1 iators of ER stress pathway in promoting lipid
accumulation remain poorly understood.
2 a, but the molecular mechanisms that initiate this
activity remain poorly understood.
3 age-specific TE insertions to recent regulatory
adaptations remain poorly understood.
4 The mechanisms mediating
AMC remain poorly understood.
5 I, their neurochemical and behavioral consequences, if
any,
remain poorly understood.
6 ecognition, some fundamental aspects of protein-DNA
binding remain poorly understood(1,2).
7 oligomerization, and interactions with cellular
chaperones remain poorly understood.
8 ture, the mechanisms that underlie their ionic
conductivity remain poorly understood.
9 1968, the mechanisms driving sarcomatoid
dedifferentiation remain poorly understood, and information and treatment optio
10 surface expression of Tim-3, but the molecular
determinants remain poorly understood.
11 immune and inflammatory cells in neurodegenerative
diseases remain poorly understood.
12 mechanisms responsible for severe inflammatory side
effects remain poorly understood.
13 esponses, yet the complex mechanisms driving immune
evasion remain poorly understood.
14 affecting aging men, but the underlying molecular
features remain poorly understood, and therapeutic options are limited
15 the factors that regulate fascin dynamics within
filopodia remain poorly understood.
16 ytes, and their evolutionary origins and ancestral
function remain poorly understood.
17 eletal system, the mechanisms that control myotube
guidance remain poorly understood.
18 The molecular mechanisms that link these pathways,
however,
remain poorly understood.
19 and frequent as droughts, are overlooked and their
impacts remain poorly understood.
20 olecular mechanisms and the influence of exercise
intensity remain poorly understood.
21 of lignin-utilizing bacteria, but their
interrelationships remain poorly understood.
22 ilability and whether it recapitulates embryonic
mechanisms remain poorly understood.
23 ess for As-induced neurotoxicity, the underlying
mechanisms remain poorly understood.
24 with premature disease onset but the underlying
mechanisms remain poorly understood.
25 ciples and dynamic changes of the bacterial wine
microbiota remain poorly understood, especially in the context of red an
26 genetic architecture and neurogenetics of animal
migration remain poorly understood.
27 nal importance, the mechanisms controlling digit
morphology remain poorly understood.
28 echanisms underlying the persistent accumulation of Mo/
MPhi remain poorly understood.
29 r, the molecular processes underpinning flowering in
nature remain poorly understood.
30 wever, the neurophysiological processes modified by
nesting remain poorly understood.
31 ible for this antibody-mediated immunity (AMI) to Y.
pestis remain poorly understood.
32 plasticity, and the mechanisms linking these two
phenomena,
remain poorly understood.
33 egrating light and temperature signaling pathways in
plants remain poorly understood.
34 by which viable interneurons are produced from
progenitors remain poorly understood.
35 ects of lipid mixing on the membrane biophysical
properties remain poorly understood.
36 contextual extinction behavior and subsequent
reinstatement remain poorly understood.
37 kably, the plant Nat machinery and its biological
relevance remain poorly understood, although NTA has gained recognition
38 gnostic indicator in T-ALL, the mechanisms of GC
resistance remain poorly understood.
39 affect the gammaherpesvirus-driven germinal center
response remain poorly understood.
40 ver, mechanisms underlying this so-called foraging
response remain poorly understood.
41 derlying the relationship between cell number and body
size remain poorly understood.
42 to remain flexible and adapt to a particular internal
state remain poorly understood.
43 ture of charge carriers and their transfer mechanisms
still remain poorly understood, especially for the cases of liquid-
44 atterns of transmission of drug-resistant tuberculosis (
TB)
remain poorly understood, despite over half a million inciden
45 re effector functions during development by mechanisms
that remain poorly understood.
46 sms that help myofibroblasts to persist in fibrotic
tissues remain poorly understood.
47 cesses that underpin mutational selection in normal
tissues remain poorly understood.
48 biquity, the possible health risks associated with ENDS
use remain poorly understood.
49 the production of these cis-isomers from trans-
violaxanthin remain poorly understood.
50 carbon (SOC) sequestration mechanisms in estuarine
wetlands remain poorly understood.