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1 e deacetylase HDAC1 when bound to the HLA-B7 repressor element.
2  demonstrating that CDP/cut binds the HLA-B7 repressor element.
3 may be partially mediated through a G/C-rich repressor element.
4 truct containing multiple copies of the VIPR repressor element.
5 t, possibly as a DNA-binding transcriptional repressor element.
6 activator element while helix II serves as a repressor element.
7 otein complex dependent on the SM22alpha G/C Repressor element.
8 ecause it is repressed by an upstream distal repressor element.
9 lex (G4) that functions as a transcriptional repressor element.
10 ored Pur alpha protein and binding to the AR repressor element.
11  of the previously identified In100 splicing repressor element.
12 ter of UBX sites that can also function as a repressor element.
13 omoter activity, suggesting that CACCC was a repressor element.
14 -MYC promoter functions as a transcriptional repressor element.
15  transcription of the hAT1R gene through the repressor element.
16 RNA by promoting the removal of a cis-acting repressor element.
17 GLD-1 binds directly and specifically to the repressor element.
18 nce of removing the downstream translational repressor element.
19 omoter region functions as a transcriptional repressor element.
20 nd defined regions that contain enhancer and repressor elements.
21 '-flanking region suggesting the presence of repressor elements.
22 t and does not require upstream activator or repressor elements.
23 for genes in which the E2F sites function as repressor elements.
24 sting interaction between the activators and repressor elements.
25 sequential binding of pELK-1 and KLF4 to G/C Repressor elements.
26  cells, resulting in deletion of translation repressor elements.
27 ledge of TFs bound to promoter, enhancer and repressor elements.
28 so contains a variety of known activator and repressor elements.
29 ession in neurons, whereas Sp1/Sp3 bound the repressor elements.
30 he TATA box of the PTP-oc contains potential repressor elements.
31 accompanied by increased AP-1 binding to the repressor elements.
32 zed to a neuron-restrictive silencer element/repressor element 1 (NRSE/RE-1) sequence within the chol
33 on is repressed in non-neuronal cells by the repressor element 1 (RE-1)-silencing transcription facto
34  has been proposed to restrict expression of repressor element 1 (RE1) bearing genes to differentiate
35  analyses, we show that the binding of REST (repressor element 1 (RE1) silencing transcription factor
36                  They expressed the neuronal repressor element 1 (RE1) silencing transcription factor
37                                          The repressor element 1 (RE1) silencing transcription factor
38                                              Repressor element 1 (RE1) silencing transcription factor
39                                          The repressor element 1 (RE1) silencing transcription factor
40  report the light-mediated regulation of the repressor element 1 (RE1)-silencing transcription factor
41                                              Repressor element 1 (RE1)-silencing transcription factor
42                                          The repressor element 1 is a long, conserved transcription f
43 ough the cis-regulatory element known as the repressor element 1 or neural restrictive silencer (RE1/
44 so stabilized binding of the Corepressor for Repressor Element 1 Silencing Transcription Factor (CoRE
45                                      Loss of repressor element 1 silencing transcription factor (REST
46 f SCF(betaTrCP)-dependent destruction of the repressor element 1 silencing transcription factor (REST
47  we show that the transcriptional repressor, repressor element 1 silencing transcription factor (REST
48                           In the case of the repressor element 1 silencing transcription factor (REST
49       We find that the transcription factor, Repressor Element 1 Silencing Transcription factor (REST
50 he gene silencing transcription factor REST (repressor element 1 silencing transcription factor), whi
51 he gene silencing transcription factor REST [repressor element 1 silencing transcription factor]/NRSF
52 r miR-124a promoters despite the presence of repressor element 1 sites, indicating REST target specif
53                Here, we demonstrate that the Repressor Element 1-Silencing Transcription factor (REST
54           Here we show that induction of the repressor element 1-silencing transcription factor (REST
55                                          The repressor element 1-silencing transcription factor (REST
56 promoter was effectively counteracted by the repressor element 1-silencing transcription factor (REST
57 racts with the master neuronal gene-silencer repressor element 1-silencing transcription factor (REST
58                          The transcriptional repressor element 1-silencing transcription factor (REST
59 nes mediated by the interaction of TRF2 with repressor element 1-silencing transcription factor (REST
60 r that recruits BRG1 as a corepressor is the repressor element 1-silencing transcription factor (REST
61 demonstrate that nuclear localization of the Repressor Element 1-Silencing Transcription factor (REST
62                                          The repressor element 1-silencing transcription factor (REST
63  whereas the transcriptional repressor REST (repressor element 1-silencing transcription factor) repr
64                Conditional knockout of REST (repressor element 1-silencing transcription factor) resu
65 tors implicated in vertebrate development is repressor element 1-silencing transcription/neuron-restr
66 in sequence and position dependence from the repressor element 1/neuron-restrictive silencer element
67 on factor neuron-restrictive silencer factor/repressor element-1 (RE-1) silencing transcription facto
68                     Here we identify a novel repressor element-1 (RE-1) site in the 5' regulatory reg
69                                              Repressor Element-1 (RE1) Silencing Transcription Factor
70                                          The repressor element-1 (RE1) silencing transcription factor
71      Methamphetamine exposure also increased repressor element-1 silencing transcription factor (REST
72  that NED of PCa requires down-regulation of repressor element-1 silencing transcription factor (REST
73                    Expression levels for the repressor element-1 silencing transcription factor (REST
74         Dysregulation of the transcriptional repressor element-1 silencing transcription factor (REST
75 gene silencing transcription factor neuronal repressor element-1 silencing transcription factor (REST
76 w that the lysine-specific demethylase 1 and repressor element-1 silencing transcription factor corep
77 ilencer factor (NRSF; also known as REST for repressor element-1 silencing transcription factor) is a
78          The transcriptional repressor REST (repressor element-1 silencing transcription factor) is a
79 lencer factor (NRSF; also known as REST, for repressor element-1 silencing transcription factor) to 1
80 he gene silencing transcription factor REST (repressor element-1 silencing transcription factor)/NRSF
81 recruitment of the transcriptional repressor repressor element-1 silencing transcription factor/neuro
82 element, neuron-restrictive silencer element/repressor element-1.
83                                          The repressor element-1/neuron-restrictive silencing element
84 that miR-9* and miR-124 are repressed by the repressor-element-1-silencing transcription factor (REST
85                                   REST/NRSF (repressor-element-1-silencing transcription factor/neuro
86 04 to -29 bp flanking a previously described repressor element (-137 to -130 bp).
87 nd K562 cells demonstrated the presence of a repressor element 650 base pairs upstream of the first e
88  is mediated through KLF4 binding to the G/C Repressor element and (2) the transcriptional repressor
89 sults in destabilization of the G-quadruplex repressor element and an increase in basal transcription
90 n requires integration of typical CDE/CHR G1 repressor elements and basal transcriptional activity by
91 hift assays indicate that the activation and repressor elements are bound by AP1 and an LBP1-related
92                                      The two repressor elements are functionally redundant in that th
93                                 The upstream repressor elements are located within the polypyrimidine
94 , suggesting that additional HSF-1-dependent repressor elements are present upstream of the minimal -
95 protein specifically interacts with the VIPR repressor element as demonstrated by gel shift assays.
96 ent located between -717 and -757, and a new repressor element at -780 to -821.
97                         The RBE acted like a repressor element at most positions in the presence of b
98  and its serial deletions identified a 15-bp repressor element at the 3'-UTR of CDKN1A, which contain
99                                          The repressor element bears sequence homology to an activato
100 ession of the reporter gene, and suggested a repressor element between -1848 and -1510.
101             We delineate the IRF1-responsive repressor element between nt -68 to -31 of the CDK2 prom
102                                          The repressor elements bound Fos family proteins; associatio
103 of reporter constructs containing the HLA-B7 repressor element but not the corresponding region of th
104  show that the Z-DNA forming transcriptional repressor element, by interacting with these putative Z-
105 g Smads 2 and 3, Rb/E2F4, and the cell-cycle repressor elements CDE and CHR.
106                              We identified a repressor element (CDE/CHR) in the region of the transcr
107 tential activator protein 1 (AP1) site and a repressor element containing a potential binding site fo
108 oteins; association of the proteins with the repressor elements correlated negatively with FR-beta ex
109 shift assays that Sp1 binding to the GC-rich repressor element did not prevent SRF binding to the adj
110                  Since E2F sites function as repressor elements during G(1) (due to the association o
111 rgenic region functions as a tissue-specific repressor element, forming an integral part of the compl
112 R4 promoter identified a seven-base pair p53-repressor element homologous to cyclic AMP/AP-1 response
113 bstitution mutations identified a functional repressor element I RE1-like silencer and functional Sp1
114     We have reported the presence of another repressor element in exon 1 that interacted with a prote
115 nstrated DNA binding activity to the labeled repressor element in hPTEC nuclear extracts.
116 ncreased DNA binding activity to the labeled repressor element in nuclear extracts treated with high
117 HoxA10 DNA-binding consensus is similar to a repressor element in the CYBB promoter.
118        Previously, we identified an intronic repressor element in the GAA gene and demonstrated that
119 s-acting factor(s) binding to the cis-acting repressor element in the hAT1R promoter, which may parti
120 ctor that interacts with the tissue-specific repressor element in the rat serum amyloid A1 (SAA1) pro
121                                              Repressor elements in the gp91(phox) promoter are necess
122                         Dd-STATa binds these repressor elements in vitro and the ectopic expression o
123 at PAC sites function as Dot6/Tod6-dependent repressor elements in vivo.
124 ssor complex that binds a specific sequence (repressor element) in the AR gene 5'-untranslated region
125 estern blot analysis indicated that the VIPR repressor element interacts specifically with a nuclear
126                We propose that this intronic repressor element is important for the restricted expres
127       In the present study, we show that the repressor element is located at position -872 to -860 ba
128 5 promoter is regulated by a silencer (Kv1.5 repressor element; KRE) containing a dinucleotide-repeti
129                  Mutation or deletion of the repressor element led to enhanced induction by TPA or ep
130                  The removal of the putative repressor elements led to the apparent loss of tissue sp
131 romoter also reveals a novel transcriptional repressor element located approximately 60 bp 5' of the
132  29-bp (5'-AACTGATTTTTGTATATTGATCTTGTATT-3') repressor element located between -1582 and -1610 bp was
133 gnificant sequence homology with an intronic repressor element located downstream of the alpha-exon.
134 ols Pax6 transcription by interacting with a repressor element located in the 5'-flanking region upst
135                           This suggests that repressor elements may be missing in the promoter used i
136 o begin to assess mechanisms whereby the G/C repressor element mediates suppression of SM22alpha in a
137                               Functional Nru repressor elements (NREs) could not be generated by mult
138 ic protein that binds to the CUCUCU splicing repressor element of the DCS RNA.
139 ed to be under negative control, mediated by repressor elements present in the promoters of stalk cel
140 we report that the zinc-finger gene-specific repressor element RE-1 silencing transcription factor/ne
141 covery of an element designated H1t promoter repressor element (RE) located between -130 and -106 bp
142                            We identified the repressor element (RE)-1 silencing transcription factor
143 ' elements of AS1 are competed by a putative repressor element 'RE1' defined previously in the oat ph
144 regions of neuron-specific genes possess the repressor element repressor element 1/neuron-restrictive
145   These studies suggest that transcriptional repressor element(s) located in PLP intron 1 are importa
146  factors, and the action of tissue-selective repressor element(s) may combine to enable a wide variet
147 cells, and suggest that there is a secondary repressor element(s) that is located in the terminal reg
148 ) mediates transcriptional repression by the repressor element silencing transcription factor/neuron-
149              We now show that these upstream repressor elements specifically interact with the polypy
150 0 kb from the proximal tethering sequence, a repressor element that excludes activation of Scr by ina
151 ikingly, mutations in a previously described repressor element that overlaps the TATA box restored pr
152 PA-dependent binding of USF1 and USF2 to the repressor element that required nucleotides within the E
153 however, is limited due to the presence of a repressor element that shows similarity to a non-canonic
154 s expression in dorsolateral epidermis; 2) a repressor element that suppresses expression in the epid
155 hus consists of closely linked activator and repressor elements that function collectively to cause e
156 (IRE) that shares sequence homology with the repressor element, the cardiac specific sequence, in the
157  3' intergenic loop formations competed with repressor elements to access promoter-proximal sequences
158 initiation site and the possible presence of repressor elements upstream of bp -223.
159 TPA-inducible binding to the imperfect E box repressor element was also apparent.
160  Gel-shift analysis showed that this GC-rich repressor element was recognized by both Sp1 and Sp3.
161 ific binding complex with the HLA-B7 and Cw2 repressor elements was demonstrated by EMSA.
162 y-old mouse brain nuclear extracts bound the repressor elements, whereas both dephosphorylated and ph
163  are similar to those of the adenovirus IIIa repressor element, which has been shown to inhibit use o
164 egulate Pax6 transcription by binding to the repressor element, which in turn blocks the effect of th
165 ATA-less promoters predominantly function as repressor elements, while in other genes constitutively
166 e of the MOR gene is a functionally synergic repressor element with NRSE in NS20Y cells, but not in t
167 t the identification of a cell type-specific repressor element within the proximal promoter.
168  these results support a model whereby a G/C repressor element within the SM22alpha promoter mediates
169 leus and binds to cell cycle homology region repressor elements within the cyclin A promoter.
170 gs indicate that the combination of enhancer/repressor elements within the proximal 5'-flanking regio
171  The data indicate that there are additional repressor elements within the vWf promoter region suppre

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