ライフサイエンスコーパス: reproductive tract

1 ent in which sperm compete (e.g., the female reproductive tract).

2 y tract, large intestine), and reproduction (reproductive tract).

3 means to reduce ZIKV persistence in the male reproductive tract.

4 genes that are selectively expressed in the reproductive tract.

5 ne disruptors on the development of the male reproductive tract.

6 ment is dependent on factors secreted by the reproductive tract.

7 rus (HPV)-driven cancer including the female reproductive tract.

8 n epithelial barrier protection of the lower reproductive tract.

9 an oocyte as they travel through the female reproductive tract.

10 trols human sperm function within the female reproductive tract.

11 over long distances in the mammalian female reproductive tract.

12 how that MISO and 20E interact in the female reproductive tract.

13 input of these sensory neurons to the female reproductive tract.

14 responses to Chlamydia muridarum within the reproductive tract.

15 gation is driven by elongation of the female reproductive tract.

16 t both IgG and SIgA are present in the human reproductive tract.

17 SP is mediated by SP retention in the female reproductive tract.

18 hus avoiding ejection by the female from her reproductive tract.

19 this capacity during migration in the female reproductive tract.

20 nology for drug delivery to the lower female reproductive tract.

21 natural selective environment of the female reproductive tract.

22 ated in an AF-2-dependent manner in the male reproductive tract.

23 ons of the sex accessory tissues in the male reproductive tract.

24 in metabolism, transport, or activity in the reproductive tract.

25 tween the sexes that occur within the female reproductive tract.

26 stitial cells in other organs, including the reproductive tract.

27 determine its pathogenic role in the female reproductive tract.

28 S colonization and persistence in the female reproductive tract.

29 in the Caenorhabditis elegans hermaphrodite reproductive tract.

30 o activation once introduced into the female reproductive tract.

31 will differentiate into the internal female reproductive tract.

32 ntains SP's ability to act within the female reproductive tract.

33 required for sperm activation in the female reproductive tract.

34 ectly observing live sperm inside the female reproductive tract.

35 plicate in epithelial cells lining the human reproductive tract.

36 s critical for the development of the female reproductive tract.

37 e most common malignancy of the lower female reproductive tract.

38 ominantly in the epithelial cells lining the reproductive tract.

39 C. sordellii spore activation in the female reproductive tract.

40 varian and endometrial cancers in the female reproductive tract.

41 tilising sperm inside the spermicidal female reproductive tract.

42 nancies at other sites, including the female reproductive tract.

43 sponsive tissues, such as the breast and the reproductive tract.

44 malian spermatozoa are quiescent in the male reproductive tract.

45 lerian duct into various parts of the female reproductive tract.

46 an epithelial tube and eventually the female reproductive tract.

47 on, like the mammalian epididymis and female reproductive tract.

48 rdinately regulate development of the female reproductive tract.

49 g, eye, intestine, uterus, oviduct, and male reproductive tract.

50 ectopic activation of sperm within the male reproductive tract.

51 le during ejaculation and swim up the female reproductive tract.

52 l to study RNA virus persistence in the male reproductive tract.

53 ze fertilization inside the mammalian female reproductive tract.

54 ation of patients with cancers of the female reproductive tract.

55 ely in a sensitive mucosal site, such as the reproductive tract.

56 (2+)-concentration environment of the female reproductive tract.

57 uding the gastrointestinal tract, heart, and reproductive tract.

58 olonizes healthy human skin, mucosa, and the reproductive tract.

59 ated in inflammatory syndromes of the female reproductive tract.

60 ghters, indicating accelerated ageing in the reproductive tract.

61 eds progress of the sperm through the female reproductive tract.

62 e with oocytes despite direct contact in the reproductive tract.

63 osal epithelium in the rhesus macaque female reproductive tract.

64 ame intersex-possessing both female and male reproductive tracts.

65 iated with diseases of the brain, airway and reproductive tracts.

66 es in the development of the male and female reproductive tracts.

67 licle cells of ovary and epithelial cells of reproductive tracts.

68 ent and homeostasis of the airway, brain and reproductive tracts.

69 hts into the Y chromosome's roles beyond the reproductive tract--a theme that promises to broaden the

70 ed constellation of increasingly common male reproductive tract abnormalities (including hypospadias,

71 ed during the 1950s and 1960s, is related to reproductive tract abnormalities, infertility, and vagin

72 ured the amount of SP retained in the female reproductive tract after mating and female egg laying af

73 gg laying, or the amount of SP in the female reproductive tract after mating.

74 mammals, sperm are stored within a female's reproductive tract after mating.

75 y before mating but also within the female's reproductive tract after mating.

76 istic effects of raloxifene persisted in the reproductive tract after treatment had ceased.

77 In utero exposure to a xenoestrogen produces reproductive tract alterations by imprinting essential d

78 by regulating sperm maturation in the female reproductive tract and by triggering key sperm physiolog

79 The presence of infectious virus in the reproductive tract and in a vaginal swab implies an alte

80 on (PMCA)-competent prions within the female reproductive tract and in fetal tissues harvested from C

81 italium can establish long-term infection of reproductive tract and joint tissues, with preliminary e

82 ry efficient at navigating the hermaphrodite reproductive tract and locating oocytes.

83 expressed by epithelial cells in the female reproductive tract and may play a role in regulating bac

84 The tissues and organs of the female reproductive tract and pelvic floor undergo significant

85 hormone profile, which controls human female reproductive tract and peripheral tissue dynamics in sin

86 conducted a thorough analysis of the female reproductive tract and report that the primary cause of

87 ary tissues, affects the virilization of the reproductive tract and results in genitourinary birth de

88 n mixtures are separately stored in the male reproductive tract and sequentially transferred to the f

89 , including persistent infection in the male reproductive tract and sexual transmission, an ability t

90 ational response to mating within the female reproductive tract and suggest a bimodal model of postma

91 nti-Neu5Gc antibodies could enter the female reproductive tract and target Neu5Gc-positive sperm or f

92 These systems simulate the in vivo female reproductive tract and the endocrine loops between organ

93 e association between the cranial end of the reproductive tract and the kidney.

94 gastrointestinal tract, nasopharynx, female reproductive tract and the surface of the eye.

95 inning with the insemination into the female reproductive tract and, finally, leading to embryogenesi

96 CYP7B1 is expressed in the liver, reproductive tract, and brain and performs different phy

97 h occurs on the ocular surface, oral cavity, reproductive tract, and gut, requires a specialized apic

98 distinct but overlapping patterns within the reproductive tract, and in haf bee1 bee3 triple mutants

99 ese stallions, EAV is detectable only in the reproductive tract, and viral persistence occurs despite

100 Congenital reproductive tract anomalies could impair fertility.

101 data of human sperm bound to excised female reproductive tract are also presented and are found to b

102 ility and reproduction, organs of the female reproductive tract are also regulated by the hypothalami

103 rse over which plugs survive in the female's reproductive tract are lacking.

104 Mucosal surfaces of the female reproductive tract are the main routes of heterosexual t

105 infections that ascend from the lower female reproductive tract are the most common route of uterine

106 Female and male reproductive tracts are developed from Mullerian ducts a

107 s in the breast, prostate, cervix, and upper reproductive tract-are shared by embryo and adult.

108 mpanied by epididymitis, suggesting the male reproductive tract as 1 site of antigen persistence.

109 blish persistent infection in the stallion's reproductive tract as a mechanism to ensure its maintena

110 Using the male reproductive tract as a model system in which water and

111 he growth of pollen tubes through the female reproductive tract as they seek out unfertilized ovules.

112 ce of >1700 genes is altered in the female's reproductive tract as well as in other tissues.

113 Mucosal surfaces of the reproductive tract as well as their secretions have impo

114 ll bowel, select lymphoid areas and the male reproductive tract, as confirmed by quantitative reverse

115 E6 contributed to carcinogenesis of the reproductive tract, as E6/E7 double transgenic mice trea

116 might contribute to dysfunction of the male reproductive tract associated with these mutations.

117 In Arabidopsis, the reproductive tract begins with the stigma, where pollen

118 eases in plasma cells not only in the female reproductive tract but also at other mucosal sites, and

119 It is plausible that MHM can affect the reproductive tract but the specific infections, the stre

120 nsgenic mice developed cancer throughout the reproductive tract, but E6 transgenic mice did not.

121 r penis into the female and ejaculate in her reproductive tract; but in some species, males are more

122 xually transmitted infections, treatment for reproductive tract cancer, and treatment of fungal and b

123 t recurrence of neoplastic disease and lower reproductive tract cancers.

124 RNA-loaded nanoparticles to the mouse female reproductive tract caused efficient and sustained gene s

125 port on a study of the alterations of murine reproductive tract collagen resulting from pregnancy and

126 Upper reproductive tract colonization was observed as early as

127 er but detectable oncogenic potential in the reproductive tract compared with the E7 oncogene.

128 We hypothesized that each part of the reproductive tract contains a characteristic combination

129 Immunohistochemistry of the reproductive tract corroborated testis-specific expressi

130 now find that mucus released from the murine reproductive tract critically depends upon concurrent bi

131 sion to test whether the evolution of female reproductive tract design might have driven the evolutio

132 to survive as allogeneic cells in the female reproductive tract despite female immunity.

133 Several tissues, known collectively as the reproductive tract, develop within the carpel to facilit

134 Normal reproductive tract development and anogenital distance (

135 Genetic basis solely for distal reproductive tract development is largely unknown.

136 our understanding of the genetic control of reproductive tract development.

137 T), a more potent androgen required for male reproductive tract development.

138 ce of an additional merging phase for proper reproductive tract development.

139 duct epithelial progenitor cells for female reproductive tract development.

140 factor that is essential for male and female reproductive tract development.

141 LHFPL2 and a novel genetic basis for distal reproductive tract development; they also emphasize the

142 exually transmitted pathogen associated with reproductive tract disease in men and women, and it can

143 Mycoplasma genitalium is associated with reproductive tract disease in women and may persist in t

144 en associated with several acute and chronic reproductive tract disease syndromes in men and women.

145 ential component to managing lower and upper reproductive tract disease syndromes in women.

146 talium is an underappreciated cause of human reproductive tract disease, characterized by persistent,

147 e Notch1 intracellular domain (N1ICD) in the reproductive tract driven by a progesterone receptor (Pg

148 equirement for FGF8 in establishing the male reproductive tract ducts and implicate Lhx1 signaling in

149 s transferred with the sperm into the female reproductive tract during copulation.

150 to limit pathogen infiltration of the lower reproductive tract during pregnancy and thereby is prote

151 n which production is exaggerated within the reproductive tract during pregnancy, might be an importa

152 t and sequentially transferred to the female reproductive tract during spermatophore assembly.

153 CD8(+) T cells residing in the female mouse reproductive tract encountered cognate antigen, they exp

154 exosomes and sperm) and interact with female reproductive tract epithelia.

155 ction requires the virus to penetrate female reproductive tract epithelial barriers to infect underly

156 verexpressing cells and TLR2-competent human reproductive tract epithelial cell lines.

157 s can recognize and be activated by infected reproductive tract epithelial cells and block Chlamydia

158 c CD4 T-cell clones recognize infected upper reproductive tract epithelial cells as early as 12 h pos

159 ential evidence for a key role of the female reproductive tract epithelium in facilitating Ab product

160 on CD4 T-cell interactions with the infected reproductive tract epithelium.

161 everely impaired in the cystic fibrosis F508 reproductive tract, even though stimulated fluid secreti

162 We find infectious foci throughout the reproductive tract, from labia to ovary.

163 sible for pathogen clearance from the female reproductive tract (FRT) are incompletely defined; in pa

164 ler (NK) cells derived from the human female reproductive tract (FRT) are phenotypically and function

165 virological events that occur in the female reproductive tract (FRT) during ART that result in such

166 rt, 2011), the earliest events in the female reproductive tract (FRT) during heterosexual HIV-1 trans

167 es, which are known to infiltrate the female reproductive tract (FRT) following semen exposure.

168 nic bacteria ascending from the lower female reproductive tract (FRT) is associated with many gynecol

169 Disruption of the epithelium in the female reproductive tract (FRT) is hypothesized to increase HIV

170 The female reproductive tract (FRT) is one of the major mucosal inv

171 We show that the gut and female reproductive tract (FRT) of humanized DRAG mice have a h

172 2 T cells from blood, rectum, and the female reproductive tract (FRT) of rhesus macaques to determine

173 To establish infection in the female reproductive tract (FRT), HIV-1 in male ejaculate must o

174 re to SIVmac251 in the rhesus macaque female reproductive tract (FRT).

175 (GR) is also an integral part of maintaining reproductive tract function; disruption of GR signaling

176 Seminal fluid, which affects female reproductive tract gene expression as well as sperm surv

177 d sequence tag analysis of Drosophila female reproductive tract genes identified 169 candidate female

178 ncing on dissected tissues revealed that the reproductive tracts harbor a complex microbiome characte

179 nvolved in the formation and function of the reproductive tract have largely remained a mystery but a

180 The results indicate that female reproductive tracts have undergone extensive diversifica

181 sed IFN has suggested a function for IFNs in reproductive tract homeostasis and protection from infec

182 and precancerous lesions in the female lower reproductive tracts (ie, cervix and vagina) in the human

183 and may regulate muscle contractions in the reproductive tract, if it is expressed there.

184 lting in complete and incomplete blockage of reproductive tract in infertile and fertile males, respe

185 differentially regulated in the female lower reproductive tract in response to conspecific mating.

186 ntrolling gynecological disease in the lower reproductive tracts in women.

187 ment of many conditions affecting the female reproductive tract, including sexually transmitted disea

188 gus, which innervates portions of the female reproductive tract, including the ovaries.

189 m) in sensory neurons innervating the female reproductive tract indicate that some of these candidate

190 ng studies of intrauterine contraception and reproductive tract infection.

191 d which looked at health outcomes, primarily reproductive tract infections (RTI).

192 For decades reproductive tract infections (RTIs) have been hypothesi

193 Malaria and sexually transmitted infections/reproductive tract infections (STIs/RTIs) in pregnancy a

194 laria infection and sexually transmitted and reproductive tract infections (STIs/RTIs).

195 , cervicovaginal and systemic HIV-1 RNA, and reproductive tract infections every 3-6 months over 8 ye

196 athogen associated with highly lethal female reproductive tract infections following childbirth, abor

197 of biological specimens that were tested for reproductive tract infections.

198 The female reproductive tract is particularly sensitive to early-li

199 e outcome of infectious diseases outside the reproductive tract is rarely considered.

200 The persistence of SP in the mated female's reproductive tract is thought to be a consequence of its

201 estrogen exposure and defects in the female reproductive tract is well established.

202 red by passage of the egg through the female reproductive tract ; it is unknown whether calcium signa

203 und that six to eight sensory neurons on the reproductive tract labeled by both ppk-GAL4 and fruitles

204 but are not limited to genes influencing the reproductive tract, mammary glands, bone, brain, fat dif

205 hich androgen action is essential for normal reproductive tract masculinization and has highlighted t

206 xhibit multiple defects in gonad morphology, reproductive tract maturation and gonad positioning.

207 d with mating and the final stages of female reproductive tract maturation and later with the declini

208 Upon mating, regions of the female reproductive tract mature and alter their function [1-3]

209 innate immune activation in the lower female reproductive tract may also affect adaptive immunity, we

210 a man other than her long-term partner, her reproductive tract may contain the sperm of both men, in

211 Secretions within the adult female reproductive tract mediate sperm survival, storage, acti

212 rtodiopsis dalmanni, sperm length and female reproductive tract morphology have coevolved across spec

213 ovulation inhibition, menses induction, and reproductive tract morphology.

214 sialyl T-antigen varied in bovine and ovine reproductive tract mucins, and terminal N-acetylgalactos

215 as) are the most common tumors of the female reproductive tract, occurring in up to 77% of reproducti

216 rm of multiple males concurrently occupy the reproductive tract of a female and compete to fertilize

217 ted molecules ("signaling molecules") in the reproductive tract of Drosophila melanogaster females be

218 mating transcriptional response of the lower reproductive tract of Drosophila mojavensis females foll

219 nitored cancer incidence in the female lower reproductive tract of FA-deficient mice expressing HPV16

220 s most renowned as venereal pathogens of the reproductive tract of humans and cattle.

221 nges involved in observing events within the reproductive tract of internally fertilizing species whi

222 ly and silently colonizes the intestinal and reproductive tract of laying hens, resulting in contamin

223 cern about persistence of Ebola virus in the reproductive tract of men who have survived EVD.

224 tein expression were increased by 25% in the reproductive tract of mice exposed in utero.

225 ed the consequences of infection in the male reproductive tract of mice.

226 s, was highly expressed and regulated in the reproductive tract of the mouse, and its expression was

227 expanded blastocyst were collected from the reproductive tract of zinc deficient animals on day 3.5

228 eumovirus (AMPV) infects the respiratory and reproductive tracts of domestic poultry, resulting in su

229 The reproductive tracts of female fireflies showed increased

230 indicate that E5 acts as an oncogene in the reproductive tracts of female mice.

231 is indicated by the presence of sperm in the reproductive tracts of queens.

232 The reproductive tracts of the classified heifers were obtai

233 dua and placenta by ascending from the lower reproductive tract or via hematogenous transmission.

234 The female reproductive tract organs of mammals, including the ovid

235 Mullerian ducts, the primordia of the female reproductive tract organs.

236 s, with preliminary evidence of pathological reproductive tract outcomes.

237 issues including intestine, stomach, kidney, reproductive tract, pancreas, brain, heart, and salivary

238 n provide evidence that they are involved in reproductive tract pathologies.

239 lowed the discovery of Hoxd9,10,11 redundant reproductive tract patterning function.

240 out human pregnancy, suggesting that reduced reproductive-tract progesterone receptor (PR) initiates

241 Recent studies of female reproductive tract proteins indicate they also are extre

242 cate that spermathecae and parovaria secrete reproductive tract proteins required for sperm maturatio

243 ent evidence of adaptive evolution in female reproductive tract proteins suggests this pattern may re

244 jaculates and their interactions with female reproductive tracts remain poorly understood.

245 women, as the organism ascends to the upper reproductive tract, resulting in pelvic inflammatory dis

246 and changes in muscle contraction within the reproductive tract (RT).

247 However, it is difficult to reconcile the reproductive tract's many changing but coordinated event

248 on (STI) that is potentially associated with reproductive tract sequelae in women.

249 nd TNF-alpha production to Chlamydia-induced reproductive tract sequelae.

250 ce this competition occurs inside the female reproductive tract, she often influences the outcome thr

251 Our results suggest that the reproductive tract signals to the ovary using glandular

252 competition should not be limited to female reproductive tract-specific genes, but should focus also

253 e found that glands of the Drosophila female reproductive tract, spermathecae and/or parovaria, are r

254 No obvious morphological differences in reproductive tract structures and histology of the uteru

255 selection, where readily quantifiable female reproductive tract structures are capable of biasing pat

256 Wolffian duct, the precursor of several male reproductive tract structures.

257 Each reproductive tract subregion displays a characteristic c

258 ium has been implicated in several important reproductive tract syndromes in women, including pelvic

259 tion and in women is associated with notable reproductive tract syndromes such as cervicitis, pelvic

260 n Mycoplasma genitalium infection and female reproductive tract syndromes through meta-analysis, Engl

261 function for the MVB-exosome pathway in the reproductive tract that appears to be conserved across e

262 te modulator of sperm motility in the female reproductive tract that increases sperm flagellar beat f

263 r coregulator in normal tissues of the human reproductive tract that is expressed at higher levels in

264 conformational changes in the mated female's reproductive tract that occur during and immediately aft

265 In the reproductive tract, the enzyme metabolizes androgens tha

266 In the female reproductive tract, the spermatozoan cytoplasm alkaliniz

267 muscle cells and the smooth muscle cells of reproductive tracts, the oviduct and ductus epididymis.

268 igration through and localization within the reproductive tract, thereby promoting their access to oo

269 inal tract, airways, nasopharynx, and female reproductive tract, thereby strongly limiting opportunit

270 ributed to carcinogenesis, but in the female reproductive tract, these activities were manifested onl

271 and during their transit through the female reproductive tract, they undergo changes that enable the

272 ented the expression of p16 in tumors of the reproductive tract through a mechanism mediated by the i

273 odeling of tenofovir (TFV) in plasma, female reproductive tract tissue, cervicovaginal lavage fluid a

274 entiating estrogen-target tissues, including reproductive tract tissues and adipocytes.

275 gene expressed at low levels in normal human reproductive tract tissues and at higher levels in castr

276 The reproductive tract tissues and peripheral organs integra

277 d suggest that M. genitalium colonization of reproductive tract tissues may result in inflammatory se

278 Seminal fluid proteins (SFPs) produced in reproductive tract tissues of male insects and transferr

279 and SEVI to intact human and rhesus macaque reproductive tract tissues to determine how it influence

280 Ascension to upper reproductive tract tissues was not detected, even among

281 ogens and showed that masculinization of all reproductive tract tissues was programmed by androgen ac

282 rs of oxidative stress and cellular aging in reproductive tract tissues were assessed at 3 and 6 mo o

283 was limited to hemolymphatic tissues, female reproductive tract tissues, kidney, and liver, potential

284 tion factors that act redundantly to specify reproductive tract tissues.

285 and cellular effects have been identified in reproductive tract tissues.

286 pregnancy reduces the capacity of the female reproductive tract to prevent bacterial infection of the

287 epithelial-immune system axis in the female reproductive tract to respond to exposure to mucosal pat

288 ts that guide spermatozoa through the female reproductive tract to the mature oocyte.

289 fication of interacting ejaculate and female reproductive tract traits that mediate sperm competition

290 l plasticity is characteristic of the female reproductive tract: vaginal sensory innervation density

291 In the reproductive tract, virK was required in early colonizat

292 The overall development of the female reproductive tract was stunted at birth with a decrease

293 Animals were euthanized at week 16 and the reproductive tracts were examined histologically.

294 tissues such as skin, gut, lung, tongue and reproductive tract where they provide a first line of de

295 activated motility as they ascend the female reproductive tract, which enables them to overcome barri

296 molecules that endow different parts of the reproductive tract with unique temporal and spatial iden

297 l opening, persistent diestrus, and atrophic reproductive tracts with absent corpora lutea.

298 synergistic homeotic transformations of the reproductive tracts, with the uterus anteriorized toward

299 m is established by default because the male reproductive tracts (Wolffian ducts) in the female degen

300 cuss how the sperm interacts with the female reproductive tract, zona pellucida, and the oolemma.