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1 n and disease are major complications in the renal transplant recipient.
2 and OGTT were performed in 1,619 nondiabetic renal transplant recipients.
3 alent to the original formulation in elderly renal transplant recipients.
4 t-line Pneumocystis pneumonia prophylaxis in renal transplant recipients.
5 curs in 16% to 20% of low-risk, CMV-positive renal transplant recipients.
6 onitoring in a large, multi-ethnic cohort of renal transplant recipients.
7 and cytokine plasma levels in 30, potential renal transplant recipients.
8 We studied a cohort of 570 consecutive renal transplant recipients.
9 n unfavorable cardiovascular risk profile in renal transplant recipients.
10 rm renal function and the overall outcome of renal transplant recipients.
11 rt study of 80 cases occurring in 4189 adult renal transplant recipients.
12 inuria is associated with poorer outcomes in renal transplant recipients.
13 obulin (rATG; Thymoglobulin) in living donor renal transplant recipients.
14 e incidence and severity of CMV infection in renal transplant recipients.
15 ides management and outcome of BK viremia in renal transplant recipients.
16 d with a triple immunosuppressive therapy in renal transplant recipients.
17 pact on peri- and posttransplant measures in renal transplant recipients.
18 nown about its effects on the bone health of renal transplant recipients.
19 A antibodies (dnDSA) may cause graft loss in renal transplant recipients.
20 to attenuate bone loss can be recommended in renal transplant recipients.
21 o potentiate DSA removal by PE in sensitized renal transplant recipients.
22 tion of, pretransplant dialysis in pediatric renal transplant recipients.
23 rapid DSA elimination in early acute AMR in renal transplant recipients.
24 in diminished patient and graft survivals in renal transplant recipients.
25 with mortality has not been well studied in renal transplant recipients.
26 te set of genes for wider-scale screening of renal transplant recipients.
27 s, and chronic allograft injury in pediatric renal transplant recipients.
28 mmunosuppressive regimen in HLA-identical LD renal transplant recipients.
29 al, and patient survival in HLA-identical LD renal transplant recipients.
30 n of MS with CAC presence and progression in renal transplant recipients.
31 ministration of everolimus and tacrolimus in renal transplant recipients.
32 up of the waiting list, the donor pool, and renal transplant recipients.
33 critical role in the elevated rate of SCC in renal transplant recipients.
34 nt in the patient with JCPyVAN and in stable renal transplant recipients.
35 ependent of plasma HDL cholesterol levels in renal transplant recipients.
36 rence in this cohort of long-term, high-risk renal transplant recipients.
37 ed with better patient and graft survival in renal transplant recipients.
38 ffness, oxidative stress, or inflammation in renal transplant recipients.
39 fness, oxidative stress, and inflammation in renal transplant recipients.
40 transplant immune surveillance for pediatric renal transplant recipients.
41 ally in the increasing population of elderly renal transplant recipients.
42 ogression of kidney failure and mortality in renal transplant recipients.
43 s, correlates with delayed graft function in renal transplant recipients.
44 allowing early glucocorticoid withdrawal in renal-transplant recipients.
47 We analyzed T cells from 57 living-donor renal transplant recipients (12 reactive and 45 quiescen
49 of AR and after AR treatment in 35 pediatric renal transplant recipients: 17 patients with AR who rec
51 t study and case-control study in 50 de novo renal transplant recipients, 50 chronic kidney disease (
53 , we systematically analyzed HDL from stable renal transplant recipients, according to graft function
54 nt Registry, primary live and deceased donor renal transplant recipients aged 18 years or older betwe
56 ed States Renal Data System data for primary renal transplant recipients (ages 0-21 years, transplant
57 ed 12-hour everolimus (EVL) PK in 16 elderly renal transplant recipients (all whites; 10 men; mean ag
58 pheral blood samples from 348 HLA-mismatched renal transplant recipients and 101 nontransplant contro
60 010 and 2011) influenza vaccine in 23 stable renal transplant recipients and 22 healthy controls.
61 velopment and long-term graft function in 16 renal transplant recipients and 32 age- and gender-match
62 l carcinoma is the most common malignancy in renal transplant recipients and a major cause of morbidi
63 We retrospectively studied 922 consecutive renal transplant recipients and analyzed patients with T
65 These data will further inform prospective renal transplant recipients and donors during pretranspl
66 for the presence of antidonor antibodies in renal transplant recipients and is usually associated wi
67 on accounts for around half of all pediatric renal transplant recipients and results in improved rena
68 reshold value likely needs to be lowered for renal transplant recipients and supports continued use o
69 sured in a longitudinal cohort of 699 stable renal transplant recipients and the associations of T50
70 cells obtained from a new cohort of tolerant renal transplant recipients and those from age- and sex-
71 k factors for visceral leishmaniasis (VL) in renal transplant recipients and to analyze the impacts o
72 entify clinically significant de novo DSA in renal transplant recipients and to define the properties
73 ant candidates, 8.02 (95% CI, 7.29-8.83) for renal transplant recipients, and 13.7 (95% CI, 11.5-16.3
74 ed in 2,851 Caucasian and 570 Afro-Caribbean renal transplant recipients, and in 236 transplant recip
75 CPyV) is reactivated in approximately 20% of renal transplant recipients, and it may rarely cause JCP
76 ct development of squamous cell carcinoma in renal transplant recipients, and undertook a prospective
78 or the prevention of rejection in sensitized renal transplant recipients are not well established.
81 reported cohort to our knowledge of tolerant renal transplant recipients, as defined by stable graft
83 finding could help in the identification of renal transplant recipients at high risk of this cancer,
85 w, we evaluated the records of all pediatric renal transplant recipients at our center from 1995 to 2
86 approximately 800 patients in the cohort of renal transplant recipients at our institution, 15 subje
87 lin (TMG) and basiliximab (BAS) induction in renal transplant recipients at risk for delayed graft fu
88 d tomography scans of the tibia in pediatric renal transplant recipients at transplantation and 3, 6,
89 and treatment strategies for dyslipidemia in renal transplant recipients based on a literature review
90 e and plasma samples were collected from 112 renal transplant recipients before and after transplanta
91 ogic and infection-related parameters in 499 renal transplant recipients between 1 month and 33 years
92 ystems patient and claims data for all adult renal transplant recipients between 2000 and 2010 with c
93 be the effects of AL induction therapy on AA renal transplant recipients beyond the first posttranspl
95 isease remains the leading cause of death in renal transplant recipients, but the underlying causativ
96 that with time the large majority of stable renal transplant recipients can be safely reduced to dua
98 based immunosuppressive treatment in de novo renal transplant recipients caused marginal changes in L
102 sfusions are generally avoided for potential renal transplant recipients due to risk of human leukocy
103 tation of PTTB and may show delayed onset in renal transplant recipients due to the use of lower dose
104 this pooled data analysis in more than 2000 renal transplant recipients, EVR versus MPA resulted in
108 (EBV) DNAemia by monthly PCR in 55 pediatric renal transplant recipients for the first 2 years after
109 arge national data registry used a cohort of renal transplant recipients from the United States Renal
110 cruited 640 consecutive incident nondiabetic renal transplant recipients from three academic centers
112 ing efficacy and safety of converting stable renal transplant recipients from twice-daily tacrolimus
115 eline, compared with reference participants, renal transplant recipients had significantly lower mean
116 ugh the proportion of elderly patients among renal transplant recipients has increased, pharmacokinet
120 tein-Barr virus (EBV) infection in pediatric renal transplant recipients have been characterized insu
125 ective population-based cohort study of 5970 renal transplant recipients in Australia registered on t
126 We describe the results of 11 ABOi pediatric renal transplant recipients in the 2 largest centers in
127 or the progression of glucose intolerance in renal transplant recipients in the late posttransplant p
129 tive cohort of 41,705 adult Medicare primary renal transplant recipients in the United States Renal D
131 this study, outcomes of adult deceased-donor renal transplant recipients included in the United Netwo
132 In moderately sensitized deceased donor renal transplant recipients, induction with ATG is assoc
134 A retrospective study was conducted in 379 renal transplant recipients initiated on EC-MPS or MMF t
136 dity associated with malignancy in long-term renal transplant recipients is due to cutaneous squamous
137 BKV) infection and associated nephropathy in renal transplant recipients is not clearly understood.
138 ment of hepatitis C virus (HCV) infection in renal transplant recipients is possible, but limited dat
140 living with cats increased the risk of VL in renal transplant recipients living in VL endemic areas.
142 ave been reported in ABO-incompatible (ABOi) renal transplant recipients managed solely with antibody
143 use of hereditary and acquired risk factors, renal transplant recipients manifest features of a chron
144 sion in patients on CNI therapy, most stable renal transplant recipients may benefit from late CNI wi
145 A, after B-lymphocyte depletion treatment in renal transplant recipients may contribute to the effica
146 Thus, early testing of urine samples from renal transplant recipients may identify those at risk f
147 sttransplant serum samples from 18 pediatric renal transplant recipients, measured against 5,056 uniq
149 he azathioprine dosing patterns of 180 adult renal transplant recipients monitored up to 4 years.
150 etabolism in nondiabetic, tacrolimus-treated renal transplant recipients more than 6 months posttrans
151 4 chronic kidney disease (CKD) (n = 46), in renal transplant recipients (n = 22), in patients treate
152 epidemiology in a large cohort of pediatric renal transplant recipients (n = 242) and assessed the i
154 mortality, and graft failure in a cohort of renal transplant recipients (n=495, median follow-up 7.0
155 a single-center, prospective cohort study of renal transplant recipients (N=81), urinary clusterin wa
156 ejection (BPAR) in 216 moderately sensitized renal transplant recipients (negative flow crossmatch an
157 t a rare survival case of isolated GITB in a renal transplant recipient, occurring seven years after
161 trolled withdrawal study enrolled 177 stable renal transplant recipients on maintenance CNI-based imm
165 blood from healthy volunteers and in de novo renal transplant recipients participating in studies com
167 drawal, from a triple-drug regimen in stable renal transplant recipients, prevented progressive deter
169 phase IIIb, open-label trial of 833 de novo renal transplant recipients randomized to everolimus, ta
170 PS) versus mycophenolate mofetil (MMF) among renal transplant recipients receiving a tacrolimus-based
171 c and preemptive strategies were compared in renal transplant recipients receiving alemtuzumab induct
172 MPS (group B, n=75) was performed in primary renal transplant recipients receiving combined thymoglob
174 h acute cellular rejection (ACR) in 43 adult renal transplant recipients receiving steroid-free tacro
175 estimated glomerular filtration rate, and 23 renal transplant recipients referred for parathyroidecto
179 apy for Pneumocystis pneumonia prevention in renal transplant recipients, reported adverse drug react
180 proteinuria, diabetes, or CKD stages 1-4 or renal transplant recipients reporting >/=10 ESRD events
183 intake, with graft failure and mortality in renal transplant recipients (RTR) and potential effect m
184 e potential role of SDMA as a risk marker in renal transplant recipients (RTR) has not been investiga
190 tabolites with cardiometabolic parameters in renal transplant recipients (RTRs) and analyzed their pr
191 f pyridoxal 5'-phosphate (PLP) are common in renal transplant recipients (RTRs) and confer increased
196 sought to explore the prevalence of GE among renal transplant recipients (RTRs) in relation to cyclos
197 ors (ACE-i) on the cardiovascular outcome of renal transplant recipients (RTRs) receiving calcineurin
198 cancer (NMSC) is substantially higher among renal transplant recipients (RTRs) than in the general p
199 essing left ventricular hypertrophy (LVH) in renal transplant recipients (RTRs) with chronic allograf
200 to modulate cardiovascular complications in renal transplant recipients (RTRs), and a relationship b
202 on viral infections that affect up to 10% of renal transplant recipients (RTRs), causing allograft dy
207 ral blood monocyte cells and CD3+ T cells of renal transplant recipients (RTX) receiving tacrolimus (
209 compartments from 20 HCMV-infected patients (renal transplant recipients, stem cell transplant recipi
211 carcinoma development in long-term, at-risk renal transplant recipients than previously identified c
212 was tested on >1000 samples from a cohort of renal transplant recipients to assess its performance in
213 cept clinical trial, we randomly assigned 50 renal transplant recipients to immediate-postoperative i
214 10,190 UK adult and pediatric deceased donor renal transplant recipients transplanted between 2000 an
215 o 24 months posttransplant in solitary adult renal transplant recipients transplanted between 2003 an
216 rospective cohort of 40,821 Medicare primary renal transplant recipients transplanted from January 1,
217 pective cohort study of 2749 adult Norwegian renal transplant recipients, transplanted between 1999 a
219 We performed a retrospective analysis of renal transplant recipients treated with rapamycin from
220 linical characteristics, and outcomes of 378 renal transplant recipients treated with SRL-based immun
221 M) on cardiorespiratory fitness in pediatric renal transplant recipients (TX) has not been establishe
222 d pressure monitoring (ABPM) in 33 pediatric renal transplant recipients (TXP), aged 8 to 19 years, m
223 , point to the need for careful follow-up of renal transplant recipients undergoing intravitreal ther
225 an leukocyte antigen-mismatched living donor renal transplant recipients underwent low-intensity cond
226 retrospective study in which 76 non-diabetic renal transplant recipients underwent oral glucose toler
227 d standard (iohexol plasma clearance) in 193 renal transplant recipients using concordance correlatio
228 (NODM) after kidney transplant in pediatric renal transplant recipients using Organ Procurement Tran
229 pared cancer incidence in PKD versus non-PKD renal transplant recipients using Poisson regression, an
230 ucted a retrospective cohort study of 32,757 renal transplant recipients using the United States Rena
232 tcomes of ST-segment elevation MI (STEMI) in renal transplant recipients vs the stage 5D CKD group or
233 ulations, plasma neopterin, and cytokines in renal transplant recipients was investigated in this stu
234 ation-based, nationwide cohort of first-time renal transplant recipients was matched by age and sex w
235 group, comparative study in primary de novo renal transplant recipients was planned for 48 months bu
236 e maintenance immunosuppression in pediatric renal transplant recipients, we enrolled 35 participants
237 invasive squamous cell carcinomas (SCCs) in renal transplant recipients, we investigated whether con
238 ousand three hundred seventy-eight pediatric renal transplant recipients were analyzed; 804 (58%) rec
244 Between August 2009 and October 2010, 40 renal transplant recipients were prospectively included
245 hundred twenty-three live or deceased donor renal transplant recipients were randomized 2:1 to recei
249 ction against early acute rejection in black renal transplant recipients, whereas sensitized patients
255 phosphate was significantly higher in those renal transplant recipients who died at follow-up when c
257 nd and comparable to that seen in a group of renal transplant recipients who had received intravenous
260 ulating anti-PLA2R antibodies in a cohort of renal transplant recipients who prospectively developed
261 cell depletion and recovery among pediatric renal transplant recipients who receive alemtuzumab indu
262 th a functioning graft (DWFG) is affected in renal transplant recipients who receive prophylaxis for
264 s a substudy of a randomized trial in stable renal transplant recipients who were on a triple CNI-bas
265 , we describe the case of a 63-year-old male renal transplant recipient with a remote history of pulm
268 sease (CVD) is the leading cause of death in renal transplant recipients with a functioning allograft
269 ective randomized trial to determine whether renal transplant recipients with a positive CMV serostat
274 In a 2-year randomized controlled trial, 155 renal transplant recipients with at least one biopsy-con
278 pective, double-blind, parallel-group trial, renal transplant recipients with GI symptoms receiving M
279 dies evaluating treatment with cinacalcet in renal transplant recipients with hyperparathyroidism.
280 es from 1 patient with JCPyVAN and 20 stable renal transplant recipients with JCPyV viruria was attem
281 munosuppression failures among 129 pediatric renal transplant recipients with mean follow-up of 5 yea
282 clusion criteria permitting analysis of 3312 renal transplant recipients with median follow-up of 12
286 infection has been reported in 10% to 60% of renal transplant recipients with progression to BK nephr
288 deling and mineral loss, and reduced eGFR in renal transplant recipients with secondary hyperparathyr
291 is of rejection rates and graft outcomes for renal transplant recipients with such preformed DSAs, de
293 receptor-activating antibodies (AT1R-Abs) in renal transplant recipients with vascular rejection and
294 nduction results in good efficacy in de novo renal transplant recipients with very well-preserved ren
295 withdrawal has been demonstrated in selected renal-transplant recipients with haematopoietic chimeris
296 (PVAN) occurs in a significant percentage of renal transplant recipients, with BK virus reactivation
297 s were found in urine samples from 19 stable renal transplant recipients, with JCPyV quasispecies det
299 urately detect graft artery abnormalities in renal transplant recipients without the risk of nephroto
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