ライフサイエンスコーパス: reproductive cell

1 he marked asymmetric polarity of the budding reproductive cell.

2 in LHr-mediated gonococcal invasion of human reproductive cells.

3 unique process that allows the generation of reproductive cells.

4 d are critical for self-fertility in diploid reproductive cells.

5 Gcna gene expression is enriched in reproductive cells across eukarya - either just prior to

6 S1 mediates signals that control the fate of reproductive cells and their contiguous somatic cells.

7 reveal a two-way relationship between early reproductive cells and their helpers involving complex e

8 The molecular interactions between reproductive cells are critical for determining whether

9 a few days old, whereas its gonidia (asexual reproductive cells) are nonmotile, specialized for growt

10 entric cell layers that surround and support reproductive cells as they progress through meiosis and

11 In Volvox carteri adults, reproductive cells called gonidia are enclosed within a

12 ousand cells, but just two cell types: large reproductive cells called gonidia, and small, biflagella

13 matic cells and potentially immortal asexual reproductive cells called gonidia.

14 len grains) are generated in the anther from reproductive cells called microsporocytes.

15 Caenorhabditis elegans of radiation-induced reproductive cell death ("Radelegans") in isolation of a

16 romosomal instability (P < 0.1), and delayed reproductive cell death (the persistent reduction in pla

17 s of the biological end points of persistent reproductive cell death and apoptosis were consistent, s

18 g-held tenet that DNA damage is the cause of reproductive cell death and further validating this mode

19 These results suggest that persistent reproductive cell death can, in part, be explained by th

20 For example, after 9.5 Gy, which causes reproductive cell death in 99% of both types of cells, t

21 med that cell death in this model represents reproductive cell death in isolation from apoptotic cell

22 hway of EGFR is critical for protection from reproductive cell death in Radelegans.

23 eveloped a tissue model of radiation-induced reproductive cell death in the nematode Caenorhabditis e

24 Reproductive cell death is the primary mode of death in

25 Chromosomal instability and persistent reproductive cell death show a significant correlation a

26 ing delayed HR showed no evidence of delayed reproductive cell death, and there was no correlation be

27 lones for sister chromatid exchange, delayed reproductive cell death, delayed mutation, mismatch repa

28 nse pathway is necessary for protection from reproductive cell death, supporting the long-held tenet

29 he EGFR signaling pathway in protection from reproductive cell death, the primary form of tumor stem

30 the possible role of apoptosis in persistent reproductive cell death, we analyzed subsets of chromoso

31 enetic pathways required for protection from reproductive cell death.

32 by UCN-01 do not necessarily correlate with reproductive cell death.

33 omosomal instability correlated with delayed reproductive cell death.

34 s undergo alternation of generation in which reproductive cells develop in the plant body ("sporophyt

35 ission, indicating TTI2's importance in male reproductive cell development.

36 ANT1) play a fundamental role in somatic and reproductive cell differentiation during early anther de

37 e functions in addition to their role during reproductive cell division.

38 across different tissues revealed that male reproductive cells express a phylogenetically younger tr

39 sterile mutants have defects in somatic and reproductive cell fate acquisition.

40 ein play key roles in regulating somatic and reproductive cell fate determination in Arabidopsis anth

41 t one branch partitions the main body of the reproductive cell from the prosthecum and swarm cell.

42 uses abnormal differentiation of somatic and reproductive cells in anthers.

43 ial germ cells (PGCs) are the progenitors of reproductive cells in metazoans and are an important mod

44 n, polarity establishment and setting up the reproductive cell line can readily be recognized, they d

45 bolic changes in two different types of male reproductive cell lines.

46 n and the differentiation of PH-like asexual reproductive cells (metulae and phialides) are normal in

47 iophores with branching chains of reiterated reproductive cells (metulae), delayed conidial different

48 nduced enhanced gonococcal invasion of human reproductive cells that utilizes the lutropin receptor (

49 e prosthecum, distal to the main body of the reproductive cell; thus, the chromosome must travel thro