ライフサイエンスコーパス: renal blood flow

1 only seen with decreased cardiac output and renal blood flow.

2 re rapid dynamic alterations in differential renal blood flow.

3 ) during angiotensin II-induced reduction in renal blood flow.

4 cant change in glomerular filtration rate or renal blood flow.

5 closely related to discharge energy than is renal blood flow.

6 n in the CD that may contribute to decreased renal blood flow.

7 nal perfusion pressure but not of changes in renal blood flow.

8 mortality, impaired ventilation, and reduced renal blood flow.

9 erfusion pressure but poorly with changes in renal blood flow.

10 otection is partly related to maintenance of renal blood flow.

11 ns in methods for measuring rapid changes in renal blood flow.

12 istance blood vessels and failed to increase renal blood flow.

13 y and have been implicated in the control of renal blood flow.

14 c oxide synthase expressions correlated with renal blood flow.

15 y injury occurs in the presence of increased renal blood flow.

16 d flow, without changing portal pressure and renal blood flow.

17 largely because of a substantial increase in renal blood flow.

18 ery narrowness and can result to decrease in renal blood flow.

19 extracellular volume depletion or decreased renal blood flow.

20 evosimendan induced significant increases in renal blood flow (12%, p<0.05) and glomerular filtration

21 t (2.1+/-0.2 to 2.3+/-0.2 L/min; P<0.05) and renal blood flow (131+/-17 to 162+/-18 mL/min; P<0.05) i

22 differ, there was a significant decrease of renal blood flow 2 hr after the intake of CsA compared w

23 nterval] 0.51 [0.28-0.92]) despite increased renal blood flow (241 to 343 mL/min, difference [95% con

24 7 versus 57 +/- 11 mL/min, P < or = 0.0001), renal blood flow (3.4 +/-0.7 versus 8.4 +/- 1.9 mL/min L

25 d animals, ANG II produced a 40% decrease in renal blood flow, a level between untreated dietary grou

26 gonist of the dopamine-1 receptor, preserves renal blood flow after iodinated contrast administration

27 ericytes in vitro Initial studies monitoring renal blood flow after IRI did not find significant effe

28 There was no significant change in mean renal blood flow after PTA (P=.44).

29 of renal hypoxia and partial restoration of renal blood flow after revascularization, inflammatory c

30 After UNX, renal blood flow and amino acid delivery to the remainin

31 st magnetic resonance imaging measurement of renal blood flow and cardiac output.

32 in mild CHF in association with increases in renal blood flow and decreases in renal vascular resista

33 usion preserves spinal cord, mesenteric, and renal blood flow and eliminates the potential catastroph

34 y allows for enhanced resolution of regional renal blood flow and functional evaluations in patients.

35 Given the differences in renal blood flow and GFR in control and postischemic kid

36 rom 141 +/- 3 (SE) mm Hg to 101 +/- 2 mm Hg; renal blood flow and GFR were not significantly altered

37 sodium excretion; however, the increases in renal blood flow and GFR were not sustained as systemic

38 re characterized by progressive reduction in renal blood flow and glomerular filtration rate and show

39 Reductions in renal blood flow and glomerular filtration rate are unde

40 Renal blood flow and glomerular filtration rate exhibite

41 Renal blood flow and glomerular filtration rate were mea

42 omerular resistance vessels, increasing both renal blood flow and glomerular filtration rate without

43 I/R markedly depressed renal blood flow and increased the production in O2, PGE

44 on was evaluated by parallel measurements of renal blood flow and inulin clearance.

45 Renal blood flow and mean arterial pressure responses to

46 l hypertension, and a progressive decline in renal blood flow and renal function.

47 de plays an important role in the control of renal blood flow and renal function.

48 ith renal vasoconstriction, but no change in renal blood flow and substantial femoral vasodilatation

49 on during endotoxic shock actually increases renal blood flow and that this effect is not the result

50 ay be a valuable tool to further investigate renal blood flow and the effects of therapies on renal b

51 The mean renal blood flow and total urine output were 68.0 mL/min

52 AM reflect unique actions of the peptide on renal blood flow and tubular function.

53 ic volume expansions reversed the changes in renal blood flow and urine output, but impaired creatini

54 scored based on the macroscopic appearance, renal blood flow and urine output.

55 We developed a technique for measuring renal blood flow and used a pig model to determine wheth

56 a role in reversing LPS-induced decreases in renal blood flow and volume, although the effects on PAR

57 en demand while lower extremity, splanchnic, renal blood flows and arteriovenous oxygen content diffe

58 ilability in renal vascular cells, augmented renal blood flow, and decreased systemic blood pressure

59 e imaged with a 1.5-T imager to estimate EF, renal blood flow, and glomerular filtration rate.

60 es systemic blood pressure, reduces regional renal blood flow, and increases platelet counts and proc

61 zed sheep, decrements in hemodynamic status, renal blood flow, and kidney function incurred following

62 on of renal microvasculature, improvement in renal blood flow, and less tissue hypoxia than TbetaRII(

63 rapidly decreases blood pressure, increases renal blood flow, and maintains or improves the glomerul

64 nsion would reduce portal pressure, increase renal blood flow, and produce insignificant changes in a

65 ynamic parameters, including cardiac output, renal blood flow, and vascular resistance.

66 y renal nerve stimulation, the reductions in renal blood flow at each stimulation frequency were grea

67 has been shown under conditions of efficient renal blood flow autoregulation.

68 ributes importantly to the early decrease in renal blood flow (basal versus handgrip, 4.2 +/- 0.2 ver

69 boreflex contributes to further decreases in renal blood flow (basal versus posthandgrip circulatory

70 Obstruction of renal blood flow before and during RF ablation resulted

71 AKI), but there were no changes over time in renal blood flow between groups (P > 0.30) or over time

72 model, NE was associated with an increase in renal blood flow both before and after endotoxin adminis

73 BQ-123 increased effective renal blood flow (BQ-123, -0.1 +/- 2.4%; BQ-123+E, 10.9

74 in normotensive F2s (n = 3) without altering renal blood flow but was inactive in hypertensive F2s (n

75 and a high dose of aminoguanidine normalized renal blood flow, but did not alter creatinine clearance

76 Sepsis increased cardiac output by 60%, renal blood flow by 35%, and arterial lactate by approxi

77 mg kg-1 bis in die (b.i.d.)) rats decreased renal blood flow by 46 and 29 % (both P < 0.001), respec

78 0 mg/kg) to untreated control mice increased renal blood flow by 55% (from 1.8+/-0.2 to 2.8+/-0.2 ml/

79 ute extracellular volume expansion increased renal blood flow by 84% and reduced renal vascular resis

80 ors in VSMCs are essential for regulation of renal blood flow by Ang II and highlight the capacity of

81 on of liver disease and portal hypertension, renal blood flow declines because of the hepatorenal ref

82 Renal blood flow decreased after reperfusion but was not

83 rotid and iliac circulations, mesenteric and renal blood flows decreased markedly.

84 sartan IC50 values for percentage changes in renal blood flow did not differ in the two groups of mic

85 njection of S1P in anesthetized rats reduced renal blood flow dose dependently.

86 of lipid peroxidation by U74389G maintained renal blood flow during acute CsA administration.

87 anced harmonic US imaging depicts changes in renal blood flow during acute obstruction.

88 hyperspectral imaging facilitates monitoring renal blood flow during animal surgery and holds conside

89 formed to clarify the mechanisms controlling renal blood flow during static exercise.

90 c, diuretic, glomerular filtration rate, and renal blood flow enhancing actions than native ANP in vi

91 uretic, with glomerular filtration rate- and renal blood flow-enhancing actions.

92 Sildenafil improved the renal blood flow for the first 30 min in the 2-hr group

93 ecule that participates in the regulation of renal blood flow, GFR, and mesangial matrix accumulation

94 We assessed BP, urinary protein, stenotic renal blood flow, GFR, microvascular structure, and oxyg

95 que and complex functional interplay between renal blood flow, GFR, O2 consumption, and arteriovenous

96 ach nephron is crucial for the regulation of renal blood flow, GFR, urine concentration, and other sp

97 al artery stenosis (ARAS) is known to reduce renal blood flow, glomerular filtration rate (GFR) and a

98 enous infusion of Hcys was found to decrease renal blood flow, glomerular filtration rate, and sodium

99 ore evaluated the effects of levosimendan on renal blood flow, glomerular filtration rate, renal oxyg

100 and generate paracrine signals that control renal blood flow, glomerular filtration, and release of

101 to control vital kidney functions, including renal blood flow, glomerular filtration, and renin relea

102 total kidney volume and decreases in GFR and renal blood flow greater than expected for a given age a

103 a high-salt diet, Tmem27(Y/-) mice had lower renal blood flow, higher abundance of renal sodium-hydro

104 hed nitric oxide bioavailability and reduced renal blood flow; however, the mechanisms leading to the

105 Fenoldopam increased renal blood flow in a dose-dependent manner compared wit

106 aluate rapid dynamic changes in differential renal blood flow in an experimental animal model.

107 in urinary sodium excretion, urine flow and renal blood flow in association with reductions in cardi

108 low in wild-type mice and an 8% reduction of renal blood flow in AT1A receptor-knockout mice.

109 ent, AngII (2 ng) produced 40% reductions in renal blood flow in both rat strains, without affecting

110 cantly reduced the effects of vasopressin on renal blood flow in control but not in endotoxemic rats.

111 l blood flow and the effects of therapies on renal blood flow in critical illness.

112 pmol/kg, IV) produced increased reduction in renal blood flow in endotoxemic rats.

113 increases in oxygen demand on splanchnic and renal blood flow in hemorrhaged dogs.

114 ninvasive method for the mapping of regional renal blood flow in humans using PET and H(2)(15)O.

115 The decrease in renal blood flow in patients with heart failure was simi

116 nic saline (7.5% NaCl) solution may maximize renal blood flow in prolonged pneumoperitoneum, but it d

117 ught to overcome such obstacles by measuring renal blood flow in septic patients with acute kidney in

118 on pressure of perfused kidneys in vitro and renal blood flow in situ were evaluated.

119 However, the accurate measurement of renal blood flow in such patients is problematic and inv

120 in a greater transfer of input signals into renal blood flow in the 0.1 to 1.0 Hz range.

121 more, laser Doppler assessment showed higher renal blood flow in the CD47mAb-treated kidneys.

122 renal AngII (1 ng) caused a 32% reduction of renal blood flow in wild-type mice and an 8% reduction o

123 Renal blood flow increased with F+T but this was not sig

124 reduction in glomerular filtration rate and renal blood flow, increased renal cytokine expression, a

125 st dose (i.e., 0.03 ILg/kg/min), significant renal blood flow increases occurred without changes in s

126 There was no rank correlation between renal blood flow index and creatinine clearance in patie

127 Renal blood flow indexed to body surface area was 244 mL

128 ow doses (0.004 to 0.02 units/min) increased renal blood flow (indicator-dilution technique), reduced

129 Because the renal blood flow is not affected by the increased cardia

130 Renal blood flow is often reduced in patients with chron

131 increased arterial pressure without reducing renal blood flow, leading to an improved renal function.

132 (maximal reduction 55+/- 6%) and increase in renal blood flow (maximal increase 136 +/- 54%).

133 of the renal arteries provided quantitative renal blood flow measurements.

134 roups in systemic vascular resistance index, renal blood flow, mesenteric blood flow, systemic oxygen

135 there was a significant correlation between renal blood flow obtained before drug administration and

136 at the lowest dose, significantly increased renal blood flow occurred without changes in systemic bl

137 longed CO2 pneumoperitoneum caused decreased renal blood flow, oliguria, and impaired creatinine clea

138 ycardia, increased cardiac output, increased renal blood flow, oliguria, decreased creatinine clearan

139 nthase inhibitor did not reduce the elevated renal blood flow or improve renal function.

140 d water retention caused by abnormalities of renal blood flow, or as a hemodynamic problem associated

141 early AKI was not associated with changes in renal blood flow, oxygen delivery, or histological appea

142 models with low cardiac output and decreased renal blood flow (p < 0.0001).

143 th high-sodium diet (16% vs. 56% decrease in renal blood flow, P < 0.001).

144 leads to relative hypovolemia and decreased renal blood flow, patients with decompensated cirrhosis

145 Pre-stent single kidney renal blood flow, perfusion, and GFR were reduced in the

146 ontrol rats, there was a 10-fold increase in renal blood flow power over the frequency range of 0.01

147 Although this did not affect the renal blood flow power spectrum in control rats, there w

148 d that is fully tissue protective, increases renal blood flow, promotes sodium excretion, reduces inj

149 crobubbles and ultrasonic flow probe-derived renal blood flow (r = 0.82, p < 0.001) over a wide range

150 in significantly higher cardiac outputs and renal blood flow rates in treated animals compared with

151 therosclerotic renal artery stenosis reduces renal blood flow (RBF) and amplifies stenotic kidney hyp

152 GMPV), glomerular filtration rate (GFR), and renal blood flow (RBF) and decreased distal fractional s

153 sclerotic renovascular disease (RVD) reduces renal blood flow (RBF) and GFR and accelerates poststeno

154 oconstrictors contributes to the decrease in renal blood flow (RBF) and GFR observed during LPS-induc

155 Basal renal blood flow (RBF) and glomerular filtration rate (G

156 mboxane A2 (TxA(2)) may mediate decreases of renal blood flow (RBF) and/or GFR associated with LPS-in

157 rmed in anesthetized dogs (n = 9) to examine renal blood flow (RBF) autoregulatory efficiency before

158 vely evaluate the feasibility of determining renal blood flow (RBF) by using a technique based on int

159 e myogenic response and the TGF mechanism in renal blood flow (RBF) control at the very earliest stag

160 mean arterial pressure (MABP), no change in renal blood flow (RBF) due to an increase in renal vascu

161 decreased by 50% after infusion of AngII and renal blood flow (RBF) fell by 3.3 ml min(-1) .

162 Renal blood flow (RBF) is often reduced in patients with

163 This rat renal blood flow (RBF) study quantified the impact of ni

164 hrectomy results in an immediate increase in renal blood flow (RBF) to the remnant kidney, followed b

165 Renal blood flow (RBF) was measured using an ultrasonic

166 reperfusion, plasma [ET-1] increased 66% and renal blood flow (RBF) was reduced by 38% compared with

167 II (ANGII) on the dynamic characteristics of renal blood flow (RBF) was studied in conscious dogs by

168 Hippuran clearance and renal blood flow (RBF) were measured twice, before and a

169 owed by decreases in mean arterial pressure, renal blood flow (RBF), and renal capillary perfusion at

170 ature, participates in the autoregulation of renal blood flow (RBF), but the underlying mechanisms ar

171 Renal blood flow (RBF), mean arterial pressure (MAP), an

172 +/-7 mmHg; the control decreases in cortical renal blood flow (RBF), measured with laser Doppler flow

173 But arterial pressure (ABP), renal blood flow (RBF), renal vascular conductance (RVC)

174 LPS on glomerular filtration rate (GFR) and renal blood flow (RBF).

175 olume, glomerular filtration rate (GFR), and renal blood flow (RBF).

176 sure (MABP, Delta = +18 to 26 mmHg), reduced renal blood flow (RBF, Delta = -1.8 to 2.9 ml min(-1)),

177 icantly improved renal function by restoring renal blood flow, reducing nicotinamide adenine dinucleo

178 Renal blood flow remained unaffected by all treatment fo

179 Renal blood flow response to Ach was blunted in pigs tha

180 The aim of this study was to determine the renal blood flow response to static exercise in healthy

181 (by electromagnetic or ultrasonic flowmetry) renal blood flow responses to AngII in rats and mice.

182 was assessed in vivo by measuring transient renal blood flow responses to bolus injections of ANG II

183 After an ischemic insult, total renal blood flow returns toward normal, but marked, regi

184 Renal blood flow seems consistently reduced as a fractio

185 ptors elicited significant increases in GFR, renal blood flow, sodium excretion, and fractional sodiu

186 hase and neuronal nitric oxide synthase with renal blood flow suggest in this experimental model that

187 e of an associated low cardiac output or low renal blood flow syndrome.

188 patorenal syndrome may improve by increasing renal blood flow through the use of vasoconstrictors (va

189 and high ureteral pressure reduced cortical renal blood flow to 88% and 66%, respectively, of baseli

190 ies and may play a role in the regulation of renal blood flow under physiological and patho-physiolog

191 Infusion of 1400W did not change renal blood flow, urine output, or creatinine clearance,

192 irst described in the 19th century, and GFR, renal blood flow, urine production, and electrolyte excr

193 le renal resistance increased moderately and renal blood flow usually was maintained above control le

194 nder this normalized curve was compared with renal blood flow values.

195 nt signaling (including pain), regulation of renal blood flow, vascular endothelium, and inflammatory

196 Beat-to-beat changes in renal blood flow velocity (RBV; Duplex Ultrasound), mean

197 e 148 ([Cl] 98 mmol/L, Baxter Healthcare) on renal blood flow velocity and perfusion in humans using

198 ter Healthcare, Thetford, United Kingdom] on renal blood flow velocity and renal cortical tissue perf

199 sion of 0.9% saline results in reductions in renal blood flow velocity and renal cortical tissue perf

200 s measured with intravascular ultrasound and renal blood flow velocity with the aid of an intravascul

201 ), 289 (171-477), and 70 (51-91) mumol/L and renal blood flow was 270 +/- 42, 653 +/- 210, and 250 +/

202 Median renal blood flow was 482 mL/min (range 335-1137) in sept

203 The mean renal blood flow was 93.6 mL/min/100 g and the kidney pr

204 The increased renal blood flow was accompanied by more rapid intrarena

205 Renal blood flow was determined with a flowprobe, and th

206 Renal blood flow was determined with a flowprobe.

207 Renal blood flow was gradually increased from 5 min to 3

208 s 1.4 +/- 0.1 ml/min), and autoregulation of renal blood flow was maintained to a pressure level of a

209 Renal blood flow was measured by dynamic positron emissi

210 Renal blood flow was measured in all patients to calcula

211 Cerebral and renal blood flow was measured with colored microspheres.

212 Renal blood flow was measured with electron-beam compute

213 Total renal blood flow was not different between UT-A1/3(-/-)

214 At comparable levels of arterial BP, renal blood flow was not significantly different between

215 cant change in glomerular filtration rate or renal blood flow was observed.

216 In contrast, renal blood flow was reduced by infusion of a nonselecti

217 Basal renal blood flow was similar among the groups, whereas G

218 Hg higher and glomerular filtration rate and renal blood flow were approximately 30% lower (P<0.001)

219 ar filtration rate, extraction fraction, and renal blood flow were assessed during PTA.

220 l perfusion, glomerular filtration rate, and renal blood flow were calculated.

221 oups, whereas glomerular filtration rate and renal blood flow were decreased less in uni-x sheep (PIn

222 med, and interlobar resistive index (RI) and renal blood flow were determined at baseline and during

223 al pressure, glomerular filtration rate, and renal blood flow were measured before and during NO inhi

224 n arterial pressure, forearm blood flow, and renal blood flow were measured during mental stress test

225 le kidney cortical, medullary perfusion, and renal blood flow were measured using multidetector compu

226 ood pressure, blood and urine chemistry, and renal blood flow were not different between e-5'NT/CD73(

227 reflex renal vasoconstriction and decreased renal blood flow, which may implicate endogenous adenosi

228 els dilate, causing increased filtration and renal blood flow with decreased vascular resistance as a

229 etermine a dose of fenoldopam that increases renal blood flow without inducing hypotension in normote

230 hen determined: glomerular filtration rate = renal blood flow x (1 - hematocrit level) x EF.