ライフサイエンスコーパス: renovascular hypertension

1 roximal tubular cells, and it may ameliorate renovascular hypertension.

2 cerebral infarctions, and 1269 instances of renovascular hypertension.

3 cardial infarction, cerebral infarction, and renovascular hypertension.

4 rlying aberrant central neuronal function in renovascular hypertension.

5 thoexcitation and elevated blood pressure in renovascular hypertension.

6 rtery angioplasty in patients with suspected renovascular hypertension.

7 m, have improved the level of BP control for renovascular hypertension.

8 utility of a noninvasive test for detecting renovascular hypertension.

9 and collagen deposition in the rat heart in renovascular hypertension.

10 r disease among patients suspected of having renovascular hypertension.

11 RABG were equally efficacious for control of renovascular hypertension.

12 ion, the swine model closely resembles human renovascular hypertension.

13 modynamics and segmental tubular function in renovascular hypertension.

14 e past 10 yr as a useful diagnostic test for renovascular hypertension.

15 potential therapeutic target for controlling renovascular hypertension.

16 ncerning the identification and treatment of renovascular hypertension.

17 al chemoreflex sensitization is a feature of renovascular hypertension.

18 eflex may be a viable therapeutic target for renovascular hypertension.

19 cardial infarction, cerebral infarction, and renovascular hypertension.

20 n hypertrophied ventricles from animals with renovascular hypertension (1.6-fold, P < 0.05) and aorti

21 en during arteriographic study for suspected renovascular hypertension (42%).

22 esults indicate a possible role for GPR91 in renovascular hypertension, a disease closely linked to a

23 imately 5% of all hypertensive patients have renovascular hypertension, although its true incidence i

24 al angiograms obtained to evaluate suspected renovascular hypertension and (b) angiographically docum

25 lar tissue from neonatal and adult rats with renovascular hypertension and aortic banding, whereas ba

26 odel an absence of HO-1 leads to more severe renovascular hypertension and cardiac hypertrophy.

27 ow-up (range, 1-44 months), two patients had renovascular hypertension and none had recurrence of hor

28 hat not only can renal artery stenosis cause renovascular hypertension, but it can also lead to progr

29 s simplified the management of patients with renovascular hypertension, but long-term results are not

30 es of myocardial and cerebral infarction and renovascular hypertension by 9% (3% to 16%), 13% (7% to

31 These results indicate that renovascular hypertension causes specific reductions in

32 cardial infarction, cerebral infarction, and renovascular hypertension, consistent with either shared

33 all accuracy of baseline parenchymal MTT for renovascular hypertension detection ranged from 54% to 5

34 On the contrary, renovascular hypertension developed to the same extent (

35 es were used to examine the cost efficacy of renovascular hypertension diagnosis and treatment.

36 medicine: microalbuminuria, renal function, renovascular hypertension, dialysis (hemodialysis and co

37 We show that CSD in renovascular hypertension halts further increases in blo

38 The pathophysiology of renovascular hypertension has been linked to other intra

39 Most children presenting with renovascular hypertension have few if any symptoms, but

40 We studied chronic renovascular hypertension in mice deficient in the induc

41 Screening for renovascular hypertension is not cost-effective at a pre

42 Renovascular hypertension is the most common cause of se

43 week low-energy shockwave regimen attenuated renovascular hypertension, normalized stenotic kidney mi

44 mice would exhibit an early exacerbation of renovascular hypertension, NSE-AT(1a) and nontransgenic

45 cal indications of suspected obstruction and renovascular hypertension; part 2 also summarizes the st

46 cardial infarction, cerebral infarction, and renovascular hypertension, respectively, as women with n

47 truction and 48 patients suspected of having renovascular hypertension (RVH) were randomly selected f

48 myocardial microvascular structure in swine renovascular hypertension (RVH) would be improved by sim

49 llular hyperplasia or remodeling, whereas in renovascular hypertension there is hypertrophy of vascul

50 Renovascular hypertension was produced by placing a silv

51 Effects of renovascular hypertension were then determined by using

52 rtery stenting is an effective treatment for renovascular hypertension, with a low angiographic reste