ライフサイエンスコーパス: renal pelvis

1 ers of the small bowel, ovaries, breast, and renal pelvis.

2 nary tract, such as the bladder, ureter, and renal pelvis.

3 adder, causing dilation of the ureter and/or renal pelvis.

4 16, 25, 30, or 35 cm above the level of the renal pelvis.

5 illae (2 vertical and 3 horizontal), and the renal pelvis.

6 e medulla, where they release urine into the renal pelvis.

7 rine attenuation was lower in the obstructed renal pelvis (7.4 HU) than in the bladder (11.4 HU) (P <

8 ly removed by peristaltic contraction of the renal pelvis, a smooth muscle structure unique to placen

9 mon disease characterized by dilation of the renal pelvis and calices, resulting in loss of kidney fu

10 with highest posttransplant recognition for renal pelvis and cortex specific antigens.

11 ee transitional cell carcinomas (TCC) of the renal pelvis and five Wilms' tumors were compared with n

12 dynamic injection of a DNA solution into the renal pelvis and found that luciferase expression was st

13 inner and outer medulla, papillary tips, and renal pelvis and from glomeruli isolated by sieving.

14 a were observed in groups at the wall of the renal pelvis and in the angular space formed by the pole

15 y devoid of AT1 receptor fail to develop the renal pelvis and the ureteral peristaltic movement.

16 in humans that drinking-water arsenic causes renal pelvis and ureter cancer.

17 For renal pelvis and ureter cancers, the adjusted odds ratio

18 included 76 renal cell, 24 transitional cell renal pelvis and ureter, and 22 other kidney cancers.

19 iferation causes abnormal development of the renal pelvis and ureter, leading to defective pyelourete

20 The contrast medium in the renal pelvis and ureters was virtually removed from excr

21 lionic fibers projected into the wall of the renal pelvis and/or to the interlobar arteries extending

22 er the induction of elevated pressure in the renal pelvis, and after the pressure was returned to nor

23 In severe cases dilatation of the ureter, renal pelvis, and calyces might be seen.

24 gauge needle was inserted into the opacified renal pelvis, and double-contrast pyelography was perfor

25 In contrast, the incidence rates for renal pelvis cancer declined among white men and remaine

26 The corresponding rates for renal pelvis cancer were 1.5, 0.7, 0.8, and 0.5 per 1000

27 en with cancer (renal-cell cancer in 759 and renal-pelvis cancer in 136) were identified by cross-lin

28 a greater risk of both renal-cell cancer and renal-pelvis cancer than men who were not smokers.

29 mass index or blood pressure and the risk of renal-pelvis cancer.

30 brosis, development of glomerular cysts, and renal pelvis dilation.

31 n of alimentary tract, retention of urine in renal pelvis, distension of bladder, and the development

32 or (AT(1)R)-deficient mice exhibit a dilated renal pelvis (hydronephrosis) and a small papilla.

33 ient with transitional cell carcinoma of the renal pelvis, hydronephrosis proximal to the tumor may c

34 stalsis and point to the central role of the renal pelvis in urine transport.

35 Hydrodynamic renal pelvis injection enables transposon mediated-kidne

36 erase expression quickly decreased following renal pelvis injection, the use of the piggyBac transpos

37 To assess renal injury, we performed the renal pelvis injections on uninephrectomised mice and fo

38 Urothelial carcinoma of the renal pelvis is a deadly disease with an unclear tumorig

39 cassette to drive human CAII cDNA, into the renal pelvis of CAII-deficient mice results in expressio

40 expressed in nascent urothelia in ureter and renal pelvis of human embryos, and it is suggested that

41 Transitional cell carcinoma of the renal pelvis or distal ureter has been extirpated with s

42 cluding renal cell cancer and cancers of the renal pelvis or ureter.

43 thyroid gland but not in the liver, spleen, renal pelvis, or salivary glands.

44 Formation of a lymphoid aggregate in the renal pelvis precedes the invasion of the kidney by infl

45 olon, non-Hodgkin lymphoma, pancreas, kidney/renal pelvis, rectum, and melanoma.

46 ensin type 1 receptor genes do not develop a renal pelvis, resulting in the buildup of urine and prog

47 s, but HB-EGF expression did not increase in renal pelvis smooth muscle cells under identical conditi

48 eter actively propels tubular fluid from the renal pelvis to the bladder, and this peristalsis, which

49 istal peristaltic waves, pump urine from the renal pelvis toward the urinary bladder.

50 eteropelvic junction (UPJ) region, where the renal pelvis transitions to the ureter, is the most comm

51 cidence for all urothelial cancers combined (renal pelvis, ureter, and bladder cancers: adjusted IRR

52 n development affecting the renal parechyma, renal pelvis, ureter, bladder and urethra; they show evi

53 /=18 years) with urothelial carcinoma of the renal pelvis, ureter, bladder, or urethra at 16 sites in

54 urothelial cancer, including cancers of the renal pelvis, ureter, bladder, or urethra, from eight ho

55 d disease (OR, 1.6; 95% CI, 0.8-3.0) and for renal pelvis/ureter cancers (OR, 1.7; 95% CI, 0.5-5.4).

56 ptoms, the association of malrotation of the renal pelvis with calculus increases the risk of hematur