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1 a confirmed independent cancer risk factor, remain poorly understood.
2 underlying MEF2C haploinsufficiency syndrome remain poorly understood.
3 the mechanisms underlying this extensive HGT remain poorly understood.
4 g transcription during productive elongation remain poorly understood.
5 fying their identities and functional states remain poorly understood.
6 oughout the metazoan, the functions of which remain poorly understood.
7 ounding line, but their formation mechanisms remain poorly understood.
8 ariations in other markers of sexual health, remain poorly understood.
9 molecular processes guiding ITC development remain poorly understood.
10 mutations to core behavioral symptoms of ASD remain poorly understood.
11 ntegrating the stemness with drug resistance remain poorly understood.
12 sion, but neuronal acid clearance mechanisms remain poorly understood.
13 nisms of chondrocyte recognition by NK cells remain poorly understood.
14 en diversity and impact on clinical outcomes remain poorly understood.
15 direct therapeutic responses of cancer cells remain poorly understood.
16 echanisms of parasite entry into hepatocytes remain poorly understood.
17 the underlying molecular signaling pathways remain poorly understood.
18 lying this type of stimulus-control learning remain poorly understood.
19 linking the infection to cancer development remain poorly understood.
20 iruses and the bee antiviral immune response remain poorly understood.
21 derlying sepsis-induced immune dysregulation remain poorly understood.
22 man genome, yet their RNA-binding properties remain poorly understood.
23 Its neural mechanisms remain poorly understood.
24 but the initial cyanophage-host interactions remain poorly understood.
25 ational control during mammalian development remain poorly understood.
26 ted, yet the events underlying its induction remain poorly understood.
27 d the forces that govern their establishment remain poorly understood.
28 isms by which they carry out these functions remain poorly understood.
29 he neural mechanisms underlying this process remain poorly understood.
30 lysosome movement in these domains, however, remain poorly understood.
31 molecular dynamics and internal organization remain poorly understood.
32 functional consequences of such interactions remain poorly understood.
33 ory pathways promote sensations such as itch remain poorly understood.
34 n on ocean biological and chemical processes remain poorly understood.
35 unction, the underlying molecular mechanisms remain poorly understood.
36 xtensively, the mechanisms of ECM remodeling remain poorly understood.
37 ate decisions in early embryonic development remain poorly understood.
38 s and developmental trajectory underlying SG remain poorly understood.
39 uvenile and adult fish to acute oil exposure remain poorly understood.
40 ts of cocaine, but the underlying mechanisms remain poorly understood.
41 g synaptic vesicle dynamics during recycling remain poorly understood.
42 the mechanisms that inhibit such plasticity remain poorly understood.
43 he pathogenesis of AP-4 deficiency, however, remain poorly understood.
44 e underlying biological mechanisms, however, remain poorly understood.
45 nisms regulating the non-AUG initiation rate remain poorly understood.
46 me dominates within a newly formed polyploid remain poorly understood.
47 ith the hybridization of the oligonucleotide remain poorly understood.
48 orphometric changes in ET, but these changes remain poorly understood.
49 the mechanisms underlying community assembly remain poorly understood.
50 isms underlying nuclear dysmorphia in cancer remain poorly understood.
51 the molecular mechanisms regulating immunity remain poorly understood.
52 rs benefits on processes related to learning remain poorly understood.
53 reprogramming in preimplantation development remain poorly understood.
54 on the electrical conductivity of antigorite remain poorly understood.
55 (MaSC) activity and breast cancer formation remain poorly understood.
56 mechanisms and functions of RGS2 proteolysis remain poorly understood.
57 al functions of most DEG/ENaC-encoding genes remain poorly understood.
58 ors in coordination with mitotic progression remain poorly understood.
59 contribute to the life cycle of S. pyogenes remain poorly understood.
60 slational activation by RNA-binding proteins remain poorly understood.
61 Cellular mechanisms of DVT initiation remain poorly understood.
62 stromal signals that promote B lymphopoiesis remain poorly understood.
63 ophil response to Mycobacterium tuberculosis remain poorly understood.
64 pathophysiology, whereas related mechanisms remain poorly understood.
65 e transfer at polymer/electrolyte interfaces remain poorly understood.
66 alpha (ERalpha) in breast cancer development remain poorly understood.
67 ry influences on mental body-representations remain poorly understood.
68 fields on protein stability and conformation remain poorly understood.
69 ever, the factors that promote DCIS invasion remain poorly understood.
70 build damaged or missing cellular structures remain poorly understood.
71 sms by which these molecules are transported remain poorly understood.
72 polarization with atherosclerosis regression remain poorly understood.
73 However, thalamocortical synaptic properties remain poorly understood.
74 functions as a tumor suppressor in pre-B ALL remain poorly understood.
75 ures of postnatal spinal circuit development remain poorly understood.
76 -benefits of species and carbon conservation remain poorly understood.
77 lly, patient perceptions toward such reports remain poorly understood.
78 naling pathways that mediate podocyte injury remain poorly understood.
79 prevalence in autism spectrum disorder (ASD) remain poorly understood.
80 for which the neurobiological underpinnings remain poorly understood.
81 neural mechanisms underlying this modulation remain poorly understood.
82 and differentiation of human HSCs remain poorly understood.
83 ignalling pathways that cause these diseases remain poorly understood.
84 tionary consequences of high-order epistasis remain poorly understood.
85 ignaling networks controlling beta-cell fate remain poorly understood.
86 ehavioural states affect stimulus perception remain poorly understood.
87 ms through which junctions influence folding remain poorly understood.
88 epigenetic marks, including DNA methylation, remain poorly understood.
89 derlying the interaction between both metals remain poorly understood.
90 ers, but the underlying molecular mechanisms remain poorly understood.
91 recycling to its NO-sensitive, reduced state remain poorly understood.
92 ar mechanisms underlying their neurotoxicity remain poorly understood.
93 erties of these multicellular configurations remain poorly understood.
94 L2; however, their physiological function(s) remain poorly understood.
95 , but precise determinants of the phenomenon remain poorly understood.
96 terval timing, yet the underlying mechanisms remain poorly understood.
97 nderlying recognition in natural populations remain poorly understood.
98 f the different factors giving rise to them, remain poorly understood.
99 erapeutic effects, the underlying mechanisms remain poorly understood.
100 he core behavioral features of mood disorder remain poorly understood.
101 ory, but the underlying molecular mechanisms remain poorly understood.
102 mechanisms underlying such behavioral choice remain poorly understood.
103 fic mechanisms underlying these interactions remain poorly understood.
104 of T-cells in rVSV-EBOV mediated protection remain poorly understood.
105 xtinction of these context-specific memories remain poorly understood.
106 uloskeletal tissues, the mechanisms for this remain poorly understood.
107 ence of experience on interneuron plasticity remain poorly understood.
108 ting to the specificity of such interactions remain poorly understood.
109 heir functions in hematological malignancies remain poorly understood.
110 chanisms underlying sex-specific development remain poorly understood.
111 h the mechanisms favoring PMN-MDSC responses remain poorly understood.
112 w alteration of this process leads to cancer remain poorly understood.
113 However, its mechanisms of action remain poorly understood.
114 roglia alter the course of AD neuropathology remain poorly understood.
115 lterations underlying this aberrant behavior remain poorly understood.
116 /activation in idiopathic pulmonary fibrosis remain poorly understood.
117 walls facilitate dynamic stomatal responses remain poorly understood.
118 he binding properties of its small RNA guide remain poorly understood.
119 by which tau organizes microtubule networks remain poorly understood.
120 both calcifying and silicifying haptophytes remain poorly understood.
121 mote IL-22 expression in the human intestine remain poorly understood.
122 enerate and maintain gut bacterial diversity remain poorly understood.
123 Mechanisms that orchestrate these events remain poorly understood.
124 but the drivers of fine-root trait diversity remain poorly understood.
125 osition and structure of these cytoskeletons remain poorly understood.
126 but the mechanisms underlying this toxicity remain poorly understood.
127 ver, the mechanisms underlying these effects remain poorly understood.
128 process, the underlying molecular mechanisms remain poorly understood.
129 cycling in relation to dry season intensity remain poorly understood.
130 ction and the mechanism of N-WASP activation remain poorly understood.
131 decidua during this early stage of infection remain poorly understood.
132 n cascade density on radiation damage in SiC remain poorly understood.
133 "hot spots" within the metal nanostructures, remain poorly understood.
134 However, neurological functions of POMTs remain poorly understood.
135 erning this flow and its impact on symbiosis remain poorly understood.
136 s of alien introduction and species richness remain poorly understood.
137 s, the mechanism of action, and target cell, remain poorly understood.
138 through which DISC1 affects stress responses remain poorly understood.
139 ts their ability to combat tumor progression remain poorly understood.
140 nsory ganglia, but the underlying mechanisms remain poorly understood.
141 ses underlying this heightened vulnerability remain poorly understood.
142 e intrinsic effects of force on tumour cells remain poorly understood.
143 ization, although the controlling mechanisms remain poorly understood.
144 al cord injury, the role of cortical targets remain poorly understood.
145 nce of this evolutionary conserved diversity remains poorly understood.
146 Extension from DNA damage at primer termini remains poorly understood.
147 2 expression is regulated in each ILC subset remains poorly understood.
148 r interior to find their functional partners remains poorly understood.
149 eg) cells in the loss of tolerance to gluten remains poorly understood.
150 in the development of ccRCC via inflammation remains poorly understood.
151 individual mast cells to cumulative stimuli remains poorly understood.
152 mic-resolution mechanism of its inactivation remains poorly understood.
153 microcircuits control long-range projections remains poorly understood.
154 strain, how HDs respond to mechanical stress remains poorly understood.
155 lar pathway, but sensing of physical stimuli remains poorly understood.
156 fects simple spike activity (and vice versa) remains poorly understood.
157 late stent thrombosis (VLST) to these events remains poorly understood.
158 lex and variable natural resource landscapes remains poorly understood.
159 polymerases gain access to the DNA template remains poorly understood.
160 th viral sensing and autoimmune pathogenesis remains poorly understood.
161 ; however, the receptor activation mechanism remains poorly understood.
162 C-terminal MF domain (CMF) of these myosins remains poorly understood.
163 n high-dimensional potential energy surfaces remains poorly understood.
164 effect, critical to enhancing public health, remains poorly understood.
165 eir most characteristic feature, the flower, remains poorly understood.
166 Their mode of action, however, remains poorly understood.
167 , its contribution to epithelial homeostasis remains poorly understood.
168 KdpA is coupled with ATP hydrolysis by KdpB remains poorly understood.
169 n of its individual domains to this activity remains poorly understood.
170 this alteration contributes to tumorigenesis remains poorly understood.
171 sed and integrated by the downstream network remains poorly understood.
172 ry syndrome (IRIS), but its underlying cause remains poorly understood.
173 ecies, the pathogenesis of Babesia infection remains poorly understood.
174 rtually all organisms, but how they function remains poorly understood.
175 conserved behavioral state whose regulation remains poorly understood.
176 r, the molecular basis of viral pathogenesis remains poorly understood.
177 ng between cell size, shape and constriction remains poorly understood.
178 e pathogenesis of severe asthma in childhood remains poorly understood.
179 yet how LDs form and function during hypoxia remains poorly understood.
180 herin loss to the gain of mesenchymal traits remains poorly understood.
181 ationship between dysbiosis and constipation remains poorly understood.
182 ing, the precise mechanism underlying gating remains poorly understood.
183 ts of CBs on the dynamics of neural circuits remains poorly understood.
184 olecular basis for tumor cell response to CQ remains poorly understood.
185 LC2 responses are regulated by other stimuli remains poorly understood.
186 f differentially present genes in eukaryotes remains poorly understood.
187 ogy, the mechanistic basis for mental effort remains poorly understood.
188 cal mechanism of social interactive deficits remains poorly understood.
189 anin fulfills its controlling role, however, remains poorly understood.
190 ecise function in the gastrointestinal tract remains poorly understood.
191 rvation prioritization, but the relationship remains poorly understood.
192 ason for such focal susceptibility to cancer remains poorly understood.
193 industrial applications; however, its origin remains poorly understood.
194 to vary >500 mV, its impact on HCO activity remains poorly understood.
195 regnancy, amniotic sac development in humans remains poorly understood.
196 nst diabetes-related cardiovascular diseases remains poorly understood.
197 y which they increase risk for these cancers remains poorly understood.
198 n peritoneal anchoring of metastatic lesions remains poorly understood.
199 expression of HER2, the underlying mechanism remains poorly understood.
200 However, the underlying circuit mechanism remains poorly understood.
201 ree-dimensional (3D) chromatin architectures remains poorly understood.
202 cell fate determination, but their interplay remains poorly understood.
203 tion advances under such complex stimulation remains poorly understood.
204 How neural activity creates this motivation remains poorly understood.
205 membrane permeabilizing activity of ceramide remains poorly understood.
206 how these diverse processes are coordinated remains poorly understood.
207 , how serotonin release is regulated in vivo remains poorly understood.
208 to the pathophysiology of persistent asthma remains poorly understood.
209 stantial skin irritation, a side effect that remains poorly understood.
210 nd segregation of chromosomes during mitosis remains poorly understood.
211 acy is modulated by other molecular contexts remains poorly understood.
212 functional sites leading to loss-of-function remains poorly understood.
213 nism of the natural development of tolerance remains poorly understood.
214 e nucleation centres, its role in nucleation remains poorly understood.
215 ues, but their tissue-restricted homeostasis remains poorly understood.
216 matin structure and dynamics in this process remains poorly understood.
217 ntrols the abundance of active Lck molecules remains poorly understood.
218 local protein synthesis in developing axons remains poorly understood.
219 Chronic fatigue syndrome (CFS) remains poorly understood.
220 structural basis underlying these base pairs remains poorly understood.
221 s, but their relevance in the nervous system remains poorly understood.
222 g of mRNA, the structural mechanism of which remains poorly understood.
223 e mechanistic underpinning of this crosstalk remains poorly understood.
224 sistence of animals in changing environments remains poorly understood.
225 gulate mRNA gene expression in a single cell remains poorly understood.
226 regulate cancer development and progression remains poorly understood.
227 (ECM) contribute to MSC phenotype in cancer remains poorly understood.
228 echanism underlying lysosomal deficits in AD remains poorly understood.
229 uced life expectancy, the underlying biology remains poorly understood.
230 structure of R5 during silica precipitation remains poorly understood.
231 fter a single AD trial, and this variability remains poorly understood.
232 factor (TF) occupancy and enhancer activity remains poorly understood.
233 The impact of SCNAs on tumour biology remains poorly understood.
234 ependent processes, the role of the daughter remains poorly understood.
235 es (MTs), but the mechanism of this activity remains poorly understood.
236 meostasis, communication between these cells remains poorly understood.
237 rolling proliferation potential of LRP cells remains poorly understood.
238 ia to the response to chemotherapeutic drugs remains poorly understood.
239 for mixed stands of varying species richness remains poorly understood.
240 er-massive black holes in the early universe remains poorly understood.
241 l genome, transcription initiation by mtRNAP remains poorly understood.
242 ial contribution to plant volatile phenotype remains poorly understood.
243 are propagated throughout the kinase domain remains poorly understood.
244 ver, how TARPs modulate AMPA receptor gating remains poorly understood.
245 ession are determined during differentiation remains poorly understood.
246 ction on S. aureus antibiotic susceptibility remains poorly understood.
247 and how this in turn shapes long range flow, remains poorly understood.
248 olecular pathogenesis of these complications remains poorly understood.
249 programming induced by persistent DNA damage remains poorly understood.
250 ternal forcing during the last deglaciation, remains poorly understood.
251 morphology is maintained in the adult brain remains poorly understood.
252 nal heterogeneity between glioblastoma cells remains poorly understood.
253 very of an ecosystem following such an event remains poorly understood.
254 most cases, though their mechanism of action remains poorly understood.
255 behind nanoscale thermal transport, however, remains poorly understood.
256 sms underlying this MT-actin cross talk have remained poorly understood.
257 igands as well as their functional relevance remained poorly understood.
258 tly, immune responses in filovirus survivors remained poorly understood.
259 c mechanism of its degenerative pathogenesis remains poorly understood, a subcellular spatial alterat
262 y strategies driving intraspecific variation remain poorly understood and have not been evaluated in
263 regulation of this pathway in breast tissue remains poorly understood and the consequences of immedi
264 hotosensitized reactions in ambient aerosols remains poorly understood and unaccounted for in atmosph
265 anic matter (CDOM) and humic substances (HS) remains poorly understood and yet to be investigated ade
266 ved, and potential for therapeutic targeting remain poorly understood, and are unknown in severe, ste
267 on is normally regulated in epithelial cells remain poorly understood, and its potential roles in reg
269 inflammatory diseases; however, these cells remain poorly understood because of the absence of speci
270 mechanism of Rap1 transcriptional activation remains poorly understood because Rap1 is encoded by a s
271 ss a range of flows, helicity in real fluids remains poorly understood because the entire quantity is
272 isms through which GDF15 reduces body weight remain poorly understood, because the cognate receptor f
273 ms by which they affect mammalian physiology remain poorly understood, but bacterial metabolites are
274 c history of ZIKV in Brazil and the Americas remain poorly understood, despite the value of this info
275 and ecosystem dynamics, but its full impacts remain poorly understood due to the difficulty of manipu
282 ll lineage have been extensively studied, it remains poorly understood how tissues that contain multi
283 controls, spanning scales of space and time, remain poorly understood in arctic tundra wetlands, part
284 key species in marine environments, but they remain poorly understood in part because of their large,
287 ng in finitely repeated games of cooperation remains poorly understood in part because their dynamics
288 ensively studied in suspension cultures, but remains poorly understood in substrate-dependent cells.
289 h prevalence of chronic musculoskeletal pain remain poorly understood, in part because little is know
290 ructural, and taxonomic diversity of viruses remain poorly understood, in part because sparse samplin
291 cale patterns and drivers of shrub expansion remain poorly understood, inhibiting accurate incorporat
293 f excitatory and inhibitory synapse function remain poorly understood; no proteins that regulate exci
294 g behaviour, and its impacts on individuals, remain poorly understood, particularly for short-term fo
296 ficking, and function, particularly in vivo, remain poorly understood, though recent studies have imp
297 otor neurons but their combinatorial actions remain poorly understood; to address this, we here scree
300 w these effects influence the fate of groups remains poorly understood, which limits our understandin
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