ライフサイエンスコーパス: repetitive stimulation

1 ious reduced responsiveness to continuous or repetitive stimulation.

2 ocytosis evoked by single depolarizations or repetitive stimulation.

3 nes the efficacy of neurotransmission during repetitive stimulation.

4 ed them to undergo temporal summation during repetitive stimulation.

5 when Ca(2+) concentrations accumulate during repetitive stimulation.

6 (50), WT=318.0 and DG=176 micromol/L) during repetitive stimulation.

7 n the cytosolic [Ca(2+)] transient evoked by repetitive stimulation.

8 root and inhibit wind-up response evoked by repetitive stimulation.

9 miting the rise of cytosolic [Ca(2+)] during repetitive stimulation.

10 otransmitter release and rapid fatigue after repetitive stimulation.

11 their ability to sustain transmission during repetitive stimulation.

12 ermeable AMPA receptors are facilitated upon repetitive stimulation.

13 lex effects on transmitter release evoked by repetitive stimulation.

14 ned with either continuous or infrequent but repetitive stimulation.

15 ferent synapses drives greater LTP following repetitive stimulation.

16 s weaken and even silence ASIC1a currents to repetitive stimulation.

17 es Pr from the steady state amplitude during repetitive stimulation.

18 ked to account for NGIC desensitization upon repetitive stimulation.

19 ritical in understanding how channels act to repetitive stimulation.

20 greater loss of channel availability during repetitive stimulation.

21 a depressed postsynaptic depolarization with repetitive stimulation.

22 a stimulus-history-dependent recovery after repetitive stimulation.

23 to enhanced postsynaptic depolarization with repetitive stimulation.

24 in a repressed state and are activated upon repetitive stimulation.

25 responses to single-pulse and high-frequency repetitive stimulation.

26 l transmission failure during high-frequency repetitive stimulation.

27 s accompanied by increased depression during repetitive stimulation.

28 on either quantal content or the response to repetitive stimulation.

29 he sarcoplasmic reticulum calcium store with repetitive stimulation.

30 n ensuring effective transmitter output with repetitive stimulation.

31 ynapse and an impaired response to prolonged repetitive stimulation.

32 ificant inhibition of exocytosis only during repetitive stimulation.

33 ially reduced, appearing as sensitization to repetitive stimulations.

34 ique in that its activity is sensitized upon repetitive stimulations.

35 alcium ions in the sensitization of TRPV3 to repetitive stimulations.

36 in synaptic response to a prolonged train of repetitive stimulation (12.5 Hz, 300 pulses) capable of

37 Synaptic inputs were reliably induced by repetitive stimulation, although with large variation in

38 Cutaneous depolarizations summed during repetitive stimulation and > 0.05 Hz.

39 tamate release from axons was facilitated by repetitive stimulation and could be inhibited through ac

40 ty that increases transmitter release during repetitive stimulation and decays thereafter with a time

41 ulfils the requirements for voltage control, repetitive stimulation and high temporal resolution, but

42 AA-mediated IPSCs are strongly influenced by repetitive stimulation and neuromodulation.

43 Evoked D2-IPSCs could be driven by repetitive stimulation and were not occluded by backgrou

44 Bursts appeared after repetitive stimulation and were stable for the life of t

45 t fused by compound exocytosis during strong repetitive stimulation and, thus, that vesicles tethered

46 1(-/-) mice showed robust proliferation with repetitive stimulations and strong resistance to stimula

47 s in the extent of depression in response to repetitive stimulation, and (4) release failures at some

48 kinetics, progressive EPSC inhibition during repetitive stimulation, and extrasynaptic NMDAR inhibiti

49 The somatic APs declined in amplitude with repetitive stimulation, and modest reduction of AP ampli

50 in single units were depleted of glycogen by repetitive stimulation, and studied histologically in fr

51 IPSCs decreased during repetitive stimulation, and the depression increased at

52 t on [Ca2+]i, exhibited desensitization with repetitive stimulation, and was regulated by PKCzeta.

53 ow membrane potentials, and the responses to repetitive stimulation are explained by the known prefer

54 t reflect a decrease in the sensitization to repetitive stimulation are most likely centrally mediate

55 en exposed to prolonged tonic GABA or during repetitive stimulation, as may occur during learning and

56 an in large cells ( approximately 2 ms), and repetitive stimulation at 20-150 Hz evoked greatly summa

57 e compound muscle action potential (CMAP) on repetitive stimulation at 3 Hz, and increased jitter and

58 a2+ that enters motor nerve terminals during repetitive stimulation at frequencies exceeding 10-20 Hz

59 In contrast, repetitive stimulation at restrictive temperatures revea

60 ion and dramatic facilitation in response to repetitive stimulation at the gamma frequency.

61 smission fails to respond to high-frequency, repetitive stimulation at the NMJs of UCH-L1 knockout mi

62 With high-frequency repetitive stimulation, both components of the IPSP show

63 e factors governing synaptic strength during repetitive stimulation, both in control conditions and d

64 he slow component becomes prominent during a repetitive stimulation, but its time constant is unchang

65 Repetitive stimulation by LSS of oocytes expressing ENaC

66 A new paper in Science reveals how repetitive stimulation can identify and help to repair f

67 calcium decay and augmented response during repetitive stimulation can serve as in vivo imaging biom

68 l recording from granule cells revealed that repetitive stimulation causes a calcium- and Ih-dependen

69 Frequency facilitation, during which repetitive stimulation causes a reversible growth in syn

70 lar synapses revealed that in the absence of repetitive stimulation, comt synapses exhibit wild-type

71 resynaptic boutons are mostly functional and repetitive stimulation did not induce additional enhance

72 pentyladenosine or baclofen, suggesting that repetitive stimulation does not relieve the G-protein in

73 We find that repetitive stimulation dramatically alters the amplitude

74 Repetitive stimulation elicited spikes within a 0.1 ms t

75 ally fully occupied receptors become active, repetitive stimulation elicits currents with distinct wa

76 ctive transmitter output with high-frequency repetitive stimulation, exhibiting both severe initial d

77 ay activity-dependent slowing (ADS), whereby repetitive stimulation (>/=1 Hz) results in a progressiv

78 The majority of vesicles, released during repetitive stimulation, have low release probability (p

79 rom the changes in transmitter release after repetitive stimulation, identification of augmentation w

80 manipulation of Doc2 but was enhanced during repetitive stimulation in Doc2 knockdown neurons, potent

81 on the pattern of responses recorded during repetitive stimulation in either strain.

82 al mechanism for tension potentiation due to repetitive stimulation in fast-twitch skeletal muscle.

83 icant increase in axonal F-actin level after repetitive stimulation in immature but not mature neuron

84 In contrast, repetitive stimulation in the gamma frequency range ( ap

85 lity of maintaining contractile force during repetitive stimulation in the presence of 2.5 mM extrace

86 dent pathology was observed in response to a repetitive stimulation in which subsequent stimuli were

87 This repetitive stimulation, in general, increased the area o

88 tory responses that quickly plateaued during repetitive stimulation, indicating that the degree of fa

89 input and this pattern remained unchanged by repetitive stimulations, indicating that vagal input ste

90 at triggering of asynchronous release during repetitive stimulation involves expansion of the same ca

91 inal mitochondrial function brought about by repetitive stimulation is a rapid acceleration of electr

92 Synaptic depression produced by repetitive stimulation is likely to be particularly impo

93 Inactivation during brief, repetitive stimulation is primarily attributed to closed

94 but the background activity increased during repetitive stimulation leading to a prolonged after-disc

95 s for short-term enhancement may explain why repetitive stimulation more readily induces LTP in the s

96 rbated by a single dose and is attenuated by repetitive stimulation of AhR.

97 Furthermore, repetitive stimulation of BLA afferents at low (2 Hz) or

98 Ca2+ imaging and found that properly timed, repetitive stimulation of both pathways results in the g

99 ple spike (SS) activity of the cell, in that repetitive stimulation of CFs causes a decrease in SS ac

100 ate potential size ([integral]MEPP) followed repetitive stimulation of contracting preparations.

101 Repetitive stimulation of either the presynaptic neuron

102 Repetitive stimulation of excitatory premotoneurons mimi

103 appeared on the ribbon in cells fixed during repetitive stimulation of exocytosis, and in some cases

104 Repetitive stimulation of glutamatergic synapses in olfa

105 fails entirely, in a cyclical manner, after repetitive stimulation of motor axons in CD24 mutant mic

106 Repetitive stimulation of parallel fibers caused a long-

107 Repetitive stimulation of presynaptic auditory nerve fib

108 In patients with IBS, repetitive stimulation of sigmoid splanchnic afferents r

109 Repetitive stimulation of synaptic inputs at frequencies

110 Repetitive stimulation of T-cell receptor (TCR) with cog

111 Repetitive stimulation of the dorsal thalamus at 7-14 Hz

112 temporal lobe epilepsy, is developed through repetitive stimulation of the hippocampus and leads to i

113 tivated from the LRN showed late EPSPs after repetitive stimulation of the pyramid.

114 oneurones (19%) responded with late EPSPs to repetitive stimulation of the pyramid; only 3% had segme

115 Our investigations revealed that repetitive stimulation of the T-cell receptor (TCR) indu

116 Repetitive stimulation of the ventral medial geniculate

117 (1) Layers II/III responses to repetitive stimulation of the white matter become increa

118 fficient antigen presenting cells (APCs) for repetitive stimulations of antigen-specific T cells in v

119 Repetitive stimulations of climbing fibers resulted in a

120 al with relatively low quantal variance, but repetitive stimulation often induced substantial changes

121 al periodic total transmission failures with repetitive stimulation point to a defect in vesicle mobi

122 Repetitive stimulation potentiates contractile tension o

123 ze in the continuous presence of agonists, a repetitive stimulation protocol was used to evaluate the

124 uscular junction, facilitation elicited by a repetitive stimulation reaches a plateau level that is p

125 l for maintaining transmitter release during repetitive stimulation, regulation of endocytosis could

126 st augmentation of spontaneous release after repetitive stimulation relative to the D2 strain.

127 s, and the actions of neuromodulators during repetitive stimulation result from their inhibition of i

128 Modelling autoreceptor activation during repetitive stimulation revealed that as P declines, the

129 in proper synaptic transmission at NMJs upon repetitive stimulation, similar to Drp1 fission mutants.

130 During repetitive stimulation, Slo effectively compensates for

131 ive clinical measures of muscle strength and repetitive stimulation studies as end points.

132 e use of small interfering RNA knockdown and repetitive stimulation studies, to show that cannabidiol

133 Repetitive stimulation suggested partial desensitization

134 stic, use-dependent inhibition during rapid, repetitive stimulation, suggesting that the drug prefere

135 nt synaptic responses showed potentiation to repetitive stimulation, suggestive of a lower transmitte

136 n in action current in IB4 +ve neurones with repetitive stimulation supports a novel hypothesis expla

137 steeper short-term depression in response to repetitive stimulation than those on Pyr neurons.

138 With repetitive stimulation, the peak amplitude of the GABAA

139 On repetitive stimulation, the strength of a reflex control

140 the cell pairs studied, we applied a direct repetitive stimulation to the real cell, making it the "

141 he vagally evoked IOS after 'training' using repetitive stimulation trials.

142 nit were distinguished by their responses to repetitive stimulation: type 1 units slowed progressivel

143 the TTS lumen during, and immediately after, repetitive stimulation under physiological conditions.

144 The local spillover produced by a repetitive stimulation was compared with the long-range

145 Repetitive stimulation was imposed at selected BZ sites.

146 Repetitive stimulation was not required for E2-induced i

147 imulation was increased and sensitization to repetitive stimulations was decreased by increasing the

148 all three drugs, use-dependent block during repetitive stimulations was sharply reduced, and the rat

149 To investigate mitochondrial responses to repetitive stimulation, we measured changes in NADH fluo

150 The postsynaptic effects of (1S,3R)-ACPD and repetitive stimulation were both antagonized by MCPG, su

151 Responses during repetitive stimulation were far more sustained for matri

152 pses to maintain effective transmission with repetitive stimulation whereas the 140 and/or 120 isofor

153 of presynaptic functional boutons induced by repetitive stimulation, whereas actin polymerizer jaspla

154 caused increased synaptic depression during repetitive stimulation, whereas the D238N mutation did n

155 t synaptic fatigue phenotype within 20 ms of repetitive stimulation, which cannot be explained by ves

156 in the amplitude of Ca 2+ signals evoked by repetitive stimulation with ATP.

157 y factor 3 and NF-kappaB signaling, and that repetitive stimulation with pIC, but not IL-1, further i

158 Repetitive stimulation with spaced patterns, however, ca

159 Synapses respond to brief, repetitive stimulation with synaptic depression when ini

160 Repetitive stimulation with the same agonist concentrati

161 On the other hand, repetitive stimulation with the same agonist induced Ca2

162 Repetitive stimulation with trains of multiple pulses (5

163 Repetitive stimulations with HNE in 2 of 5 HLA-A*0201+ i

164 urotransmitter-gated ion channels (NGICs) to repetitive stimulation without interference from presyna