ライフサイエンスコーパス: renal cortex

1 -inducible factor-1alpha upregulation in the renal cortex.

2 mouse renal inner medulla but much lower in renal cortex.

3 d only NADH-dependent O(2)(.-) generation by renal cortex.

4 epithelial cells cultured from normal human renal cortex.

5 higher values in regions composed largely of renal cortex.

6 lack of terminal branches in the midventral renal cortex.

7 ding protein from a cytosolic extract of rat renal cortex.

8 and degradation of nitric oxide (NO) in the renal cortex.

9 hione and accumulation of malondialdehyde in renal cortex.

10 ased AQP2 protein expression particularly in renal cortex.

11 tion of inducible NO synthase protein in the renal cortex.

12 is indicated that PTP-1D is expressed in the renal cortex.

13 fluorescence was used to detect gamma in rat renal cortex.

14 ted in marked homogeneous enhancement of the renal cortex.

15 ase-2 (COX-2) at restricted sites in the rat renal cortex.

16 be located to the tubular epithelium of the renal cortex.

17 nal outer stripe of the outer medulla and/or renal cortex.

18 d to proximal tubule epithelial cells in the renal cortex.

19 ding type IV collagen and fibronectin in the renal cortex.

20 l membrane vesicles isolated from the rabbit renal cortex.

21 n apical membrane vesicles isolated from rat renal cortex.

22 nephrons to cortical collecting ducts in the renal cortex.

23 accumulation of type III collagen within the renal cortex.

24 odialysis catheter was placed in the lateral renal cortex.

25 sured levels of HO-1 mRNA and protein in the renal cortex.

26 hment of their corresponding antigens in the renal cortex.

27 lateral membranes of proximal tubules in the renal cortex.

28 fewer CD3(+) and proliferating cells in the renal cortex.

29 m normal renal cortex and abundant in Alport renal cortex.

30 specific binding in the pancreas than in the renal cortex.

31 had evidence for increased apoptosis in the renal cortex.

32 me cytokines were increased significantly in renal cortex.

33 led DOTA-biotin localized selectively in the renal cortex.

34 are identified in the outer medulla and the renal cortex.

35 FC was minimally elevated in renal cortex (0 to 15%), the majority apparently localiz

36 dulla, 1702 msec +/- 205 and 60 msec +/- 21; renal cortex, 1314 msec +/- 77 and 47 msec +/- 10; splee

37 ammonium sulfate-mediated injury; and 3) in renal cortex 18 hours after induction of glycerol-induce

38 m (93%), cerebrum (183%), brain stem (177%), renal cortex (53%), ileal mucosa (69%), gastric antral m

39 f angiotensin converting enzyme (ACE) in the renal cortex (942 +/- 130 versus 464 +/- 60 arbitrary pi

40 wise, had a strong positive correlation with renal cortex ACE protein expression (170-kDa band) (r =

41 In renal cortex, ACE2 activity was increased in both models

42 icromol Pi/mg protein per h while decreasing renal cortex activity from 10.9 +/- 0.9 to 6.5 +/- 0.7.

43 analyzed, we identified 6 as enriched in the renal cortex and 11 in the renal medulla.

44 entified 58 proteins as more abundant in the renal cortex and 72 in the renal medulla.

45 sent in the vascular endothelium from normal renal cortex and abundant in Alport renal cortex.

46 ediates initial trafficking to the brain and renal cortex and contributes to fungal persistence in th

47 Renal cortex and cultured GEnC were examined by microsco

48 Human renal cortex and heart cDNA libraries were screened for

49 Biopsies were obtained from renal cortex and hybridized to Affymetrix HG-U133A GeneC

50 ormed in four regions of interest within the renal cortex and in three regions within the medulla.

51 rt the hypothesis that CAA is accumulated in renal cortex and is responsible for nephrotoxicity.

52 Both renal cortex and liver have similar developmental patter

53 Renal cortex and medulla AA metabolism were not signific

54 he expression of these genes was analyzed in renal cortex and medulla after ischemic-reperfusion inju

55 ide wasting, we analyzed their expression in renal cortex and medulla of animals placed on KD diet.

56 The non-heme iron content both in the renal cortex and medulla of Heph/Cp KO mice was signific

57 Mean ADCs of renal cortex and medulla were significantly higher in gr

58 ver lobe, gallbladder, pancreas, spleen, and renal cortex and medulla).

59 ulmonary and renal artery flow probes in the renal cortex and medulla, combination fiber-optic probes

60 ons at 3.0 T (left liver lobe, pancreas, and renal cortex and medulla, P < .008), intervendor differe

61 cCD8alpha and in CD11cCD8alpha DC within the renal cortex and medulla.

62 eys with regions of interest assigned to the renal cortex and medulla.

63 ed a major 30-kDa band in membranes from rat renal cortex and medulla.

64 Src, and tyrosine phosphorylation of ROMK in renal cortex and outer medulla in wild-type (WT) mice.

65 Immunoprecipitation of tissue lysates from renal cortex and outer medulla or 293T cells transfected

66 mmunoprecipitation of proteins obtained from renal cortex and outer medulla with ROMK antibody reveal

67 the tyrosine phosphorylation of ROMK in the renal cortex and outer medulla.

68 s abundant CYP2J5 mRNA within tubules of the renal cortex and outer medulla.

69 sociation between c-Src and RPTPalpha in the renal cortex and outer medulla.

70 hosphorylation of c-JUN N-terminus kinase in renal cortex and outer medulla.

71 ylation of c-Jun, a transcription factor, in renal cortex and outer medulla.

72 On the basis of renographic findings, renal cortex and renal medulla enhancement curves and no

73 f the liver and relatively low (<10%) in the renal cortex and renal medulla.

74 Although found at high level in liver, renal cortex and small intestine, fructokinase activity

75 tes a gradient of oxygen tension between the renal cortex and the papillary tip that results in a sta

76 l profile of microRNA expression between the renal cortex and the renal medulla and greatly expands t

77 ed by Rho-kinase, were increased in both the renal cortex and the renal medulla of endotoxemic rats.

78 Time-activity curves were derived from the renal cortex and were analyzed by the Gjedde-Patlak plot

79 p47(phox), Nox2, and Nox4 mRNA levels in the renal cortex and were unchanged in diabetic PKC-beta(-/-

80 xylases was localized in renal microvessels, renal cortex, and a striated muscle microvascular bed (c

81 osis with hemorrhage of both the adrenal and renal cortex, and an intense influx of neutrophils into

82 s of the liver, and in the pancreas, spleen, renal cortex, and renal medulla.

83 diac tissue, ACE2 activity was lower than in renal cortex, and there were no significant differences

84 dopamine-derived radioactivity in the heart, renal cortex, and thyroid gland but not in the liver, sp

85 alizes sympathetic innervation in the heart, renal cortex, and thyroid gland.

86 thelial cells of the liver, lung, intestine, renal cortex, and urinary tract.

87 isplaceable uptake, was calculated using the renal cortex as a reference tissue devoid of specific VM

88 BP(ND)) was estimated voxelwise by using the renal cortex as reference tissue.

89 PLA2 activity was increased in the renal cortex as well as in the renal medulla.

90 lant, which revealed multiple lesions in the renal cortex as well as liver and spleen.

91 the proximal convoluted tubules of the outer renal cortex, assessed by Western blotting and immunohis

92 165-kDa band in membrane fractions from the renal cortex but not from the renal medulla.

93 restricted to stromal cells in the embryonic renal cortex, but it mediates its effects on the adjacen

94 ays indicated that cytosolic extracts of rat renal cortex contain a protein that binds to the R-2 and

95 hibitor of matrix degradation, PAI-1, in the renal cortex, contributing to significant reversal of me

96 ACE2 activity in renal cortex correlated positively with ACE2 protein in

97 We have previously shown that in renal cortex, COX-2 expression is localized to macula de

98 We have previously shown that in rat renal cortex, cyclooxygenase-2 (COX-2) expression is loc

99 Both renal neoplasms and normal renal cortex demonstrated significantly greater enhancem

100 njury and formation of giant vacuoles in the renal cortex, die from renal failure, a phenotype that r

101 hat are rapidly filtered in the kidneys, the renal cortex dose is approximately one-half of that pred

102 ults demonstrate a desensitization of sGC in renal cortex during endotoxemia, which may contribute to

103 hase subtypes increased significantly in the renal cortex during septic acute kidney injury but tende

104 dative stress promotes NO degradation in the renal cortex during the early stage of diabetes mellitus

105 itionally, FC/CE levels were measured in rat renal cortex either 10 days after CSA or tacrolimus ther

106 inal vesicles, pituitary, thyroid, pancreas, renal cortex, enteric epithelium, muscles, myocardium an

107 For the adult, the absorbed dose to the renal cortex for (90)Y-labeled compounds retained within

108 nterstitial monocytes that accumulate in the renal cortex from Alport mice are immunopositive for int

109 NADPH-dependent superoxide production in the renal cortex from Asm(+/+) mice compared with that from

110 ACE2 mRNA levels in renal cortex from db/db and STZ-induced diabetic mice, b

111 beta1 subunits, is increased in glomeruli or renal cortex from diabetic animals or in mesangial cells

112 We conclude that in renal cortex from diabetic mice, ACE2 expression is incr

113 levels or their distribution were evident in renal cortex from diabetic rats.

114 y revealed an approximately 70-kD protein in renal cortex from sham rats, the nitrotyrosine content o

115 erns for the gluconeogenic enzymes, although renal cortex generally shows greater activity than liver

116 a global downregulation of microRNAs in the renal cortex; however, these animals were remarkably res

117 Renal cortex immunoblot analysis using anti-peptide anti

118 l moving blood volume estimates of the right renal cortex in a volunteer when simulating different bo

119 pressed in the differentiation domain of the renal cortex in an overlapping manner with the vasopress

120 inant fibroblast chemoattractant produced by renal cortex in anti-GBM disease.

121 situ hybridization localized the mRNA to the renal cortex in both mice and confirmed equal message le

122 bular glomeruli by electrocoagulation of the renal cortex in mice.

123 eeks of age and vacuolization throughout the renal cortex in older mice.

124 endothelin-1 (ET-1) and ET receptors in the renal cortex in short-term diabetes.

125 eceptor, and signaling levels throughout the renal cortex in this animal model of type 2 diabetes.

126 onstrated in tubular epithelial cells in the renal cortex, in spermatogenic cells in the testis, and

127 fibronectin protein was markedly reduced in renal cortex including glomeruli of AS-treated diabetic

128 reases CD3(+) cells and proliferation in the renal cortex independent of changes in BP, but changes i

129 tored interleukin-6 to control levels in the renal cortex, indicating the protective effects on the p

130 FC and CE levels in renal cortex, isolated glomeruli, and proximal tubule se

131 xpression and phosphorylation of both hsp in renal cortex, isolated glomeruli, outer medulla, and inn

132 ADC and eADC values in the renal cortex measured at b1000 present a relationship wi

133 ium, lungs, hepatic parenchyma, jejunum, and renal cortex/medulla) and potentially adverse (hepatobil

134 he aorta, inferior vena cava, liver, spleen, renal cortex, muscle, and fat.

135 n and diuresis associated with inhibition of renal cortex Na,K-ATPase activity and redistribution of

136 ogenous lithium clearance, 33% inhibition of renal cortex Na,K-ATPase activity, and redistribution of

137 A/I total NO production, with no decrease in renal cortex NOS activity despite a decrease in remnant

138 Core-needle biopsy of the renal cortex obtained during surgical implantation of th

139 AL-DNA adducts were present in the renal cortex of 83% of patients with A:T to T:A mutation

140 t-off point, 30,000 D) were implanted in the renal cortex of anesthetized rats and were perfused at 2

141 respectively, than those of NOS1alpha in the renal cortex of C57BL/6 mice.

142 xcept for the tumor of case 1 (5.9%) and the renal cortex of case 2 (5.6%).

143 s in vivo decreases expression of p53 in the renal cortex of control and streptozotocin-injected diab

144 ence for chronic NF-kappaB activation in the renal cortex of db/db mice and suggest a novel, diabetes

145 Status of GSK3beta was examined in vivo in renal cortex of db/db mice with type 2 diabetes at 2 wee

146 microRNA-192 (miR-192) are increased in the renal cortex of diabetic mice, and this is associated wi

147 P-1 activation in brain pericytes and in the renal cortex of diabetic mice.

148 and noncanonical activation pathways in the renal cortex of diabetic mice.

149 In the renal cortex of diabetic rats, the increase in Akt phosp

150 WNK1 but increased expression of WNK4 in the renal cortex of mice.

151 ug resistance gene 1 was up-regulated in the renal cortex of mutant mice.

152 a low basal expression of RANTES mRNA in the renal cortex of nephrotic rats that did not differ from

153 istolactam (AL) and dG-AL DNA adducts in the renal cortex of patients with EN but not in patients wit

154 te mRNA expression of IP-10 and MCP-1 in the renal cortex of rats 6 to 8 days after the administratio

155 ed a large increase in TSC expression in the renal cortex of rats on a low-NaCl diet (207 +/- 21% of

156 Extracts prepared from the renal cortex of rats that were made acutely acidotic als

157 as malondialdehyde content was lower, in the renal cortex of SOD-Tg-db/db compared with NTg-db/db mic

158 erone, BiP, was observed in the cells of the renal cortex of the male transgenic mice, suggesting ER

159 1 protein was significantly increased in the renal cortex of these animals.

160 nd C5b-9 deposition were demonstrated in the renal cortex of two TTP patients, by immunofluorescence

161 synthase protein levels were upregulated in renal cortex of uni-x sheep (P<0.05).

162 the partial pressure of oxygen (PO2) in the renal cortex of unrestrained rats, which might in turn c

163 Diabetes increased PKC activity in renal cortex of wild-type mice and was significantly red

164 paralleled by a decrease in its abundance in renal cortex on immunoblot.

165 y, SNR measurements in the liver, aorta, and renal cortex on pre- and postcontrast images were signif

166 mal ablation zone, perirenal fat, and normal renal cortex on the MR images.

167 in levels were not increased in either mouse renal cortex or medulla after either 2 or 7 days of oral

168 radioactivity in the heart (P < 0.0001) and renal cortex (P = 0.02 and P = 0.005, respectively).

169 Increased neutrophils were observed in the renal cortex, particularly within the glomeruli where a

170 1.7% was observed between the sphere and the renal cortex phantoms.

171 d cholesteryl ester (CE) accumulation within renal cortex/proximal tubules.

172 following tissues of Brown Norway rats: the renal cortex, renal outer medulla, liver, cardiac left v

173 the site of erythropoietin production in the renal cortex, showing the greatest accumulation of renox

174 T1 and T2 SI ratio (ratio of tumor to renal cortex SI on T1- and T2-weighted images, respectiv

175 bserved transgene expression confined to the renal cortex (specifically proximal and distal tubules)

176 hat transfected shRNAs were expressed in the renal cortex starting on day 3 and continued for 24 days

177 Gsalpha expression in the renal cortex (the site of PTH action) is markedly reduce

178 reas interior structures are defined for the renal cortex, the medullary pyramids with papillae (2 ve

179 of COMT to nuclei of epithelial cells in the renal cortex, the site of CE biosynthesis and where the

180 ygen tension and that when injected into the renal cortex, they incorporated into the renal parenchym

181 ameters offer quality ADC measurement in the renal cortex using b1000.

182 ELV], smart region of interest [ROI] volume, renal cortex volume [RCV]) were performed in 101 living

183 Overall renal cortex volumetry seems to be the most accurate tec

184 average difference between haematoma and the renal cortex was 5 dB.

185 Renal cortex was analyzed after sorbitol density gradien

186 days with 50 mg/kg, the level of CAA in the renal cortex was approximately 15 micromol/L.

187 2, Tie2, and VEGF expression in normal human renal cortex was examined with immunofluorescence and im

188 , and alpha1-(IV) collagen observed in OVE26 renal cortex was significantly reduced in OVE26 RKO kidn

189 RSOR expression, normally confined to the renal cortex, was markedly increased and extended into t

190 A, VEGFR2, and nephrin protein expression in renal cortex were determined by Western immunoblotting;

191 Consequently, apoptotic cells in the renal cortex were significantly decreased by 47%, wherea

192 est measurements within tumor and uninvolved renal cortex were used to calculate percentage signal in

193 These lesions concentrate in the renal cortex, where they serve as a sensitive and specif

194 wn to induce net vasoconstriction within the renal cortex while increasing medullary blood flow and o

195 ate distinct binding of 11C-L-159,884 in the renal cortex with a specific binding component suitable

196 of HB-EGF on renal function, we infused the renal cortex with active rHB-EGF, prepared from transfec

197 ghest mean absorbed dose was received by the renal cortex, with 1.9 mGy per MBq of (86)Y- 6: CONCLUSI