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1 ed bone formation and slightly hindered bone resorption.
2 ncreasing bone formation and decreasing bone resorption.
3 t osteoclast precursors to induce local bone resorption.
4 lasts, is a major negative regulator of bone resorption.
5 1 antisense RNA to control pathological bone resorption.
6 ase increased when cells underwent flagellar resorption.
7 sertion to minimize future peri-implant bone resorption.
8 ariables that cause higher or lower rates of resorption.
9 such as hydroxyapatite affecting the rate of resorption.
10 in pathway and Runx2 that contribute to bone resorption.
11 often eliminated in a process of allogeneic resorption.
12 w stromal cells and systemic effects on bone resorption.
13 static bone remodeling and pathological bone resorption.
14 in osteoclasts, a process required for bone resorption.
15 a key role in bacteria-induced alveolar bone resorption.
16 ces and extracting teeth with extensive root resorption.
17 osteocytes to orchestrate bone formation and resorption.
18 n rhythm helps coordinate bone formation and resorption.
19 tomography analysis was used to assess bone resorption.
20 egulates osteoclast differentiation and bone resorption.
21 mode of bone formation and pathological bone resorption.
22 ot sufficient stimuli for bone deposition or resorption.
23 al calcification and dental inflammation and resorption.
24 entiation and inhibition of OC-directed bone resorption.
25 under climatic stress could interfere with N resorption.
26 transient lumen support followed by complete resorption.
27 hat successfully mated, and increased embryo resorption.
28 lated RANKL in osteoblasts and parietal bone resorption.
29 ide (LPS) from P. gingivalis stimulates bone resorption.
30 ugmented osteoclast differentiation and bone resorption.
31 turn, increases osteoclastogenesis and bone resorption.
32 -induced osteoclast differentiation and bone resorption.
33 as the result of defective osteoclastic bone resorption.
34 essential for L. monocytogenes-induced fetal resorption.
35 strogen deficiency leads to accelerated bone resorption.
36 mation that was accompanied by elevated bone resorption.
37 ScT may occur at advanced stages of scaffold resorption.
38 ary base, is important for regulating cilium resorption.
39 e formation is not as clear as its effect on resorption.
40 h in the bone environment and inhibited bone resorption.
41 multinucleated OCs and decreases bone matrix resorption.
42 titis in mice, as evidenced by alveolar bone resorption.
43 c MTs and podosomes interact to control bone resorption.
44 hages into gingival tissue and alveolar bone resorption.
45 creased bone formation rate and reduced bone resorption.
46 zone resulting in reduced mineralized matrix resorption.
47 lls (MGCs) of the monocytic lineage, is bone resorption.
48 ic mechanisms, such as control of growth and resorption.
49 osteoclasts, plays an important role in bone resorption.
50 nized in a belt, a feature critical for bone resorption.
51 Ts changes during flagellar regeneration and resorption.
52 sorption and could be an indicator of lamina resorption.
53 microCT analysis was used to assess bone resorption.
54 tant gatekeepers of estrogen-controlled bone resorption.
55 tment, reduced IL-6 and RANKL, and less bone resorption.
56 undergoes continual cycles of formation and resorption.
57 ific genes via NFATc1, which facilitate bone resorption.
58 of IFT-B through cilia decapitation precedes resorption.
59 ied data that provide new insights into root resorption.
60 tegerin (OPG) signaling associated with bone resorption.
61 d strength by uncoupling bone formation from resorption.
62 severely osteopenic because of enhanced bone resorption.
63 al knockout mice alleviated progressive bone resorption.
64 ml CSC neonatally had increased rates of pup resorption.
65 increases bone formation, and decreases bone resorption.
66 anced osteoclastogenesis, and increased bone resorption.
67 to bone tissue by inducing osteoclastic bone resorption.
68 ish demonstrate that shedding involved tooth resorption, a primitive feature in bony fishes, but abse
69 teoclasts are the cells responsible for bone resorption, a process that is essential for the maintena
72 d bone, periosteal reaction, serpentine bone resorption, abscess formation, and root penetration of t
75 n functional osteo-assays, we show that bone resorption activity of D2J osteoclasts is dramatically r
78 expression of osteoclastic markers and bone resorption activity, as well as decreased expression of
81 ean duration to the first sign suggestive of resorption among patients of SJS was 36.7 months and amo
82 sociated with a significant decrease in bone resorption and a marked reduction in number of osteoclas
83 sociated with a significant decrease in bone resorption and a marked reduction in the number of osteo
85 h an increase in osteoclastogenesis and bone resorption and an increase in the pool of monocytes.
87 tein (PTHrP) is a critical regulator of bone resorption and augments osteolysis in skeletal malignanc
91 hometry measurements revealed that both bone resorption and bone formation parameters were increased
93 osis results from the imbalance between bone resorption and bone formation, and restoring the normal
96 d impaired ability to protect mice from bone resorption and bone loss in response to high-dose recept
97 of osteocytes and osteoblasts precedes bone resorption and bone loss with reduced mechanical stimula
98 ignificantly inhibits regional alveolar bone resorption and contributes to periodontal healing in an
99 the most common presenting feature of lamina resorption and could be an indicator of lamina resorptio
100 e remodeling, as confirmed by increased bone resorption and decreased bone formation, and significant
102 eous ozone application accelerates xenograft resorption and enhances bone regeneration, especially in
104 t activation and unbalanced coupling between resorption and formation, which induces a thinning of tr
107 t osteoclasts (OCLs) block both pagetic bone resorption and formation; therefore, PD offers key insig
108 , there were no differences in alveolar bone resorption and gingival RANKL expression between mice tr
109 (ATV) are known to inhibit osteoclastic bone resorption and have been proposed to have osteostimulati
110 ic matrix deposition and osteoclastic tissue resorption and immunomodulation for tissue development.
112 decrease calcium excretion by reducing bone resorption and increasing renal calcium reabsorption.
113 hich are characterized by high rates of bone resorption and loss of bone mass, may benefit from treat
115 phosphonates used for treatment inhibit bone resorption and prevent bone loss but fail to influence b
116 ation by inhibiting osteoclast-mediated bone resorption and promoting osteoblast-mediated osteogenesi
119 (+) M2-like macrophages associated with root resorption and root surface repair processes linked to t
123 ises two processes: the removal of old bone (resorption) and the laying down of new bone (formation).
124 y related to proinflammatory burden, infarct resorption, and adverse left ventricular remodeling in t
125 collagen-degrading enzyme activity, infarct resorption, and adverse structural remodeling (r>0.5).
126 stogenic cytokine production, stimulate bone resorption, and cause trabecular bone loss, demonstratin
127 nd low strains on apoptosis, mineralization, resorption, and collagen matrix deposition in peri-impla
129 xtensive peri-implantitis with advanced bone resorption, and extensive inflammation with granulation
132 ionships among PMO, iron deposition, infarct resorption, and left ventricular remodeling between day
133 very high interfacial strains, marginal bone resorption, and no improvement in implant stability.
134 ops plant, on osteoclastogenesis, osteoclast resorption, and RANKL-induced signaling pathway using bo
135 h Aurora-A (AURKA) kinase to control ciliary resorption, and with Src and other partners to influence
136 ays a relevant role in inflammation and bone resorption associated with the LPS model of experimental
137 ssels and osteoclasts and promoted cartilage resorption at the repair site during the periosteal endo
142 NKL and BMPs, in osteoclastogenesis and bone resorption by ablating p38alpha MAPK in LysM+monocytes.
143 of bone increased CatK-mediated osteoclastic resorption by approximately 27%, and negligible resorpti
145 ks PTH-induced osteoclast formation and bone resorption by its additional effect to inhibit RANKL-med
152 ature OCs but is critically involved in bone resorption by stimulating extracellular acidification an
153 tes periosteal osteoclast formation and bone resorption by stimulating RANKL in osteoblasts via TLR2.
155 ted that the inhibition of osteoclastic bone resorption by these compounds did not result from their
156 one matrix, pharmacologic inhibition of bone resorption by zoledronate attenuates inflammasome activa
157 ith blood lead and plasma biomarkers of bone resorption (C-terminal telopeptides of type I collagen (
158 xisting leaf-trait databases, since nutrient resorption can cause traits of litter and green leaves t
161 ymal stem cell-derived osteoblasts, and bone resorption, carried out by monocyte-derived osteoclasts.
162 eased loss of alveolar bone with patterns of resorption characteristic of aggressive forms of periodo
163 e had elevated cancellous bone formation and resorption compared to other treatment groups as well as
164 y (p < 0.01) less P. gingivalis-induced bone resorption compared with controls in BALB/c and C57BL/6
165 cular bone in vivo was due to decreased bone resorption, consistent with the reduced receptor activat
166 arthritis was associated with increased bone resorption, decreased bone formation, and significant bo
167 l sources of variability, including feeding (resorption decreases) and recent fracture (all markers i
168 idea, Pkd1(-/-);Nedd9(-/-) mice had ciliary resorption defects, and treatment of Pkd1(-/-) mice with
169 to be a reduction front that releases As for resorption downflow, yielding a high load of labile As s
174 Osteopenia occurs where the rate of bone resorption exceeds that of bone formation, so we investi
175 at there was a significant reduction in bone resorption following 3 months of SPI supplementation tha
176 rthermore, the CMP materials showed signs of resorption from 4 weeks, and no graft materials were obs
177 enisci; and (2) the bubble contracted as the resorption front advanced, dissolving air along the way.
179 ylated on arginine residues during flagellar resorption; however, the function is not understood.
181 ubhyaloid blood into vitreous cavity and its resorption, improvement in visual acuity, need for furth
183 to assess its role in inflammation and bone resorption in a murine model of lipopolysaccharide (LPS)
184 to assess its role in inflammation and bone resorption in a murine model of lipopolysaccharide (LPS)
187 overexpression results in premature ciliary resorption in cultured cells dependent on function of th
189 Dentin in permanent teeth rarely undergoes resorption in development, homeostasis, or aging, in con
196 itive feedback mechanism that amplifies bone resorption in pathologic conditions of accelerated bone
197 l pool, osteoclast differentiation, and bone resorption in response to receptor activator of nuclear
198 zed osteopenia associated with enhanced bone resorption in the cancellous bone compartment and with s
201 urthermore, CX3CR1 knockout mice resist bone resorption in the oral cavity following challenge with P
202 The importance of osteoclast-mediated bone resorption in the process of osseointegration has not be
203 resorption may have implications in internal resorption in the root canal, pulp/dentin regeneration,
204 rbate synovial inflammation in vivo and bone resorption in vitro, suggesting that LTB4 and BLT1 could
205 tly decarboxylated and activated during bone resorption, inactivation of furin in osteoblasts in mice
206 odel of P. gingivalis-induced calvarial bone resorption, injection of mmu-miR-155-5p or anti-mmu-miR-
209 -implant bone develops micro-fractures, bone resorption is increased, and bone formation is decreased
212 to superfluous osteoclast formation and bone resorption, is widespread in the pathologic bone loss an
213 ough chloroquine had no effect on basal bone resorption, it inhibited parathyroid hormone- and ovarie
217 mice exhibited high serum levels of the bone resorption marker C-telopeptide fragments of type I coll
218 as well as a transient decrease in the bone resorption marker C-telopeptide of type I collagen (CTX-
222 ent and modeling, rather than excessive bone resorption, may be the underlying pathophysiology of the
223 emodeling due to balanced bone formation and resorption mediated by osteoblasts and osteoclasts, resp
225 mouse model and RANKL-injection-induced bone resorption model, we found that administration of XN mar
227 ad increased mechanical loading-induced bone resorption, number of osteoclasts, and expression of pro
228 Our data indicate that the increase in bone resorption observed in states of estrogen deficiency in
229 plants demonstrated a peri-implant mean bone resorption of 2.96 mm increased bone loss, yielding a cu
232 der, characterized by defective osteoclastic resorption of bone that results in increased bone densit
234 ng intensities of drought and warming on the resorption of different classes of plant metabolites.
235 s in embryonic/neonatal lethality with rapid resorption of homozygous mutants, hampering additional s
237 reduces Src activity in osteoclasts, reduces resorption of mineralized matrix both in vivo and in cel
243 llary growth remodelling (bone formation and resorption) of the Devil's Tower (Gibraltar 2) and La Qu
244 alendronic acid, a potent inhibitor of bone resorption, optimally linked through a differentially hy
246 plementation also did not affect the rate of resorptions or the size of litters, but instead skewed t
247 nt to restrain osteoclast formation, inhibit resorption, or stop bone loss induced by skeletal unload
248 l (e.g. the nutrient uptake from litter, the resorption, or the storage of nutrients in the biomass),
249 periodontal pocket formation, alveolar bone resorption, osteoclast activation, bacterial invasion of
250 ngivalis and four other TLR2 ligands on bone resorption, osteoclast formation, and gene expression in
252 promoting bone formation and inhibiting bone resorption, our results suggest that Wnt4 signaling coul
253 of 17 patients who showed evidence of lamina resorption out of the 85 eyes (87 laminae) of 82 patient
255 -resistant acid phosphatase (TRAP) staining, resorption pit assays, and real-time polymerase chain re
256 uced osteoclastogenesis, leading to enhanced resorption pit formation compared with wild-type control
259 or IG9402, a BP analog that does not inhibit resorption, prevented the increase in osteocyte apoptosi
260 ic cell adhesion in the external apical root resorption process and the specific role of alpha/beta i
261 fold discontinuity and restenosis during the resorption process, which appear delayed in humans; thes
263 portant role not only in the degradation and resorption properties of the materials, but also in cons
264 gradation of ARL13b that occurs during cilia resorption, raising the possibility that the sensitivity
265 ta T cells but were designed to inhibit bone resorption rather than treating cancer and have limited
267 The ZOL arm had a 65% reduction in bone resorption relative to the placebo arm at 24 weeks (0.11
270 hways; and (3) regulatory mechanisms of root resorption repair by cementum at the proteomic and trans
272 en (CTX-I) are markers of bone formation and resorption, respectively, that are recommended for clini
274 last activity and diminishes osteoclast bone resorption, shifting the balance of bone homeostasis and
275 sed increased RANKL and produced an abnormal resorption-stimulating bone matrix high in BSP content.
276 ceted processes of immunoregulation and bone resorption such as they occur in rheumatoid arthritis (R
277 se in sympathetic output that increases bone resorption sufficiently to counteract its local anti-res
279 a rare entity distinct from pathologic root resorption that occurs as a result of several local and
283 and this protective effect extended to fetal resorption triggered by partial ablation of immune-suppr
286 re: complications such as ankylosis and root resorption up to the tooth exfoliation have occurred fre
287 orption by approximately 27%, and negligible resorption was observed when osteoclasts were cultured o
288 rease in osteoclast differentiation and bone resorption was observed with an increase in IL-17 levels
289 Phagocytosis and infarcted brain tissue resorption was reduced in TREM2 knock-out (KO) mice comp
290 TNF-alpha-induced model of inflammatory bone resorption, we determined that RBP-J deficiency enables
291 raditionally thought to occur solely through resorption, we show that an acute loss of IFT-B through
292 y and map fields of bone deposition and bone resorption, which affect the development of the facial s
293 n of these bone defects revealed active bone resorption, which is suppressed by Wnt activation in ost
294 upted c-Kit signaling couples increased bone resorption with bone formation through osteoclast-derive
295 eting senescent cells were due to lower bone resorption with either maintained (trabecular) or higher
297 ic drug-eluting stents, followed by complete resorption with recovery of more normal vascular structu
298 microbiota increases both bone formation and resorption, with the net effect of colonization varying
299 an increased number of osteoclasts and bone resorption, without a decrease in osteoblast number or b
300 pharmacologic action as an inhibitor of bone resorption, yet CT-deficient mice display increased bone
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