ライフサイエンスコーパス: resistant bacteria

1 rtance given the alarming growth of pan-drug-resistant bacteria.

2 will most limit the emergence and spread of resistant bacteria.

3 tibacterials are needed to tackle antibiotic-resistant bacteria.

4 antibiotics and selectively kill antibiotic-resistant bacteria.

5 l platform for developing AMPs to treat drug-resistant bacteria.

6 efore help to treat infections of antibiotic resistant bacteria.

7 ible, for study of infections caused by drug-resistant bacteria.

8 re needed to treat infections caused by drug-resistant bacteria.

9 ons were to increase and may also select for resistant Bacteria.

10 s the main driver for carriage of antibiotic-resistant bacteria.

11 for development and enrichment of antibiotic resistant bacteria.

12 rates of genome rearrangements in radiation-resistant bacteria.

13 tive vaccine and the emergence of multi-drug-resistant bacteria.

14 it (ICU) decreases acquisition of antibiotic-resistant bacteria.

15 due to transmission of particular antibiotic-resistant bacteria.

16 s to antibiotics and therapies for multidrug-resistant bacteria.

17 were directly attributable to extremely drug-resistant bacteria.

18 for new antimicrobial drugs to combat these resistant bacteria.

19 Pro were more likely to be colonized with FQ-resistant bacteria.

20 s to prevent the spread of highly antibiotic-resistant bacteria.

21 ent outcomes while reducing the emergence of resistant bacteria.

22 n of virulent toxin producing and antibiotic resistant bacteria.

23 s well as the potential for the evolution of resistant bacteria.

24 ening of antimicrobials for use against drug-resistant bacteria.

25 ic resistance through selection of naturally resistant bacteria.

26 type bacteria but not in the number of drug resistant bacteria.

27 a promising strategy for combating multidrug-resistant bacteria.

28 e with new agents to combat the rise of drug resistant bacteria.

29 xposure to pesticide residues and antibiotic-resistant bacteria.

30 life-threatening infections caused by highly resistant bacteria.

31 from intestinal colonization with antibiotic-resistant bacteria.

32 nd help prevent the development of multidrug-resistant bacteria.

33 have varying efficacy against the multidrug-resistant bacteria.

34 of antimicrobial agents against 2 multidrug-resistant bacteria.

35 rapy and prophylaxis in the war against drug-resistant bacteria.

36 les, which could serve as the source of drug-resistant bacteria.

37 mising antibacterial properties against drug-resistant bacteria.

38 y against vancomycin-sensitive or vancomycin-resistant bacteria.

39 oping new aminoglycoside antibiotics against resistant bacteria.

40 hanism increased the emergence of antibiotic-resistant bacteria.

41 and vital for the battle against multi-drug-resistant bacteria.

42 rategies for combating the emergence of drug-resistant bacteria.

43 d by penicillin-resistant or chloramphenicol-resistant bacteria.

44 oside antibiotics with activity against drug-resistant bacteria.

45 s of the transmission dynamics of antibiotic-resistant bacteria.

46 iii) the removal of pathogens and antibiotic resistant bacteria.

47 trains, including methicilin- and vancomycin-resistant bacteria.

48 o be potent against macrolide-sensitive and -resistant bacteria.

49 found to be inactive against aminoglycoside resistant bacteria.

50 simply inhibited the enzyme expressed by the resistant bacteria.

51 ion of patients colonized by emerging highly resistant bacteria.

52 these strains generate both nonresistant and resistant bacteria.

53 ost often detected enzymes in aminoglycoside-resistant bacteria.

54 ment of infections caused by susceptible and resistant bacteria.

55 ed, infections; and the spread of antibiotic-resistant bacteria.

56 present a new weapon against infections with resistant bacteria.

57 e colonization and infection with antibiotic-resistant bacteria.

58 tening infections due to multiply antibiotic resistant bacteria.

59 ve a high frequency of mutation in quinolone-resistant bacteria.

60 n the treatment of infections by beta-lactam-resistant bacteria.

61 y reduce the prevalence of colonization with resistant bacteria.

62 not appear to impede wound healing or induce resistant bacteria.

63 th potent activity toward Gram-positive drug-resistant bacteria.

64 al agents to treat people infected with drug-resistant bacteria.

65 hat responds to cell-wall alteration in drug-resistant bacteria.

66 r clinical efficacy through the selection of resistant bacteria.

67 ics against the growing number of antibiotic-resistant bacteria.

68 ons, particularly those caused by antibiotic-resistant bacteria.

69 evaluated as adjuvants for the treatment of resistant bacteria.

70 ns, especially in cases involving antibiotic-resistant bacteria.

71 e to find new therapies to combat antibiotic-resistant bacteria.

72 iated infections caused by highly antibiotic-resistant bacteria.

73 , are predominantly active against many drug-resistant bacteria.

74 ent antibacterial activity towards multidrug-resistant bacteria.

75 y medicine causes the emergence of multidrug resistant bacteria.

76 d (Cas) system into the genome of antibiotic-resistant bacteria.

77 articularly those caused by extensively drug-resistant bacteria.

78 s for the selection and spread of antibiotic-resistant bacteria.

79 e metallopolymer hydrogels to kill multidrug resistant bacteria.

80 atment, toxicity, and selection of multidrug-resistant bacteria.

81 pecially infection from the extensively drug-resistant bacteria, A. baumannii.

82 In the fight against drug-resistant bacteria, accurate and high-throughput detecti

83 ial transmission and dominance of antibiotic-resistant bacteria among cohoused untreated mice.

84 U) on the subsequent emergence of antibiotic-resistant bacteria among humans.

85 With current concerns of antibiotic-resistant bacteria and biodefense, it has become importa

86 peptides with proved activity to antibiotic resistant bacteria and biofilms.

87 Therefore, it seems advisable to quantify resistant bacteria and characterize their MICs and resis

88 elected extended-spectrum beta-lactam (ESBL) resistant bacteria and genes in 12 hospital wastewater o

89 manure increase the density of antimicrobial resistant bacteria and genes in the environment.

90 omes leads to emergence of new antimicrobial-resistant bacteria and global dissemination of them and

91 ces, especially with the growth of multidrug-resistant bacteria and interest in the microbiome.

92 g/mL) against both vancomycin-sensitive and -resistant bacteria and likely benefit from two independe

93 veloped for use as a drug against multi-drug resistant bacteria and protists.

94 als into the aquatic environment selects for resistant bacteria and resistance genes and stimulates b

95 adult patients not colonized with antibiotic-resistant bacteria and reviewed them for adverse events

96 f conventional antibiotics to kill multidrug-resistant bacteria and superbugs.

97 ure has led to the development of antibiotic-resistant bacteria and the accumulation of antibiotics i

98 The emergence of antimicrobial-resistant bacteria and viruses is a new threat to managi

99 sing daily with the development of multidrug-resistant bacteria and viruses that do not respond to st

100 To address the increase of drug-resistant bacteria and widespread inappropriate use of a

101 ents are enriched in antibiotics, antibiotic resistant bacteria, and antibiotic resistance genes, and

102 ions in immunization practices, emergence of resistant bacteria, and changes in patterns of immune mo

103 is of the emergence and spread of antibiotic-resistant bacteria, and demonstrates that early initiati

104 th blood disorders colonized with antibiotic-resistant bacteria (ARB) are prone to systemic infection

105 f patients already colonized with antibiotic-resistant bacteria (ARB) at the time of admission.

106 Infections with antibiotic-resistant bacteria (ARB) in hospitalized patients are be

107 tream infections (BSIs) caused by antibiotic-resistant bacteria (ARB) replace antibiotic-susceptible

108 ir efficiencies in removing three antibiotic-resistant bacteria (ARB), namely, blaNDM-1-positive Esch

109 r time, allows for development of antibiotic-resistant bacteria (ARB); b) identifying and describing

110 arrier for limiting the spread of antibiotic-resistant bacteria (ARBs) and antibiotic resistance gene

111 Emerging multidrug-resistant bacteria are a challenge for modern medicine,

112 Penicillin-resistant and chloramphenicol-resistant bacteria are a considerable threat in resource

113 Infections caused by drug-resistant bacteria are a major problem worldwide.

114 Multi-drug resistant bacteria are a persistent problem in modern he

115 Infections caused by antibiotic-resistant bacteria are a rising public health threat and

116 The emergence and spread of antibiotic-resistant bacteria are aggravated by incorrect prescript

117 Antibiotic-resistant bacteria are associated with increased patient

118 g septic emergency department patients, drug-resistant bacteria are covered infrequently.

119 Among these amoeba resistant bacteria are numerous members of the genus Myc

120 Resistant bacteria are one of the leading causes of hosp

121 Multidrug-resistant bacteria are responsible for substantial morbi

122 The emergence and spread of multidrug resistant bacteria are widely believed to endanger human

123 us diseases, especially those caused by drug-resistant bacteria, are a major problem worldwide.

124 which are natural products of aminoglycoside-resistant bacteria, as inhibitors of the in vitro activi

125 tible lineage 2 strain will harbor multidrug-resistant bacteria at the time of diagnosis.

126 d vancomycin compounds are effective against resistant bacteria because they interact directly with b

127 sidues are not only conserved in WT and drug-resistant bacteria but also significant in enzymatic act

128 risk areas for transmission of antimicrobial-resistant bacteria, but no controlled study has tested t

129 in a population and the frequent recovery of resistant bacteria, but whether a reduction in antimicro

130 cs has been proposed as a means to eliminate resistant bacteria by allowing sensitive clones to sweep

131 he National Action Plan to Combat Antibiotic Resistant Bacteria calls for all US hospitals to improve

132 chanism by which drug-target interactions in resistant bacteria can be enhanced.

133 e of antibiotics in animals raised for food; resistant bacteria can be transmitted to humans through

134 ental releases of antibiotics and antibiotic-resistant bacteria can in many cases be reduced at littl

135 Penicillin-resistant bacteria can often be treated through the co-a

136 In theory, these phenotypically resistant bacteria can retard the rate of microbiologica

137 ting of empiric antimicrobials for multidrug-resistant bacteria, Candida species, methicillin-resista

138 tion in Deinococcus radiodurans, a radiation-resistant bacteria capable of accumulating high concentr

139 Antibiotic-sensitive and -resistant bacteria coexist in natural environments with

140 The emergence of multidrug resistant bacteria compounded by the depleting arsenal o

141 Our strategy to combat resistant bacteria consisted of targeting the GyrB/ParE

142 uld provide new antibiotic functions against resistant bacteria, contain disordered N-terminal transl

143 ritically important, especially as multidrug-resistant bacteria continue to emerge.

144 As the global threat of drug- and antibiotic-resistant bacteria continues to rise, new strategies are

145 otics due to the urgent threat of antibiotic resistant bacteria coupled with the reduced effort in de

146 The ruthless selection for resistant bacteria, coupled with insufficient investment

147 The heavy metal-resistant bacteria Cupriavidus metallidurans CH34 relies

148 n proteins, lipids, and nucleic acids of the resistant bacteria described in this study.

149 ary outcome was colonization with antibiotic-resistant bacteria, determined by surveillance cultures

150 n, the growth of wild type bacteria and drug resistant bacteria differ significantly.

151 t potent antimicrobial activity against VanA resistant bacteria ( E. faecalis , VanA VRE) at a level

152 +) bacteria (e.g., S. aureus) and antibiotic-resistant bacteria (e.g., MRSA).

153 fficacy, without the emergence of antibiotic-resistant bacteria either orally or extra-orally.

154 Epidemics of drug-resistant bacteria emerge worldwide, even as resistant s

155 When phage-resistant bacteria emerge, mutation may generate phage c

156 e infection and the likelihood of antibiotic-resistant bacteria emerging and ascending.

157 The emergence of multidrug-resistant bacteria emphasizes the urgent need for novel

158 ty relationship (SAR) against aminoglycoside resistant bacteria equipped with various aminoglycoside-

159 ntibiotics or strategies to combat multidrug resistant bacteria, especially Gram-negative bacteria fo

160 Infections caused by antibiotic-resistant bacteria, especially the "ESKAPE" pathogens, c

161 supplemented with chloramphenicol (Cm) when resistant bacteria expressing Cm acetyltransferase (CAT)

162 from this research suggested that antibiotic resistant bacteria expressing OMP TolC could spread more

163 is a plasmid-encoded DHFR from trimethoprim-resistant bacteria, extensive studies on R67 with variou

164 imal models to control pathogenic antibiotic resistant bacteria found on mucosal surfaces and in bloo

165 imal models to control pathogenic antibiotic resistant bacteria found on mucosal surfaces and infecte

166 opportunities for the cross-transmission of resistant bacteria from patient to patient, and patients

167 The continuous emergence of resistant bacteria has become a major worldwide health t

168 t years, the increasing number of antibiotic-resistant bacteria has become a serious health concern.

169 The emergence of drug-resistant bacteria has compromised the use of many conve

170 therapeutics against notoriously antibiotic-resistant bacteria has led to a quest for novel antibact

171 The rise of antibiotic-resistant bacteria has led to an urgent need for rapid d

172 ocedures, prosthetic material and antibiotic-resistant bacteria has made the management of these card

173 The rapid emergence of multidrug-resistant bacteria has renewed interest in developing an

174 iseases titled, Bed bugs as Vectors for Drug-Resistant Bacteria has sparked a renewed interest in bed

175 crease in the prevalence of multi-antibiotic-resistant bacteria has threatened the physician's abilit

176 Mercury resistant bacteria have developed a system of two enzyme

177 ted animals can contribute to persistence of resistant bacteria in agricultural environments.

178 rate the development and spread of multidrug-resistant bacteria in cirrhosis.

179 Using a biofilm of heavy metal resistant bacteria in combination with this special desi

180 lonisation and transmission of antimicrobial-resistant bacteria in European ICUs.

181 bedding reservoir compared with shedding of resistant bacteria in faeces.

182 Diseases caused by antibiotic-resistant bacteria in hospitals are the outcome of compl

183 n on the clinical epidemiology of antibiotic-resistant bacteria in hospitals.

184 any significant role in the success of drug-resistant bacteria in human populations.

185 , and indicated that the higher frequency of resistant bacteria in manure-amended soil was attributab

186 of life-threatening infections by antibiotic-resistant bacteria in recent years have provided strong

187 ndment induced a bloom of certain antibiotic-resistant bacteria in soil that was independent of antib

188 cystic fibrosis patients, highly antibiotic-resistant bacteria in the BCC have emerged as problemati

189 it may be further developed to fight against resistant bacteria in the clinic.

190 er understanding of how to manage antibiotic-resistant bacteria in the future.

191 etermination of the actual proportion of PZA-resistant bacteria in the isolate and it is simple and i

192 Inoculation with Ni-resistant bacteria in the rhizosphere of L. emarginata h

193 evaluate the risk of exposure to antibiotic-resistant bacteria in treated wastewater.

194 Selection of resistant bacteria in vitro reveals that two of the pred

195 and are highly active against some quinolone-resistant bacteria including quinolone-resistant MRSA.

196 f-sterilizing fabrics (which kill antibiotic-resistant bacteria, including methicillin-resistant Stap

197 antibiotic development in increasingly drug-resistant bacteria, including Mycobacterium tuberculosis

198 treatment of infections caused by multidrug resistant bacteria, including S. aureus.

199 The emergence of antimicrobial resistant bacteria increases the risk of treatment failu

200 nergy will diminish as the frequency of drug-resistant bacteria increases.

201 urthermore, antibiotic treatment selects for resistant bacteria, increases opportunities for horizont

202 iated with a higher occurrence of antibiotic-resistant bacteria, indicating that AB treatment in wome

203 al sources rarely contribute to the entry of resistant bacteria into the community.

204 ize the levels of antibiotics and antibiotic-resistant bacteria introduced into the environment.

205 al bacteria promotes infection by antibiotic-resistant bacteria is a fertile area for speculation wit

206 prevalence of infections caused by multidrug-resistant bacteria is a global health problem that has b

207 The rapid rise of multi-drug-resistant bacteria is a global healthcare crisis, and ne

208 well-tolerated therapies against antibiotic-resistant bacteria is a global public health problem lea

209 The spread of antibiotic resistant bacteria is a global threat that shakes the fo

210 The increasing prevalence of antibiotic-resistant bacteria is a global threat to public health.

211 The widespread development of multidrug-resistant bacteria is a major health emergency.

212 nce in living cells while fighting multidrug-resistant bacteria is a paramount topic.

213 The development of antibiotic-resistant bacteria is associated with significant morbid

214 Tracking the nosocomial spread of resistant bacteria is critical to infection control.

215 tivity of pyranmycins against aminoglycoside resistant bacteria is less than expected, the synthetic

216 The emergence of multi-drug resistant bacteria is limiting the effectiveness of comm

217 The increase in multi-drug-resistant bacteria is limiting the effectiveness of curr

218 e prevalence and proliferation of antibiotic resistant bacteria is profoundly important to human heal

219 With the advent of drug-resistant bacteria, it is necessary to understand the fu

220 a (Gram-positive and Gram-negative) and drug-resistant bacteria (methicillin-resistant Staphylococcus

221 nst both vancomycin-sensitive and vancomycin-resistant bacteria (MICs = 0.06-0.005 and 0.5-0.06 mug/m

222 found in vancomycin-sensitive and vancomycin-resistant bacteria: N-acetylglycine, D-lactic acid, and

223 Network provides annual reports on monitored resistant bacteria, national surveillance efforts are st

224 heir potential to treat infections caused by resistant bacteria or their unique value for specific cr

225 ges in individual risks of colonization with resistant bacteria over calendar time were consistent wi

226 howed more often colonization with multidrug resistant bacteria (P = 0.02).

227 washing reduced acquisition of antimicrobial-resistant bacteria, particularly MRSA.

228 The primary outcome was acquisition of resistant bacteria per 100 patient-days at risk, for whi

229 ophage can enhance the killing of antibiotic-resistant bacteria, persister cells, and biofilm cells,

230 t one patient death was lowest for multidrug-resistant bacteria (PNNT = 20) followed by Candida speci

231 tal-acquired infections caused by antibiotic-resistant bacteria pose a grave and growing threat to pu

232 tam specificity and metal content.Carbapenem-resistant bacteria pose a major health threat by express

233 The spread of antibiotic-resistant bacteria poses a substantial threat to morbidi

234 The increase in antibiotic resistant bacteria poses a threat to the continued use o

235 Antibiotic-resistant bacteria present an ongoing challenge to both

236 t result in a change in either the number of resistant bacteria present or the acquisition of antibio

237 he prevalence of infection due to antibiotic-resistant bacteria (Pseudomonas aeruginosa, Acinetobacte

238 ), blaNDM-1 (r = 0.934, p = 0.009), and ESBL-resistant bacteria (r = 0.913, p = 0.010) levels across

239 but not in the actual counts, of doxycycline-resistant bacteria recovered from both plaque and saliva

240 theory of competitive release predicts, drug-resistant bacteria replicate fastest when their drug-sus

241 Infections due to multidrug-resistant bacteria represent a major global health chall

242 Drug-resistant bacteria represent a significant global threat

243 s for infections caused by highly antibiotic-resistant bacteria represents a new, and as yet, unteste

244 s a consequence of problematic antimicrobial-resistant bacteria, research is now directed towards nar

245 he global strategy should include antibiotic-resistant bacteria responsible for community-acquired in

246 gens, whereas the addition of phage to phage-resistant bacteria resulted in significantly reduced lev

247 erall exposure to antibiotics and antibiotic-resistant bacteria (resulting from close proximity to ot

248 acterial growth, especially against the drug-resistant bacteria Salmonella typhimurium.

249 ational Action Plan for Combating Antibiotic-Resistant Bacteria set a goal of reducing inappropriate

250 hypoxia and the appearance of the multidrug resistant bacteria Staphylococcus simulans in the fetal

251 yze the dynamic elements of nonresistant and resistant bacteria strains in epidemic populations in ho

252 AIs) and the increasing number of antibiotic-resistant bacteria strains remain significant public hea

253 so showed high activity against various drug-resistant bacteria such as methicillin-resistant Staphyl

254 cluding drugs and vaccines against multidrug-resistant bacteria such as Neisseria gonorrhoeae The fir

255 to restore the susceptibility of multi-drug-resistant bacteria, such as MRSA, to clinically availabl

256 Bloodstream infection by highly antibiotic-resistant bacteria, such as vancomycin-resistant Enteroc

257 Infection with antibiotic-resistant bacteria, such as vancomycin-resistant Enteroc

258 ct on an individual's carriage of antibiotic-resistant bacteria than does direct consumption of antib

259 contained a higher abundance of beta-lactam-resistant bacteria than soil treated with inorganic fert

260 inical care has driven the emergence of drug-resistant bacteria that are adapted to thrive in hospita

261 ofilm cells, reduce the number of antibiotic-resistant bacteria that arise from an antibiotic-treated

262 tion may retain small subpopulations of drug-resistant bacteria that can flourish once the host is tr

263 ed soils, indicating that the high number of resistant bacteria that enter the soils from the TWW are

264 mber of environmental pathogens and for drug-resistant bacteria, the environmental mimics developed h

265 the face of the clinical challenge posed by resistant bacteria, the present needs for novel classes

266 hese enzymes are expressed constitutively in resistant bacteria, the turnover of ATP is continuous du

267 s may be our best defense against antibiotic-resistant bacteria, there has been little progress in th

268 With rapid emergence of multidrug-resistant bacteria, there is often a need to perform sus

269 ion analysis method to prioritise antibiotic-resistant bacteria; this method involved the identificat

270 ient, there was no evidence for emergence of resistant bacteria to any of the tested antibiotics (inc

271 erb substrate promiscuity of AMEs allows the resistant bacteria to cope with diverse structural modif

272 a National Action Plan to Combat Antibiotic-Resistant Bacteria to curb the rise of "superbugs," bact

273 o human pathogens, to decrease the spread of resistant bacteria to people and animals via foodstuffs,

274 o create a priority list of other antibiotic-resistant bacteria to support research and development o

275 in and were found to re-sensitize vancomycin-resistant bacteria to the antibiotic.

276 The results indicated that, antibiotic resistant bacteria turned the methylene blue in to white

277 le for acquired resistance but essential for resistant bacteria under therapeutic concentrations of a

278 shing molecular features of lead and cadmium resistant bacteria using Attenuated Total Reflectance-Fo

279 The final priority ranking of the antibiotic-resistant bacteria was established after a preference-ba

280 alence of children colonized with antibiotic-resistant bacteria was from 29% to 24% (p =.41).

281 esence of infection attributed to antibiotic-resistant bacteria was similar for patients who survived

282 Considering the current dilemma of multidrug-resistant bacteria we consider it particularly prudent t

283 eration cephalosporin against AmpC-producing resistant bacteria, we designed a novel broad-spectrum n

284 To discover reagents to fight against resistant bacteria, we initiated a chemical approach by

285 For trends in acquisition of antimicrobial-resistant bacteria, weekly incidence rate ratio (IRR) wa

286 ciated pneumonia, and infection by multidrug-resistant bacteria were independently associated to incr

287 althcare worker contamination with multidrug-resistant bacteria were positive environmental cultures

288 do not reduce acquisition rates of multidrug-resistant bacteria, whether or not screening is done wit

289 re needed to treat infections caused by drug-resistant bacteria, which constitute a major growing thr

290 d-spectrum cephalosporins and monobactams in resistant bacteria, which lead to treatment problems in

291 ming of lytic phages to kill only antibiotic-resistant bacteria while protecting antibiotic-sensitize

292 sing in vitro activity against wild-type and resistant bacteria while retaining the favorable bacteri

293 itiated soon, our inability to treat totally resistant bacteria will halt other developments in medic

294 bations that can enhance the killing of drug-resistant bacteria with antibiotic treatment.

295 rectly monitor the phenotypic switch in drug-resistant bacteria with temporal resolution.

296 potently active against pathogenic and drug-resistant bacteria, with minimal inhibitory concentratio

297 effective at preventing spread of antibiotic-resistant bacteria within hospitals.

298 iminate intestinal colonization by multidrug-resistant bacteria, without profound disruption of the i

299 challenge posed by increasing levels of drug-resistant bacteria world-wide is manifest, and must be d

300 Increase in drug-resistant bacteria worldwide and the dearth of new antib