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1 ld standard for the measurement of effective renal plasma flow.
2 ld standard for the measurement of effective renal plasma flow.
3 ld standard for the measurement of effective renal plasma flow.
4 lomerular hyperfiltration and an increase in renal plasma flow.
6 anied by declines in plasma NO and effective renal plasma flow and an increase in renal vascular resi
9 h chronic heart failure, serelaxin increased renal plasma flow and reduced the increase in filtration
11 ession, greater body mass index, lower basal renal plasma flow, and higher diastolic blood pressure t
12 s associated with lower GFR, lower effective renal plasma flow, and higher filtration fraction, even
14 rate; GFR) and paraaminohippurate (effective renal plasma flow) clearances were measured in individua
15 tracer with a capacity to measure effective renal plasma flow comparable to that of the clinical gol
17 still substantial and sustained increases in renal plasma flow, cortical blood flow (20 +/- 4% increa
19 partmental analysis for estimating effective renal plasma flow (ERPF in units of milliliters per minu
20 nstrumented rats increases GFR and effective renal plasma flow (ERPF) and decreases effective renal v
21 r recombinant human RLX 2 (rhRLX), effective renal plasma flow (ERPF) and glomerular filtration rate
22 ronic inhibition of NO synthase on effective renal plasma flow (ERPF) and glomerular filtration rate
23 n 3 d) that includes estimation of effective renal plasma flow (ERPF) from a single plasma sample in
24 dy, we retrospectively review 1626 effective renal plasma flow (ERPF) measurements in 197 patients wi
26 n cortical blood flow (34 +/- 12% increase), renal plasma flow, GFR, and sodium excretion; however, t
27 iltration rate (I-iothalamate) and effective renal plasma flow (I-hippuran) measured 4 months before
30 ficant changes in mean arterial pressure and renal plasma flow in either C or D rats but reduced glom
31 renin inhibition led to a larger increase in renal plasma flow in healthy human volunteers studied on
32 perfiltrating and showed the largest rise in renal plasma flow in response to irbesartan, whereas ren
36 on GFR (measured by iothalamate clearance), renal plasma flow (measured by para-amino hippurate clea
38 creased plasma renin activity (P < 0.01) and renal plasma flow (P < 0.01); the latter effect plateaue
42 difference between serelaxin and placebo on renal plasma flow (para-aminohippuric acid clearance) an
43 hanges from baseline mean arterial pressure, renal plasma flow, plasma renin activity, aldosterone, u
45 ced the glomerular filtration rate (GFR) and renal plasma flow rate in diabetic rats, despite variabl
48 asma flow in response to irbesartan, whereas renal plasma flow rose less and GFR fell in patients wit
49 mide on glomerular filtration rate (GFR) and renal plasma flow (RPF) in heart failure patients are co
50 intrarenal renin activity, we have examined renal plasma flow (RPF) responses in a standardized prot
53 easured glomerular filtration rate (GFR) and renal plasma flow (RPF), and analysed renal structural i
55 ong the three centers, but systolic BP, GFR, renal plasma flow (RPF), and filtration fraction were lo
58 s showed parallel increases in single-kidney renal plasma flow (RPF), renocortical volume, and glomer
60 There were no differences in GFR, effective renal plasma flow, urine output, or sodium excretion for
61 y [GC]/mass spectrometry [MS] analysis), and renal plasma flow was determined with paraaminohippurate
64 sured with infusion of [6-(2)H2]glucose, and renal plasma flow was measured by para-aminohippurate cl
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