ライフサイエンスコーパス: resistance to antibiotics

1 pumps significantly contribute for bacteria resistance to antibiotics.

2 f enzymes that plays a key role in bacterial resistance to antibiotics.

3 ences in phenotypes, including virulence and resistance to antibiotics.

4 garnering notoriety in an era of increasing resistance to antibiotics.

5 ble by vaccination and is rapidly developing resistance to antibiotics.

6 ficult to eradicate because of its intrinsic resistance to antibiotics.

7 ion, and potential contribution to bacterial resistance to antibiotics.

8 d with mutations, can contribute to adaptive resistance to antibiotics.

9 "intrinsic resistome," which provides innate resistance to antibiotics.

10 a major health threat because of increasing resistance to antibiotics.

11 elements responsible for spreading bacterial resistance to antibiotics.

12 nificant mechanism by which bacteria develop resistance to antibiotics.

13 fication as well as their ability to develop resistance to antibiotics.

14 ons continues to be complicated by expanding resistance to antibiotics.

15 pid spread of genes such as those conferring resistance to antibiotics.

16 gly difficult to treat due to high levels of resistance to antibiotics.

17 ructural basis of catalysis and mechanism of resistance to antibiotics.

18 ulated genes points to mechanisms of biofilm resistance to antibiotics.

19 problematic outbreaks and acquire high-level resistance to antibiotics.

20 racellular residency, biofilm formation, and resistance to antibiotics.

21 inhibitors and often contribute to multidrug resistance to antibiotics and biocides.

22 Efflux pumps have been shown to mediate resistance to antibiotics and cationic peptides in other

23 ellular superstructure can display increased resistance to antibiotics and cause serious, persistent

24 omplex multicellular assemblies that exhibit resistance to antibiotics and contribute to the pathogen

25 ts prominence as a pathogen is its intrinsic resistance to antibiotics and disinfectants.

26 Its predilection to develop resistance to antibiotics and expression of multiple vir

27 In developing countries, increased resistance to antibiotics and its cost make eradication

28 the same genetic determinant responsible for resistance to antibiotics and metals).

29 Bacillus subtilis are broadly implicated in resistance to antibiotics and other cell envelope stress

30 e selecting drugs, chromosomal mutations for resistance to antibiotics and other chemotheraputic agen

31 sion of multiple chromosomal genes affecting resistance to antibiotics and other environmental hazard

32 ial MDRs had previously been associated with resistance to antibiotics and other toxic compounds.

33 to play an important role in the bacteria's resistance to antibiotics and the host immune response.

34 The emerging and sustained resistance to antibiotics and the poor pipeline of new a

35 an isolate, testing its properties, such as resistance to antibiotics and virulence, and monitoring

36 ), and sigma(X)) are induced by, and provide resistance to, antibiotics and other agents eliciting ce

37 ere pneumonia in young children), increasing resistance to antibiotics, and changes in HIV prevalence

38 ay the dominant role in conferring intrinsic resistance to antibiotics, and provide initial insights

39 outcomes of increased infectivity, intrinsic resistance to antibiotics, and subversion of the host im

40 bidity and mortality and, because of biofilm resistance to antibiotics, are difficult to treat.

41 only display distinct phenotypes, including resistance to antibiotics, but also, serve as building b

42 entical bacteria display differing levels of resistance to antibiotics, clonal yeast populations demo

43 he typical ARG profiles suggest a prevailing resistance to antibiotics commonly used in human health

44 Increasing resistance to antibiotics creates the need for prudent a

45 The rise in bacterial resistance to antibiotics demonstrates the medical need

46 eradicate because of their unusually robust resistance to antibiotics, disinfectants, and desiccatio

47 The continued evolution of resistance to antibiotics has led to wide ranging consul

48 ly related to these shifts include those for resistance to antibiotics, heavy metals, and phage.

49 Mutations that cause resistance to antibiotics in bacteria often reduce growt

50 a challenge for treatment due to its evolved resistance to antibiotics, including carbapenems.

51 icult to treat due to intrinsic and acquired resistance to antibiotics, including vancomycin.

52 The emergence of bacterial resistance to antibiotics is a major health problem and,

53 The emergence of resistance to antibiotics is a serious problem often rel

54 The incidence of resistance to antibiotics is increasing; therefore, it i

55 jejuni is a major zoonotic pathogen, and its resistance to antibiotics is of great concern for public

56 of P. aeruginosa and its ability to develop resistance to antibiotics, it continues to be problemati

57 With the rapid increase of infection resistance to antibiotics, it is urgent to find novel in

58 he survival of bacteria and the evolution of resistance to antibiotics make it an attractive target f

59 animal-adapted S. aureus lineages exhibiting resistance to antibiotics must be considered a major thr

60 Bacterial resistance to antibiotics, particularly plasmid-encoded

61 Bacterial resistance to antibiotics, particularly to multiple drug

62 We postulate that phenotypic resistance to antibiotics, persistence, is not an evolve

63 The spread of bacterial resistance to antibiotics poses the need for antimicrobi

64 With increase in the incidence of resistance to antibiotics, probiotics are emerging as a

65 sion as measured by mtrCDE transcription and resistance to antibiotics, progesterone and antimicrobia

66 It has long been assumed that resistance to antibiotics reduces the fitness of disease

67 The increasing threat of pathogen resistance to antibiotics requires the development of no

68 Genes for resistance to antibiotics such as acriflavin, bacitracin

69 With the rise in resistance to antibiotics such as methicillin, there is

70 NA) modulates ribosomal function and confers resistance to antibiotics targeted to the ribosome.

71 in mixed cultures was associated with higher resistance to antibiotics than in either monoculture.

72 ncluding cell wall remodeling (and therefore resistance to antibiotics that target bacterial cell wal

73 aminoglycoside O-phosphotransferases, confer resistance to antibiotics that were not administered in

74 The emergence of bacterial multidrug resistance to antibiotics threatens to cause regression

75 omised by the ability of bacteria to develop resistance to antibiotics through mutations or through t

76 Due to their resistance to antibiotics, treatment is often very chall

77 inistered drug, as well as genes that confer resistance to antibiotics unrelated to the administered

78 Strains with rRNA mutations conferring resistance to antibiotics used in acne treatment were do

79 Bacterial resistance to antibiotics usually incurs a fitness cost

80 Bacteria can rapidly evolve resistance to antibiotics via the SOS response, a state

81 estimating the degree of E. coli JM109 cells resistance to antibiotics within 2-5h using disposable s