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1 gnosed as having TINU syndrome (confirmed by renal biopsy).
2 ith viremia without evidence of nephritis on renal biopsy.
3 propriate treatment and typically requires a renal biopsy.
4 tored by urine output, serum creatinine, and renal biopsy.
5 postprocedural hemorrhage after hepatic and renal biopsy.
6 ecipients, and definitive diagnosis requires renal biopsy.
7 urrent and de novo disease were diagnosed by renal biopsy.
8 rare finding of glomerular CMV inclusions on renal biopsy.
9 inal differentiation may be possible only by renal biopsy.
10 nt laboratory and US analysis at the time of renal biopsy.
11 ent and degree of glomerulonephritis seen on renal biopsy.
12 creatinine values obtained near the time of renal biopsy.
13 acute rejection in kidney transplants is the renal biopsy.
14 R potentially avoiding the need for invasive renal biopsy.
15 IGN in patients >/=65 years old diagnosed by renal biopsy.
16 sed or untreated BKN, which was confirmed by renal biopsy.
17 ithelial cells is commonly observed in human renal biopsies.
18 s found histologically in seven (9%) of open renal biopsies.
19 elated to the histopathologic changes in the renal biopsies.
20 All patients had baseline renal biopsies.
21 of six urinary cell samples, and two of four renal biopsies.
22 of six urinary cell samples, and two of four renal biopsies.
23 nction was monitored by serum creatinine and renal biopsies.
24 in most centers in the evaluation of native renal biopsies.
25 gnostic information in nearly half of native renal biopsies.
26 s, and renal function was performed, as were renal biopsies.
27 ormalin-fixed, paraffin-embedded tissue from renal biopsies.
28 and flare are defined and the role of repeat renal biopsies.
30 verity of glomerulosclerosis was assessed by renal biopsy 8 wk later, and rats were divided into four
33 routine use of electron microscopy in native renal biopsies also examined by immunofluorescence and r
35 was correlated with pathology in concomitant renal biopsies and BK viruria (decoy cell shedding and v
36 the largest study evaluated 213 consecutive renal biopsies and found that electron microscopy was ne
38 patients with lupus nephritis documented by renal biopsy and 26 with a history of lupus nephritis.
39 nce of peritubular capillary C4d staining on renal biopsy and donor-specific anti-human leukocyte ant
40 of polyomavirus infection was established by renal biopsy and EM of urine in five patients, by biopsy
43 uences of light chain GLA extracted from the renal biopsy and light chain CHO from myocardial tissue
45 -associated systemic vasculitis confirmed by renal biopsy and serum creatinine >500 micromol/L (5.8 m
46 mputed tomography (CT)-guided native medical renal biopsy and to evaluate its efficacy and safety com
47 ined as >/=10,000 viral copies/mL) underwent renal biopsy and treated with 30% to 50% reduction in do
49 e levels, increased interstitial fibrosis on renal biopsy, and increased fractional excretion of immu
50 osis of "osmotic nephrosis" was confirmed by renal biopsy, and the condition was reversed by cessatio
52 undant tubular calcium phosphate deposits on renal biopsy are referred to as nephrocalcinosis, a cond
55 dividuals with IgA nephropathy who underwent renal biopsy at our institution between 1973 and 1995.
56 From the total of patients, 151 underwent a renal biopsy because of renal dysfunction, whereas the 2
59 ommon with older age and is characterized on renal biopsy by global glomerulosclerosis, tubular atrop
63 ulointerstitial transcriptomes from protocol renal biopsy cores were analyzed for differential and co
66 us nephritis were identified from 5 sources: renal biopsy databases, dialysis/transplant databases, n
68 A consultation through the Internet after a renal biopsy demonstrated crescentic, necrotizing glomer
69 cted before the use of immunofluorescence in renal biopsy diagnosis became widespread and before seve
77 on of renal failure, glofil measurement, and renal biopsy findings, offers a practical approach to th
86 on of SGLT2 mRNA and protein is increased in renal biopsies from human subjects with diabetic nephrop
90 hropathy, an immunohistochemical analysis of renal biopsies from patients with diabetic nephropathy (
91 me-wide expression profiles of more than 200 renal biopsies from patients with different CKD stages r
94 tes from HIV-1 transgenic mice as well as in renal biopsies from patients with HIV-associated nephrop
99 ted in situ within endothelial cells both in renal biopsies from transplantation patients with chroni
102 In both patients, immunocytochemistry of renal biopsy frozen sections with an anti-H(+)-ATPase mo
104 ve polymerase chain reaction (PCR) assay for renal biopsy has not been evaluated as a diagnostic test
108 patterns of glomerulonephritis (GN) seen on renal biopsy impact upon the prognosis of these patients
110 staining that best predicts renal outcome in renal biopsies in a multicenter study in which local and
112 on in HSCT patients that can be diagnosed by renal biopsy in patients with hematuria and adenoviruria
113 rlying glomerular lesion, and therefore, the renal biopsy is an essential clinical tool in the approa
114 may provide clues to the presence of HCV-GD, renal biopsy is essential to differentiate HCV-GD from H
115 deposition or endocapillary proliferation on renal biopsy is more likely a manifestation of SLE than
121 d to renal tubular epithelial cell nuclei in renal biopsies of patients with FSGS by in situ hybridiz
124 dy, we show glomerular C5b-9 deposits in the renal biopsy of a child with EHEC-associated hemolytic u
125 ent with autosomal dominant transmission and renal biopsy of at least one individual showed C3 glomer
127 without COPD, 32 nonsmokers who underwent a renal biopsy or nephrectomy, and in CS-exposed mice, we
130 tions were reviewed in 431 CT-guided medical renal biopsies performed between July 2007 and September
132 ast 10 years of diabetes duration that had a renal biopsy performed for research purposes were studie
133 ncreased significantly (P <0.001) in a first renal biopsy performed within 3 months from transplantat
134 executor enzyme caspase-3 in preimplantation renal biopsies (PIB) as markers for delayed graft functi
140 ollow-up visits, emergency hospitalizations, renal biopsies, rejection episodes, renal function, and
142 iewed the posttransplant clinical course and renal biopsy results in 97 consecutive SLE patients who
143 ch, when applied to CT-guided native medical renal biopsies, results in higher rates of sample adequa
144 a known or suspected infectious process, and renal biopsies revealed an immune complex glomerulonephr
153 kidney, we performed microarray analyses of renal biopsy samples from patients with ANCA-associated
154 antibodies and glomerular target antigens in renal biopsy samples from patients with LN and determine
155 3.4 +/- 1.7 copies/cell) were observed in 74 renal biopsy samples from renal allograft recipients wit
156 pression and microRNA expression profiles in renal biopsy samples from tolerance-induced FCRx recipie
159 ents with BKV viruria, but 58 (50.4%) of 115 renal biopsy samples tested negative for BKV DNA, reflec
165 in this study is evaluation of surveillance renal biopsies (SB) and clinically indicated biopsies (C
167 rmed via the ipsilateral femoral vein with a renal biopsy set designed for transjugular renal biopsy
168 ed semiquantitative histologic evaluation of renal biopsies showed better preserved morphology in bot
169 acute deterioration in renal function whose renal biopsies showed typical viral cytopathic changes i
171 h donors had renal failure and pretransplant renal biopsies showing 100% of the glomeruli containing
172 n/day after transplantation (P=0.05) and had renal biopsies showing MPGN than did HCV- recipients (4/
176 ars) had pronounced cerebellar atrophy and a renal biopsy specimen that showed focal segmental glomer
179 nd inflammation classification, with >90% of renal biopsy specimens adequately classified by FTIR ima
180 nd validated a classification model using 49 renal biopsy specimens and subsequently tested the robus
181 increased IL-36alpha expression detected in renal biopsy specimens and urine samples from patients w
183 with biopsy specimens from control patients, renal biopsy specimens from 44 patients with acute AAV h
184 In conclusion, the AGN classification for renal biopsy specimens is a practical and informative sc
185 ased level of active TGF-beta1 expression in renal biopsy specimens of patients receiving CsA may ind
186 Snail/nephrin axis were similar to those in renal biopsy specimens of Zucker diabetic fatty rats and
189 nophenotyped the inflammatory infiltrates in renal biopsy specimens with BK polyomavirus-associated n
198 mononuclear cells (PBMC), urinary cells, and renal biopsy tissue was performed using specific primers
199 Shc in peripheral blood monocytes (PBMs) and renal biopsy tissues from DN patients and then analysed
202 m archived formalin-fixed, paraffin-embedded renal biopsies, until recently considered an unsuitable
208 pients, 126 protocol, serial, posttransplant renal biopsies were examined by centralized, blinded Ban
211 The most common pathological diagnoses on renal biopsies were membranoproliferative glomerulonephr
212 From January 1996 to June 1996, 288 native renal biopsies were received, and all were evaluated by
215 ipients without PVN on simultaneous protocol renal biopsy were analyzed by PCR; BKV genome was demons
218 and mass spectrometry in further evaluating renal biopsies when routine assessment fails to reach an
219 We evaluated 10 serial sections from 15 renal biopsies with a range of fibrosis extent and diagn
220 te rejection within the first 3 mo and had a renal biopsy with available frozen tissue at acute rejec
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