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1 ed, concomitant with ameliorated cardiac and renal hypertrophy.
2 tool to examine primary cilia regulation of renal hypertrophy.
3 synthesis during development of compensatory renal hypertrophy.
4 mycin also completely inhibited compensatory renal hypertrophy.
5 the remnant kidney, followed by compensatory renal hypertrophy.
6 eases in the early diabetic kidney to enable renal hypertrophy.
7 Thus, p8 is a novel marker for diabetic renal hypertrophy.
8 play an important role in the development of renal hypertrophy.
9 expansion, basement membrane thickening, and renal hypertrophy.
10 t diet exhibited an increase in body weight, renal hypertrophy, an increase in urine H(2)O(2) and uri
11 , unilateral nephrectomy triggered increased renal hypertrophy and accelerated renal cyst formation,
16 2 kinase phosphorylation in association with renal hypertrophy and glomerular and tubular increase in
20 n plays a central role in the development of renal hypertrophy and in modulating the production of th
21 and establish that Nox4-derived ROS mediate renal hypertrophy and increased fibronectin expression.
23 EF2 kinase, and p70S6 kinase and ameliorated renal hypertrophy and laminin-beta1 protein content, wit
26 duced urinary Agt and angiotensin II levels, renal hypertrophy, and glomerulotubular fibrosis in Akit
28 latory role and can ameliorate hypertension, renal hypertrophy, and interstitial fibrosis in diabetes
29 systemic hypertension, Agt gene expression, renal hypertrophy, and interstitial fibrosis were studie
31 ely, organ failure; and cardiac and diabetic renal hypertrophy are major causes of morbidity and mora
33 ation of eIF2Bepsilon, which correlated with renal hypertrophy at 2 weeks, and increased laminin beta
34 ptors had significantly less albuminuria and renal hypertrophy, despite similar degrees of hyperglyce
35 ll-cycle proteins in regulating pathological renal hypertrophy, diabetes was induced in mice expressi
36 t kidney mass to lean body mass also mediate renal hypertrophy following unilateral nephrectomy (UNX)
37 zole-4-carboxamide-1-riboside also prevented renal hypertrophy, glomerular basement thickening, foot
38 betic PKC-beta(-/-) mice were protected from renal hypertrophy, glomerular enlargement, and hyperfilt
39 e activation of Fcgamma receptors alleviates renal hypertrophy, inflammation, and fibrosis in hyperch
40 -beta antibodies prevents the development of renal hypertrophy, mesangial matrix expansion, and the d
41 se in RBF seen at 2 and 7 d and retarded the renal hypertrophy that should have developed by 7 d.
42 Diabetic nephropathy is characterized by renal hypertrophy, thickening of basement membranes, and
43 not abrogate the weight gain, it reduced the renal hypertrophy, urine H(2)O(2), and urine and renal M
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