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1 kidney cancer (hereditary leiomyomatosis and renal cell cancer).
2 ients with metastatic malignant melanoma and renal cell cancer.
3 and lymphoid malignancies and in metastatic renal cell cancer.
4 romosome 10q deletion in primary bladder and renal cell cancer.
5 lected patients with metastatic melanoma and renal cell cancer.
6 on itself, may contribute to the etiology of renal cell cancer.
7 metastatic malignant melanoma or metastatic renal cell cancer.
8 Odds ratios were not elevated for renal cell cancer.
9 mas as well as hereditary leiomyomatosis and renal cell cancer.
10 th melanoma, non-small-cell lung cancer, and renal cell cancer.
11 ond-line therapy in patients with metastatic renal cell cancer.
12 st, lung, ovarian, pancreatic, prostate, and renal cell cancer.
13 ts and therefore may also reduce the risk of renal cell cancer.
14 lescence, is associated with reduced risk of renal cell cancer.
15 cence were associated with a reduced risk of renal cell cancer.
16 for human bladder, head and neck, lung, and renal cell cancer.
17 ition syndrome hereditary leiomyomatosis and renal cell cancer.
18 significant activity of PS-341 in metastatic renal cell cancer.
19 e inhibitor PS-341 in patients with stage IV renal cell cancer.
20 ydratase cause hereditary leiomyomatosis and renal cell cancer.
21 myoma syndrome (MCL) and MCL associated with renal cell cancer.
22 cated as risk factors for the development of renal-cell cancer.
23 ith non-small-cell lung cancer, melanoma, or renal-cell cancer.
24 nhibitors approved for treatment of advanced renal-cell cancer.
25 ng non-small-cell lung cancer, melanoma, and renal-cell cancer.
26 ssion of disease in patients with metastatic renal-cell cancer.
27 n 65 of 285 (23%) bladder and 15 of 60 (25%) renal cell cancers.
28 herin-6 expression in a series of 32 primary renal cell cancers.
29 mary head and neck, non-small cell lung, and renal cell cancers.
30 odels and in some patients with melanoma and renal cell cancers.
31 melanoma, 18 with colorectal cancer, 17 with renal-cell cancer, 17 with ovarian cancer, 14 with pancr
33 9 of 52 patients with melanoma, 2 of 17 with renal-cell cancer, 5 of 49 with non-small-cell lung canc
37 y, further etiologic research is needed into renal cell cancer, an increasingly common form of cancer
38 isk of upper urinary tract cancer, including renal cell cancer and cancers of the renal pelvis or ure
39 udies in untreated patients with melanoma or renal cell cancer and in other rhIL-12-responsive malign
40 mutation in von Hippel-Lindau (VHL) develop renal cell cancers and hypervascular tumors of the brain
43 or former smokers had a greater risk of both renal-cell cancer and renal-pelvis cancer than men who w
44 nal stromal tumours, neuroendocrine cancers, renal-cell cancer and sarcoma is associated with longer
45 derlying malignancy (renal cell cancer v non-renal cell cancer) and TKI (sunitinib v sorafenib), but
46 substantially lower the overall incidence of renal cell cancer, and 2) intervention measures may have
48 ghths had a 30 to 60 percent greater risk of renal-cell cancer, and men in the highest two eighths ha
51 ed for treatment of patients with metastatic renal-cell cancer, but no validated biomarkers are avail
53 de that survival in patients with metastatic renal cell cancer can be correlated with the expression
54 rate among blacks compared with whites with renal cell cancer can be explained largely by the increa
56 thors present estimates of the proportion of renal cell cancer cases attributable (or population attr
59 l common malignancies, for example colon and renal cell cancer, code for ubiquitin ligase components.
62 ts, 21 of 23 with melanoma and 34 of 38 with renal cell cancer developed at least PRs after the first
63 open radical nephrectomy for the majority of renal cell cancers due to equivalent oncologic control,
64 d a unique association of MN expression with renal cell cancers, especially those of the clear cell v
65 patient with melanoma, and one patient with renal cell cancer had complete regression of pulmonary m
71 mata (MCL) and hereditary leiomyomatosis and renal cell cancer (HLRCC), and recently, FH mutations ha
72 a rare cancer, hereditary leiomyomatosis and renal cell cancer (HLRCC), characterised by benign smoot
73 CUL1), and the hereditary leiomyomatosis and renal cell cancer (HLRCC), implicated FH, a gene on chro
74 FH predispose individuals to leiomyomas and renal cell cancer (HLRCC), whereas mutations in SDH caus
77 ar relative survival rates for patients with renal cell cancer improved among whites but not among bl
78 rs examined physical activity in relation to renal cell cancer in a large, prospective US cohort stud
80 ypertensive medications to the risk of fatal renal cell cancer in a prospective cohort study of 998,9
83 uting to the rapidly increasing incidence of renal cell cancer in the United States, particularly amo
88 roscopic retroperitoneal lymphadenectomy for renal cell cancer is safe and allows for adequate stagin
89 itors (SMIs) are commonly used in metastatic renal cell cancer (mRCC) patients; patients often develo
90 also known as hereditary leiomyomatosis and renal cell cancer), NRF2 activation is a direct conseque
92 higher blood pressures and a higher risk of renal-cell cancer (P for trend, <0.001 for diastolic pre
93 n of the Fas(CD95)-R expression on PBLs from renal cell cancer patients compared with normal healthy
100 Genome-wide association studies (GWASs) of renal cell cancer (RCC) have identified four susceptibil
103 overall survival of patients with metastatic renal cell cancer (RCC) receiving high-dose or one of tw
106 we also performed a comparison analysis with renal cell cancer (RCC)) studies that evaluated sorafeni
107 ast cancer, prostate cancer, bladder cancer, renal cell cancer (RCC), and malignant melanoma data in
108 ile of genes silenced by hypermethylation in renal cell cancer (RCC), we did an expression microarray
113 pared the transcriptional profile of primary renal cell cancers (RCCs) with that of normal kidney tis
114 s mDC of patients with advanced melanoma and renal cell cancer reduced the activation and Th1 cytokin
115 nce for aldosterone serving a causal role in renal cell cancer regulation via its GPER receptor; thus
116 a (relative risk 3.10, 95% CI 1.26-7.63) and renal cell cancer (relative risk 5.67, 0.66-48.42).
118 tivariate Cox regression models adjusted for renal cell cancer risk factors, the authors observed tha
120 size, lifestyle, and medical conditions with renal cell cancer risk was examined among 161,126 Hawaii
123 nger survival among patients with metastatic renal-cell cancer than does interferon therapy alone.
124 on-free survival in patients with metastatic renal cell cancer; the phase II data were confirmed in a
125 ith non-small-cell lung cancer, melanoma, or renal-cell cancer; the adverse-event profile does not ap
126 ccumulation in hereditary leiomyomatosis and renal cell cancer tumors is thought to result from fumar
127 so stratified for the underlying malignancy (renal cell cancer v non-renal cell cancer) and TKI (suni
128 A population-based case-control study of renal cell cancer was conducted in Minnesota between 198
129 PET/CT imaging of 10 subjects with stage IV renal cell cancer was performed after intravenous admini
130 effects of preclinical disease, the risk of renal-cell cancer was still consistently higher in men w
133 f immunotherapeutic approaches in metastatic renal cell cancer, which have produced a consistent demo
134 Most patients with metastatic melanoma and renal cell cancer who achieved PRs or CRs to intravenous
136 patients with either metastatic melanoma or renal cell cancer who were treated with high-dose bolus
137 ates were higher in patients with metastatic renal-cell cancer who received sunitinib than in those r
138 e randomly assigned patients with metastatic renal-cell cancer who were acceptable candidates for nep
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