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1 feration induced by the 5-HT(2A) receptor in renal mesangial cells.
2 ase in P-gp activity in Cox-2 overexpressing renal mesangial cells.
3 d expression of iNOS and production of NO in renal mesangial cells.
4 nase kinase-4 (MKK4)/SEK1, MKK3, and MKK6 in renal mesangial cells.
5 ith TGF-beta receptors type I and type II in renal mesangial cells.
6 protein, is strongly induced in ET-1-treated renal mesangial cells and in an animal model of diabetic
7 e to quantify TNFalpha-mediated apoptosis in renal mesangial cells and to determine whether expressio
8 ular smooth muscle cells, endothelial cells, renal mesangial cells, and tubular cells.
9                                              Renal mesangial cell apoptosis is a crucial repair mecha
10                                              Renal mesangial cells are responsible for glomerular PAF
11 (COX-2) is an early response gene induced in renal mesangial cells by interleukin-1beta (IL-1beta).
12        Some major pathobiologic processes in renal mesangial cells, elicited in response to immunoinf
13                                           In renal mesangial cells, high glucose increased the expres
14                                     In human renal mesangial cells, high glucose induced ROS producti
15 nvestigated in an in vitro model using human renal mesangial cells (HRMCs).
16                                              Renal mesangial cell hypertrophy is a characteristic of
17 ns into the glomerular basement membrane and renal mesangial cell hypertrophy.
18 ed expression of CCN2/CTGF in human lung and renal mesangial cells is inhibited by 10 nm PGE(2), wher
19 on of HMMP13 and RMMP13 protein synthesis in renal mesangial cells is strikingly different.
20 ansforming growth factor-beta1 (TGF-beta) in renal mesangial cells (MC) are hallmark features of diab
21 al tropism of some light chains, an in vitro renal mesangial cell model of amyloid formation was used
22                                           In renal mesangial cells, overexpression of FXR or treatmen
23                                              Renal mesangial cells produce excess extracellular matri
24 he contribution of TxNIP was investigated in renal mesangial cell reactive oxygen species (ROS) gener
25                                              Renal mesangial cells regulate their expression of the p
26                                              Renal mesangial cells (RMCs) constitute a population of
27 h the WT bone marrow-derived macrophages and renal mesangial cells stimulated with S100A8/A9 secrete
28 ta can activate MKK4/SEK1, MKK3, and MKK6 in renal mesangial cells; therefore, we examined the role o
29                      We used cultures of rat renal mesangial cells to study the regulation of EGF rec

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