ライフサイエンスコーパス: respiratory acidosis

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1 nate and base excess, indicating compensated respiratory acidosis.

2 g is limited to sheep akin to sheep maternal respiratory acidosis.

3 riation, or differences in the management of respiratory acidosis.

4 anges in cerebrovascular tone in response to respiratory acidosis.

5 unction and may blunt renal compensation for respiratory acidosis.

6 phragm force output, despite hypercapnia and respiratory acidosis.

7 However, lowering Vts may result in respiratory acidosis.

8 In medullary raphe neurones, respiratory acidosis (5%--> 9% CO(2)) caused a rapid fal

9 Maternal respiratory acidosis, a common finding in sheep studies

10 Respiratory acidosis, a decrease in blood pH caused by a

11 tracellular pH levels such as those found in respiratory acidosis--a known risk factor for SIDS--prod

12 levels (mean +/- SEM) from term babies with respiratory acidosis (acute hypoxia), normal babies, and

13 ive ventilation due to refractory hypoxia or respiratory acidosis and high plateau airway pressures.

14 Oxygen-breathing conscious mice sustained respiratory acidosis and, consequently, reduced cell pro

15 Episodes of hypoxia, respiratory acidosis, and bradycardia occurred more freq

16 resuscitation results in profound hypoxemia, respiratory acidosis, and significantly worse 24-hr neur

17 th groups had similar (mean +/- SD) baseline respiratory acidosis (arterial pH, 7.22 +/- 0.08; Pa(CO(

18 All animals developed a metabolic and a respiratory acidosis during the infusion of oleic acid.

19 Heliox improves respiratory acidosis, encephalopathy, and the respirator

20 increase in the arterial-alveolar gradient, respiratory acidosis, histological changes similar to th

21 nction in pulmonary disorders in relation to respiratory acidosis, impaired gas exchange, systemic co

22 an pCO2 above 1,000 muatm by 2100 - inducing respiratory acidosis in fish that must be corrected thro

23 t maintains arterial CO2 levels and prevents respiratory acidosis in the face of increased metabolism

24 channel, and suggests an important role for respiratory acidosis in triggering the fatal arrhythmia

25 Tactics to reduce respiratory acidosis include reductions in ventilation c

26 ity (n = 3), inadequate oxygenation (n = 3), respiratory acidosis (n = 3), and undocumented (n = 1).

27 nts with hypercapnic respiratory failure and respiratory acidosis nonresponsive to noninvasive ventil

28 tuations associated with profound hypoxemia, respiratory acidosis, or hemodynamic compromise.

29 moderate bronchoconstriction; more profound respiratory acidosis (PaCO2 137+/-41 torr (18.2+/-5.5 kP

30 injection and after the PET scan implicated respiratory acidosis that was induced by oxygen breathin

31 Despite respiratory acidosis, there was no decrease in trandiaph

32 In brainstem slices of wild-type mice, respiratory acidosis triggered robust elevations in [Ca(

33 As risk factors for SIDS include apnea and respiratory acidosis, Y1103 and wild-type channels were

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