ライフサイエンスコーパス: renal artery

1 ltiple renal arteries (n = 106 with multiple renal arteries).

2 mbrane, and can always be traced back to the renal artery.

3 clips must not be used to control the donor renal artery.

4 surgery typically requires clamping the main renal artery.

5 result of an occluding thrombus in the main renal artery.

6 1 x 10(10)/ml infectious units) through the renal artery.

7 (ARF) induced by temporary occlusion of the renal artery.

8 on that provides maximal length of the right renal artery.

9 hemodynamically significant stenosis of any renal artery.

10 ctive treatment for revascularization of the renal artery.

11 than conventional treatment of only the main renal artery.

12 utes of contralateral (left) clamping of the renal artery.

13 mannitol 30 min or more before clamping the renal artery.

14 rom 30 to 15 min or less before clamping the renal artery.

15 perivascular fat compartment located around renal arteries.

16 eri-arterial sympathetic nerves around human renal arteries.

17 , angiotensin II, or acetylcholine in native renal arteries.

18 r direct bilateral infusion of BNP into both renal arteries.

19 ten impossible to optimize depiction of both renal arteries.

20 ikely to accurately depict the origin of the renal arteries.

21 is required to extend this technique to the renal arteries.

22 appears to be feasible for MR angiography of renal arteries.

23 e tool for restenosis and its application to renal arteries.

24 iod to identify patients with precaval right renal arteries.

25 t kidney should prompt a search for precaval renal arteries.

26 een patients with and those without precaval renal arteries.

27 d 39 additional patients with precaval right renal arteries.

28 y significant stenoses of accessory and main renal arteries.

29 Fifteen kidneys had 16 accessory renal arteries.

30 aortic labeling pulse was developed for the renal arteries.

31 mented with flow probes on the pulmonary and renal arteries.

32 ndable bare metal stents) were placed in 692 renal arteries, 156 superior mesenteric arteries (SMA),

33 In the 48 patients with precaval renal arteries, 52 precaval arteries were found, of whic

34 In three renal arteries, a signal void was found at the origin in

35 aused a pronounced (>10-fold) contraction of renal arteries after ischemia and after allogeneic trans

36 erial-enhanced MR imaging of the kidneys and renal arteries and added 8 minutes or less to the total

37 Two radiologists assessed visualization of renal arteries and detection of vascular disease.

38 e CO-releasing molecule (CORM)-3 constricted renal arteries and increased O2*- production in a dose-d

39 Multiple renal arteries and proximal calcifications may require e

40 lvement may occur, including stenosis of the renal arteries and veins, renal atrophy, and different t

41 al abdominal aorta halfway between the right renal artery and aortic trifurcation into the iliac and

42 Renal artery and peri-arterial soft tissue injury is gre

43 mplantation were performed four times in the renal artery and twice in the iliac artery.

44 s 100% safe and effective in controlling the renal artery and vein during HALDN, allowed for addition

45 efficacy of the NPL clip for control of the renal artery and vein during hand-assisted laparoscopic

46 llel NPL clips were used to control both the renal artery and vein.

47 allic clips simplified final division of the renal artery and vein.

48 an artifact, quality scores for the proximal renal arteries, and overall image quality scores between

49 ty of renal stenting after suboptimal/failed renal artery angioplasty in patients with suspected reno

50 n the basis of these results, precaval right renal arteries appear to be more common than previously

51 Going forward, clamping the main renal artery appears unnecessary during most partial nep

52 Eighteen (35%) of the 52 precaval renal arteries arose from the anterior aspect of the aor

53 diofrequency ablation was observed in 17% of renal arteries at 60 days and 71% of 180 days.

54 es after commencement of infusion to measure renal artery blood flow velocity and renal cortical perf

55 es after commencement of infusion to measure renal artery blood flow velocity and renal cortical perf

56 dpoint was the change from baseline in total renal artery blood flow.

57 To date, the literature on renal artery brachytherapy for restenosis consists of se

58 es in ultrasound dilution measurements after renal artery branch clamping did not correlate with chan

59 erior mesenteric arteries) and mediolateral (renal arteries) branch vessels in a cryogel abdominal ao

60 In contrast, targeted treatment of the renal artery branches or distal segment of the main rena

61 y comparing volume changes after clamping of renal artery branches.

62 caused small contractions only in native rat renal arteries but not in mesenteric arteries.

63 study was to assess the associations between renal artery calcification (RAC) and mortality in a heal

64 We tested the hypothesis that renal artery calcium (RAC), a marker of atherosclerotic

65 igation through either a ureteral stent or a renal artery cannulation, and the application of ice slu

66 mpaired endothelium-dependent relaxations in renal arteries, carotid arteries and aortae, and flow-me

67 ion The MARC catheter system is feasible for renal artery catheterization and embolization under real

68 (n = 2) receiving 9000 PFOB capsules through renal artery catheterization were imaged with a clinical

69 Male Sprague-Dawley rats underwent bilateral renal artery clamp for 30 min followed by reperfusion.

70 Renal artery clamping immediately caused significantly d

71 ysfunction and tissue damage after bilateral renal artery clamping, compared with wild-type mice.

72 h H2O2 and in whole kidneys after unilateral renal artery clamping.

73 SD (n = 9) or sham CSD (n = 9) 5 weeks after renal artery clipping, in comparison with normal Wistar

74 CT depicted 107 of 114 renal arteries confirmed at surgery; seven accessory art

75 Control angiographiy of the left renal artery confirmed the effective closure of all vasc

76 ested the hypothesis that AT1R-Abs can cause renal artery contraction by AT1R activation with renal i

77 st, atrophic kidneys beyond totally occluded renal arteries demonstrated low levels of R2* that did n

78 Prior studies of catheter-based renal artery denervation have not systematically perform

79 randomized study was to assess the impact of renal artery denervation in patients with a history of r

80 This trial did not demonstrate a benefit of renal artery denervation on reduction in ambulatory BP i

81 Renal artery denervation reduces systolic and diastolic

82 PVI only, and 13 were randomized to PVI with renal artery denervation.

83 ents were randomized to PVI only or PVI with renal artery denervation.

84 s with resistant hypertension 6 months after renal-artery denervation as compared with a sham control

85 d studies have suggested that catheter-based renal-artery denervation reduces blood pressure in patie

86 The measurements were repeated with renal arteries derived from native kidneys subjected to

87 Recognizing renal artery disease and directing revascularization pro

88 Atherosclerotic renal artery disease is present in 7% of the general pop

89 y adults and seven consecutive patients with renal artery disease, real-time navigator technology was

90 Reported procedural complications included 1 renal artery dissection and 4 femoral pseudoaneurysms.

91 In the crossover group, there was 1 renal artery dissection during guide catheter insertion,

92 n of an aortic balloon implanted between the renal arteries; during the same period, the RPP to the r

93 1) AMLs, nephrectomy (25% vs 7%, P<.001), or renal artery embolization (9% vs 2%, P<.05).

94 ted by means of vascular clamping (n = 5) or renal artery embolization (n = 3).

95 forming a simple endovascular procedure (ie, renal artery embolization) in vivo and to compare with x

96 options for reconstruction of the transplant renal artery exist, although no single technique has bee

97 itially based on histology, the diagnosis of renal artery fibromuscular dysplasia (FMD) is now based

98 s from failure of vascular clips used on the renal artery, first documented in 2006, have continued d

99 Changes in renal artery flow and perfusion were measured before and

100 Fifteen sheep were instrumented with a renal artery flow probe and renal vein cannula.

101 e surgically instrumented with pulmonary and renal artery flow probes in the renal cortex and medulla

102 tion times, renal parenchymal perfusion, and renal artery flow rates were measured for MR-guided and

103 There was a significant reduction in mean renal artery flow velocity (P = 0.045) and renal cortica

104 strong ion difference (P = 0.219), and mean renal artery flow velocity (P = 0.319) were similar.

105 ic "string of beads" that may be observed in renal artery FMD does not occur in coronary arteries, po

106 c classification into unifocal or multifocal renal artery FMD is straightforward and discriminates 2

107 Of 337 patients with established renal artery FMD, 276 (82%) were classified as multifoca

108 synthase inhibitor (aminoguanidine) into the renal artery for 2 hrs after the induction of sepsis, an

109 ocyanate (for smoking), intimal thickness of renal arteries (for hypertension), glycohemoglobin (for

110 Nine of 186 patients had precaval right renal arteries, for a prevalence of 5%.

111 A total of 49 renal arteries from 28 animals with 4 different time poi

112 and rats of both sexes, as well as in small renal arteries from female tammar wallabies (an Australi

113 ted the reduced myogenic reactivity of small renal arteries from relaxin-treated nonpregnant and midt

114 eased pro and active MMP-2 activity in small renal arteries from relaxin-treated nonpregnant and midt

115 al reduction in myogenic reactivity of small renal arteries from relaxin-treated nonpregnant rats was

116 1 to 133+/-1 mm Hg, P<0.001), and interlobar renal arteries from these rats displayed decreased relax

117 adiofrequency catheter (15 W/60 s) treated 8 renal arteries (group 1).

118 With SSFP MR angiography, 39 of 41 renal arteries in 19 patients were correctly detected.

119 adequate or excellent portrayal of the main renal arteries in 21 of 22 studies.

120 ed for their ability to depict the origin of renal arteries in patent vessels and for any signs of ti

121 that chymase is upregulated in coronary and renal arteries in patients with diabetes by immunohistoc

122 (SSFP) with inversion recovery for assessing renal arteries in patients with renal transplants.

123 daver organ donors to reconstruct transplant renal arteries in patients with specific lesions and fol

124 ne and 13 (33%) of 39 patients with precaval renal arteries in the retrospective and prospective grou

125 FMD was identified in the renal artery in 294 patients, extracranial carotid arter

126 e-3 siRNA was administered directly into the renal artery in hyperosmolar citrate solution (3 mug/ml)

127 e needed to maximize the length of the right renal artery in LDN.

128 ntil the time of release of the clamp on the renal artery in the recipient.

129 s more commonly performed, the prevalence of renal artery in-stent restenosis will increase.

130 Renal artery infusions of 2',3'-cAMP in vivo increased u

131 Renal artery injury secondary to blunt abdominal trauma

132 We hereby report two interesting cases of renal artery injury sustained in polytrauma patients.

133 the CT findings so as to accurately identify renal artery injury.

134 hours) was defined as the time of the donor renal artery interruption or aortic clamp, until the tim

135 quency of male subjects, abdominal aorta and renal artery involvement, and hypertension.

136 resolution, unenhanced MR angiography of the renal arteries is feasible with 3D radial undersampling.

137 Small renal arteries isolated from relaxin-1 gene-deficient mi

138 king (50% and 26%), prevalence of unilateral renal artery lesions (79% and 38%), presence of kidney a

139 e patients with aorto-ostial atherosclerotic renal artery lesions.

140 sin II-treated adult cardiac fibroblasts and renal artery-ligated rat heart, suggests that AA-driven

141 o cardiac hypertrophy model was generated by renal artery ligation in adult male Wistar rats (Rattus

142 mesenteric vasoconstriction was achieved by renal artery ligation.

143 e causes of pathological connections between renal arteries may be congenital or iatrogenic - mainly

144 hown to provide a more reliable depiction of renal artery morphology than older techniques.

145 heterologously expressing Kv7.4, and in rat renal artery myocytes.

146 none of whom received kidneys with multiple renal arteries (n = 106 with multiple renal arteries).

147 3, 10%), infrarenal aorta (n = 10, 32%), and renal artery (n = 1, 3%).

148 Furthermore, vasculitic changes can lead renal artery narrowness and can result to decrease in re

149 mal Care and Use Committee protocol, in vivo renal artery navigation and embolization were tested in

150 apy, specifically targeting and ablating the renal artery nerves with radiofrequency waves without pe

151 function and injury caused by I/R (bilateral renal artery occlusion [30 min] followed by reperfusion

152 After 30 min of bilateral renal artery occlusion and 24 h of reperfusion, mortalit

153 Twenty-four hours after bilateral, total renal artery occlusion for 15 minutes, transgenic sickle

154 Traumatic renal artery occlusion is a rare occurrence with devasta

155 ypertension and chronic renal failure due to renal artery occlusion was treated by endovascular recan

156 ffective procedure in the treatment of total renal artery occlusion which also led to a significant r

157 fixation with respect to renal dysfunction, renal artery occlusion, or endoleaks (P > .05).

158 ARF was induced in rats by renal artery occlusion.

159 ce, employing models of transient hepatic or renal artery occlusion.

160 of acute tubular injury caused by 30 min of renal artery occlusion.

161 eta1 or non-immune IgY were infused into the renal arteries of 3-d-old rats, and the kidneys were exa

162 ition, direct injection of histones into the renal arteries of mice demonstrated that histones induce

163 ute repercussion of renal denervation on the renal arteries of patients treated with balloon-based an

164 ction was enhanced approximately 1.5-fold in renal arteries of uni-x sheep (P<0.05).

165 ed by placing a silver clip (0.25 mm) on the renal artery of the retained contralateral native kidney

166 as injected sequentially into each segmental renal artery of the right kidney until capillary stasis

167 llografts with preservation solution via the renal artery or arteries is standard practice.

168 aortic patch (n=8), aortic conduit (n=1), or renal artery orifice (n=5).

169 verestimation of an existing stenosis at the renal artery origin can be caused by timing errors of th

170 ming errors impair the accurate depiction of renal artery origins.

171 tients with ARVD, neither renal function nor renal artery patency predicted a difference in echocardi

172 alyzed according to renal function, residual renal artery patency, and unilateral or bilateral ARVD.

173 ney function beyond occlusive disease of the renal arteries poses a major clinical challenge.

174 se contrast material-enhanced imaging of the renal arteries provided quantitative renal blood flow me

175 imaging- and conventional fluoroscopy-guided renal artery PTA in terms of success and complication ra

176 xcretion, and potential mediators, including renal artery pulsatility index, renal vascular resistanc

177 IVUS), and renal arteriography in diagnosing renal artery (RA) fibromuscular dysplasia (FMD) and corr

178 However, long-term safety of the renal artery (RA) is of concern.

179 In selected patients with hypertension, renal artery (RA) stenting is used to treat significant

180 hemodynamically significant lesion following renal artery reconstruction was considered a recurrence.

181 denervation using phenol application to the renal arteries reduced renal norepinephrine levels and b

182 t myogenic responses of mouse mesenteric and renal arteries rely on ligand-independent, mechanoactiva

183 uring pregnancy, is involved in systemic and renal artery remodeling and activates PPARgamma in vitro

184 rtery branches or distal segment of the main renal artery resulted in markedly less variability of re

185 for the development of a randomised trial of renal artery revascularisation versus medical therapy in

186 We aimed to compare clinical outcomes for renal artery revascularisation with medical therapy for

187 MSC infusion without main renal artery revascularization associated with increased

188 esuscitated sudden death, coronary artery or renal artery revascularization, lower-extremity arterial

189 Thus, failure to detect accessory renal arteries should not unduly affect the utility of a

190 sduced with Ad-IkappaB and injected into the renal artery significantly reduced inducible nitric oxid

191 agamma subunits colocalized in HEK cells and renal artery smooth muscle cells.

192 In kidneys with multiple arteries, localized renal artery stenoses produced focal elevations of R2*,

193 e novo or restenotic > or = 70% aorto-ostial renal artery stenoses, who underwent implantation of a b

194 68 patients had hemodynamically significant renal artery stenoses.

195 , we recruited patients with atherosclerotic renal artery stenosis (>50% as judged by CT, MR, or dire

196 Background: Atherosclerotic renal artery stenosis (ARAS) is associated with high blo

197 Atherosclerotic renal artery stenosis (ARAS) is known to reduce renal bl

198 was to assess the impact of atherosclerotic renal artery stenosis (ARAS) on outcomes after open-hear

199 esses the clinical syndromes associated with renal artery stenosis (RAS) and the published data guidi

200 that statins would decrease renal injury in renal artery stenosis (RAS) by restoring angiogenesis an

201 Renal artery stenosis (RAS) is a common disorder in adul

202 The selection of patients with renal artery stenosis (RAS) likely to improve glomerular

203 Renal artery stenosis (RAS) may impair renal function by

204 l Care and Use Committee approval, bilateral renal artery stenosis (RAS) was created surgically in 12

205 ic nephropathy compared with other causes of renal artery stenosis (RAS), but the underlying mechanis

206 To prospectively test--in a swine model of renal artery stenosis (RAS)--the hypothesis that magneti

207 g hypertension improvement after stenting of renal artery stenosis (RAS).

208 netic resonance (MR) angiography to diagnose renal artery stenosis (RAS).

209 study, we analyze the outcomes of transplant renal artery stenosis (TRAS), determine the different an

210 As observed in other patient cohorts, renal artery stenosis and adrenal causes of hypertension

211 NF1 patients develop renal artery stenosis and arterial occlusions resulting

212 stenting (PTRAS) is frequently used to treat renal artery stenosis and renovascular disease (RVD); ho

213 both for the identification of patients with renal artery stenosis and to follow patients with known

214 The risks of renal artery stenosis are related both to declining kidn

215 y revascularisation with medical therapy for renal artery stenosis associated with heart failure as t

216 First, is the realization that not only can renal artery stenosis cause renovascular hypertension, b

217 Recent studies indicate that atherosclerotic renal artery stenosis develops as a function of age and

218 Interest in identifying patients with renal artery stenosis has been stimulated recently by ad

219 After surgically inducing bilateral renal artery stenosis in 11 swine, the authors performed

220 sory artery stenosis unaccompanied by a main renal artery stenosis in either kidney; this patient had

221 Revascularisation of renal artery stenosis in heart failure is associated wit

222 e patients with aorto-ostial atherosclerotic renal artery stenosis in whom PTRA is unsuccessful, Palm

223 Renal artery stenosis is a common cause of secondary hyp

224 Atherosclerotic renal artery stenosis is increasingly common in an aging

225 Microvascular rarefaction distal to renal artery stenosis is linked to renal dysfunction and

226 Atherosclerotic renal artery stenosis reduces renal blood flow (RBF) and

227 One new renal artery stenosis requiring stenting and three death

228 rmed in patients with severe atherosclerotic renal artery stenosis scheduled for PTRA.

229 failure, and suggest that investigation for renal artery stenosis should be considered more frequent

230 Renal artery stenosis surgery (n = 10) or sham surgery (

231 on, and RBF in patients with atherosclerotic renal artery stenosis undergoing PTRA.

232 Normal pigs and pigs subjected to 3 weeks of renal artery stenosis were treated with six sessions of

233 st renal MR angiography and visualization of renal artery stenosis without exogenous contrast agent o

234 Eight pigs (two with induced unilateral renal artery stenosis) were studied with both electron-b

235 weeks of chronic RVD (induced by unilateral renal artery stenosis), established renal damage, and hy

236 y stenosis and to follow patients with known renal artery stenosis, has simplified the diagnostic asp

237 lated by concurrent hypercholesterolemia and renal artery stenosis, n = 7), RVD daily supplemented wi

238 In additional pigs with prolonged (6 weeks) renal artery stenosis, shockwave therapy also decreased

239 ther potential contributing factors, such as renal artery stenosis, valvular heart disease, and ische

240 on of renal fibrosis in mice with unilateral renal artery stenosis.

241 on of renal fibrosis in mice with unilateral renal artery stenosis.

242 emonstrate the hemodynamic significance of a renal artery stenosis.

243 angiography in patients suspected of having renal artery stenosis.

244 nt approach over another for atherosclerotic renal artery stenosis.

245 release and promoting hypertension following renal artery stenosis.

246 an internal carotid artery anomaly also had renal artery stenosis.

247 and alleviated fibrosis in pigs subjected to renal artery stenosis.

248 ular ejection fraction of less than 40% have renal artery stenosis.

249 revascularization for human atherosclerotic renal artery stenosis.

250 ned 947 participants who had atherosclerotic renal-artery stenosis and either systolic hypertension w

251 dical therapy in people with atherosclerotic renal-artery stenosis and hypertension or chronic kidney

252 Atherosclerotic renal-artery stenosis is a common problem in the elderly

253 ns, or hypertensive crisis at 1 month or new renal-artery stenosis of more than 70% at 6 months.

254 ultiple randomized clinical trials comparing renal artery stent placement plus medical therapy with m

255 disease from the largest randomized trial of renal artery stent placement, the CORAL (Cardiovascular

256 seline blood pressure affects outcomes after renal artery stent placement.

257 nce of a significant treatment effect of the renal artery stent procedure compared with medical thera

258 renal function is an important objective of renal artery stent procedures.

259 efficacy, and long-term clinical benefits of renal artery stent revascularization in hypertensive pat

260 Renal artery stenting alone, stenting with embolic prote

261 One hundred patients undergoing renal artery stenting at 7 centers were randomly assigne

262 As renal artery stenting becomes more commonly performed, t

263 insertion, before denervation, corrected by renal artery stenting, and 1 hypotensive episode, which

264 Renal-artery stenting did not confer a significant benef

265 ronic kidney disease to medical therapy plus renal-artery stenting or medical therapy alone.

266 omized trials that did not show a benefit of renal-artery stenting with respect to kidney function, t

267 lure of surgical clips to sustain closure of renal artery stumps in live donor nephrectomies were rec

268 rupted this colocalization, contracted whole renal arteries to a similar degree as the Kv7 inhibitor

269 Pure ethanol was then injected into the main renal artery to achieve complete arterial stasis.

270 01), and volume was measured from the lowest renal artery to the aortic bifurcation (P = .03) and to

271 tic aneurysm sac; and volume from the lowest renal artery to the aortic bifurcation and to the common

272 7%, respectively) and volume from the lowest renal artery to the common iliac artery bifurcation (57.

273 nterline diameter and volume from the lowest renal artery to the iliac bifurcation were the most sens

274 Arteriovenous grafts were created from the renal artery to the renal vein in 10 swine.

275 Twenty-five patients (50 renal arteries) underwent bilateral renal denervation wi

276 ephrectomy surgery without clamping the main renal artery/vein.

277 9%, for identification of variant anatomy of renal arteries, veins, and ureters, respectively.

278 ion and reduced myogenic reactivity of small renal arteries via the endothelial ETB receptor subtype.

279 Renal artery volume flow in patients with a duration of

280 stems are introduced, the incidence of acute renal artery wall injury with relation to the denervatio

281 opic projection (VIPR) MR angiography of the renal arteries was performed with a 1.5-T clinical MR sy

282 ignificant stenosis isolated to an accessory renal artery was 1.5% in our study.

283 The overall prevalence of calcium in either renal artery was 17.1%, with men having a significantly

284 ber of RF lesions (4, 8, and 12) in the main renal artery was not sufficient to yield a clear dose-re

285 asonographic flow probe encircling the right renal artery was surgically implanted in each pig to obt

286 y significant isolated stenoses of accessory renal arteries were calculated.

287 Bilateral renal arteries were collected from human autopsy subject

288 e, 0.64 second per section), the splenic and renal arteries were consecutively catheterized by using

289 Isolated rat renal arteries were cultured for 2 days in low K+ (4 mmo

290 The quality scores for the segmental renal arteries were higher for phase-contrast VIPR than

291 When small renal arteries were incubated with either of the general

292 Locking and standard clips applied to the renal artery were associated with the greatest risks.

293 acute in the ostium but holds throughout the renal artery, which requires further definition.

294 In 19 renal arteries with an average stenosis of 62% (range, 0

295 uthors successfully dilated nine (82%) of 11 renal arteries with MR guidance and all 11 arteries (100

296 e to endovascular radiofrequency ablation of renal arteries with nerve and ganglia distributions.

297 ins without stents; intimal hyperplasia in a renal artery with a stent was identified on 12 images.

298 Surgical reconstruction of the transplant renal artery with blood type-matched iliac artery grafts

299 antegrade VB perfusion of the kidney via the renal artery would restore urine output (UO) and glomeru

300 Histological analysis of renal arteries yielded a mean neurofilament score of hea