH), which is released from inflamed
111 , we have virally traced local corticotropin-releasing hormone (CRH)-expressing inhibitory interneuro
114 the cortisol-regulating genes, corticotropin releasing hormone (CRH; P=0.05) and glucocorticoid recep
116 n addition, we discovered that corticotropin-releasing hormone-expressing cells contain GABAergic and
117 tion resulted from diminished growth hormone-releasing hormone expression in the hypothalamus and, su
118 tance, resulting in increased corticotrophin-releasing hormone expression, chronic hypercortisolism,
120 chum salmon (Oncorhynchus keta), thyrotropin-releasing hormone gene expression increased immediately
123 th inadequate compensation by Growth hormone-releasing hormone (GHRH) and Growth hormone (GH), undera
124 fects of agonistic analogs of growth hormone-releasing hormone (GHRH) and their mechanism of action w
125 clinical studies suggest that growth hormone-releasing hormone (GHRH) antagonists effectively shrink
126 in, called SXN101742, contains a modified GH-releasing hormone (GHRH) domain and the endopeptidase do
127 growth-stimulatory peptides, growth hormone-releasing hormone (GHRH) for GHRH-neurons and somatostat
130 act of targeted disruption of growth hormone-releasing hormone (GHRH) in mice on longevity and the pu
131 Antagonists of hypothalamic growth hormone-releasing hormone (GHRH) inhibit growth of various malig
134 MC), neuropeptide Y (NPY) and growth hormone releasing hormone (GHRH) neurons, regulates feeding, ene
137 ed serum GH levels, a greater response to GH releasing hormone (GHRH) stimulation along with lower we
138 ion using a potent agonist of growth-hormone-releasing hormone (GHRH) to promote islet viability and
139 CSCs) express the receptor of growth hormone releasing hormone (GHRH), activation of which improves i
140 the hypothalamic polypeptide growth hormone-releasing hormone (GHRH), as well as for GHRH itself, in
141 in-releasing factor (CRF) and growth hormone-releasing hormone (GHRH), suggesting novel interactions
142 of the multiple activities of growth hormone-releasing hormone (GHRH), we hypothesized that pretreatm
143 nthetic nonapeptide analogue of gonadotropin-releasing hormone (GnRH or LHRH), is the active pharmace
146 ART treatment cycles receiving gonadotropin-releasing hormone (GnRH) agonists and recombinant follic
148 iandrogens (AA), 26,959 were on gonadotropin-releasing hormone (GnRH) agonists, and 3,747 underwent s
149 ized trial evaluated the use of gonadotropin-releasing hormone (GnRH) analog triptorelin to preserve
153 te meiosis, TGF-beta signaling, gonadotropin-releasing hormone (GnRH) and epidermal growth factor rec
156 diol (E2) regulating release of gonadotropin releasing hormone (GnRH) and luteinizing hormone (LH) ar
157 Elagolix, an oral, nonpeptide, gonadotropin-releasing hormone (GnRH) antagonist, produced partial to
160 with an inherited deficiency in gonadotropin-releasing hormone (GnRH) have impaired sexual reproducti
161 pressed basal transcription and gonadotropin-releasing hormone (GnRH) induction of both the murine an
162 minence (ME) where they release gonadotropin-releasing hormone (GnRH) into a specialized capillary ne
171 We have previously shown that gonadotropin-releasing hormone (GnRH) ligand-independently activates
173 rons have been shown to inhibit gonadotropin-releasing hormone (GnRH) neuronal activity and hence rep
174 lases (HDACs) in the control of gonadotropin releasing hormone (GnRH) neuronal development is unknown
175 nfluence on the activity of the gonadotropin-releasing hormone (GnRH) neuronal network controlling fe
177 Estradiol feedback regulates gonadotropin-releasing hormone (GnRH) neurons and subsequent luteiniz
183 ons act cooperatively to excite gonadotropin-releasing hormone (GnRH) neurons during positive feedbac
184 tioning in mammals depends upon gonadotropin-releasing hormone (GnRH) neurons generating a pulsatile
185 controlling the activity of the gonadotropin-releasing hormone (GnRH) neurons in vivo to establish th
187 ypogonadism to study effects of gonadotropin releasing hormone (GnRH) neurons on neuronal circuits co
190 and electrophysiology, that the gonadotropin-releasing hormone (GnRH) neurons that control mammalian
191 radiol on proestrus to activate gonadotropin-releasing hormone (GnRH) neurons that, in turn, trigger
192 which estradiol (E2) regulates gonadotropin-releasing hormone (GnRH) neurons to control fertility ar
193 erminal Ca(2+) is controlled in gonadotropin-releasing hormone (GnRH) neurons via action potentials a
194 tes reproduction by stimulating gonadotropin-releasing hormone (GnRH) neurons via the kisspeptin rece
203 ure of information transfer via gonadotropin-releasing hormone (GnRH) receptors (GnRHR) to extracellu
206 ion by brain-secreted pulses of gonadotropin-releasing hormone (GnRH) represents a longstanding puzzl
207 uisition of a mature pattern of gonadotropin-releasing hormone (GnRH) secretion from the CNS is a hal
210 sm had a measurable response to gonadotropin-releasing hormone (GnRH) stimulation, suggesting that it
211 e is an inherited deficiency of gonadotropin-releasing hormone (GnRH) that is characterized by hypogo
212 IKK-beta and NF-kappaB inhibit gonadotropin-releasing hormone (GnRH) to mediate ageing-related hypot
213 ropin hormones are regulated by gonadotropin-releasing hormone (GnRH) via MAPK signaling pathways tha
214 neurons that express genes for gonadotropin-releasing hormone (GnRH), a G-protein-coupled receptor f
215 express an antibody that binds gonadotropin-releasing hormone (GnRH), a master regulator of reproduc
217 hypothalamic neurons secreting gonadotropin-releasing hormone (GnRH), the neuropeptide controlling r
219 ry-gonadal axis is dependent on gonadotropin-releasing hormone (GNRH)-stimulated synthesis and secret
226 he receptor of the neuropeptide gonadotropin-releasing hormone (GnRHR) is unique among the G protein-
227 type D with a fragment of the growth hormone releasing hormone, has previously revealed promising res
228 ex restrains the amygdala, the corticotropin-releasing hormone/hypothalamic-pituitary-adrenal (CRH/HP
229 de homologous to the vertebrate gonadotropin-releasing hormone, is downregulated as workers become ga
232 ents on BPH therapy with luteinizing hormone-releasing hormone (LHRH) antagonist are also reviewed an
233 fied synthetic analog of luteinizing hormone-releasing hormone (LHRH) as a targeting moiety specific
234 able fragment (scFv) and luteinizing hormone releasing hormone (LHRH) peptide, resulting in specific
235 ortantly, SSA induced by luteinizing hormone-releasing hormone (LHRH) receptor antagonism bypassed th
237 ceived letrozole (plus a luteinizing hormone-releasing hormone [LHRH] agonist if premenopausal).
239 in or leptin on proopiomelanocortin, thyroid-releasing hormone, melanin-concentrating hormone, and or
240 aventricular hypothalamic CRH (corticotropin-releasing hormone) neuroendocrine neurons shortly after
241 in responses, suggesting normal gonadotropin-releasing hormone neuronal and gonadotrope function.
242 and send axonal projections to gonadotropin-releasing hormone neurons and regulate reproductive func
244 eus tractus solitarii (NTS) to corticotropin releasing hormone neurons in the paraventricular nucleus
245 PC1 suppressed the migration of gonadotropin-releasing hormone neurons without affecting cell prolife
249 these neurons largely express corticotrophin-releasing hormone or thyrotropin-releasing hormone and d
250 duction relies on hypothalamic gonadotrophin-releasing hormone output, and most cells producing repro
252 f RTN activity by Substance P or thyrotropin-releasing hormone, previously identified neuromodulators
254 ficial effects of agonists of growth hormone-releasing hormone receptor (GHRH-R) in heart failure mod
255 r (L-NAME) and stimulation of growth hormone-releasing hormone receptor (GHRHR) with GHRH agonists au
258 rtitioning mutant conformers of gonadotropin-releasing hormone receptor (GnRHR), a G protein-coupled
259 stimulates transcription of the thyrotropin-releasing hormone receptor (TRHR) by negative modulation
260 -binding site defined--for the corticotropin-releasing hormone receptor 1 (CRF1R)--which was located
265 ein 2)), chromosome 17 (CRHR1 (corticotropin-releasing hormone receptor 1) and MAPT (microtubule-asso
268 ve ganglion cells (dsGCs): TRHR (thyrotropin-releasing hormone receptor) and Drd4 (dopamine receptor
269 mbrane progestin receptor, the corticotropin releasing hormone receptor, and the 5HT1a serotonin rece
270 hours significantly increases corticotropin-releasing hormone receptor-1 (CRHR-1) mRNA and protein e
271 icant three-way interaction on corticotropin-releasing hormone receptor-1 (Crhr1) gene expression, in
272 gold nanorods (gGNRs) promotes gonadotropin releasing hormone receptor-mediated internalization and
276 s1 promoter disrupted pulsatile gonadotropin-releasing hormone release, delayed puberty and compromis
278 g sensitization of the NKB-NK3R-gonadotropin-releasing hormone signaling pathway under metabolic dist
279 itial and replication samples: corticotropin-releasing hormone signaling, cardiac beta-adrenergic sig
280 nalyses revealed enrichment of corticotropin-releasing hormone signaling, GNRH signaling, and/or CDK5
284 availability via leptin-induced thyrotropin-releasing hormone/thyroid-stimulating hormone expression
285 er factors control secretion of gonadotropin-releasing hormone to determine initiation of puberty.
286 satile, coordinated delivery of gonadotropin-releasing hormone to the pituitary and the resulting sur
287 om subsets of neurons expressing thyrotropin-releasing hormone (TRH) and pituitary adenylate cyclase-
288 yroid (HPT) axis hormones, i.e., thyrotropin-releasing hormone (TRH) and thyrotropin (TSH), are expre
289 loop that inhibits production of thyrotropin-releasing hormone (TRH) in the mediobasal hypothalamus (
293 phe-derived serotonin (5-HT) and thyrotropin-releasing hormone (TRH) play important roles in fundamen
296 ma-aminobutyric acid (GABA), and thyrotropin-releasing hormone (TRH) were colocalized with histamine
297 nin-concentrating hormone (MCH), thyrotropin-releasing hormone (TRH), gonadotropin-releasing hormone
298 activated thyroid-stimulating hormone (TSH)-releasing hormone (TRH)-positive neurons in the paravent
300 on requires timely secretion of gonadotropin-releasing hormone, which is controlled by a complex exci
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