ライフサイエンスコーパス: replication control

1 the link between chromatin assembly and DNA replication control.

2 upon prolonged growth suggesting a defect in replication control.

3 /6 inhibition had no effect on cell cycle or replication control.

4 d be unambiguously linked to its role in DNA replication control.

5 imers can bind to iterons, a new paradigm in replication control.

6 y partially due to alterations in centrosome replication control.

7 recG mutant that shows defects in repair and replication control.

8 OB1 bypass activity and other aspects of DNA replication control.

9 ation origin, thus elucidating a key step of replication control.

10 ssing DnaBS371P are consistent with aberrant replication control.

11 plex picture emerging for papillomavirus DNA replication control.

12 that were proposed to play distinct roles in replication control.

13 iral ISG expression, which results in better replication control.

14 e I elements, the only known determinant for replication control.

15 h is believed to be the major determinant of replication control.

16 sults is discussed in the context of plasmid replication control.

17 evidence in support of the coupling model of replication control.

18 hase, illustrating the dynamic nature of DNA replication controls.

19 Cdks may inhibit ORC function as part of re-replication control and show that DmORC activity may be

20 tion between facultative heterochromatin and replication control and show that heterochromatin machin

21 oth inhibitory and facilitating roles in DNA replication control and that the sole essential function

22 ight be associated with its function in cell replication control, and that p53-independent mechanisms

23 of the F plasmid as a model system to study replication control, and these investigations led to the

24 conventional mice for investigation of viral replication, control, and pathogenesis.

25 rogrammed changes in chromosome structure in replication control are discussed.

26 tivators; (3) high-copy plasmids with sloppy replication control arise because intracellular selectio

27 1 degradation plays an essential role in DNA replication control, as overexpression of Cdt1 leads to

28 We propose that perturbation of the re-replication control by overexpression of HsORC1 is due t

29 s of data support the hypothesis of negative replication control by pi binding to the non-iteron site

30 We discuss new insights into plasmid replication control by pi dimers.

31 within the Rif1 sequence, it is likely that replication control by Rif1 through PP1 is a conserved m

32 CDC16 and CDC27 may contribute to replication control by targeted proteolysis of an S phas

33 Ad-sPSA-E1, an adenoviral vector with viral replication controlled by a strong super prostate-specif

34 These suppressors suppressed the replication control defects of mutants in seqA alone and

35 os, revealing an unexpected role for Cdk1 in replication control during development.

36 that compounds that do not bind to this RNA replication control element fail to induce plasmid loss

37 ces upstream of rctBp, implicated earlier in replication control, enhanced the repression.

38 Nucleosome assembly following DNA replication controls epigenome maintenance and genome in

39 The replication control genes of bacterial plasmids face sel

40 to determine if viral reprogramming of host replication controls had an impact on host DNA replicati

41 aster host, we have tracked the evolution of replication control in both somatic and reproductive tis

42 , E2f2, Dp, and Rbf1 genes all contribute to replication control in ovarian follicle cells, which bec

43 DNA replication control involves DnaA, which promotes initia

44 romosome generally match in copy number, the replication control is apparently lost over several mega

45 tensively studied, the role of CDK4/6 in DNA replication control is less understood.

46 ticular it is hypothesized that: (1) tighter replication control is more vulnerable to selfishness; (

47 It is also discussed how replication control may be subject to a third level of s

48 We propose a replication control mechanism whereby pi would directly

49 firming the involvement of the host cellular replication control mechanism, and prevented rereplicati

50 A replication control model, designated handcuffing or cou

51 Escherichia coli mutant highly perturbed for replication control, obgE::Tn5-EZ seqADelta, we isolated

52 mGBP2-deficient cells display defects in the replication control of T. gondii.

53 ar processes such as cell cycle control, DNA replication, control of gene expression, tumor suppresso

54 ich are known to be directly involved in DNA replication, control of viral and cellular transcription

55 s to be a well-conserved and central mode of replication control, optimal replication might require a

56 PLpromoter of phage lambda; (iv) and (v) the replication control promoters PRNAIand PRNAIIof plasmid

57 hypothesis that the N-terminal region of the replication control protein Cdc6 acts as an inhibitor of

58 d identified three up-regulated genes: a DNA replication control protein, a basic helix-loop-helix tr

59 nalysis of the DNA fragment representing the replication control region of the rodent mitochondrial g

60 A potential candidate for these replication control sequences was the 3' regulatory regi

61 after DNA damage, it relocalizes to selected replication control sites and suppresses abnormal, postd

62 of neuromuscular disease, exhibited at mtDNA replication control sites in muscle an accumulation of t

63 To assess the role of conserved sequences in replication control, structural and functional studies w

64 binding to ORC provides an origin-localized replication control switch that specifically prevents re

65 cycled, a necessary feature of the plasmid's replication control system.

66 have very different chromosome copy numbers, replication control systems and even employ distinct mac

67 we demonstrate a novel role for p16ink4a in replication control that is distinct from that of RB.

68 ndent kinases (Cdks) are critical for the re-replication controls that inhibit the activities of comp

69 iple stages of viral infection including DNA replication, controlling the cell cycle, and virion matu

70 nin knockdown 4pX-1 cell line used as DNA re-replication control, the Cdt1/geminin ratio is greater i

71 Acute deregulation of replication control ultimately causes extensive DNA dama

72 ta correlate the pppGpp/ppGpp ratio with DNA replication control under bacterial starvation condition

73 s were dependent on T cell degranulation for replication control with only a minor additional contrib