ライフサイエンスコーパス: renovascular

1 nking water) or renin/angiotensin-dependent (renovascular 2-kidney-1-clip).

2 Although renovascular abnormalities were mild, their detection in

3 Renovascular abnormalities were noted in the dictated re

4 e exhibited a strong hypertensive phenotype, renovascular abnormalities, persistent constriction of t

5 es and their relationship to renal function, renovascular anatomy, and BP are delineated.

6 Renovascular and adrenal responses to infused angiotensi

7 The blunted renovascular and functional responses to acetylcholine i

8 tetraenoic acid (20-HETE), a metabolite with renovascular and tubular functions.

9 pendent factors protect against vascular and renovascular calcification, and vitamin K antagonists ma

10 ctable in intrinsic glomerular, tubular, and renovascular cells in all analyzed cases.

11 ctable in intrinsic glomerular, tubular, and renovascular cells in native kidneys.

12 mage as a result of warm ischemia, affecting renovascular circulating volume.

13 d dilution techniques may be used to measure renovascular circulating volumes during hypothermic mach

14 dilution is a reproducible method to assess renovascular circulating volumes in machine-perfused kid

15 Renovascular circulating volumes of machine-perfused por

16 Renovascular circulating volumes significantly increased

17 ility of ultrasound dilution measurements of renovascular circulating volumes was good (mean coeffici

18 mbolic events resulting in end-organ damage, renovascular complications, or hypertensive crisis at 1

19 One patient had CT evidence of renovascular compromise.

20 tion and realignment leading to fibrosis and renovascular constriction in diabetes.

21 ng II)-induced BP, dipsogenic responses, and renovascular contractility were significantly attenuated

22 Magnetic-resonance angiography showed severe renovascular disease (>50% renal-artery stenosis or occl

23 enal donors, the arteriographic incidence of renovascular disease (10.9%) was higher than previously

24 egarding the epidemiology of atherosclerotic renovascular disease (ARVD) in dialysis populations are

25 Atherosclerotic renovascular disease (ARVD) is associated with heart dis

26 the other (n = 60) with a high prevalence of renovascular disease (group II).

27 Atherosclerotic renovascular disease (RVD) amplifies damage in a stenoti

28 Renovascular disease (RVD) induces renal microvascular (

29 Atherosclerotic renovascular disease (RVD) reduces renal blood flow (RBF

30 ntly used to treat renal artery stenosis and renovascular disease (RVD); however, renal function is r

31 s; range, 27-62 years) and six patients with renovascular disease (three women; average age, 48 years

32 We investigated the frequency of renovascular disease among elderly people with heart fai

33 upport its routine use for the evaluation of renovascular disease among patients suspected of having

34 nction and size in patients with obstructive renovascular disease and chronic renal insufficiency.

35 ing renal function and size in patients with renovascular disease and chronic renal insufficiency.

36 may limit progressive CKD in atherosclerotic renovascular disease and may apply to other conditions i

37 tudies indicate that whereas atherosclerotic renovascular disease can accelerate both systemic hypert

38 Progressive renovascular disease during medical therapy can produce

39 Patients with renovascular disease had worse renal function (mean crea

40 investigation and appropriate management of renovascular disease has remained a controversial topic.

41 Many children with renovascular disease have abnormalities of other blood v

42 Renovascular disease is a frequent cause of severe hyper

43 Renovascular disease is an uncommon but important cause

44 Chronic azotemic renovascular disease is common in patients with atherosc

45 Treating patients with renovascular disease is complex, particularly as imaging

46 The anatomic diagnosis of renovascular disease is increasing in frequency due to t

47 reflected in the proportion of patients with renovascular disease listed as cause of ESRD on the Medi

48 Atherosclerotic renovascular disease may augment deterioration of renal

49 Some elderly patients with occult renovascular disease on ACE inhibitors will be at risk o

50 The effect of detecting renovascular disease on donor selection was determined i

51 cular disease, and the effect of identifying renovascular disease on subsequent donor surgery was asc

52 Kidney injury in atherosclerotic renovascular disease reflects complex interactions among

53 of ESRD, rates of end-stage atherosclerotic renovascular disease seem to be on the rise in older pat

54 , and were more likely than patients without renovascular disease to have peripheral arterial disease

55 Most children with renovascular disease will need interventional or surgica

56 yperuricaemia itself causes hypertension and renovascular disease, and that lowering of serum urate m

57 gh technical success rate in atherosclerotic renovascular disease, but little is known about the clin

58 the other (n = 60) with a high prevalence of renovascular disease, defined with angiograms.

59 iency and global obstructive atherosclerotic renovascular disease, renal artery stenting improves or

60 rculation and alleviate renal dysfunction in renovascular disease.

61 We used captopril renography to screen for renovascular disease.

62 ficity of 94.3% (67.6-97.3) for detection of renovascular disease.

63 e placed in 32 patients with atherosclerotic renovascular disease.

64 oninvasive intervention in the management of renovascular disease.

65 R-26a might be a novel therapeutic target in renovascular disease.

66 c targets for recovery of kidney function in renovascular disease.

67 ic intervention for preserving the kidney in renovascular disease.

68 nal microcirculation in chronic experimental renovascular disease.

69 sport on tissue oxygenation in subjects with renovascular disease.

70 d help identify patients suspected of having renovascular disease.

71 acellular matrix (ECM) metabolism and reduce renovascular fibrosis.

72 n hypertrophied ventricles from animals with renovascular hypertension (1.6-fold, P < 0.05) and aorti

73 en during arteriographic study for suspected renovascular hypertension (42%).

74 truction and 48 patients suspected of having renovascular hypertension (RVH) were randomly selected f

75 myocardial microvascular structure in swine renovascular hypertension (RVH) would be improved by sim

76 al angiograms obtained to evaluate suspected renovascular hypertension and (b) angiographically docum

77 lar tissue from neonatal and adult rats with renovascular hypertension and aortic banding, whereas ba

78 odel an absence of HO-1 leads to more severe renovascular hypertension and cardiac hypertrophy.

79 ow-up (range, 1-44 months), two patients had renovascular hypertension and none had recurrence of hor

80 es of myocardial and cerebral infarction and renovascular hypertension by 9% (3% to 16%), 13% (7% to

81 These results indicate that renovascular hypertension causes specific reductions in

82 all accuracy of baseline parenchymal MTT for renovascular hypertension detection ranged from 54% to 5

83 On the contrary, renovascular hypertension developed to the same extent (

84 es were used to examine the cost efficacy of renovascular hypertension diagnosis and treatment.

85 We show that CSD in renovascular hypertension halts further increases in blo

86 The pathophysiology of renovascular hypertension has been linked to other intra

87 Most children presenting with renovascular hypertension have few if any symptoms, but

88 We studied chronic renovascular hypertension in mice deficient in the induc

89 Screening for renovascular hypertension is not cost-effective at a pre

90 Renovascular hypertension is the most common cause of se

91 llular hyperplasia or remodeling, whereas in renovascular hypertension there is hypertrophy of vascul

92 Renovascular hypertension was produced by placing a silv

93 Effects of renovascular hypertension were then determined by using

94 esults indicate a possible role for GPR91 in renovascular hypertension, a disease closely linked to a

95 imately 5% of all hypertensive patients have renovascular hypertension, although its true incidence i

96 hat not only can renal artery stenosis cause renovascular hypertension, but it can also lead to progr

97 s simplified the management of patients with renovascular hypertension, but long-term results are not

98 cardial infarction, cerebral infarction, and renovascular hypertension, consistent with either shared

99 medicine: microalbuminuria, renal function, renovascular hypertension, dialysis (hemodialysis and co

100 week low-energy shockwave regimen attenuated renovascular hypertension, normalized stenotic kidney mi

101 mice would exhibit an early exacerbation of renovascular hypertension, NSE-AT(1a) and nontransgenic

102 cardial infarction, cerebral infarction, and renovascular hypertension, respectively, as women with n

103 rtery stenting is an effective treatment for renovascular hypertension, with a low angiographic reste

104 RABG were equally efficacious for control of renovascular hypertension.

105 ion, the swine model closely resembles human renovascular hypertension.

106 modynamics and segmental tubular function in renovascular hypertension.

107 e past 10 yr as a useful diagnostic test for renovascular hypertension.

108 potential therapeutic target for controlling renovascular hypertension.

109 al chemoreflex sensitization is a feature of renovascular hypertension.

110 ncerning the identification and treatment of renovascular hypertension.

111 eflex may be a viable therapeutic target for renovascular hypertension.

112 cardial infarction, cerebral infarction, and renovascular hypertension.

113 cerebral infarctions, and 1269 instances of renovascular hypertension.

114 cardial infarction, cerebral infarction, and renovascular hypertension.

115 rlying aberrant central neuronal function in renovascular hypertension.

116 thoexcitation and elevated blood pressure in renovascular hypertension.

117 rtery angioplasty in patients with suspected renovascular hypertension.

118 m, have improved the level of BP control for renovascular hypertension.

119 roximal tubular cells, and it may ameliorate renovascular hypertension.

120 utility of a noninvasive test for detecting renovascular hypertension.

121 and collagen deposition in the rat heart in renovascular hypertension.

122 r disease among patients suspected of having renovascular hypertension.

123 cal indications of suspected obstruction and renovascular hypertension; part 2 also summarizes the st

124 iology and real-time PCR in MNCs in sham and renovascular hypertensive (RVH) rats.

125 magnocellular neurosecretory cells (MNCs) in renovascular hypertensive (RVH) rats.

126 NCs obtained from sham, but not in MNCs from renovascular hypertensive (RVH) rats.

127 similar inward currents in MNCs of sham and renovascular hypertensive (RVH) rats.

128 imbalanced excitatory/inhibitory function in renovascular hypertensive rats (RVH).

129 subunit genes was examined in ventricles of renovascular hypertensive rats.

130 e mice but only in the clipped kidney of the renovascular knockout mice.

131 ographic techniques have been used to detect renovascular lesions, but none has yet demonstrated cons

132 ilar changes in GluR1 density are found in a renovascular model of hypertension, the DOCA-salt rat, a

133 lnerability to ventricular fibrillation in a renovascular model of LVH.

134 ency (creatinine >1.5 mg. dL(-1)) and global renovascular obstruction (bilateral renal artery stenosi

135 e performed in selected patients for primary renovascular pathology.

136 this study, we hypothesized that HHcy causes renovascular remodeling by DNA hypermethylation, leading

137 n selected patients slows the progression of renovascular renal failure and may delay the need for re

138 e and after stent insertion in patients with renovascular renal failure.

139 The responses of renovascular resistance and arterial pressure to norepin

140 etween plasma prorenin concentration and the renovascular response to captopril in diabetes supports

141 correlation between plasma prorenin and the renovascular response to captopril in patients with diab

142 ubjects, OSA subjects demonstrated decreased renovascular sensitivity (ERPF, -153 +/- 79 vs. -283 +/-

143 Future studies should evaluate the value of renovascular volume in pretransplantation kidney viabili