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1 an pCO2 above 1,000 muatm by 2100 - inducing respiratory acidosis in fish that must be corrected thro
2 nctional analysis showed that such a reduced respiratory activity influenced chloroplast electron tra
3 tory proteins, with unknown implications for respiratory activity.
4 enous rate of aerobic or anaerobic microbial respiratory activity.
5 rgic manifestations, and was associated with respiratory allergies (P < .002) or respiratory allergie
6 ted with respiratory allergies (P < .002) or respiratory allergies coexistent with atopic eczema (P <
7 ts potential role in secondary prevention of respiratory allergy progression.
8 odies may be efficacious in the treatment of respiratory and autonomic dysfunction in heart failure.
9 real membrane oxygenation (ECMO) is used for respiratory and cardiac failure in children but is compl
10 -based RSV reports submitted to the National Respiratory and Enteric Virus Surveillance System during
11 ration of sedation, sevoflurane consumption, respiratory and hemodynamic data, Simplified Acute Physi
12 ove survival without BPD at 36 weeks' PMA or respiratory and neurodevelopmental outcomes at 18 to 24
13 ronchopulmonary dysplasia that best predicts respiratory and neurodevelopmental outcomes in preterm i
14                     Most adverse events were respiratory, and in some patients it led to drug discont
15                                The phagocyte respiratory burst is crucial for innate immunity.
16            CBD treatment also attenuated the respiratory burst of neutrophils isolated from chronic p
17          Priming neutrophils for an enhanced respiratory burst together with promoting granule conten
18  cholesterol-dependent reduced mitochondrial respiratory capacity and release of mitochondrial DNA in
19 chondrial respiratory gene expression, spare respiratory capacity, and ATP levels.
20  biological clock status, photosynthetic and respiratory capacity, cell-cycle phase, and environmenta
21 tions in whole-plant leaf area and increased respiratory carbon losses.
22 tical event was defined as the occurrence of respiratory, cardiac, allergic, or neurological complica
23 ion of at least 1 organ system of the renal, respiratory, cardiovascular, coagulation, and neurologic
24                      The contribution of non-respiratory causes of death to global influenza-associat
25                       We fed KD to mice with respiratory chain complex III (CIII) deficiency and prog
26 mbalanced stoichiometry of the mitochondrial respiratory chain complexes and skin inflammation and su
27 The genomic data suggest that C. trachomatis respiratory chain could produce a sodium gradient that m
28 nd skin inflammation and suggest that severe respiratory chain dysfunction, as observed in few cells
29 ce and restored mtDNA copy number as well as respiratory chain enzyme activities and levels.
30 is required for the insertion of heme b into respiratory chain enzymes.
31 genase (Na(+)-NQR) is a key component of the respiratory chain of diverse prokaryotic species, includ
32 ctron-accepting complex of the mitochondrial respiratory chain.
33  a role in the assembly of the mitochondrial respiratory chain.
34 in the Kolliker-Fuse (KF), a key area in the respiratory circuit, was examined.
35  3 years of screening from 183 hospitals and respiratory clinics in 14 countries.
36 gnaling and the posttranslational control of respiratory CO2 refixation and anaplerotic photosynthate
37                  Initially we confirmed that respiratory-competent cells can be produced following in
38 d hygiene, and overnight hospital stays with respiratory complaints.
39                           Energy-transducing respiratory complex I (NADH:ubiquinone oxidoreductase) i
40 ndrial oxygen consumption in response to the respiratory complex II substrate succinate.
41 three classes independently acquired aerobic respiratory complexes, supporting the hypothesis that ae
42 sthetic dose on risk of severe postoperative respiratory complications.
43 The children involved in these incidents had respiratory conditions (n = 387; 18%), injuries (n = 289
44 onal mechanism for a direct influence of the respiratory cycle on memory function.
45                                 Standardized respiratory cycles consisted of a deep standardized insp
46 ate annual O3 exposures, and estimated total respiratory deaths in 2010 that were attributable to lon
47  (c.352 G>A) associated with early childhood respiratory deficiency.
48 s with the relative lack of tolerance to the respiratory depressant effect of opioids.
49  heretofore have been associated with opiate respiratory depression, which may have clinical applicat
50 ainkillers, like morphine, can be limited by respiratory depression.
51  29,468 participants provided information on respiratory, dermal, and eye irritation health.
52 tial exposure to the dispersants and adverse respiratory, dermal, and eye irritation symptoms.
53 ment in Primary Care to Identify Undiagnosed Respiratory Disease and Exacerbation Risk), was used to
54 er less than or equal to 2.5 mum (PM2.5)) on respiratory disease and lung cancer mortality is poorly
55 ramyxoviruses include global causes of lower respiratory disease like the parainfluenza viruses, as w
56                              The presence of respiratory disease may affect tissue oxygenation and en
57  in mammals.IMPORTANCE Canine influenza is a respiratory disease of dogs caused by two CIV subtypes,
58 IOPRED (Unbiased Biomarkers in Prediction of Respiratory Disease Outcomes) study.
59 n, tuberculosis infection, diabetes, chronic respiratory disease, and blindness health programmes; pr
60 th, including cancer, heart disease, stroke, respiratory disease, and infection.
61 nic and ischemic heart disease, nonmalignant respiratory disease, and lung, skin, bladder, and kidney
62 ymphoid cells (ILC2s) in aspirin-exacerbated respiratory disease-like Ptges(-/-) mice.
63 and at distances greater than 7 km for fatal respiratory disease.
64 may eventually lead to tissue remodeling and respiratory disease.Cystic fibrosis is caused by mutatio
65 apy, the aetiological treatment for allergic respiratory diseases, has demonstrated to be an effectiv
66 itively associated with the risk of incident respiratory diseases, hypertension and myocardial infarc
67 are critical processes in the development of respiratory diseases, including asthma.
68 unction is critical in the pathology of many respiratory diseases, including cystic fibrosis.
69  approved for the management of allergic and respiratory disorders and biologics currently in develop
70 re also present in several critical neonatal respiratory disorders.
71  paradigm for developing strategies to treat respiratory distress in SMA.
72                             RATIONALE: Acute respiratory distress syndrome (ARDS) is caused by widesp
73                   RATIONALE: Following acute respiratory distress syndrome (ARDS), joblessness is com
74 cal ventilation in adult patients with acute respiratory distress syndrome (ARDS).
75  is increasingly used in patients with acute respiratory distress syndrome (ARDS).
76  Mechanically ventilated children with acute respiratory distress syndrome (Berlin).
77  469 patients (18 tuberculosis-related acute respiratory distress syndrome and 451 acute respiratory
78                     After induction of acute respiratory distress syndrome by hydrochloric acid insti
79  Cdyn, and PaO2/FIO2 were collected at acute respiratory distress syndrome onset and at 24 hours in 3
80                                     At acute respiratory distress syndrome onset, neither mechanical
81     In a multisite cohort of long-term acute respiratory distress syndrome survivors, better annual p
82                  One hundred fifty-six acute respiratory distress syndrome survivors.
83                                        Acute respiratory distress syndrome was induced by repeated lu
84 iratory distress syndrome-others) with acute respiratory distress syndrome were admitted.
85 ower incidence of acute kidney injury, acute respiratory distress syndrome, and need for vasopressors
86 ggest hypothermia may be beneficial in acute respiratory distress syndrome, but cooling causes shiver
87                             During the acute respiratory distress syndrome, epithelial cells, primari
88 r of significant human pathologies including respiratory distress syndrome, lung adenocarcinoma, and
89  respiratory distress syndrome and 451 acute respiratory distress syndrome-others) with acute respira
90 the severity of rodent E. coli-induced acute respiratory distress syndrome.
91 ean Consensus Conference definition of acute respiratory distress syndrome.
92 rotective ventilation in patients with acute respiratory distress syndrome.
93 greater than or equal to 2 years after acute respiratory distress syndrome.
94 h the emergency department at risk for acute respiratory distress syndrome.
95              Consecutive subjects with acute respiratory distress syndrome.
96 ciated with a lower incidence of early acute respiratory distress syndrome.
97 4 months thereafter, until 5 years postacute respiratory distress syndrome.
98                       Low birthweight and/or respiratory distress were reported in 11 (29%) infected
99 hest indrawing pneumonia and signs of severe respiratory distress, oxygen saturation <93% (when not a
100 onth to 5 years of age with undifferentiated respiratory distress.
101 ccessful owing to lung disease or inadequate respiratory drive.
102 certain clinical features, like dysphonia or respiratory dysfunction, were exclusively detected in th
103 ion manifested later than cardiovascular and respiratory dysfunction.
104 Other changes, such as the appearance of the respiratory eggshell ridges, are caused by changes in th
105 l types, suggesting that thermal response of respiratory enzymes in leaves could be conserved.
106 ch as heme chaperones, that insert heme into respiratory enzymes.
107 piratory mucosal explants and primary equine respiratory epithelial cells (EREC), grown at the air-li
108 r findings demonstrate that integrity of the respiratory epithelium is crucial in the host's innate d
109  It specializes in infection of the ciliated respiratory epithelium, causing disease of variable seve
110 Ps were not readily taken up into or through respiratory epithelium, with very low silver levels foun
111  in subjects with mild COPD or similar acute respiratory events in smokers without airflow obstructio
112  subjects without airflow obstruction, acute respiratory events were not associated with additional F
113 were higher in participants with two or more respiratory exacerbations per year than in those with ze
114                             Furthermore, the respiratory exchange rate was significantly lower in LER
115 mia (14 [6%]; one related to treatment), and respiratory failure (14 [6%]).
116 with extracorporeal membrane oxygenation for respiratory failure and sepsis between the service being
117 orporeal membrane oxygenation in adults with respiratory failure and sepsis is steadily increasing, b
118 domized Evaluation of Sedation Titration for Respiratory Failure clinical trial.
119          Further analysis revealed increased respiratory failure in nonsurvivors.
120  in one patient, and acute renal failure and respiratory failure in one patient) were suspected to be
121 d to be related to everolimus treatment, but respiratory failure was suspected to be related.
122 Understand the Global Impact of Severe Acute Respiratory Failure) study described the management of p
123 nous extracorporeal membrane oxygenation for respiratory failure, 2) veno-arterial extracorporeal mem
124  attributed to treatment (pneumonitis, acute respiratory failure, and cardiovascular failure).
125 poreal membrane oxygenation for severe acute respiratory failure.
126 la is increasingly used in the management of respiratory failure.
127  used to sustain life in patients with acute respiratory failure.
128 l benefits in unselected patients with acute respiratory failure.
129 rectly affects acid-base and ion regulation, respiratory function and aerobic performance in aquatic
130  treated with neonatal caffeine had improved respiratory function at 11 years of age compared with ch
131 ed for those interested in cardiovascular or respiratory function in addition to motor function and c
132 poorer birth outcomes, neurodevelopment, and respiratory function in children.
133 onchopulmonary dysplasia, but its effects on respiratory function in later childhood are unknown.
134 ities of Fe-S cluster enzymes, a decrease in respiratory function, and an increase in oxidative stres
135 ng was defined as good cognitive, motor, and respiratory functioning, along with absence of disabilit
136 es, namely cardiovascular, neuronal, immune, respiratory, gastrointestinal, liver, and endocrine syst
137 nd p53 signaling, and diminish mitochondrial respiratory gene expression, spare respiratory capacity,
138  wheezing illnesses during infancy influence respiratory health for years.
139 xposure to select phthalates and phenols and respiratory health in boys.
140              Clinicians should be aware that respiratory health often deteriorates during reproductiv
141 ects of tobacco smoke exposure on children's respiratory health.
142 mmatory effects and thereby adversely affect respiratory health.
143  exposure may underestimate true effects for respiratory hospitalizations.
144                                              Respiratory ICU of a tertiary care hospital in North Ind
145 uring sentinel surveillance for severe acute respiratory illness (SARI) hospitalization conducted in
146                                              Respiratory illness caused by viral infection is associa
147 V1 are major viral causes of acute pediatric respiratory illness for which no vaccines or suitable an
148 because of impaired lung function or whether respiratory illness reduces lung function.
149            Controls were subjects with acute respiratory illness testing negative for influenza.
150  type 1 (HPIV1) also causes severe pediatric respiratory illness, especially croup.
151 e East respiratory syndrome-CoV, cause acute respiratory illness.
152 virus (CoV) HKU1 is a pathogen causing acute respiratory illnesses and so far little is known about i
153 breathing problems, tiring easily, and acute respiratory illnesses.
154                                Unexpectedly, respiratory impairment by mdivi-1 occurs without mitocho
155 ver- and ever-smokers without obstruction or respiratory impairment, PRM(FSA) increased 2.7% per deca
156 iddle East respiratory syndrome severe acute respiratory infection (adjusted odds ratio, 5.87; 95% CI
157  22% of all episodes of (severe) acute lower respiratory infection (ALRI) resulting in 55 000 to 199
158 [95% confidence interval {CI}, .76-.95]) and respiratory infection (aOR, 0.82 [95% CI, .75-.90]) and
159   Previous studies have suggested that acute respiratory infection (ARI) and nonsteroidal anti-inflam
160 mbers of the microbiota that protect against respiratory infection by the major human pathogens Strep
161 iddle East respiratory syndrome severe acute respiratory infection from other etiologies; therefore,
162 NALE: Bronchiolitis is the most common lower respiratory infection in infants; however, it remains un
163 sting is essential component of severe acute respiratory infection investigation for at-risk patients
164 enza death across countries by comparing GHE respiratory infection mortality rates from countries wit
165 e animals after intravenous infection, while respiratory infection results in virus replication in th
166 ountries with high rates of mortality due to respiratory infection using the same methods.
167 age or a less severe syndrome, such as upper respiratory infection).
168 s between early-life exposures (infant lower respiratory infection, manual social class, home overcro
169 naling axis they activate to protect against respiratory infection.
170 t can cause a multitude of diseases, such as respiratory infections and conjunctivitis.
171 eutrophil migratory accuracy with age during respiratory infections and determine if and how a statin
172                           The spread of many respiratory infections is determined by contact patterns
173 /182 prescriptions), while it was non-severe respiratory infections using the control algorithm (ALMA
174 ient data from trials in which patients with respiratory infections were randomly assigned to receive
175 aryngeal niche, a reservoir for ME and upper respiratory infections.
176 ng antibiotic therapy in patients with acute respiratory infections.
177 ces multidrug tolerance in S. aureus through respiratory inhibition and reduction of cellular ATP.
178 akness of proximal dominance, hypotonia, and respiratory insufficiency but typically not cardiac dysf
179 f ventilator-induced lung injury in terms of respiratory mechanics, blood gases, and pulmonary edema.
180 = 0.83) indicated that OA patients possess a respiratory metabolic profile fully divergent from those
181 ributes to sustain the increased activity of respiratory metabolism to fuel plant defences.
182 sive reorganization of the transcriptome and respiratory metabolism.
183 these aberrant developmental trajectories of respiratory microbiota members were mode of delivery, in
184  which we reconstruct volumetric cardiac and respiratory motion phases, contrast-agent dynamics, and
185 nly for scan-rescan examinations affected by respiratory motion.
186 at synaptic scaling sustains strength of the respiratory motor output following months of inactivity,
187 3A) ) we previously demonstrated that spinal respiratory motor plasticity elicited by acute intermitt
188 erpesvirus family, was used to infect equine respiratory mucosal explants and primary equine respirat
189 PV in children with acute severe asthma with respiratory muscle fatigue and failure of medical treatm
190 r atrophy in limb muscles when compared with respiratory muscle.
191 tion over ca. 2.5 cycles of the short-period respiratory oscillation.
192                                              Respiratory outcomes on discharge to home, at 1 year, an
193  tube defects, harms of treatment (twinning, respiratory outcomes).
194                                    The ePlex respiratory pathogen panel (RP panel) is a novel molecul
195                        As an important human respiratory pathogen, molecular detection is an indispen
196 T cells (TRMs) mediate optimal protection to respiratory pathogens, and we hypothesized that reduced
197 nsults, including coinfections by recognized respiratory pathogens.
198 are a critical first line of defense against respiratory pathogens.
199                                              Respiratory polygraphy was the most cost-effective proto
200 ic, physiological, or chemical inhibition of respiratory processes elicited increased biofilm formati
201  grasslands will release less C through soil respiratory processes under the projected seasonal redis
202 ssion but an increase in nuDNA expression of respiratory proteins, with unknown implications for resp
203 mean change from baseline in the St George's Respiratory Questionnaire (SGRQ) total score at week 24
204 fied as the apnoea-hypopnoea index (AHI) and respiratory rate variability index (RRVI).
205 hted average heart rate, blood pressure, and respiratory rate, along with changes-over-time for each.
206 ecord oxygen requirement, oxygen saturation, respiratory rate, consciousness level, and other evidenc
207                                              Respiratory rate, pH, PaCO2, and encephalopathy score im
208 cipients failed to afford protection against respiratory reinfection with influenza virus.
209 irst COPD hospitalization, 964 (11%) a first respiratory-related and 342 (4%) a first CV-related hosp
210  pirfenidone had a lower risk of nonelective respiratory-related hospitalization over the course of 1
211 of brainstem interneurons in the specialized respiratory rhythmogenic site dubbed preBotzinger Comple
212          Measles virus is transmitted by the respiratory route and illness begins with fever, cough,
213 ion have been reported, yet the bioenergetic respiratory status of mitochondria from prion-infected a
214 red with those using the criterion of oxygen/respiratory support (RS) (receiving supplemental oxygen
215 demonstrate a strong correlation between the respiratory suppression/antinociception therapeutic wind
216 dren and adults-is characterised by variable respiratory symptoms and variable airflow limitation.
217                                    Thus, the respiratory symptoms improved by the use of NPPV in chil
218    Specifically, asthma medication usage and respiratory symptoms increased [OR=3.51; 95% confidence
219              All subjects were monitored for respiratory symptoms, lung function, and nasal viral loa
220 Vaccines and prophylactic antibodies against respiratory syncytial virus (RSV) are in development and
221                                              Respiratory syncytial virus (RSV) belongs to the family
222                                              Respiratory syncytial virus (RSV) infection induces asth
223 ligand 4 (Dll4) was upregulated on APC after respiratory syncytial virus (RSV) infection, and its inh
224                                              Respiratory syncytial virus (RSV) is the most common cau
225                                        Human respiratory syncytial virus (RSV) is the most prevalent
226                                              Respiratory syncytial virus (RSV) lower respiratory trac
227                                              Respiratory syncytial virus (RSV) remains a major human
228 associated with enhanced rhinovirus (RV) and respiratory syncytial virus (RSV) replication.
229            We have previously estimated that respiratory syncytial virus (RSV) was associated with 22
230 ch, we discovered that plasma membrane-bound respiratory syncytial virus G rapidly recycles from the
231 ynamic formation of filamentous and branched respiratory syncytial virus particles, and assembly with
232 ydia, Haemophilus influenzae, influenza, and respiratory syncytial virus respiratory tract infections
233 experimentally challenged with either bovine respiratory syncytial virus, infectious bovine rhinotrac
234 A respiratory viruses (NIRVs), which include respiratory syncytial virus, parainfluenza viruses, coro
235 so includes rabies virus, measles virus, and respiratory syncytial virus.
236 hogens hepatitis C virus (HCV), Severe acute respiratory syndrome (SARS), coxsackie viruses, and rhin
237 ronaviruses (CoVs), such as the severe acute respiratory syndrome (SARS)-CoV and the Middle East resp
238 .12; 95% CI: 0.06-0.26) against severe acute respiratory syndrome (SARS).
239                                  Middle East respiratory syndrome coronavirus (MERS-CoV) emerged in 2
240 ERS-CoV pathogenesis in vivo The Middle East respiratory syndrome coronavirus (MERS-CoV) is a highly
241                                  Middle East respiratory syndrome coronavirus (MERS-CoV) is an import
242                                 Severe acute respiratory syndrome coronavirus (SARS-CoV) causes letha
243                                 Severe acute respiratory syndrome coronavirus (SARS-CoV) is a highly
244 iated with death compared to non-Middle East respiratory syndrome severe acute respiratory infection
245 omorbidities among patients with Middle East respiratory syndrome severe acute respiratory infection
246 r potential confounding factors, Middle East respiratory syndrome was independently associated with d
247 tory syndrome (SARS)-CoV and the Middle East respiratory syndrome-CoV, cause acute respiratory illnes
248 s of these forms of OP(DTT) deposited in the respiratory system may have differing health impacts.
249                  Given the resistance of the respiratory system to develop tolerance, desensitization
250                              Rounds included respiratory therapists in 26 of 43 (60.5%) and pharmacis
251  aerosols of virus would penetrate the lower respiratory tract and blanket alveoli where target cells
252 ssis is a human pathogen that can infect the respiratory tract and cause the disease known as whoopin
253 pha, interleukin-6, and interleukin-8 in the respiratory tract and central nervous system.
254 ility to early allergic sensitization, upper respiratory tract colonization with bacterial pathogens,
255 ng invasive mechanical ventilation for lower respiratory tract disease.
256 nd resistance to treatment of multiple human respiratory tract diseases including otitis media, chron
257 s the most common cause of viral acute lower respiratory tract illness (LRTI) in young children, and
258 coccal load in blood was not associated with respiratory tract illness in controls (P = .32).
259 act of early life exposures, including lower respiratory tract illness, on lung function during infan
260  common reason for hospitalization was lower respiratory tract infection (LRTI).
261  months and 12 years diagnosed with an acute respiratory tract infection and prescribed an oral antib
262 re present during symptoms of an acute viral respiratory tract infection in human subjects.
263 the most prevalent worldwide cause of severe respiratory tract infection in infants and young childre
264      Respiratory syncytial virus (RSV) lower respiratory tract infection is implicated in asthma deve
265 g for viruses in children who present with a respiratory tract infection is to differentiate between
266 or duration or severity of other acute lower respiratory tract infection symptoms, duration of abnorm
267 thelial secretome participating in RSV lower respiratory tract infection-induced airway remodeling.
268 two studies, 1 681 020 events) and for lower respiratory tract infections (-18.48% [-32.79 to -4.17];
269 s), and rates of hospital attendance for all respiratory tract infections (-3.45% [-4.64 to -2.25]; t
270   Common grade 1-2 toxicities included upper respiratory tract infections (in 28 [57%] of 49 patients
271 , influenza, and respiratory syncytial virus respiratory tract infections and ovalbumin-induced, seve
272 e number of laboratory-confirmed viral upper respiratory tract infections based on parent-collected n
273                           HBoV1 causes acute respiratory tract infections in young children and has a
274 single most important cause of serious lower respiratory tract infections in young children, yet no h
275 oV1) is a human parvovirus that causes acute respiratory tract infections in young children.
276                              Atopy and viral respiratory tract infections synergistically promote ast
277 a exacerbations, and hospital attendance for respiratory tract infections.
278 ide and represent the leading cause of upper respiratory tract infections.
279 the appropriate use of antibiotics for acute respiratory tract infections.
280 rum antibiotics for most children with acute respiratory tract infections.
281 escribing of antibiotics for childhood upper respiratory tract infections.
282                                   Among 3263 respiratory tract samples, 24.5% (798) and 37.3% (1216)
283 ificity (eg, detection of pathogens in upper respiratory tract specimens, which may indicate asymptom
284 adults with acute cough and at least 1 lower respiratory tract symptom not requiring immediate antibi
285 nd CYP2F1 in NA bioactivation and NA-induced respiratory tract toxicity in mouse models.
286 to check that they originated from the lower respiratory tract.
287 tion with the replication in the human upper respiratory tract.
288 e stresses, which are present throughout the respiratory tract.
289                                              Respiratory virus (RV) has been suggested to play a role
290 8637), allogeneic stem cell transplantation, respiratory virus infection, and Aspergillus PCR positiv
291 h Centers for Disease Control and Prevention respiratory virus surveillance data.
292 oronavirus (SARS-CoV) is a highly pathogenic respiratory virus that causes morbidity and mortality in
293 oronavirus (MERS-CoV) is a highly pathogenic respiratory virus that emerged from zoonotic sources in
294 ) emerged in 2012 and is a highly pathogenic respiratory virus.
295 gether with influenza, the non-influenza RNA respiratory viruses (NIRVs), which include respiratory s
296          Molecular testing was performed for respiratory viruses in nasopharyngeal aspirates collecte
297 urden, as recognised by WHO's Battle against Respiratory Viruses initiative.
298       Rapid point-of-care testing (POCT) for respiratory viruses might improve clinical care by reduc
299 fied, specifically the transmission of human respiratory viruses to wild great apes, causing high mor
300 ar localization of MS is a unique feature of respiratory yeasts such as P. pastoris and C. albicans,

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