1 However, its mechanisms of
action remain poorly understood.
2 of this interaction and the mechanism of N-WASP
activation remain poorly understood.
3 circuit-level alterations underlying this aberrant
behavior remain poorly understood.
4 hy cells and how alteration of this process leads to
cancer remain poorly understood.
5 ment for decades, the mechanism of action, and target
cell,
remain poorly understood.
6 ever, the mechanisms of chondrocyte recognition by NK
cells remain poorly understood.
7 e or exacerbate inflammatory diseases; however, these
cells remain poorly understood because of the absence of specific m
8 molecular composition and structure of these
cytoskeletons remain poorly understood.
9 ular mechanisms linking the infection to cancer
development remain poorly understood.
10 amics of translational control during mammalian
development remain poorly understood.
11 ntal-genetic mechanisms underlying sex-specific
development remain poorly understood.
12 rocesses that generate and maintain gut bacterial
diversity remain poorly understood.
13 tem processes, but the drivers of fine-root trait
diversity remain poorly understood.
14 However, the mechanisms underlying these
effects remain poorly understood.
15 Mechanisms that orchestrate these
events remain poorly understood.
16 er, landscape-scale patterns and drivers of shrub
expansion remain poorly understood, inhibiting accurate incorporation o
17 overnutrition, a confirmed independent cancer risk
factor,
remain poorly understood.
18 differentiation/activation in idiopathic pulmonary
fibrosis remain poorly understood.
19 mmary stem cell (MaSC) activity and breast cancer
formation remain poorly understood.
20 l the balance of excitatory and inhibitory synapse
function remain poorly understood; no proteins that regulate excitator
21 te to reshape the binding properties of its small RNA
guide remain poorly understood.
22 neralisation in both calcifying and silicifying
haptophytes remain poorly understood.
23 ivers of caching behaviour, and its impacts on
individuals,
remain poorly understood, particularly for short-term food ca
24 actions in the decidua during this early stage of
infection remain poorly understood.
25 and carbon (C) cycling in relation to dry season
intensity remain poorly understood.
26 anisms contributing to the specificity of such
interactions remain poorly understood.
27 lism, but specific mechanisms underlying these
interactions remain poorly understood.
28 on F-actin and the details of the intermolecular
interface remain poorly understood at atomic resolution.
29 anisms that promote IL-22 expression in the human
intestine remain poorly understood.
30 However, the factors that promote DCIS
invasion remain poorly understood.
31 olid cancers, their functions in hematological
malignancies remain poorly understood.
32 vascular disorders, but the underlying molecular
mechanisms remain poorly understood.
33 ribute to this process, the underlying molecular
mechanisms remain poorly understood.
34 a synaptic dysfunction, the underlying molecular
mechanisms remain poorly understood.
35 innervating sensory ganglia, but the underlying
mechanisms remain poorly understood.
36 e of plasmonic "hot spots" within the metal
nanostructures,
remain poorly understood.
37 The mechanisms by which tau organizes microtubule
networks remain poorly understood.
38 ms by which microglia alter the course of AD
neuropathology remain poorly understood.
39 lying the influence of experience on interneuron
plasticity remain poorly understood.
40 However, neurological functions of
POMTs remain poorly understood.
41 bject, though the neural mechanisms underlying this
process remain poorly understood.
42 gree this affects their ability to combat tumor
progression remain poorly understood.
43 models, although the mechanisms favoring PMN-MDSC
responses remain poorly understood.
44 how guard cell walls facilitate dynamic stomatal
responses remain poorly understood.
45 the mechanisms through which DISC1 affects stress
responses remain poorly understood.
46 n in the drivers of alien introduction and species
richness remain poorly understood.
47 of the collision cascade density on radiation damage in
SiC remain poorly understood.
48 mechanisms governing this flow and its impact on
symbiosis remain poorly understood.
49 cleinopathies," but the mechanisms underlying this
toxicity remain poorly understood.
50 ological processes underlying this heightened
vulnerability remain poorly understood.