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1 atterns and drivers at the continental scale remain unknown .
2 echanisms of their activation and regulation remain unknown.
3 e influence (e.g., politicians and salesmen) remain unknown.
4 , functions for the majority of ApiAP2 genes remain unknown.
5 s of prolonged exposure to vehicle emissions remain unknown.
6 ular details of their effects on the enzymes remain unknown.
7 tion, storage, and expression of such models remain unknown.
8 ysiological functions of most family members remain unknown.
9 it transendothelial crossing to proplatelets remain unknown.
10 ygen delivery, and carbon dioxide reactivity remain unknown.
11 The processes underlying such COS emissions remain unknown.
12 that initiate and aggravate fibrosis in CTS remain unknown.
13 cific sodium permeation via calcium channels remain unknown.
14 rout pIgR or trout secretory component (tSC) remain unknown.
15 hanisms for carbofuran-impaired neurogenesis remain unknown.
16 e and its role in systemic defense responses remain unknown.
17 (III) chemoreceptor and regulatory mechanism remain unknown.
18 ut the mechanisms of skeletal manifestations remain unknown.
19 s of these specific interactions and actions remain unknown.
20 icroevolution within human hosts (intrahost) remain unknown.
21 bolic dependencies of obesity-associated PDA remain unknown.
22 ut the identity and function of such neurons remain unknown.
23 atures specific to individual NSCLC subtypes remain unknown.
24 l utility with targeted anticancer therapies remain unknown.
25 strust in the care provided to their parents remain unknown.
26 p63 is down-regulated after acute OE injury remain unknown.
27 lity, but mechanisms mediating these effects remain unknown.
28 sue repair at mucosal surfaces in early life remain unknown.
29 ular distribution and physiological function remain unknown.
30 hanism(s) for cancer cell-selective toxicity remain unknown.
31 ansformation but their exact molecular roles remain unknown.
32 ) of Argonautes (AGOs) involved in herbivory remain unknown.
33 alidomide-mediated sensitivity or resistance remain unknown.
34 lution of hemangioma into fibro-fatty tissue remain unknown.
35 sequelae of these ophthalmic manifestations remain unknown.
36 ce or absence of host inflammatory responses remain unknown.
37 functional consequences of this association remain unknown.
38 on criteria that predict outcomes after MIDP remain unknown.
39 m or endoderm, but the underlying mechanisms remain unknown.
40 in one host over years of chronic infection, remain unknown.
41 cell types are key players in this response remain unknown.
42 ups or carbon matrices for electron transfer remain unknown.
43 the mechanisms underlying these interactions remain unknown.
44 ts of its aetiology and molecular mechanisms remain unknown.
45 ease; yet, the molecular mechanisms involved remain unknown.
46 however, the underlying molecular mechanisms remain unknown.
47 etails of how scission is accomplished often remain unknown.
48 >20 years ago, but disease mechanisms still remain unknown.
49 e for these metabolic alterations in PH-Fibs remain unknown.
50 er and how they might influence its activity remain unknown.
51 s of prion spread during early CWD infection remain unknown.
52 chanisms underlying these behavioral effects remain unknown.
53 the functional significance of this reaction remain unknown.
54 njury and, if so, the underlying mechanisms, remain unknown.
55 ction at the blood-brain barrier (BBB) level remain unknown.
56 mechanisms and regulation of RAN translation remain unknown.
57 hat connect excess GC to tissue inflammation remain unknown.
58 al signaling, its action and molecular basis remain unknown.
59 llular cascades whose nature and connections remain unknown.
60 to Earth surface oxidation and Fe deposition remain unknown.
61 However, the underlying molecular mechanisms remain unknown.
62 n humans, many elements of this complex area remain unknown.
63 RAFV600E(+) LCH cell accumulation in lesions remain unknown.
64 MPR-II dysfunction conspire to cause disease remain unknown.
65 reduction and fatty acid metabolic handling remain unknown.
66 e neural circuits that mediate these effects remain unknown.
67 ar players involved in Pi sensing in mammals remain unknown.
68 h central serotonin action leads to fat loss remain unknown.
69 t the targets responsible for this phenotype remain unknown.
70 ivo canonical Wnt signalling and ISC biology remain unknown.
71 nzyme in more differentiated skeletal muscle remain unknown.
72 diseased limbal epithelial stem cells (LESC) remain unknown.
73 which infiltrating lymphocytes cause disease remain unknown.
74 aberrant mRNAs and their mechanism of action remain unknown.
75 o prevent excessive leukocyte transmigration remain unknown.
76 IIT-induced hypoglycemia, but the mechanism remains unknown.
77 ormal and chronic heart failure (CHF) states remains unknown.
78 nt excitatory and inhibitory synaptic inputs remains unknown.
79 riation on the evolution of parasite genomes remains unknown.
80 classic calciphylaxis or a different entity remains unknown.
81 as a function of vitamin D status in mothers remains unknown.
82 However, their medical significance remains unknown.
83 an bypass the general control of translation remains unknown.
84 power of such models for real human subjects remains unknown.
85 lity against naturally circulating parasites remains unknown.
86 to the spindle, but its role on the spindle remains unknown.
87 cy that is observed in ectotherms, on memory remains unknown.
88 n the cortex, but their functional relevance remains unknown.
89 )-induced obesity, however the genetic cause remains unknown.
90 fect of these substitutions on DPOR activity remains unknown.
91 of this diversity to maintain self-tolerance remains unknown.
92 y elicited during negative social evaluation remains unknown.
93 However, the intrinsic function of OsbHLH068 remains unknown.
94 se pathogenesis and the paired TCRbeta-chain remains unknown.
95 lso influences auditory-perceptual processes remains unknown.
96 rdiac function in both normal and CHF states remains unknown.
97 eceptor connecting cilium where its function remains unknown.
98 n is mediated only through PR-ER interaction remains unknown.
99 ating amblyopia, yet its physiological basis remains unknown.
100 ) participating in the basolateral Pi efflux remains unknown.
101 adaptation or resistance to mTOR inhibition remains unknown.
102 change in glial cell and myelin composition, remains unknown.
103 hose cognate receptor and intracellular fate remains unknown.
104 ation with Orai1-mediated cell proliferation remains unknown.
105 low-performing and high-performing hospitals remains unknown.
106 growth phase control of speB gene expression remains unknown.
107 his dolphin population's conservation status remains unknown.
108 or plays a functional role in leukemogenesis remains unknown.
109 in platelets, but its physiological function remains unknown.
110 many cases the nature of these improvements remains unknown.
111 rather than saline affects patient outcomes remains unknown.
112 emical capabilities and biological functions remains unknown.
113 gnaling, but the cardiac function of Herpud1 remains unknown.
114 enal tubules, though its mechanism of action remains unknown.
115 ole in neurogenesis during brain development remains unknown.
116 iations differ by Hispanic/Latino background remains unknown.
117 r core stress-responsive survival pathway(s) remains unknown.
118 led receptor for which the endogenous ligand remains unknown.
119 f myeloid differentiation in the bone marrow remains unknown.
120 ic disease in bats infected with filoviruses remains unknown.
121 le of progesterone receptors in these nuclei remains unknown.
122 However, why this blocks fibrosis remains unknown.
123 However, the precise role of BM in this axis remains unknown.
124 an actual disease gene in human infertility remains unknown.
125 distress syndrome secondary to other causes remains unknown.
126 , however, its function in the heart in vivo remains unknown.
127 patients with UIAs suitable for both options remains unknown.
128 N1 phosphorylation in response to DNA damage remains unknown.
129 l antigenicity of the surface-displayed ZnT8 remains unknown.
130 e to chimera formation in non-rodent species remains unknown.
131 respond to host rhythmic physiological cues remains unknown.
132 of the envelope's multilayered architecture remains unknown.
133 but its relationship to oncogenic signaling remains unknown.
134 h these mutations impact telomerase function remains unknown.
135 mance, the effect of GHRD on human cognition remains unknown.
136 s of the Knops polymorphisms on CR1 function remains unknown.
137 DDR communicates with the autophagy pathway remains unknown.
138 CoA and histone acetylation in these tissues remains unknown.
139 ut in a subset of families the genetic cause remains unknown.
140 ulum, but the exact nature of these currents remains unknown.
141 ication to complete its life cycle, however, remains unknown.
142 The mechanism underlying this currently remains unknown.
143 cancer, but what drives immune infiltration remains unknown.
144 smitter release, and short-term facilitation remains unknown.
145 ergic enhancement on visuospatial perception remains unknown.
146 that caspase-3 could have on tau metabolism remains unknown.
147 tinct steps in CCP formation in living cells remains unknown.
148 tion of D-mannose in T cell immune responses remains unknown.
149 calpain in airway smooth muscle remodelling remains unknown.
150 However, how LDAH regulates TAG metabolism remains unknown.
151 catalytic necessity of these kinase domains remains unknown.
152 als during early life in natural populations remains unknown.
153 n the VtE-induced amelioration of DN in vivo remains unknown.
154 the molecular and circuit-level dysfunction remains unknown.
155 However, long-term toxicity of SiNPs remains unknown.
156 e efforts the underlying molecular mechanism remains unknown.
157 ion; yet the full extent of their activities remains unknown.
158 neural basis for this fundamental difference remains unknown.
159 ey, further improves cardio-renal impairment remains unknown.
160 ivo and contributes to mammalian development remains unknown.
161 ent they uniformly provide high-quality care remains unknown.
162 patients with CAKUT, the causative mutation remains unknown.
163 ating Tau's inhibition of kinesin-1 motility remains unknown.
164 heir added value in the emergency department remains unknown.
165 omal RNA (rRNA), yet its mechanism of action remains unknown.
166 n of KCTD13 in the mammalian brain, however, remains unknown.
167 lung microbiome influences IPF host defense remains unknown.
168 al rearrangements, whose oncogenic potential remains unknown.
169 ion Strategy initiative, whose effectiveness remains unknown.
170 sterolemia on hepatic T-cell differentiation remains unknown.
171 of MS cortical pathology of which the cause remains unknown.
172 uces metabolic features promoting recurrence remains unknown.
173 ective response generated by preconditioning remains unknown.
174 However, how POT1 interacts with TPP1 remains unknown.
175 this extrinsic glycosylation pathway in vivo remains unknown.
176 , with an impact on surrounding brain cells, remains unknown.
177 lating anti-PE antibodies access cellular PE remains unknown.
178 The mechanism of NE uptake, however, remains unknown.
179 ng pathway triggering Src activation of IRF4 remains unknown.
180 lost, whereas its role in regenerating liver remains unknown.
181 fold as a function of threat-related arousal remains unknown.
182 netic regulator(s) underpinning this process remains unknown.
183 gonist; however, its mechanism of antagonism remains unknown.
184 tating caspase-3 activation and HuR cleavage remains unknown.
185 ic constraints placed by the histone octamer remains unknown.
186 d via a specific cell type within the kidney remains unknown.
187 llar malformations, the underlying mechanism remains unknown.
188 nk, but how this transformation is performed remains unknown.
189 subpopulations to the injured brain largely remains unknown.
190 The nature of this rare condition remains unknown.
191 However, the underlying mechanism remains unknown.
192 that controls the upregulation of N-cadherin remains unknown.
193 se, however, the function of alpha-synuclein remains unknown.
194 romium (Cr(VI)), but the molecular mechanism remains unknown.
195 that supports this high proliferative demand remains unknown.
196 in eukaryotes, but their molecular function remains unknown.
197 et the cellular origin of the bone phenotype remains unknown.
198 publications on TLRs, the function of TLR10 remains unknown.
199 the effect of non-covalent binding to Nedd8 remains unknown.
200 particularly in HIV-coinfected individuals, remains unknown.
201 ll polarization is established and regulated remains unknown.
202 in transgender women (transwomen) in Brazil remains unknown.
203 NS genes play conserved roles in nephrocytes remains unknown.
204 ibution of each process to neuronal polarity remains unknown.
205 te to affect plant signaling and development remains unknown.
206 mulator compounds bind and how they function remains unknown.
207 level of adhesion, the importance of lectins remains unknown.
208 ut its functional importance in human cancer remains unknown.
209 onsible for this cell-type specific function remains unknown.
210 on of the vast majority of CREs in chromatin remains unknown.
211 his modification may have in septin function remains unknown.
212 rapidly recruit new AMPARs during early LTP remains unknown.
213 on subgroup of paediatric ALL, its aetiology remains unknown.
214 SR and inhibits inversional switching events remains unknown.
215 ion between these hemoglobin traits and ESRD remains unknown.
216 vance to other viruses or in vivo infections remained unknown.
217 on system, but its role in pathogenicity has remained unknown.
218 ad52 contributes to RNA-dependent DSB repair remained unknown.
219 for ligand-induced NLR activation has so far remained unknown.
220 translates those updates into choices, have remained unknown.
221 5 in patterning the oropharyngeal region has remained unknown.
222 the gene responsible for this phenotype has remained unknown.
223 on neurons, but the underlying mechanism has remained unknown.
224 rmal' BiP activity post-oxidative stress has remained unknown.
225 However, the status of the Cys residues has remained unknown.
226 mechanisms underlying these alterations have remained unknown.
227 of S. cerevisiae Ydr109c and human FGGY has remained unknown.
228 responsible, but the functional aspects had remained unknown.
229 ism by which the transposon ends are cleaved remained unknown.
230 act (17%); in 22% of cases, infection source remained unknown.
231 nce of the two types of glia at the node has remained unknown.
232 that target HLB components to histone genes remained unknown.
233 genetics and structure of LA catabolism have remained unknown.
234 ns could drive long-term synaptic plasticity remained unknown.
235 fferent olfactory inputs interact has so far remained unknown.
236 been notified, and the status of their care remained unknown.
239 Despite increased research effort, much remains unknown about these phenomena, especially the ca
240 ve attracted a great deal of attention, much remains unknown about ZIKV disease epidemiology and ZIKV
241 gin of this pain (critical to its treatment) remains unknown, although spastic motor dysfunction has
242 unctional equivalent in protostomian animals remains unknown, although TRH receptors are conserved in
244 ited epigenetic states to organismal fitness remains unknown as well-documented examples are often no
245 itial isotopic composition of atmospheric Xe remains unknown, as do the mechanisms involved in its de
246 processes of the earliest European hominins remain unknown because most of the academic attention ha
247 d thermodynamic stability of these complexes remained unknown because they are hard to probe by conve
248 mechanism of how TPX2 stimulates MT assembly remains unknown because structural information about the
249 rlying ASD in approximately half of patients remain unknown, despite many of them being screened by w
250 ell epitopes and low immunogenicity of H7 HA remains unknown due to the lack of animal models reprodu
253 airment in QoL in nonallergic rhinitis (NAR) remained unknown for a long time, due to a lack of a val
255 r dementia, but the precise biological basis remains unknown, hampering the search for novel biomarke
258 f aluminum (Al) phytotoxicity for plants, it remained unknown if, and how, calcineurin B-like calcium
259 ar cells isolated from rodent testes, but it remains unknown if PFOS has similar effects in humans.
260 sequencing studies, the cause of the disease remains unknown in most patients, and several studies su
263 re of the initial plasma-tissue interactions remains unknown, including RONS speciation and delivery
265 the exact mechanisms underlying this process remain unknown, it was speculated that mediators release
268 y basic characteristics of gustatory neurons remain unknown, partly due to the absence of specific ma
269 ry, and the gene that codes for the receptor remains unknown, precluding the use of modern biological
270 onnections between burgeoning FMRP functions remain unknown, recent advances have extended understand
271 ng molecular and cellular mechanisms of PTSD remain unknown, recent studies indicate that PTSD is ass
274 er, the directionality of the causal pathway remains unknown.The objectives of this study were to exa
275 gh the mechanism by which FAS sequesters OGA remains unknown, these data suggest that FAS fine-tunes
277 istic gene expression signature; however, it remains unknown to what degree diverse TCR-mediated inte
278 major isoforms of dysbindin-1, (A, B, and C) remain unknown, we generated a novel mutant mouse, dys-1
279 ed in most major vertebrate lineages, but it remains unknown whether all SFMs share excitation-contra
281 display altered pain-modulation efficacy, it remains unknown whether brainstem pain-modulation circui
282 utes to adaptive physiological responses, it remains unknown whether changes in SK channel function/e
284 or starting antiretroviral treatment, but it remains unknown whether either of them is more advantage
288 nsitive to extracellular metabolites, but it remains unknown whether metabolites present at an infect
290 L1 loci are present in the same genomes, it remains unknown whether non-functional L1s have any tran
292 nisms control PD-L1 expression in cancer, it remains unknown whether such regulatory loops operate al
293 nction in inhibitory synapse development, it remains unknown whether there is a unifying mechanism fo
294 quantities of expanded NK cells ex vivo, it remains unknown whether these expanded NK cells can pers
295 rons play critical roles in seizures, but it remains unknown whether these vary across interneuron su
299 f seeking information about a reward, but it remains unknown whether, and how, neurons encode choices
300 children (40.4%), whose etiology would have remained unknown without genetic testing, underwent some
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