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1 omosomal instability correlated with delayed reproductive cell death.
2 enetic pathways required for protection from reproductive cell death.
3 by UCN-01 do not necessarily correlate with reproductive cell death.
4 s of the biological end points of persistent reproductive cell death and apoptosis were consistent, s
5 g-held tenet that DNA damage is the cause of reproductive cell death and further validating this mode
6 ing delayed HR showed no evidence of delayed reproductive cell death, and there was no correlation be
8 lones for sister chromatid exchange, delayed reproductive cell death, delayed mutation, mismatch repa
10 med that cell death in this model represents reproductive cell death in isolation from apoptotic cell
12 eveloped a tissue model of radiation-induced reproductive cell death in the nematode Caenorhabditis e
14 Caenorhabditis elegans of radiation-induced reproductive cell death ("Radelegans") in isolation of a
16 nse pathway is necessary for protection from reproductive cell death, supporting the long-held tenet
17 romosomal instability (P < 0.1), and delayed reproductive cell death (the persistent reduction in pla
18 he EGFR signaling pathway in protection from reproductive cell death, the primary form of tumor stem
19 the possible role of apoptosis in persistent reproductive cell death, we analyzed subsets of chromoso
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