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1 ed bone formation and slightly hindered bone resorption.
2 ncreasing bone formation and decreasing bone resorption.
3 t osteoclast precursors to induce local bone resorption.
4 lasts, is a major negative regulator of bone resorption.
5 1 antisense RNA to control pathological bone resorption.
6 ase increased when cells underwent flagellar resorption.
7 sertion to minimize future peri-implant bone resorption.
8 ariables that cause higher or lower rates of resorption.
9 such as hydroxyapatite affecting the rate of resorption.
10 in pathway and Runx2 that contribute to bone resorption.
11  often eliminated in a process of allogeneic resorption.
12 w stromal cells and systemic effects on bone resorption.
13 static bone remodeling and pathological bone resorption.
14  in osteoclasts, a process required for bone resorption.
15 a key role in bacteria-induced alveolar bone resorption.
16 ces and extracting teeth with extensive root resorption.
17 osteocytes to orchestrate bone formation and resorption.
18 n rhythm helps coordinate bone formation and resorption.
19  tomography analysis was used to assess bone resorption.
20 egulates osteoclast differentiation and bone resorption.
21 mode of bone formation and pathological bone resorption.
22 ot sufficient stimuli for bone deposition or resorption.
23 al calcification and dental inflammation and resorption.
24 entiation and inhibition of OC-directed bone resorption.
25 under climatic stress could interfere with N resorption.
26 transient lumen support followed by complete resorption.
27 hat successfully mated, and increased embryo resorption.
28 lated RANKL in osteoblasts and parietal bone resorption.
29 ide (LPS) from P. gingivalis stimulates bone resorption.
30 ugmented osteoclast differentiation and bone resorption.
31  turn, increases osteoclastogenesis and bone resorption.
32 -induced osteoclast differentiation and bone resorption.
33 as the result of defective osteoclastic bone resorption.
34 essential for L. monocytogenes-induced fetal resorption.
35 strogen deficiency leads to accelerated bone resorption.
36 mation that was accompanied by elevated bone resorption.
37 ScT may occur at advanced stages of scaffold resorption.
38 ary base, is important for regulating cilium resorption.
39 e formation is not as clear as its effect on resorption.
40 h in the bone environment and inhibited bone resorption.
41 multinucleated OCs and decreases bone matrix resorption.
42 titis in mice, as evidenced by alveolar bone resorption.
43 c MTs and podosomes interact to control bone resorption.
44 hages into gingival tissue and alveolar bone resorption.
45 creased bone formation rate and reduced bone resorption.
46 zone resulting in reduced mineralized matrix resorption.
47 lls (MGCs) of the monocytic lineage, is bone resorption.
48 ic mechanisms, such as control of growth and resorption.
49 osteoclasts, plays an important role in bone resorption.
50 nized in a belt, a feature critical for bone resorption.
51 Ts changes during flagellar regeneration and resorption.
52 sorption and could be an indicator of lamina resorption.
53     microCT analysis was used to assess bone resorption.
54 tant gatekeepers of estrogen-controlled bone resorption.
55 tment, reduced IL-6 and RANKL, and less bone resorption.
56  undergoes continual cycles of formation and resorption.
57 ific genes via NFATc1, which facilitate bone resorption.
58 of IFT-B through cilia decapitation precedes resorption.
59 ied data that provide new insights into root resorption.
60 tegerin (OPG) signaling associated with bone resorption.
61 d strength by uncoupling bone formation from resorption.
62 severely osteopenic because of enhanced bone resorption.
63 al knockout mice alleviated progressive bone resorption.
64 ml CSC neonatally had increased rates of pup resorption.
65 increases bone formation, and decreases bone resorption.
66 anced osteoclastogenesis, and increased bone resorption.
67 to bone tissue by inducing osteoclastic bone resorption.
68 ish demonstrate that shedding involved tooth resorption, a primitive feature in bony fishes, but abse
69 teoclasts are the cells responsible for bone resorption, a process that is essential for the maintena
70         C. acnes significantly decreased the resorption ability of osteoclasts with a major impact by
71 cytokines and its influence on alveolar bone resorption (ABR) in rats.
72 d bone, periosteal reaction, serpentine bone resorption, abscess formation, and root penetration of t
73                                  During bone resorption, abundant factors previously buried in the bo
74 one formation activity nor osteoclastic bone resorption activity in vivo.
75 n functional osteo-assays, we show that bone resorption activity of D2J osteoclasts is dramatically r
76 of Bmpr1b null osteoclasts was increased but resorption activity was decreased.
77               Osteoclast differentiation and resorption activity was enhanced in Gnas(+/p-) cells.
78  expression of osteoclastic markers and bone resorption activity, as well as decreased expression of
79  aureus on osteoclastogenesis and osteoclast resorption activity.
80 nB2-expressing osteoclasts and a decrease in resorption activity.
81 ean duration to the first sign suggestive of resorption among patients of SJS was 36.7 months and amo
82 sociated with a significant decrease in bone resorption and a marked reduction in number of osteoclas
83 sociated with a significant decrease in bone resorption and a marked reduction in the number of osteo
84                  Tgif2 deletion reduces bone resorption and abolishes miR-34a regulation.
85 h an increase in osteoclastogenesis and bone resorption and an increase in the pool of monocytes.
86                         One instance of root resorption and ankylosis was noted with EMD+CAF.
87 tein (PTHrP) is a critical regulator of bone resorption and augments osteolysis in skeletal malignanc
88 and/or T cells, accompanied by enhanced bone resorption and BMD loss.
89 mice exhibited osteopenia with elevated bone resorption and bone formation at 6- and 9-week-old.
90                     The balance between bone resorption and bone formation is vital for maintenance a
91 hometry measurements revealed that both bone resorption and bone formation parameters were increased
92               We concluded that the material resorption and bone formation was highly impacted by the
93 osis results from the imbalance between bone resorption and bone formation, and restoring the normal
94 role in determining the balance between bone resorption and bone formation.
95 , thereby creating an imbalance between bone resorption and bone formation.
96 d impaired ability to protect mice from bone resorption and bone loss in response to high-dose recept
97  of osteocytes and osteoblasts precedes bone resorption and bone loss with reduced mechanical stimula
98 ignificantly inhibits regional alveolar bone resorption and contributes to periodontal healing in an
99 the most common presenting feature of lamina resorption and could be an indicator of lamina resorptio
100 e remodeling, as confirmed by increased bone resorption and decreased bone formation, and significant
101 ally sufficient to prevent increases in bone resorption and decreases in BMD in men.
102 eous ozone application accelerates xenograft resorption and enhances bone regeneration, especially in
103           CLP promoted OC formation and bone resorption and expression of OC-associated genes.
104 t activation and unbalanced coupling between resorption and formation, which induces a thinning of tr
105 ey insights into mechanisms that couple bone resorption and formation.
106  is characterized by focal and dramatic bone resorption and formation.
107 t osteoclasts (OCLs) block both pagetic bone resorption and formation; therefore, PD offers key insig
108 , there were no differences in alveolar bone resorption and gingival RANKL expression between mice tr
109 (ATV) are known to inhibit osteoclastic bone resorption and have been proposed to have osteostimulati
110 ic matrix deposition and osteoclastic tissue resorption and immunomodulation for tissue development.
111 ammation associated with aging promotes bone resorption and impairs bone formation.
112  decrease calcium excretion by reducing bone resorption and increasing renal calcium reabsorption.
113 hich are characterized by high rates of bone resorption and loss of bone mass, may benefit from treat
114                                Alveolar bone resorption and myeloperoxidase activity were statistical
115 phosphonates used for treatment inhibit bone resorption and prevent bone loss but fail to influence b
116 ation by inhibiting osteoclast-mediated bone resorption and promoting osteoblast-mediated osteogenesi
117  and heterozygous mice exhibit elevated bone resorption and reduced bone mass.
118 -like cells and unusual patterns of cementum resorption and repair.
119 (+) M2-like macrophages associated with root resorption and root surface repair processes linked to t
120 sions that rarely heal due to increased bone resorption and suppressed bone formation.
121 e is mediated by increased osteoclastic bone resorption and suppressed bone formation.
122  to pathologic changes and, ultimately, bone resorption and tooth loss.
123 ises two processes: the removal of old bone (resorption) and the laying down of new bone (formation).
124 y related to proinflammatory burden, infarct resorption, and adverse left ventricular remodeling in t
125  collagen-degrading enzyme activity, infarct resorption, and adverse structural remodeling (r>0.5).
126 stogenic cytokine production, stimulate bone resorption, and cause trabecular bone loss, demonstratin
127 nd low strains on apoptosis, mineralization, resorption, and collagen matrix deposition in peri-impla
128 hage recruitment, osteoclast formation, bone resorption, and cortical and trabecular bone loss.
129 xtensive peri-implantitis with advanced bone resorption, and extensive inflammation with granulation
130                 Bacterial colonization, bone resorption, and implant inflammation were evaluated by p
131 ment, extensive alveolar bone and tooth root resorption, and incisor malocclusion.
132 ionships among PMO, iron deposition, infarct resorption, and left ventricular remodeling between day
133 very high interfacial strains, marginal bone resorption, and no improvement in implant stability.
134 ops plant, on osteoclastogenesis, osteoclast resorption, and RANKL-induced signaling pathway using bo
135 h Aurora-A (AURKA) kinase to control ciliary resorption, and with Src and other partners to influence
136 ays a relevant role in inflammation and bone resorption associated with the LPS model of experimental
137 ssels and osteoclasts and promoted cartilage resorption at the repair site during the periosteal endo
138 out the choreography of cilia biogenesis and resorption at this organ.
139  with the severity of periodontitis and bone resorption biomarkers.
140 ween the periodontal parameters TOS and bone resorption biomarkers.
141 ssociated mutation, exhibited increased bone resorption, but not formation.
142 NKL and BMPs, in osteoclastogenesis and bone resorption by ablating p38alpha MAPK in LysM+monocytes.
143 of bone increased CatK-mediated osteoclastic resorption by approximately 27%, and negligible resorpti
144                                Enhanced bone resorption by infiltrating macrophages has been proposed
145 ks PTH-induced osteoclast formation and bone resorption by its additional effect to inhibit RANKL-med
146 ting bone formation by OBs and reducing bone resorption by OCs.
147                               Excessive bone resorption by osteoclasts (OCs) can result in serious cl
148                    OC-iTcREG also limit bone resorption by osteoclasts, forming a negative feedback l
149  both bone formation by osteoblasts and bone resorption by osteoclasts.
150  bone remodeling process and stimulates bone resorption by osteoclasts.
151 gand (RANKL), an essential cytokine for bone resorption by osteoclasts.
152 ature OCs but is critically involved in bone resorption by stimulating extracellular acidification an
153 tes periosteal osteoclast formation and bone resorption by stimulating RANKL in osteoblasts via TLR2.
154 ation of XN markedly inhibited bone loss and resorption by suppressing osteoclast activity.
155 ted that the inhibition of osteoclastic bone resorption by these compounds did not result from their
156 one matrix, pharmacologic inhibition of bone resorption by zoledronate attenuates inflammasome activa
157 ith blood lead and plasma biomarkers of bone resorption (C-terminal telopeptides of type I collagen (
158 xisting leaf-trait databases, since nutrient resorption can cause traits of litter and green leaves t
159            These cytokines enhanced the bone resorption capacity of uninfected mature osteoclasts and
160 nstream signaling resulting in impaired bone resorption capacity.
161 ymal stem cell-derived osteoblasts, and bone resorption, carried out by monocyte-derived osteoclasts.
162 eased loss of alveolar bone with patterns of resorption characteristic of aggressive forms of periodo
163 e had elevated cancellous bone formation and resorption compared to other treatment groups as well as
164 y (p < 0.01) less P. gingivalis-induced bone resorption compared with controls in BALB/c and C57BL/6
165 cular bone in vivo was due to decreased bone resorption, consistent with the reduced receptor activat
166 arthritis was associated with increased bone resorption, decreased bone formation, and significant bo
167 l sources of variability, including feeding (resorption decreases) and recent fracture (all markers i
168  idea, Pkd1(-/-);Nedd9(-/-) mice had ciliary resorption defects, and treatment of Pkd1(-/-) mice with
169 to be a reduction front that releases As for resorption downflow, yielding a high load of labile As s
170                         External apical root resorption during orthodontic treatment implicates speci
171 ople with SRA itself are predisposed to root resorption during orthodontic treatment.
172 agen degradation, is a key biomarker of bone resorption during the bone remodeling process.
173                                              Resorption efficiencies (REs) of extractable metabolites
174     Osteopenia occurs where the rate of bone resorption exceeds that of bone formation, so we investi
175 at there was a significant reduction in bone resorption following 3 months of SPI supplementation tha
176 rthermore, the CMP materials showed signs of resorption from 4 weeks, and no graft materials were obs
177 enisci; and (2) the bubble contracted as the resorption front advanced, dissolving air along the way.
178                  Although elemental nutrient resorption has been shown to respond modestly to tempera
179 ylated on arginine residues during flagellar resorption; however, the function is not understood.
180 nt bioceramics for application as controlled resorption implants.
181 ubhyaloid blood into vitreous cavity and its resorption, improvement in visual acuity, need for furth
182 ted osteoclast actin-ring formation and bone resorption in a dose-dependent manner.
183  to assess its role in inflammation and bone resorption in a murine model of lipopolysaccharide (LPS)
184  to assess its role in inflammation and bone resorption in a murine model of lipopolysaccharide (LPS)
185 aintaining soft tissues and minimizing ridge resorption in all dimensions after ARP.
186 nts compromised their ability to induce bone resorption in an ex vivo organ culture system.
187  overexpression results in premature ciliary resorption in cultured cells dependent on function of th
188 lpha to induce osteoclast formation and bone resorption in DAP12-deficient animals.
189   Dentin in permanent teeth rarely undergoes resorption in development, homeostasis, or aging, in con
190 ic melatonin administration on alveolar bone resorption in experimental periodontitis in rats.
191 ymal dental pulp cells in attenuating dentin resorption in homeostasis are also reviewed.
192 may be responsible for the absence of dentin resorption in homeostasis.
193 th stimulate bone formation and inhibit bone resorption in humans.
194 ot canal, pulp/dentin regeneration, and root resorption in orthodontic tooth movement.
195 brae, suggesting enhanced bone formation and resorption in OVX-Diet rats.
196 itive feedback mechanism that amplifies bone resorption in pathologic conditions of accelerated bone
197 l pool, osteoclast differentiation, and bone resorption in response to receptor activator of nuclear
198 zed osteopenia associated with enhanced bone resorption in the cancellous bone compartment and with s
199  Nek2 is a switch balancing ciliogenesis and resorption in the development of LR asymmetry.
200                              Therefore, bone resorption in the mother becomes elevated during these p
201 urthermore, CX3CR1 knockout mice resist bone resorption in the oral cavity following challenge with P
202   The importance of osteoclast-mediated bone resorption in the process of osseointegration has not be
203 resorption may have implications in internal resorption in the root canal, pulp/dentin regeneration,
204 rbate synovial inflammation in vivo and bone resorption in vitro, suggesting that LTB4 and BLT1 could
205 tly decarboxylated and activated during bone resorption, inactivation of furin in osteoblasts in mice
206 odel of P. gingivalis-induced calvarial bone resorption, injection of mmu-miR-155-5p or anti-mmu-miR-
207       The process of bone mineralization and resorption is complex and is affected by numerous factor
208 in modern humans extensive osteoclastic bone resorption is found in the same regions.
209 -implant bone develops micro-fractures, bone resorption is increased, and bone formation is decreased
210                                 Because bone resorption is largely unaltered, OSM could represent a n
211 late scaffold thrombosis (VLScT) occurs when resorption is presumed to be nearly complete.
212 to superfluous osteoclast formation and bone resorption, is widespread in the pathologic bone loss an
213 ough chloroquine had no effect on basal bone resorption, it inhibited parathyroid hormone- and ovarie
214  into "sealing-zones" (SZs) that confine the resorption lacuna.
215             After 8 wk, multiple dentin/root resorption lacunae were present in root dentin with robu
216         The 11 days of ligature induced bone resorption, low levels of BALP, leukocyte infiltration;
217 mice exhibited high serum levels of the bone resorption marker C-telopeptide fragments of type I coll
218  as well as a transient decrease in the bone resorption marker C-telopeptide of type I collagen (CTX-
219                          The pattern of bone resorption markers was consistent with accelerated bone
220                   Our results suggest that N resorption may be controlled not only by plant demand, b
221            Mesenchymal attenuation of dentin resorption may have implications in internal resorption
222 ent and modeling, rather than excessive bone resorption, may be the underlying pathophysiology of the
223 emodeling due to balanced bone formation and resorption mediated by osteoblasts and osteoclasts, resp
224            Multiple idiopathic cervical root resorption (MICRR) is a rare entity distinct from pathol
225 mouse model and RANKL-injection-induced bone resorption model, we found that administration of XN mar
226 s after parathyroidectomy suggests that bone resorption normalizes earlier than bone formation.
227 ad increased mechanical loading-induced bone resorption, number of osteoclasts, and expression of pro
228  Our data indicate that the increase in bone resorption observed in states of estrogen deficiency in
229 plants demonstrated a peri-implant mean bone resorption of 2.96 mm increased bone loss, yielding a cu
230 d improve organ function by accelerating the resorption of amyloid deposits.
231 ersal of uremia, because of a lack of active resorption of apatite.
232 der, characterized by defective osteoclastic resorption of bone that results in increased bone densit
233                                          The resorption of brushite-based bone cements has been shown
234 ng intensities of drought and warming on the resorption of different classes of plant metabolites.
235 s in embryonic/neonatal lethality with rapid resorption of homozygous mutants, hampering additional s
236  altered precipitation on the production and resorption of metabolites in Quercus rubra.
237 reduces Src activity in osteoclasts, reduces resorption of mineralized matrix both in vivo and in cel
238         We propose a molecular mechanism for resorption of NaCl by MRCs during development, and concl
239                                         Bone resorption of osteoclasts from subchondral bone and the
240 by climatic influences on the production and resorption of plant metabolites.
241 e transmigrating canine as well as ruled out resorption of roots of mandibular incisors.
242 e abdominal aneurysm was performed after the resorption of the pseudocyst.
243 llary growth remodelling (bone formation and resorption) of the Devil's Tower (Gibraltar 2) and La Qu
244  alendronic acid, a potent inhibitor of bone resorption, optimally linked through a differentially hy
245 bone formation rate but did not inhibit bone resorption or reduce tumor burden.
246 plementation also did not affect the rate of resorptions or the size of litters, but instead skewed t
247 nt to restrain osteoclast formation, inhibit resorption, or stop bone loss induced by skeletal unload
248 l (e.g. the nutrient uptake from litter, the resorption, or the storage of nutrients in the biomass),
249  periodontal pocket formation, alveolar bone resorption, osteoclast activation, bacterial invasion of
250 ngivalis and four other TLR2 ligands on bone resorption, osteoclast formation, and gene expression in
251 nomalies (early-onset periodontitis and root resorption), osteopenia, and acro-osteolysis.
252 promoting bone formation and inhibiting bone resorption, our results suggest that Wnt4 signaling coul
253 of 17 patients who showed evidence of lamina resorption out of the 85 eyes (87 laminae) of 82 patient
254 as osteoclastogenic gene expression and bone resorption pit are increased.
255 -resistant acid phosphatase (TRAP) staining, resorption pit assays, and real-time polymerase chain re
256 uced osteoclastogenesis, leading to enhanced resorption pit formation compared with wild-type control
257 sed by using an osteologic plate assay (bone resorption pit formation).
258                                         Bone resorption pits in calvaria, observed by micro-computed
259 or IG9402, a BP analog that does not inhibit resorption, prevented the increase in osteocyte apoptosi
260 ic cell adhesion in the external apical root resorption process and the specific role of alpha/beta i
261 fold discontinuity and restenosis during the resorption process, which appear delayed in humans; thes
262 olved in cartilage damage, bone erosion, and resorption processes during osteoarthritis.
263 portant role not only in the degradation and resorption properties of the materials, but also in cons
264 gradation of ARL13b that occurs during cilia resorption, raising the possibility that the sensitivity
265 ta T cells but were designed to inhibit bone resorption rather than treating cancer and have limited
266 on of matrix TGF-beta during osteoclast bone resorption recruits MSCs to bone-resorptive sites.
267      The ZOL arm had a 65% reduction in bone resorption relative to the placebo arm at 24 weeks (0.11
268 ammatory diseases, their direct role in bone resorption remains unclear.
269  in contrast to bone that undergoes periodic resorption/remodeling.
270 hways; and (3) regulatory mechanisms of root resorption repair by cementum at the proteomic and trans
271 H stimulation of RANKL, a stimulator of bone resorption, requires CRTC2.
272 en (CTX-I) are markers of bone formation and resorption, respectively, that are recommended for clini
273 geneic donors into recipients shows that the resorption response can be adoptively acquired.
274 last activity and diminishes osteoclast bone resorption, shifting the balance of bone homeostasis and
275 sed increased RANKL and produced an abnormal resorption-stimulating bone matrix high in BSP content.
276 ceted processes of immunoregulation and bone resorption such as they occur in rheumatoid arthritis (R
277 se in sympathetic output that increases bone resorption sufficiently to counteract its local anti-res
278 Sixteen out of 20 laminae showed evidence of resorption superiorly.
279  a rare entity distinct from pathologic root resorption that occurs as a result of several local and
280 that XN inhibits osteoclastogenesis and bone resorption through RANK/TRAF6 signaling pathways.
281 oblastogenesis and inhibit osteoclastic bone resorption, thus promoting tissue regeneration.
282 ase initiated by bacteria, resulting in bone resorption, tooth loss, and systemic inflammation.
283 and this protective effect extended to fetal resorption triggered by partial ablation of immune-suppr
284 d C-terminal telopeptide release during bone resorption under distinct conditions.
285 ss pathologic analysis showed gradual device resorption until 32 weeks after deployment.
286 re: complications such as ankylosis and root resorption up to the tooth exfoliation have occurred fre
287 orption by approximately 27%, and negligible resorption was observed when osteoclasts were cultured o
288 rease in osteoclast differentiation and bone resorption was observed with an increase in IL-17 levels
289      Phagocytosis and infarcted brain tissue resorption was reduced in TREM2 knock-out (KO) mice comp
290 TNF-alpha-induced model of inflammatory bone resorption, we determined that RBP-J deficiency enables
291 raditionally thought to occur solely through resorption, we show that an acute loss of IFT-B through
292 y and map fields of bone deposition and bone resorption, which affect the development of the facial s
293 n of these bone defects revealed active bone resorption, which is suppressed by Wnt activation in ost
294 upted c-Kit signaling couples increased bone resorption with bone formation through osteoclast-derive
295 eting senescent cells were due to lower bone resorption with either maintained (trabecular) or higher
296                          Incidence of severe resorption with extrusion of cylinder/requiring lamina r
297 ic drug-eluting stents, followed by complete resorption with recovery of more normal vascular structu
298 microbiota increases both bone formation and resorption, with the net effect of colonization varying
299  an increased number of osteoclasts and bone resorption, without a decrease in osteoblast number or b
300 pharmacologic action as an inhibitor of bone resorption, yet CT-deficient mice display increased bone

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