ライフサイエンスコーパス: renal excretion

1 odistribution profiles with both hepatic and renal excretion.

2 injection followed by a decline, indicating renal excretion.

3 a protein binding, a necessary condition for renal excretion.

4 and ultimate elimination of the K(+) load by renal excretion.

5 ydrodynamic diameter by >15 nm and prevented renal excretion.

6 18F-FB-[Lys3]BBN had predominant renal excretion.

7 arance from tissue compartments primarily by renal excretion.

8 e injected dose per gram]) and predominantly renal excretion.

9 ncomitant rapid disappearance from blood and renal excretion.

10 e, unless the investigational agent involves renal excretion.

11 in very high for several days due to lack of renal excretion.

12 r, first-order process and that there was no renal excretion.

13 of the radiotracer from the blood and rapid renal excretion.

14 , DS exhibited a progressive increase in MBG renal excretion (66 +/-13 pmol/24 hours at week 4 versus

15 (68)Ga-NOTA-UBI-29-41 showed high renal excretion (83.2% +/- 7.3%) of injected dose and ra

16 ion and excrete from normal tissue/organ via renal excretion after complete targeting to the tumor si

17 function may have abnormal renal transit of renal excretion agents during exercise, although their b

18 bution in the abdomen and pelvis with little renal excretion and bladder activity-characteristics ben

19 efficacy of N-BPs is hampered by their rapid renal excretion and high affinity for bone.

20 above 15 mg kg(-1) : high dose expedited the renal excretion and shortened the blood retention.

21 Renal excretion and vasodilation did not account for the

22 .21 percentage injected dose per gram), fast renal excretion, and low background; tumor-to-blood and

23 Diuresis, natriuresis, renal excretion, and tissue levels of MBG and OLC were m

24 ng and locally produced inhibitors; impaired renal excretion; and current therapies.

25 Sulfoconjugation and renal excretion are important determinants of the wide i

26 ncreased efflux of bile acids into blood for renal excretion as well as hydroxylation of bile acids b

27 sing renal activity at 1-2 min (PETinitial), renal excretion at 2-10 min (PETearly), and, subsequentl

28 N) and (99m)Tc(CO)(3)(dd,ll-LAN) showed good renal excretion, averaging 85% and 77% that of (131)I-OI

29 There is little renal excretion compared with (18)F-FDG.

30 Renal excretion fractions were measured from 10 to 14 di

31 The renal excretion fractions were measured from 12-24 discr

32 The 24-hr renal excretion fractions were measured from conjugate e

33 ac arrest and resuscitation, sepsis, reduced renal excretion, hypoxia induced cancer, decreased extra

34 Moreover, the renal excretion kinetic and intrahepatic albumin binding

35 d in 5-25% of the human population, impaired renal excretion leads to hyperuricemia.

36 Renal excretion of 8-iso PGF(2alpha) was increased in An

37 ent relies on fluid resuscitation to promote renal excretion of active metabolite, withholding the do

38 titution at the cleavage site resulted in no renal excretion of AIM.

39 lasma bile salt levels predominantly through renal excretion of bile products.

40 e, we conclude that strategies to accelerate renal excretion of bile salt and other toxins should be

41 testinal calcium may increase absorption and renal excretion of both phosphate and oxalate.

42 wider range of values for serum calcium and renal excretion of calcium than we observe in control li

43 e kidney proximal tubule (PT) participate in renal excretion of drugs and endogenous compounds.

44 Renal excretion of imatinib was less than 10% of the tot

45 g for 1 wk decreased PMg++ 18%, TZR 25%, and renal excretion of magnesium (UMg) and calcium (UCa) mor

46 uction is explained partly by the absence of renal excretion of metabolizable organic anions, leaving

47 Impaired renal excretion of NOx is a contributor to the high plas

48 a volume by at least 3 mechanisms: increased renal excretion of salt and water, vasodilation, and inc

49 ium excretion by the kidney is essential for renal excretion of sufficient amounts of protons and to

50 lthough the plasma clearance and the rate of renal excretion of the (99m)Tc(CO)(3)(LAN) complexes wer

51 he renal proximal tubule is critical for the renal excretion of the prototypic organic anion, para-am

52 These reactions result in enhanced renal excretion of the sulfate-conjugated reaction produ

53 Because of the renal excretion of the tracer, the absorbed dose was hig

54 aminoguanidine (AG) can improve turnover and renal excretion of these substances.

55 mplicates them as substantial players in the renal excretion of urate.

56 genes seem to be involved in regulating the renal excretion of uric acid which underscores the impor

57 These substances rapidly increase renal excretion or reduce renal tubular reabsorption and

58 ptake and urinary clearance, visualizing the renal excretion pathway from cortex to ureter.

59 from systemic blood circulation through the renal excretion route.

60 This defect in renal excretion was associated with a severe, PTH-induce

61 inetic parameters (solubility, permeability, renal excretion) were substantially improved by a bioiso

62 ly low activity and small size lead to rapid renal excretion when applied in vivo, limiting its thera

63 t showed rapid blood clearance and exclusive renal excretion, which provides a clear abdominal field

64 PET imaging and biodistribution showed rapid renal excretion with low liver activity.

65 can be easily cleared from the body through renal excretion without causing accumulation/toxicity pr