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1 The health ramifications of our findings remain to be seen.
2 leads to neurological pathology and symptoms remain to be seen.
3 th in patients receiving long-term treatment remains to be seen.
4 Whether these associations are causal remains to be seen.
5 ul this approach will ultimately prove to be remains to be seen.
6 HF risk prediction or targets for prevention remains to be seen.
7 s of these models and classification schemes remains to be seen.
8 al biological significance of these products remains to be seen.
9 ersimplification of the correlation approach remains to be seen.
10 health benefits to other countries worldwide remains to be seen.
11 d convergence of (any) two bacterial species remains to be seen.
12 s replication in seronegative human subjects remains to be seen.
13 mal translate into true benefit for patients remains to be seen.
14 rent limitations in measurement sensitivity, remains to be seen.
15 these abilities have communicative relevance remains to be seen.
16 s that are models of adaptive sophistication remains to be seen.
17 nosuppressive agents on the incidence of RLN remains to be seen.
18 ly reduced Notch signaling in vivo, however, remains to be seen.
19 are involved in the aetiology of depression remains to be seen.
20 but the overall effect on long-term survival remains to be seen.
21 ntage in designing more effective immunogens remains to be seen.
22 activity that preceded occupation of MAN-44 remains to be seen.
23 l prove to have been necessary or sufficient remains to be seen.
24 s will translate into real clinical advances remains to be seen.
25 ions of alpha-melanocyte-stimulating hormone remains to be seen.
26 Whether these favorable trends will continue remains to be seen.
27 ry, however, the realization of this promise remains to be seen.
28 s-leukemia effects and disease-free survival remains to be seen.
29 Whether this is structural and/or regulatory remains to be seen.
30 these non-vaccine types will have on disease remains to be seen, but clearly continued efforts at cha
31 strong kinesin tail-microtubule interaction remains to be seen, but it is likely to play an importan
32 h enduring effects extend to other disorders remains to be seen, but the capacity to reduce risk foll
35 ructure is the central piece in a puzzle; it remains to be seen how it will fit together with other d
39 valence-screening data are available, and it remains to be seen if CT will truly reduce mortality.
41 may be ligand-binding receptors, although it remains to be seen if phospholipids or possibly other mo
43 mber of oral macrocycles is still low and it remains to be seen if they are outliers or if macrocycle
46 uences of this substrate-induced aggregation remain to be seen, it has the potential to play a role i
52 ontrol the pace of drug release; however, it remains to be seen whether an autocatalytic-based drug d
54 different from those of other organs, and it remains to be seen whether flower waxes have composition
55 ol corrects the malabsorption of calcium, it remains to be seen whether higher amounts of vitamin D c
56 y the depletion of intracellular Cl-, but it remains to be seen whether it is mediated predominantly
61 genetic relationship of these two hosts, it remains to be seen whether or not this conservation of s
67 , as GHB can also be metabolized to GABA, it remains to be seen whether the many GABAB receptor-media
68 ting properties have been attributed, but it remains to be seen whether the PH domain is involved in
69 e carbon and nitrogen fractionations, but it remains to be seen whether the specific effects observed
76 l in a large number of diabetic patients, it remains to be seen whether those who are preparing the E
80 and pacemaker (PM) cardiomyocytes (CMs), it remains to be seen which subtypes are generated by direc
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