ライフサイエンスコーパス: renal lesion

1 function and networking, which augmented the renal lesion.

2 losclerosis (FSGS) is a common, non-specific renal lesion.

3 e centrally reviewed to assess for bilateral renal lesions.

4 lin, causing salt-sensitive hypertension and renal lesions.

5 ntiation between benign and malignant cystic renal lesions.

6 and blocked the development of all types of renal lesions.

7 dney that associated with the progression of renal lesions.

8 ) and serum creatinine level and more severe renal lesions.

9 eutically to treat Birt-Hogg-Dube-associated renal lesions.

10 ream signaling but also displayed attenuated renal lesions.

11 ents with non-fat-containing T1 hyperintense renal lesions.

12 lead to frequent serendipitous discovery of renal lesions.

13 y role for C5 activation in the induction of renal lesions.

14 ) underwent biopsy of focal liver lesions or renal lesions.

15 quired renal disease, and 178 had structural renal lesions.

16 teral, papillary macroscopic and microscopic renal lesions.

17 he ubiquitous presence of other, nondiabetic renal lesions.

18 entiation between solid benign and malignant renal lesions.

19 Three patients had indeterminate renal lesions, 1 patient had a solid renal mass, 1 patie

20 1558, 9.6%), adenopathy (103 of 1558, 6.6%), renal lesions (101 of 1558, 6.5%), and negative examinat

21 n and 41 women; mean age, 54 years) with 144 renal lesions (111 benign, 33 malignant) measuring 1-4 c

22 The 109 renal lesions--81 benign lesions and 28 RCCs--had a mean

23 The T1 signal characteristics of the renal lesions and necrotic or cystic areas were recorded

24 ignalling is not inhibited in Tsc-associated renal lesions and that by partially inhibiting the Akt/m

25 The renal lesions and the disease course were more severe in

26 ge, 66.5 years) underwent cryoablation of 11 renal lesions, and 14 patients (11 men, four women; mean

27 , we assessed pathologic and ultrastructural renal lesions, and measured urinary albumin/creatinine r

28 The T1 signal characteristics of a renal lesion appear to be related to the ADC of the lesi

29 utaneous cryoablation of small (< or = 4-cm) renal lesions appears to require less analgesia than RF

30 sly because missed or mischaracterized small renal lesions are a frequent problem in these patients.

31 ephropathy from type 2 diabetes and that the renal lesions are associated with increased oxidative st

32 background developed macroscopically visible renal lesions as early as 3-6 months, increasing to 95%

33 For each renal lesion, attenuation measurements were obtained; at

34 detection, characterization, and staging of renal lesions, but pelvic CT is probably not needed for

35 s were compared between benign and malignant renal lesions by using the two-sample t test.

36 to characterize renal lesions; the ADC of a renal lesion can be potentially used as an additional pa

37 Relatively advanced diabetic renal lesions can be present in some diabetic patients w

38 The renal lesions (casts) are directly related to the produc

39 e, diabetic mice rapidly develop morphologic renal lesions characteristic of both early and advanced

40 roves the characterization of small (1-4 cm) renal lesions compared with conventional attenuation mea

41 icity for characterization of small (1-4 cm) renal lesions compared with conventional attenuation mea

42 differentiating between benign and malignant renal lesions compared with conventional enhancement mea

43 Despite treatment improvements, associated renal lesions - congenital dysplasia, acquired scarring

44 postprocessing enabled accurate detection of renal lesion enhancement across the attenuation spectrum

45 f interest were measured in 74 patients with renal lesions evaluated by means of dynamic contrast mat

46 e, 62 years; age range, 26-87 years) with 93 renal lesions for which pathologic correlation was avail

47 Here, we show that many of the earliest renal lesions from Tsc1+/- and Tsc2+/- mice do not exhib

48 lesions and RCC, 26 microdissected archival renal lesions from two VHL disease patients were studied

49 DW imaging can be used to characterize renal lesions; however, compared with CE MR imaging, it

50 Renal lesions in 10 patients regressed (in nine, after s

51 One hundred nine renal lesions in 64 patients (46 men, 18 women; mean age

52 tenuation spectrum of frequently encountered renal lesions in a non-body habitus-restricted patient p

53 Pentosan polysulfate reduced structural renal lesions in CsA-treated, salt-depleted Sprague-Dawl

54 examine this we have evaluated the growth of renal lesions in Eker rats (Tsc2+/-) subjected to a keto

55 sgenic mouse kidneys and the exacerbation of renal lesions in these mice.

56 PDGF activity contributes to the progressive renal lesions in this animal model.

57 ence in ADC between the malignant and benign renal lesions: in patients with angiomyolipoma the ADC v

58 The renal lesions included 2 benign cysts, 5 atypical cysts,

59 e develop a spectrum of bilateral clear-cell renal lesions including cysts and renal cell carcinomas

60 We found that all renal lesions including cysts, adenomas and carcinomas e

61 albuminuria, elevated creatinine levels, and renal lesions including extensive apoptotic cell death,

62 Moreover, the severity of renal lesions induced by ER stress was dramatically grea

63 hat some MA patients have only mild diabetic renal lesions is consistent with the lower than original

64 Currently, cryoablation of small renal lesions is minimally invasive, safe, and efficacio

65 by dose-dependent decreases in albuminuria, renal lesions (mesangial expansion, leukocyte infiltrati

66 Measurements in renal lesions (n = 9) were poorly correlated (r = -0.081

67 egmental glomerulosclerosis is a nonspecific renal lesion observed both as a primary (idiopathic) ent

68 2 activation, and thereby explains the worse renal lesions observed.

69 nuria, glomerular endotheliosis (a classical renal lesion of pre-eclampsia), placental abnormalities

70 e history of renal cell carcinoma, number of renal lesions, presence of a coexistent solid renal mass

71 Renal lesions progressed from cysts through cystadenomas

72 eatment lowered blood pressure and prevented renal lesions, raising the question as to whether these

73 of HIF in the development of VHL-associated renal lesions remains to be determined.

74 Ultrastructural analyses of renal lesions revealed the presence of electron-dense de

75 Except for a single benign cyst, 25 of 26 renal lesions showed nonrandom allelic loss of the VHL g

76 and resolved more quickly for patients with renal lesions than they did for patients with liver lesi

77 insulin secretion and perhaps improving the renal lesions that develop in patients with diabetes mel

78 ease (ARPKD) is characterized by biliary and renal lesions that produce significant morbidity and mor

79 ologic studies revealed typical Randall-type renal lesions that were absent in mice expressing the co

80 more likely to have a serologic profile and renal lesions that were associated with more aggressive

81 The third case involved multiple renal lesions that were subsequently treated with radiof

82 DWI can be used to characterize renal lesions; the ADC of a renal lesion can be potentia

83 Regions of interest were placed on renal lesions to measure the ADC of whole lesions, enhan

84 studied pathology was urinary calculi (28%), renal lesion/tumor (23%), and hepatic lesion/tumor (20%)

85 y excised Bosniak IIF and Bosniak III cystic renal lesions was 25% and 54%, respectively, in our stud

86 The efficacy of enalapril on pulmonary and renal lesions was assessed in CS-exposed mice.

87 Renal lesions were characterized with use of CE MR crite

88 Renal lesions were found in two other patients during fo

89 IAL/Seventy-five patients with 75 unilateral renal lesions were included, and 75 normal contralateral

90 ristane-treated BALB/c mice, even though the renal lesions were similar in both strains.

91 o in the characterization of T1 hyperintense renal lesions, with both methods having lower sensitivit