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1 tially reduced risk of recurrence but not of renal scarring.
2 1280 included participants, 199 (15.5%) had renal scarring.
3 1 identified 44.9% of patients with eventual renal scarring.
4 coli are at high risk for the development of renal scarring.
5 mune injury but also kidney regeneration and renal scarring.
6 inary tract infection and the development of renal scarring.
7 erapy are associated with increased rates of renal scarring.
8 per versus lower urinary tract infection and renal scarring.
9 thout affecting the risk of recurrent UTI or renal scarring.
10 aphy for the diagnosis of pyelonephritis and renal scarring.
11 about its impact on recurrent infection and renal scarring.
12 designed to prevent recurrent infections and renal scarring.
13 ract infection and preventing recurrence and renal scarring.
14 aphy for the diagnosis of pyelonephritis and renal scarring.
15 I, 2.89-14.38) were also associated with new renal scarring.
16 pathogenesis of urinary tract infection and renal scarring.
17 intigraphy as the reference method to detect renal scarring.
18 ction and thereby prevent the development of renal scarring.
19 ibiotics, bowel and bladder dysfunction, and renal scarring.
20 btained six months later identify those with renal scarring.
21 is is effective in reducing reinfections and renal scarring.
22 possible therapeutic strategy in progressive renal scarring.
23 the diseased kidney and could contribute to renal scarring.
24 total nephrectomy (SNx) model of progressive renal scarring.
25 A total of 35 children (7.2%) developed new renal scarring.
26 rs that block angiotensin II protect against renal scarring and drug-induced arteriolopathy in this m
27 children is considered as a risk factor for renal scarring and each patient is treated with prudence
28 therefore play a role in the pathogenesis of renal scarring and fibrosis in patients with CRD and can
30 Increased rates of breakthrough infection, renal scarring and surgical failure have been associated
31 y albuminuria (>2 g/24 h; n=15) had the most renal scarring and the lowest endothelial CD34 staining.
34 ureteral reflux, urinary tract infection and renal scarring, as well as the efficacy of various treat
35 ernative approaches to prevention of UTI and renal scarring based on research into host-pathogen inte
39 erapy remained significantly associated with renal scarring even after adjusting for these variables.
41 between UTI-vesicoureteral reflux (VUR) and renal scarring has been challenged by several studies.
42 s such as recurrent urinary tract infection, renal scarring, hypertension, and compromised renal func
43 boratory, and imaging variables in detecting renal scarring in children and adolescents with a first
44 prevent recurrence of UTI and development of renal scarring in children with vesicoureteral reflux.
45 amyl) lysine crosslink, occur in progressive renal scarring in humans independently of the original e
46 complement inhibition with Crry might affect renal scarring in lupus nephritis, gene transcript profi
49 t understanding of the mechanisms underlying renal scarring leading to ESRD to inform on current and
52 with a complete duplication anomaly, severe renal scarring, or persistent VUR associated with an ect
53 inine clearance (P<0.01) and positively with renal scarring (P<0.05) but did not correlate with MR mR
54 or acute pyelonephritis, which can result in renal scarring (reflux nephropathy), hypertension, end-s
55 of antimicrobial therapy was associated with renal scarring; the median (25th, 75th percentiles) dura
56 eteral reflux was the strongest predictor of renal scarring, this degree of reflux was present in onl
59 antibiotic therapy in those with and without renal scarring was 72 (30, 120) and 48 (24, 72) hours, r
61 for 89 percent of the children (275 of 309); renal scarring was noted in 9.5 percent of these childre
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