ライフサイエンスコーパス: repair protein

1 otein homolog 2 (MSH2) is a key DNA mismatch repair protein.

2 is required for loading of the Ku70/Ku80 DSB repair protein.

3 calized accumulations of ectopic 53BP1-a DNA repair protein.

4 a critical nuclear DNA double-stranded break repair protein.

5 hosphate-ribose) polymerase], a critical DNA repair protein.

6 amage is mediated by FANCD2, a DNA crosslink repair protein.

7 ne dependent on the Sir, as well as on other repair proteins.

8 nick provides an entry point for downstream repair proteins.

9 litates long-range signaling between [4Fe4S] repair proteins.

10 EIL1's interactions with other base excision repair proteins.

11 nts in transporter proteins and DNA mismatch repair proteins.

12 ns that promote efficient recruitment of DNA repair proteins.

13 pathway using conserved repair and some non-repair proteins.

14 further causes degradation of these modified repair proteins.

15 ant trigger for activating at least some DNA repair proteins.

16 tical roles in the assembly of signaling and repair proteins.

17 interact with several well-characterized DNA repair proteins.

18 is known to interact with many cellular DNA repair proteins.

19 d immunoblot analysis of DNA replication and repair proteins.

20 es, DinB error-prone DNA polymerase, and DSB-repair proteins.

21 on of local chromatin and recruitment of DNA repair proteins.

22 nserved meiotic regulators, the DNA mismatch repair proteins.

23 , thought to be important for recruitment of repair proteins.

24 osome-associated DNA lesions in chromatin to repair proteins.

25 ng a recruitment platform for checkpoint and repair proteins.

26 ty of chromatin to transcription factors and repair proteins.

27 by aiding the catalytic turnover of excision repair proteins.

28 ing the importance of the putative AddAB DNA repair proteins.

29 via hydrolysis of polyADP-ribose on related repair proteins.

30 r against toxic side effects of abundant DNA repair proteins.

31 nteractions between the MR complex and other repair proteins.

32 id men without defects in genes encoding DNA-repair proteins.

33 depends on the overexpression of alkylation repair proteins.

34 s leading to synthesis of malfunctioning DNA repair proteins.

35 on of the versatility of these base excision repair proteins.

36 changes substantially in the absence of DNA repair protein 53BP1.

37 alleles based on the accumulation of the DNA-repair protein 53BP1.

38 e ubiquitin in the recruitment of DNA damage repair proteins 53BP1 and BRCA1.

39 for understanding the success and failure of repair protein activity associated with such oxidatively

40 s a sensitive, selective, and rapid assay of repair protein activity, enabling a biological interroga

41 molecular level for how this network of DNA repair proteins acts to maintain genome stability and su

42 h TNKS1 concomitant recruitment of TNKS1 and repair proteins after damage.

43 The DNA and RNA repair protein AlkB removes alkyl groups from nucleic ac

44 Rad23 was identified as a DNA repair protein, although a role in protein degradation h

45 PCNA, the MSH2 mismatch repair protein and the XPA nucleotide excision repair (N

46 asthma, we observed increased levels of DNA repair proteins and apoptosis, as shown by caspase-3 cle

47 quitinated forms of H2A, mobilization of DNA repair proteins and enhanced viral fitness.

48 ion of several DNA double-strand break (DSB) repair proteins and formation of repair complexes, and r

49 not only damages DNA but can also modify DNA repair proteins and further causes degradation of these

50 date ClpS1 substrates, including plastid DNA repair proteins and Glu tRNA reductase, which is a contr

51 activity, XRCC1 interacts with multiple DNA repair proteins and is a subunit of distinct DNA repair

52 erns of histone modification, recruiting DNA repair proteins and modulating chromatin packing during

53 of DNA breaks facilitates recruitment of DNA repair proteins and promotes chromatin relaxation in par

54 However, how H4 acetylation (H4Ac) recruits repair proteins and reorganizes chromatin during DNA rep

55 eals previously unknown interactions between repair proteins and silencing proteins and suggests insi

56 genetically competent they also activate DNA repair proteins and stop dividing.

57 interactions between PHF11 and multiple DNA repair proteins and suggests that PHF11 mediates 5' end

58 ing DNA damage to phosphorylate specific DNA repair proteins and/or that NMD inactivation may lead to

59 n early glycation intermediates and releases repaired proteins and lactate or glycolate, respectively

60 B is caused by loss of dysferlin, a membrane repair protein, and LGMD 2C is caused by loss of the dys

61 easured DNA double-strand breaks (DSBs), DNA repair proteins, and apoptosis in an HDM-induced allergi

62 nt carcinogenesis (such as p53, DNA mismatch repair proteins, and DNA base excision-repair proteins),

63 on proteins, transcription factors, mismatch repair proteins, and DNA polymerases discriminate betwee

64 ) forms complexes with CtIP as well as other repair proteins, and is essential for HR repair by regul

65 ribose) include PARP-1 itself, histones, DNA repair proteins, and transcription factors.

66 in APTX, which encodes the DNA strand-break repair protein aprataxin (APTX).

67 representing the CCHH zinc finger of the DNA repair protein aprataxin, but did bind to an aprataxin p

68 PTX gene, which encodes the DNA strand-break repair protein aprataxin.

69 A base excision repair protein, Apurinic/apyrimidinic (AP) endonuclease

70 The essential base excision repair protein, apurinic/apyrimidinic endonuclease 1 (AP

71 s proteins accumulated at DSB sites, how DNA repair proteins are assembled into damaged chromatin has

72 However, although mutations in DSB repair proteins are common in hereditary cancers, mechan

73 -glycoprotein complex and the known membrane repair proteins are expressed normally, membrane reseali

74 The presence of an active repair protein as an essential component of the polymera

75 mplexes containing DNA replication and other repair proteins, as shown by gel filtration.

76 SET led to loss of expression of several DNA repair proteins, as well as decreased recruitment of DNA

77 Our findings emphasize that damage to DNA repair proteins, as well as to DNA itself, is likely to

78 can partner with MSH2, an important mismatch repair protein associated with hereditary non-polyposis

79 mitted to SSA and show that the Rad22(Rad52) repair protein associates robustly with sequences adjace

80 quitylation events that promote retention of repair proteins at DNA lesions.

81 ine the organization and interaction of NHEJ repair proteins at DSB ends.

82 analyzed the dynamics of interaction of DNA repair proteins at individual DSB sites in living cells

83 and show via immunofluorescent detection of repair proteins at ionizing radiation-induced DNA damage

84 subunits that assist in the assembly of DNA repair proteins at sites of damage.

85 tion of nucleosomes and concentration of DNA-repair proteins at the site of damage.

86 e more dispersedly occupied by the conserved repair protein, AtBRCA1, which can also co-localise with

87 ing a positive feedback loop between the DNA repair proteins ATM and VDR.

88 difficult in the case of base extrusion DNA repair proteins because of the complexity of the repair

89 Little is known about Tpa1 as a repair protein beyond the initial observation from a hig

90 uclear exploration is regulated by the Rad51 repair protein, both at the break and throughout the gen

91 re that, together with a number of other DNA repair proteins, both ATR and its associated protein, AT

92 ites of DNA damage along with the DNA damage repair protein, BRCA1, and FUS and TDP43 participate in

93 that the mRNA and protein levels of the DNA repair protein BRIP1 [Fanconi anemia gene J (FANC J)] ar

94 D4 deficiency impairs the recruitment of DNA repair proteins BRIT1, BRCA1, and replication protein A

95 LexA represses the transcription of many DNA repair proteins by binding to SOS 'boxes' in their opera

96 Modulation of DNA repair proteins by small molecules has attracted great i

97 lomere-specific shelterin components and DNA repair proteins, (c) the nontelomeric functions of TERT

98 The nucleotide excision repair protein complex ERCC1-XPF is required for incisio

99 pose that protein stability, mediated by DNA repair protein complex formation, functions as a regulat

100 e amplification when the function of the DSB repair protein complex MRN (Mre11/Rad50/Nbs1) is impaire

101 A damage checkpoint protein 1 (MDC1) and DNA repair protein complex to sites of DNA breaks for DNA re

102 ir proteins and is a subunit of distinct DNA repair protein complexes.

103 nalysis of endonuclease III (EndoIII), a DNA repair protein containing a [4Fe-4S] cluster known to be

104 Arsenic interaction with DNA repair proteins containing functional zinc finger motifs

105 aracterization of the redox chemistry of DNA repair proteins containing redox cofactors, and multiple

106 We have proposed a model where repair proteins containing redox-active [4Fe-4S] cluster

107 DNA repair protein counteracting oxidative promoter lesions

108 Cellular mechanisms aimed at repairing protein damage and maintaining homeostasis, wi

109 The DNA repair protein damaged DNA-binding 2 (DDB2) has been imp

110 a novel function of the nucleotide excision repair protein DDB2 in the accumulation of ROS in a mann

111 o reliably identify tumors with DNA mismatch repair protein deficiency (MMR-D) on the basis of increa

112 in the nonhomologous end-joining (NHEJ) DNA repair protein DNA ligase IV (LIG4) lead to immunodefici

113 romoter methylation of the gene encoding the repair protein DNA methyltransferase MGMT, although othe

114 ction of NEIL1 with downstream base excision repair proteins DNA polymerase beta and flap endonucleas

115 Inhibition of the DNA repair proteins DNA-dependent protein kinase (DNA-PK) or

116 ensive assessment of the localization of DSB repair proteins during KSHV replication, we have determi

117 leus, including a dozen that function as DNA repair proteins (e.g., BRCA1, 53BP1) or nucleases (e.g.,

118 we found that vorinostat suppressed DNA DSB repair proteins, e.g., RAD50, MRE11, in cancer but not n

119 orectal cancer cells deficient in DNA damage repair proteins, EME1 and MUS81, were significantly more

120 uction potential found experimentally in DNA repair proteins, enabling their HiPIP-like redox behavio

121 S] clusters regulates the ability of two DNA repair proteins, Endonuclease III and DinG, to bind pref

122 The DNA repair proteins ERCC1 and XPF are needed to remove cispl

123 ing activities of two well-characterized DNA repair proteins, Escherichia coli MutS and human p53.

124 MI was found to be correlated with mismatch repair protein expression, MSI, BRAF (V600E) mutation st

125 interactions with other nucleotide excision repair protein factors of the two enzymes.

126 Intriguingly, ICL repair protein, Fanconi anemia complementation group A p

127 interaction of related double-stranded break repair proteins, finally causing the increase of unrepai

128 we demonstrated that the DNA replication and repair protein Flap endonuclease 1 (FEN1) is required fo

129 gesterone) on gene expression related to DNA repair, protein folding (chaperones), the ubiquitin-prot

130 of phospho-Smad2 (pSmad2) and Smad7 with DSB repair proteins following low and high linear energy tra

131 ate treatment, suggesting that APE1 is a key repair protein for glutamate-induced DNA damage.

132 nine-DNA alkyltransferase (MGMT) is the sole repair protein for O(6)-alkylguanine lesions in DNA and

133 low the sequential recruitment of downstream repair proteins for successful execution of NER.

134 e efficient DNA resection and recruitment of repair proteins; for example, inward translocation of th

135 cation compartments (RCs), whereas other DSB repair proteins form foci outside RCs.

136 e-transport signaling in E. coli between DNA repair proteins from distinct pathways.

137 nt mechanistic hypotheses regarding mismatch repair protein function in mediating triplet repeat expa

138 ut the extent of their interactions with DNA repair proteins has not been thoroughly characterized.

139 Multiple DNA repair proteins having ubiquitin ligase activity are rec

140 d similar telomere loss, suggesting that the repair proteins help to resolve similar problems in telo

141 The mismatch repair protein hMSH2 was found to be a major ATR-binding

142 heterogeneous nuclear levels of the mismatch repair protein hMSH3.

143 show that the mammalian nucleotide excision repair protein homolog MMS19 can simultaneously bind pro

144 physiological Ku70, an essential DNA damage repair protein in neurons whose function is known to be

145 ude that Tpa1 is a hitherto unidentified DNA repair protein in yeast and that it plays a crucial role

146 (ERFSs), by genome-wide localization of DNA repair proteins in B cells subjected to replication stre

147 Although DNA repair proteins in bacteria are critical for pathogens'

148 we examined the role of double-strand-break repair proteins in gene silencing and nuclear organizati

149 matin organization and suggest a role for HR repair proteins in genomic organization.

150 oscopy as methods to monitor kinetics of DSB repair proteins in living cells under conditions mimicki

151 rting the host defense, the role of host DNA repair proteins in response to bacterial infection is po

152 ein kinase, catalytic subunit, essential DNA repair proteins in the nonhomologous end-joining pathway

153 nce for coordinated DNA CT between different repair proteins in their search for damage in the genome

154 Nol12 co-localizes with DNA repair proteins in vivo including Dhx9, as well as with

155 tion of DNA-PK, a non-homologous end joining repair protein, in Hec-108 cells.

156 bstantial number of immunomodulatory and DNA repair proteins including CXCL9, CD3D, and RAD51 were fo

157 nates two substrates, and several downstream repair proteins including nucleases and homologous recom

158 cruitment of a subset of double-strand break repair proteins including RAD51 and 53BP1 to repair foci

159 The n-terminal tail of histone H4 recruits repair proteins, including 53BP1, to DNA double-strand b

160 Repair proteins, including dysferlin, EHD1, EHD2, MG53,

161 f BRCA1 in repair complexes; upregulation of repair proteins, including HMGN1 and RFC1; and regulatio

162 The binding of DNA repair proteins, including p53 and BRCA2, has been visua

163 lin D1 interactors revealed a network of DNA repair proteins, including RAD51, a recombinase that dri

164 ta-CASP motif, which is found in a number of repair proteins, including the DNA double-strand break (

165 ctions as a scaffold protein to assemble DNA repair proteins into chromatin followed by efficient act

166 lguanine-DNA methyltransferase (MGMT), a DNA repair protein involved in chemotherapeutic resistance o

167 owever, when a CT-deficient mutant of either repair protein is combined with the CT-proficient repair

168 Activation of DNA repair proteins is often accompanied by an arrest in cel

169 A feature of many base excision repair proteins is that they contain [4Fe4S] clusters th

170 A poorly understood aspect of DNA repair proteins is their ability to identify exceedingly

171 A common feature of DNA repair proteins is their mobilization in response to DNA

172 MutL is a mismatch repair protein known to coordinate several steps in repa

173 PAXX interacts directly with the DSB-repair protein Ku and is recruited to DNA-damage sites i

174 ecognition of each broken DNA end by the DNA repair protein Ku is the first step in NHEJ, followed by

175 man GBM cells had enhanced activation of DNA repair proteins, leading to increased DNA repair and sub

176 ing localize to sites of damage and some DNA repair proteins localize to heterochromatin, but the bio

177 All incident CRCs were analyzed for mismatch repair protein loss, supported by BRAF mutation and micr

178 The mammalian repair protein MBD4 (methyl-CpG-binding domain IV) excis

179 We identify two distinct mechanisms for repair protein-mediated silencing-via direct and indirec

180 ions differing only in expression of the DNA repair protein methyltransferase (MGMT), a TMZ-sensitivi

181 ast majority of cells, we found that the DNA repair protein Metnase (also SETMAR), which has a SET hi

182 ges and occurs independently of the membrane repair protein MG53.

183 Dysferlin interacts with a new membrane repair protein, mitsugumin 53 (MG53), an E3 ubiquitin li

184 Mlh1 (the gene that encodes the DNA mismatch-repair protein Mlh1) displayed dramatically higher incid

185 stability from reduction of the DNA mismatch repair protein MLH1, are unaffected by CENP-E-dependent

186 sites marked by the binding of the mismatch repair protein MLH1.

187 y those with dMMR, based on loss of mismatch repair proteins MLH1, MSH2, MSH6, and/or PMS2.

188 s used to analyze the presence of 4 mismatch repair proteins (MLH1, MSH2, MSH6, and PMS2) in these tu

189 E1b and E4 proteins target the cellular DNA repair protein Mre11 for redistribution and degradation,

190 Also, protein levels of DNA repair protein Mre11 were decreased in the G(2) phase of

191 otruding nonhomologous 3' tail, the mismatch repair protein Msh2 does not discourage homeologous reco

192 tivity of DNA polymerase-delta, although the repair proteins Msh2, Mlh1 and Exo1 influence the extent

193 inds cooperatively with UNG and the mismatch repair proteins Msh2-Msh6 to Ig Smu and Sgamma3 regions,

194 lusterin modulated the expression of the DNA repair proteins, MSH2, MSH6, OGG1 and BRCA1.

195 Cells lacking mismatch repair proteins Msh6 and Mlh1 form chimeric recombinants

196 DNA repair proteins must locate rare damaged sites within th

197 its weak-binding partners, the DNA mismatch repair protein MutL.

198 the long-range communication in DNA mismatch repair proteins MutS and its homologs where intramolecul

199 Here we use the postreplicative mismatch repair proteins MutSalpha and MutLalpha as model systems

200 e presence, not the absence, of the mismatch repair protein MutSbeta (Msh2-Msh3 heterodimer).

201 7, the transcriptional repressor TRIM27, DNA repair proteins NBN and MRE11A, regulators of apoptosis,

202 transmission of HSV-1 and found that the DNA repair protein Nbs1 from only some primate species is ab

203 MCM, RPA), checkpoint protein Rad26 and DNA repair protein Nbs1 to telomeres.

204 , in part, through reduced activation of the repair protein Nijmegen Breakage Syndrome-1 but not p53.

205 hylguanine due to elevated expression of the repair protein O(6)-methylguanine-DNA methyltransferase

206 (iRFP670), with the latter fused to the DNA repair protein O(6)-methylguanine-DNA-methyltransferase

207 ses S-nitrosylation and depletion of the DNA repair protein O6-alkylguanine-DNA-alkyltransferase (AGT

208 Expression of the gene encoding the DNA repair protein O6-methylguanine DNA methyltransferase (M

209 CHD4 is recruited by the excision repair protein OGG1 for oxidative damage to interact wit

210 DDAs by interfering with trafficking of DNA repair proteins on interphase microtubules.

211 investigating the influence of specific DNA repair proteins on the biological response toward DNA da

212 lutionally conserved antioxidant enzyme that repairs protein oxidation by reversing the oxidative mod

213 of shot1 thermotolerance, indicating HSP101 repairs protein oxidative damage and/or reduced oxidativ

214 The DNA repair protein p53 binding protein 1 (53BP1) protects th

215 oth CCHC DNA-binding zinc fingers of the DNA repair protein PARP-1 (poly(ADP-ribose) polymerase-1).

216 zymatic cofactor for enzymes such as the DNA repair protein PARP.

217 ficant DNA damage in host cells and that DNA repair proteins play a critical role in the host respons

218 The DNA mismatch repair protein PMS2 was recently found to encode a novel

219 otential synthetic lethal partner of the DNA repair protein polynucleotide kinase/phosphatase (PNKP).

220 We also examined expression of the DNA repair protein Rad18, which is regulated by MTDH at the

221 The human homolog of the yeast DNA repair protein RAD23, hHR23A, has been found previously

222 omplex [meiotic recombination 11 (Mre11)/DNA repair protein Rad50/Nijmegen breakage syndrome 1 protei

223 ch correlated with reduced levels of the DNA repair protein Rad51 and elevated levels of p53.

224 The DNA repair protein RAD51 and the trans-lesion synthesis DNA

225 hesis that Jab1 positively regulates the DNA repair protein Rad51 and, in turn, cellular response to

226 P = 4.51 x 10(-5)) between a SNP in the DNA repair protein RAD51 homolog 2 gene (RAD51L1; rs10483813

227 foci of DNA damage marker gammaH2AX and the repair protein Rad51 in damaged cells.

228 set of foci of the homologous recombination repair protein Rad51 that are colocalized with Nbs1 and

229 c or pharmacologic suppression of the HR DNA repair protein RAD51.

230 ous p220 aberrantly directs at least one DNA repair protein, RAD51, to damage sites, where their acti

231 es and regulates the recruitment of the core repair protein, RAD51, to hydroxyurea-induced foci.

232 The DNA repair protein RAD52 is an emerging therapeutic target o

233 protein RPA and the homologous recombination repair protein Rad52.

234 ased in the knockouts of postreplication DNA repair proteins, Rad6 and Rad5, and the DNA helicase Sgs

235 Since mismatch repair proteins recognize mismatches containing both nor

236 el reporter that allows for visualization of repair protein recruitment and local transcription in si

237 ike proteins (ATLs) are a novel class of DNA repair proteins related to O(6)-alkylguanine-DNA alkyltr

238 lcytosine (mC), and downstream base excision repair proteins restore a G.C pair.

239 on gene NTHL1, which encodes a base excision repair protein, revealed a mutational footprint (signatu

240 athway that included DNA double-strand break repair proteins RexAB, recombinase A, and polymerase V.

241 s, likely by facilitating the recruitment of repair proteins similar, but not identical, to its propo

242 How DNA repair proteins sort through a genome for damage is one

243 lation of double-stranded DNA (dsDNA) damage-repair proteins subsequent to RNF8 accrual, and for prof

244 As a result, DNA repair proteins such as BRCA1, RAP80, and Rad51 cannot b

245 A adducts, is linked to their recognition by repair proteins such as HMGB1a.

246 alyzed repair outcomes in the absence of DNA repair proteins such as KU70 and LIG4 (both involved in

247 f viral genes coding for other base excision repair proteins suggest that pol X functions in a manner

248 articipate in interaction with MLH1 mismatch-repair protein, suggesting that the FANCJ activity suppo

249 How human DNA repair proteins survey the genome for UV-induced photopr

250 (HBD, HBB, IL36G), and decreased epithelial repair proteins (TFF3, F11R).

251 1) is a multi-functional double-strand break repair protein that is essential for class switch recomb

252 However, Metnase has evolved as a DNA repair protein that is specifically involved in nonhomol

253 MutS is a DNA repair protein that recognizes unpaired and bulged bases

254 inding site for p53 and other DNA damage and repair proteins that contain amphipathic alpha helical d

255 icing activity arose to post-translationally repair proteins that had been inactivated by deleterious

256 reatments were associated with damage to DNA repair proteins that reduced the efficiency of nucleotid

257 allow the virus to evade localization of DSB repair proteins that would otherwise have a detrimental

258 Cyt b(5) is the electron-carrier "repair" protein that reduces met-Mb and met-Hb to their

259 irus (HCMV) selectively relocalizes many DNA repair proteins, thereby avoiding a potentially detrimen

260 f biological macromolecules such as mismatch repair proteins through biotinylated DNA substrates.

261 cate a general link between the ability of a repair protein to carry out DNA CT and its ability to re

262 emonstrate a link between the ability of the repair protein to carry out DNA CT and its ability to re

263 ic strength influence the binding of two DNA repair proteins to a variety of DNA structures.

264 trand breaks (DSB), promoting recruitment of repair proteins to chromatin.

265 d to modulate the DDR by affecting access of repair proteins to chromatin.

266 ng affinity regulates the ability of [4Fe4S] repair proteins to collaborate in the lesion detection p

267 histone octamers, however, impedes access of repair proteins to DNA damage.

268 In mammalian cells the accumulation of repair proteins to double-strand breaks is a phosphoryla

269 hat accelerates cell recovery by helping DNA repair proteins to locate their targets, and facilitatin

270 DNA, a property that may be explored by the repair proteins to locate these lesions.

271 Migration also causes multiple DNA repair proteins to segregate away from DNA, with cytopla

272 tion in which it directly interacts with DNA repair proteins to sensitize prostate cancer cells to th

273 e PARylation orchestrates the recruitment of repair proteins to sites of damage.

274 wn as crucial facilitators of recruitment of repair proteins to sites of DNA damage.

275 ins, as well as decreased recruitment of DNA repair proteins to sites of DNA double-strand breaks (DS

276 verse contributions of several recombination/repair proteins to telomere maintenance in Ustilago mayd

277 uitination and recruitment of additional DSB repair proteins to the break.

278 aces to recruit multiple postreplication DNA repair proteins to the CRL4-DCAF1 E3 ligase for ubiquiti

279 DNA replication and recruit other checkpoint/repair proteins to the DNA lesions.

280 tokines, caused hMSH3, but no other mismatch repair proteins, to move from the nucleus to the cytosol

281 We conclude DNA damage-repair proteins traffic on microtubules and addition of

282 match repair proteins, and DNA base excision-repair proteins), transcription factors (such as nuclear

283 bility including cell cycle checkpoints, DNA repair, protein ubiquitination, chromatin remodelling, t

284 Similar to other DNA repair proteins, USP20 is phosphorylated post DNA damage

285 e complex formed between the prokaryotic DNA repair protein UvrA2 and DNA.

286 reserves the pool of reduced thioredoxin for repairing proteins vital to survival.

287 only treated cells; the transcription of DNA repair proteins was also reduced.

288 cells; and significant up-regulation of DNA repair proteins was observed after co-exposure.

289 DNA lesions also interact with base excision repair proteins, we investigated whether CREB1 and repai

290 lguanine-DNA alkyltransferase (AGT) is a DNA repair protein which removes alkyl groups from the O-6 p

291 MSH2 is a key DNA mismatch repair protein, which plays an important role in genomic

292 inase/phosphatase (PNKP), a DNA strand break repair protein with DNA 5'-kinase and DNA 3'-phosphatase

293 utilize DNA-modified electrodes to generate repair proteins with [4Fe4S] clusters in the 2+ and 3+ s

294 redox signal resembles that of base excision repair proteins, with a DNA-bound redox potential of ~80

295 NA damage-regulated complex with the key DNA repair protein xeroderma pigmentosum A (XPA).

296 ty associated with the deficiency of the DNA repair protein xeroderma pigmentosum type A (XPA).

297 FA core complex requires nucleotide excision repair proteins XPA and XPC for its association.

298 ome group B (CSB), but not the global genome repair protein XPC.

299 2 and is required for recruitment of the ICL repair protein, XPF, to damage-induced foci at telomeres

300 inhibition abolishes the recruitment of the repair proteins XRCC1 and polymerase beta at damaged tel