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1 feration induced by the 5-HT(2A) receptor in renal mesangial cells.
2 ase in P-gp activity in Cox-2 overexpressing renal mesangial cells.
3 d expression of iNOS and production of NO in renal mesangial cells.
4 nase kinase-4 (MKK4)/SEK1, MKK3, and MKK6 in renal mesangial cells.
5 ith TGF-beta receptors type I and type II in renal mesangial cells.
6 protein, is strongly induced in ET-1-treated renal mesangial cells and in an animal model of diabetic
7 e to quantify TNFalpha-mediated apoptosis in renal mesangial cells and to determine whether expressio
11 (COX-2) is an early response gene induced in renal mesangial cells by interleukin-1beta (IL-1beta).
18 ed expression of CCN2/CTGF in human lung and renal mesangial cells is inhibited by 10 nm PGE(2), wher
20 ansforming growth factor-beta1 (TGF-beta) in renal mesangial cells (MC) are hallmark features of diab
21 al tropism of some light chains, an in vitro renal mesangial cell model of amyloid formation was used
24 he contribution of TxNIP was investigated in renal mesangial cell reactive oxygen species (ROS) gener
27 h the WT bone marrow-derived macrophages and renal mesangial cells stimulated with S100A8/A9 secrete
28 ta can activate MKK4/SEK1, MKK3, and MKK6 in renal mesangial cells; therefore, we examined the role o
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